1. A pheochromocytoma with high adrenocorticotropic hormone and a silent lung nodule
- Author
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Franco Grimaldi, Michele Bertolotto, Alessandro Proclemer, Nicoletta Sabato, Nicoletta Finato, Stella Bernardi, Sergio De Marchi, Bruno Fabris, Bernardi, Stella, Grimaldi, F, Finato, N, De Marchi, S, Proclemer, A, Sabato, Nicoletta, Bertolotto, Michele, and Fabris, Bruno
- Subjects
medicine.medical_specialty ,Lung Neoplasms ,Adrenal Gland Neoplasm ,Adrenal Gland Neoplasms ,Adrenocorticotropic hormone ,Pheochromocytoma ,ACTH ,EACS ,Nocardiasis ,Medicine (all) ,Cushing syndrome ,Adrenocorticotropic Hormone ,Fluorodeoxyglucose F18 ,medicine ,Humans ,Radionuclide Imaging ,Cushing Syndrome ,3-Iodobenzylguanidine ,Female ,Middle Aged ,Radiopharmaceuticals ,Nocardiasi ,Lung ,medicine.diagnostic_test ,business.industry ,Nodule (medicine) ,General Medicine ,medicine.disease ,medicine.anatomical_structure ,Positron emission tomography ,bacteria ,Radiology ,medicine.symptom ,business ,Emission computed tomography - Abstract
Pheochromocytoma (PCC) is a challenging and life-threatening neoplasm. Herein, the authors report an interesting and unexpected solution for a clinical case concerning a patient with a PCC, who developed delayed ectopic adrenocorticotropic hormone Cushing syndrome originating from the PCC. In addition, after a misleading 123 I-labeled metaiodobenzylguanidine single-photon emission computed tomography/computed tomography, an 18 F-fluorodeoxyglucose positron emission tomography/computed tomography, executed to confirm the diagnosis of PCC, showed a silent pulmonary nodule that unexpectedly turned out to be a lung nocardiasis.
- Published
- 2011