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1. Ni(II) Ions May Target the Entire Melatonin Biosynthesis Pathway—A Plausible Mechanism of Nickel Toxicity

2. Interactions of neurokinin B with copper(<scp>ii</scp>) ions and their potential biological consequences

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4. Dual mode of voltammetric studies on Cu(II) complexes of His2 peptides: phosphate and peptide sequence recognition

5. Aβ5–x Peptides: N-Terminal Truncation Yields Tunable Cu(II) Complexes

6. Peptide bond cleavage in the presence of Ni-containing particles

7. Copper Transporters? Glutathione Reactivity of Products of Cu–Aβ Digestion by Neprilysin

8. How trimerization of CTR1 N-terminal model peptides tunes Cu-binding and redox-chemistry

10. Copper(ii) complex of N-truncated amyloid-β peptide bearing a His-2 motif as a potential receptor for phosphate anions

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12. The N-terminal 14-mer model peptide of human Ctr1 can collect Cu(<scp>ii</scp>) from albumin. Implications for copper uptake by Ctr1

13. Cu transfer from amyloid-β4–16 to metallothionein-3: the role of the neurotransmitter glutamate and metallothionein-3 Zn(<scp>ii</scp>)-load states

14. Peptide Bond Cleavage by Ni(II) Ions within the Nuclear Localization Signal Sequence

15. Cu(II) Binding to the N-Terminal Model Peptide of the Human Ctr2 Transporter at Lysosomal and Extracellular pH

16. Cysteine and glutathione trigger the Cu–Zn swap between Cu(<scp>ii</scp>)-amyloid-β4-16peptide and Zn7-metallothionein-3

17. Resistance of Cu(Aβ4 – 16) to Copper Capture by Metallothionein‐3 Supports a Function for the Aβ4 – 42 Peptide as a Synaptic Cu II Scavenger

18. Correction: Copper(<scp>ii</scp>) complex of N-truncated amyloid-β peptide bearing a His-2 motif as a potential receptor for phosphate anions

19. Triggering Cu-coordination change in Cu( ii )-Ala-His-His by external ligands

20. Human Annexins A1, A2, and A8 as Potential Molecular Targets for Ni(II) Ions

21. A Functional Role for Aβ in Metal Homeostasis? N-Truncation and High-Affinity Copper Binding

22. Ni(ii) ions cleave and inactivate human alpha-1 antitrypsin hydrolytically, implicating nickel exposure as a contributing factor in pathologies related to antitrypsin deficiency

23. cis-Urocanic acid as a potential nickel(II) binding molecule in the human skin