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3,633 results on '"Nicolai, J"'

1. Prospective validation of ORACLE, a clonal expression biomarker associated with survival of patients with lung adenocarcinoma

Author

Biswas, Dhruva, Liu, Yun-Hsin, Herrero, Javier, Wu, Yin, Moore, David A., Karasaki, Takahiro, Grigoriadis, Kristiana, Lu, Wei-Ting, Veeriah, Selvaraju, Naceur-Lombardelli, Cristina, Magno, Neil, Ward, Sophia, Frankell, Alexander M., Hill, Mark S., Colliver, Emma, de Carné Trécesson, Sophie, East, Philip, Malhi, Aman, Snell, Daniel M., O’Neill, Olga, Leonce, Daniel, Mattsson, Johanna, Lindberg, Amanda, Micke, Patrick, Moldvay, Judit, Megyesfalvi, Zsolt, Dome, Balazs, Fillinger, János, Nicod, Jerome, Downward, Julian, Szallasi, Zoltan, Hackshaw, Allan, Jamal-Hanjani, Mariam, Kanu, Nnennaya, Birkbak, Nicolai J., and Swanton, Charles

Published
2025
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2. TRACERx analysis identifies a role for FAT1 in regulating chromosomal instability and whole-genome doubling via Hippo signalling

Author

Lu, Wei-Ting, Zalmas, Lykourgos-Panagiotis, Bailey, Chris, Black, James R. M., Martinez-Ruiz, Carlos, Pich, Oriol, Gimeno-Valiente, Francisco, Usaite, Ieva, Magness, Alastair, Thol, Kerstin, Webber, Thomas A., Jiang, Ming, Saunders, Rebecca E., Liu, Yun-Hsin, Biswas, Dhruva, Ige, Esther O., Aerne, Birgit, Grönroos, Eva, Venkatesan, Subramanian, Stavrou, Georgia, Karasaki, Takahiro, Al Bakir, Maise, Renshaw, Matthew, Xu, Hang, Schneider-Luftman, Deborah, Sharma, Natasha, Tovini, Laura, Jamal-Hanjani, Mariam, McClelland, Sarah E., Litchfield, Kevin, Birkbak, Nicolai J., Howell, Michael, Tapon, Nicolas, Fugger, Kasper, McGranahan, Nicholas, Bartek, Jiri, Kanu, Nnennaya, and Swanton, Charles

Published
2025
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5. Proteomic profiling reveals diagnostic signatures and pathogenic insights in multisystem inflammatory syndrome in children

Author

Nygaard, Ulrikka, Nielsen, Annelaura Bach, Dungu, Kia Hee Schultz, Drici, Lylia, Holm, Mette, Ottenheijm, Maud Eline, Nielsen, Allan Bybeck, Glenthøj, Jonathan Peter, Schmidt, Lisbeth Samsø, Cortes, Dina, Jørgensen, Inger Merete, Mogensen, Trine Hyrup, Schmiegelow, Kjeld, Mann, Matthias, Vissing, Nadja Hawwa, and Wewer Albrechtsen, Nicolai J.

Published
2024
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9. Exploring the molecular landscape of cancer of unknown primary: A comparative analysis with other metastatic cancers

Author

Laura Andersen, Ditte S. Christensen, Asbjørn Kjær, Michael Knudsen, Andreas K. Andersen, Maria B. Laursen, Johanne Ahrenfeldt, Britt E. Laursen, and Nicolai J. Birkbak

Subjects
cancer of unknown primary, genomic profile, immune response to cancer, transcriptomic profile, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, RC254-282
Abstract

Cancer of unknown primary (CUP) tumors are biologically very heterogeneous, which complicates stratification of patients for treatment. Consequently, these patients face limited treatment options and a poor prognosis. With this study, we aim to expand on the current knowledge of CUP biology by analyzing two cohorts: a well‐characterized cohort of 44 CUP patients, and 213 metastatic patients with known primary. These cohorts were treated at the same institution and characterized by identical molecular assessments. Through comparative analysis of genomic and transcriptomic data, we found that CUP tumors were characterized by high expression of immune‐related genes and pathways compared to other metastatic tumors. Moreover, CUP tumors uniformly demonstrated high levels of tumor‐infiltrating leukocytes and circulating T cells, indicating a strong immune response. Finally, the genetic landscape of CUP tumors resembled that of other metastatic cancers and demonstrated mutations in established cancer genes. In conclusion, CUP tumors possess a distinct immunophenotype that distinguishes them from other metastatic cancers. These results may suggest an immune response in CUP that facilitates metastatic tumor growth while limiting growth of the primary tumor.

Published
2024
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10. IDEOLOGY AND ORGANIZATIONAL DYNAMICS: CLARIFYING AND GENERALIZING OUR ARGUMENT ON “WOKE” COMPANIES.

Author

FOSS, NICOLAI J. and KLEIN, PETER G.

Subjects
ORGANIZATIONAL behavior, SOCIAL groups, DIVERSITY & inclusion policies, IDEOLOGY, ARGUMENT
Abstract

Olenick and Somaraju (O&S) criticize the arguments in our paper “Why do companies go woke?” (Foss & Klein, 2023). They claim that “woke” attitudes and behaviors are simply an extension of previous social and cultural practices in organizations and that we fail to appreciate the contributions of the existing diversity literature (Olenick & Somaraju, 2023). Our reply is twofold. First, we briefly restate our argument to show that O&S misinterpret and misrepresent our core claims concerning the nature and implications of the modern diversity, equity, and inclusion (DEI) movement. Second, we engage their broader arguments about intraorganizational dynamics and, in doing so, generalize our own analysis about the “woke company.” While O&S are correct to highlight attraction–selection–attrition processes in organizations and the conformity-inducing forces that exist in any social group, their argument omits (a) the importance of ideology in transforming DEI policies, (b) the role of internal stakeholders who pushed such policies internally, and (c) how the unique combination of these forces has made corporations “woke.” The broader point is that while research has pointed to the role of institutions as well as the role of internal dynamics in shaping organizational behaviors, the role of ideology in shaping these forces has been relatively neglected. [ABSTRACT FROM AUTHOR]

Published
2024
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13. Advocacy and the Search for Truth in Management Scholarship: Can the Twain Ever Meet?

Author

Wright, Thomas A, Emich, Kyle, Pearce, Jone L, Ramoglou, Stratos, Ashkanasy, Neal, Bartunek, Jean M, Kunisch, Sven, Denyer, David, Foss, Nicolai J, Klein, Peter G, Town, Sophia, Hollwitz, John, Barney, Chet E, Harms, Peter, Munyon, Timothy P, Seijts, Gerard, and Tsang, Eric WK

Subjects
Commerce, Management, Tourism and Services, Strategy, Management and Organisational Behaviour, academic freedom, advocacy, interestingness, kindness, scientific discourse, truth, Business and Management, Business & Management, Strategy, management and organisational behaviour
Abstract

Scholars have long debated the merits of advocacy-based research versus research considered from the quest for objective truth. Building upon reflections from multiple sources, a set of 11 brief reflections on three posed questions are presented. Tsang concludes our discussion with additional insights on how moving beyond the “interestingness” advocacy will be beneficial to the continued professional development of the management discipline.

Published
2023

18. The effect of exogenous glucagon on circulating amino acids in individuals with and without type 2 diabetes and obesity

Author

Magnus F G Grøndahl, Jonatan I Bagger, Malte P Suppli, Gerrit Van Hall, Nicolai J W Albrechtsen, Jens J Holst, Tina Vilsbøll, Mikkel B Christensen, Asger B Lund, and Filip K Knop

Subjects
amino acids, glucagon, obesity, type 2 diabetes, Diseases of the endocrine glands. Clinical endocrinology, RC648-665
Abstract

Objective: In obesity and type 2 diabetes, hyperglucagonaemia may be caused by elevated levels of glucagonotropic amino acids due to hepatic glucagon resistance at the level of amino acid turnover. Here, we investigated the effect of exogenous glucagon on circulating amino acids in obese and non-obese individuals with and without type 2 diabetes. Design: This was a post hoc analysis in a glucagon infusion study performed in individuals with type 2 diabetes (n = 16) and in age, sex, and body mass index-matched control individuals without diabetes (n = 16). Each group comprised two subgroups of eight individuals with and without obesity, respectively. Methods: All participants received a 1-h glucagon infusion (4 ng/kg/min) in the overnight fasted state. Plasma amino acid concentrations were measured with frequent intervals. Results: Compared to the control subgroup without obesity, baseline total amino acid levels were elevated in the control subgroup with obesity and in the type 2 diabetes subgroup without obesity. In all subgroups, amino acid levels decreased by up to 20% in response to glucagon infusion, which resulted in high physiological steady-state glucagon levels (mean concentration: 74 pmol/L, 95% CI [68;79] pmol/L). Following correction for multiple testing, no intergroup differences in changes in amino acid levels reached significance. Conclusion: Obesity and type 2 diabetes status was associated with elevated fasting levels of total amino acids. The glucagon infusion decreased circulating amino acid levels similarly in all subgroups, without significant differences in the response to exogenous glucagon between individuals with and without obesity and type 2 diabetes. Significance statement The hormone glucagon stimulates glucose production from the liver, which may promote hyperglycaemia if glucagon levels are abnormally elevated, as is often seen in type 2 diabetes and obesity. Glucagon levels are closely linked to, and influenced by, the levels of circulating amino acids. To further investigate this link, we measured amino acid levels in individuals with and without obesity and type 2 diabetes before and during an infusion of glucagon. We found that circulating amino acid levels were higher in type 2 diabetes and obesity, and that glucagon infusion decreased amino acid levels in both individuals with and without type 2 diabetes and obesity. The study adds novel information to the link between circulating levels of glucagon and amino acids.

Published
2024
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24. The impact of short-term eucaloric low- and high-carbohydrate diets on liver triacylglycerol content in males with overweight and obesity: a randomized crossover study

Author

London, Amalie, Richter, Michael M, Sjøberg, Kim Anker, Wewer Albrechtsen, Nicolai J, Považan, Michal, Drici, Lylia, Schaufuss, Amanda, Madsen, Lise, Øyen, Jannike, Madsbad, Sten, Holst, Jens Juul, van Hall, Gerrit, Siebner, Hartwig Roman, Richter, Erik A, Kiens, Bente, Lundsgaard, Annemarie, and Bojsen-Møller, Kirstine Nyvold

Published
2024
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25. Co-shared genomic alterations within tumors from patients with both myeloproliferative neoplasms and lymphoma

Author

Johanne M. Holst, Martin B. Pedersen, Marie B. Enemark, Marcus C. Hansen, Patrick R. Noerhave, Trine L. Plesner, Henrik Frederiksen, Michael B. Moeller, Stephen J. Hamilton-Dutoit, Peter Noergaard, Bo K. Mortensen, Hans B. Ommen, Jesper Stentoft, Wayne Tam, Maja Ludvigsen, Wing C. Chan, Nicolai J. Birkbak, Giorgio Inghirami, and Francesco d’Amore

Subjects
Diseases of the blood and blood-forming organs, RC633-647.5
Abstract

Not available.

Published
2024
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27. Hyperglucagonaemia and amino acid alterations in individuals with type 2 diabetes and non-alcoholic fatty liver disease

Author

Iben Rix, Marie L Johansen, Asger Lund, Malte P Suppli, Elizaveta Chabanova, Gerrit van Hall, Jens J Holst, Nicolai J Wewer Albrechtsen, Caroline Kistorp, and Filip K Knop

Subjects
glucagon, amino acids, type 2 diabetes mellitus, non-alcoholic fatty liver disease, humans, Diseases of the endocrine glands. Clinical endocrinology, RC648-665
Abstract

Aims: Hyperglucagonaemia contributes to the pathophysiology in type 2 diabetes (T2D), but the mechanisms behind the inappropriate glucagon secretion are not fully understood. Glucagon and amino acids are regulated in a feedback loop referred to as the liver–α cell axis. Individuals with non-alcoholic fatty liver disease (NAFLD) appear to be glucagon resistant, disrupting the liver–α cell axis resulting in hyperglucagonaemia and hyperaminoacidaemia. We investigated the associations between circulating glucagon, amino acids, and liver fat content in a cohort of individuals with T2D. Methods: We included 110 individuals with T2D in this cross-sectional study. Liver fat content was quantified using 1H magnetic resonance spectroscopy (MRS). Associations between liver fat content and plasma glucagon and amino acids, respectively, were estimated in multivariate linear regression analyses. Results: Individuals with NAFLD (n = 52) had higher plasma glucagon concentrations than individuals without NAFLD (n = 58). The positive association between plasma glucagon concentrations and liver fat content was confirmed in the multivariable regression analyses. Plasma concentrations of isoleucine and glutamate were increased, and glycine and serine concentrations were decreased in individuals with NAFLD. Concentrations of other amino acids were similar between individuals with and without NAFLD, and no clear association was seen between liver fat content and amino acids in the regression analyses. Conclusion: MRS-diagnosed NAFLD in T2D is associated with hyperglucagonaemia and elevated plasma concentrations of isoleucine and glutamate and low plasma concentrations of glycine and serine. Whether NAFLD and glucagon resistance per se induce these changes remains to be elucidated.

Published
2023
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28. Time-Based High-Pass, Low-Pass, Shelf, and Notch Filters

Author

Nicolai J. Dahl, Pere L. Muntal, and Michael A. E. Andersen

Subjects
time-based, filter, modelling, cmos, pll, Electrical engineering. Electronics. Nuclear engineering, TK1-9971
Abstract

This paper presents formulations for time-based first-order and second-order high-pass, shelf, and notch filters. These formulations are an extension to the existing literature where low-pass filters are already developed using a multiphase controlled oscillator in conjunction with a phase detector and charge pump. The presented high-pass filter expands the circuit by introducing a current-controlled delay line (CCDL) that provides a direct path from input to output. By combining the high-pass filter with the low-pass filter, we show that shelf and notch filters can be obtained without an increase in circuit complexity compared to the high-pass filter. The results show good matching between the ideal small signal and the simulated time-based large signal frequency response. The simulated of total harmonic distortion for the filters shows an increase in distortion due to the nonlinearities introduced by the CCDL for the high-pass, notch, and shelf filter compared to the existing low-pass filter. The derivation of the new filter types allows the creation of complex high-order time-based filters by combining multiple first- or second-order filters.

Published
2023
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30. 100 years of glucagon and 100 more

Author

Wewer Albrechtsen, Nicolai J., Holst, Jens J., Cherrington, Alan D., Finan, Brian, Gluud, Lise Lotte, Dean, E. Danielle, Campbell, Jonathan E., Bloom, Stephen R., Tan, Tricia M.-M., Knop, Filip K., and Müller, Timo D.

Published
2023
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31. Glucagon augments the secretion of FGF21 and GDF15 in MASLD by indirect mechanisms

Author

Richter, Michael M., Kemp, Ida M., Heebøll, Sara, Winther-Sørensen, Marie, Kjeldsen, Sasha A.S., Jensen, Nicole J., Nybing, Janus D., Linden, Frederik H., Høgh-Schmidt, Erik, Boesen, Mikael P., Madsbad, Sten, Schiødt, Frank Vinholt, Nørgaard, Kirsten, Schmidt, Signe, Gluud, Lise Lotte, Haugaard, Steen B., Holst, Jens J., Nielsen, Søren, Rungby, Jørgen, and Wewer Albrechtsen, Nicolai J.

Published
2024
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33. Effects of 3 months of 10-h per-day time-restricted eating and 3 months of follow-up on bodyweight and cardiometabolic health in Danish individuals at high risk of type 2 diabetes: the RESET single-centre, parallel, superiority, open-label, randomised controlled trial

Author

Quist, Jonas Salling, Pedersen, Hanne Enghoff, Jensen, Marie Møller, Clemmensen, Kim Katrine Bjerring, Bjerre, Natasja, Ekblond, Trine Spragge, Uldal, Sarah, Størling, Joachim, Wewer Albrechtsen, Nicolai J, Holst, Jens Juul, Torekov, Signe Sørensen, Nyeland, Martin Erik, Vistisen, Dorte, Jørgensen, Marit Eika, Panda, Satchidananda, Brock, Christina, Finlayson, Graham, Blond, Martin Bæk, and Færch, Kristine

Published
2024
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34. Author Correction: The evolution of lung cancer and impact of subclonal selection in TRACERx

Author

Frankell, Alexander M., Dietzen, Michelle, Al Bakir, Maise, Lim, Emilia L., Karasaki, Takahiro, Ward, Sophia, Veeriah, Selvaraju, Colliver, Emma, Huebner, Ariana, Bunkum, Abigail, Hill, Mark S., Grigoriadis, Kristiana, Moore, David A., Black, James R. M., Liu, Wing Kin, Thol, Kerstin, Pich, Oriol, Watkins, Thomas B. K., Naceur-Lombardelli, Cristina, Cook, Daniel E., Salgado, Roberto, Wilson, Gareth A., Bailey, Chris, Angelova, Mihaela, Bentham, Robert, Martínez-Ruiz, Carlos, Abbosh, Christopher, Nicholson, Andrew G., Le Quesne, John, Biswas, Dhruva, Rosenthal, Rachel, Puttick, Clare, Hessey, Sonya, Lee, Claudia, Prymas, Paulina, Toncheva, Antonia, Smith, Jon, Xing, Wei, Nicod, Jerome, Price, Gillian, Kerr, Keith M., Naidu, Babu, Middleton, Gary, Blyth, Kevin G., Fennell, Dean A., Forster, Martin D., Lee, Siow Ming, Falzon, Mary, Hewish, Madeleine, Shackcloth, Michael J., Lim, Eric, Benafif, Sarah, Russell, Peter, Boleti, Ekaterini, Krebs, Matthew G., Lester, Jason F., Papadatos-Pastos, Dionysis, Ahmad, Tanya, Thakrar, Ricky M., Lawrence, David, Navani, Neal, Janes, Sam M., Dive, Caroline, Blackhall, Fiona H., Summers, Yvonne, Cave, Judith, Marafioti, Teresa, Herrero, Javier, Quezada, Sergio A., Peggs, Karl S., Schwarz, Roland F., Van Loo, Peter, Miedema, Daniël M., Birkbak, Nicolai J., Hiley, Crispin T., Hackshaw, Allan, Zaccaria, Simone, Jamal-Hanjani, Mariam, McGranahan, Nicholas, and Swanton, Charles

Published
2024
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35. Glucagon, Metabolic Dysfunction-Associated Steatotic Liver Disease and Amino Acids in Humans and Animals without Diabetes Mellitus—An Evidence Map

Author

Katharina Maruszczak, Pia Koren, Konrad Radzikowski, Thomas Pixner, Malte Palm Suppli, Nicolai J. Wewer Albrechtsen, Daniel Weghuber, and Gabriel Torbahn

Subjects
MASLD, glucagon, amino acid, liver-alpha cell, pediatric, liver, Science
Abstract

Introduction: Health systems are confronted with not only the growing worldwide childhood obesity epidemic but also associated comorbidities. These subsequently cause variations in distinct metabolic pathways, leading to metabolic dysfunction-associated steatotic liver disease (MASLD). The aim of this evidence map is to systematically evaluate the evidence and to identify research gaps on glucagon-induced amino acid (AA) turnover and its metabolic interaction with MASLD. Methodology: A systematic literature search was conducted up to April 2023 in three electronic databases. Studies were required to include at least two of the main research areas, glucagon, AA metabolism and MASLD. Two independent reviewers screened titles and abstracts according to prespecified eligibility criteria, as well as full-text articles. Results are summarized in tables stratified by human and animal studies and study population age. Results: Thirty-four references were ultimately included. The publication years dated back to 1965 showed a great increase from 2012 to 2023. In total, there were 19 animal studies and 15 human studies. Among the human studies, except for two studies in adolescents, all the studies were conducted in adults. In human studies, the methods used to evaluate metabolic changes differed among hyperinsulinemic-euglycemic clamp and oral glucose tolerance tests. Thirteen studies focused on the metabolic effects of MASLD, while only two studies explored the interaction between MASLD, glucagon and AA metabolism in humans. The other 19 studies focused on metabolomics, beta cell function or just one topic of a research area and not on interactions between one another. Conclusion: Research on the interaction between MASLD, glucagon and AA metabolism in humans is sparse and complete lacking in pediatrics. Furthermore, longitudinal studies with a focus on hyperglucagonemia independent of diabetes but related to MASLD present an unambiguous research gap.

Published
2024
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39. Systematic Design of a Pseudodifferential VCO Using Monomial Fitting

Author

Nicolai J. Dahl, Pere L. Muntal, and Michael A. E. Andersen

Subjects
cmos, modeling, vco, time-based, optimisation, Electrical engineering. Electronics. Nuclear engineering, TK1-9971
Abstract

Digital integrated electronics benefits from its higher abstraction level, allowing optimisation methods and automated workflows. However, analogue integrated circuit design is still predominantly done manually, leading to lengthy design cycles. This paper proposes a new systematic design approach for the sizing of analogue integrated circuits to address this issue. The method utilises a surrogate optimisation technique that approximates a simple monomial function based on few simulation results. These monomials are convex and can be optimised using a simple linear optimisation routine, resulting in a single global optimal solution. We show that monomial functions, in many cases, have an analytic relation to integrated circuits, making them well suited for the application. The method is demonstrated by designing a 14 MHz pseudodifferential voltage-controlled oscillator (VCO) with minimised current consumption and is manufactured in a 180 nm process. The measured total current matches the predicted and is lower than that for other similar state-of-the-art VCOs.

Published
2023
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43. Neprilysin activity is increased in metabolic dysfunction-associated steatotic liver disease and normalizes after bariatric surgery or GLP-1 therapy

Author

Kjeldsen, Sasha A.S., Gluud, Lise L., Werge, Mikkel P., Pedersen, Julie S., Bendtsen, Flemming, Alexiadou, Kleopatra, Tan, Tricia, Torekov, Signe S., Iepsen, Eva W., Jensen, Nicole J., Richter, Michael M., Goetze, Jens P., Rungby, Jørgen, Hartmann, Bolette, Holst, Jens J., Holst, Birgitte, Holt, Joachim, Gustafsson, Finn, Madsbad, Sten, Svane, Maria S., Bojsen-Møller, Kirstine N., and Wewer Albrechtsen, Nicolai J.

Published
2023
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45. Antibodies against endogenous retroviruses promote lung cancer immunotherapy

Author

Ng, Kevin W., Boumelha, Jesse, Enfield, Katey S. S., Almagro, Jorge, Cha, Hongui, Pich, Oriol, Karasaki, Takahiro, Moore, David A., Salgado, Roberto, Sivakumar, Monica, Young, George, Molina-Arcas, Miriam, de Carné Trécesson, Sophie, Anastasiou, Panayiotis, Fendler, Annika, Au, Lewis, Shepherd, Scott T. C., Martínez-Ruiz, Carlos, Puttick, Clare, Black, James R. M., Watkins, Thomas B. K., Kim, Hyemin, Shim, Seohee, Faulkner, Nikhil, Attig, Jan, Veeriah, Selvaraju, Magno, Neil, Ward, Sophia, Frankell, Alexander M., Al Bakir, Maise, Lim, Emilia L., Hill, Mark S., Wilson, Gareth A., Cook, Daniel E., Birkbak, Nicolai J., Behrens, Axel, Yousaf, Nadia, Popat, Sanjay, Hackshaw, Allan, Hiley, Crispin T., Litchfield, Kevin, McGranahan, Nicholas, Jamal-Hanjani, Mariam, Larkin, James, Lee, Se-Hoon, Turajlic, Samra, Swanton, Charles, Downward, Julian, and Kassiotis, George

Published
2023
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46. The evolution of non-small cell lung cancer metastases in TRACERx

Author

Al Bakir, Maise, Huebner, Ariana, Martínez-Ruiz, Carlos, Grigoriadis, Kristiana, Watkins, Thomas B. K., Pich, Oriol, Moore, David A., Veeriah, Selvaraju, Ward, Sophia, Laycock, Joanne, Johnson, Diana, Rowan, Andrew, Razaq, Maryam, Akther, Mita, Naceur-Lombardelli, Cristina, Prymas, Paulina, Toncheva, Antonia, Hessey, Sonya, Dietzen, Michelle, Colliver, Emma, Frankell, Alexander M., Bunkum, Abigail, Lim, Emilia L., Karasaki, Takahiro, Abbosh, Christopher, Hiley, Crispin T., Hill, Mark S., Cook, Daniel E., Wilson, Gareth A., Salgado, Roberto, Nye, Emma, Stone, Richard Kevin, Fennell, Dean A., Price, Gillian, Kerr, Keith M., Naidu, Babu, Middleton, Gary, Summers, Yvonne, Lindsay, Colin R., Blackhall, Fiona H., Cave, Judith, Blyth, Kevin G., Nair, Arjun, Ahmed, Asia, Taylor, Magali N., Procter, Alexander James, Falzon, Mary, Lawrence, David, Navani, Neal, Thakrar, Ricky M., Janes, Sam M., Papadatos-Pastos, Dionysis, Forster, Martin D., Lee, Siow Ming, Ahmad, Tanya, Quezada, Sergio A., Peggs, Karl S., Van Loo, Peter, Dive, Caroline, Hackshaw, Allan, Birkbak, Nicolai J., Zaccaria, Simone, Jamal-Hanjani, Mariam, McGranahan, Nicholas, and Swanton, Charles

Published
2023
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47. The evolution of lung cancer and impact of subclonal selection in TRACERx

Author

Frankell, Alexander M., Dietzen, Michelle, Al Bakir, Maise, Lim, Emilia L., Karasaki, Takahiro, Ward, Sophia, Veeriah, Selvaraju, Colliver, Emma, Huebner, Ariana, Bunkum, Abigail, Hill, Mark S., Grigoriadis, Kristiana, Moore, David A., Black, James R. M., Liu, Wing Kin, Thol, Kerstin, Pich, Oriol, Watkins, Thomas B. K., Naceur-Lombardelli, Cristina, Cook, Daniel E., Salgado, Roberto, Wilson, Gareth A., Bailey, Chris, Angelova, Mihaela, Bentham, Robert, Martínez-Ruiz, Carlos, Abbosh, Christopher, Nicholson, Andrew G., Le Quesne, John, Biswas, Dhruva, Rosenthal, Rachel, Puttick, Clare, Hessey, Sonya, Lee, Claudia, Prymas, Paulina, Toncheva, Antonia, Smith, Jon, Xing, Wei, Nicod, Jerome, Price, Gillian, Kerr, Keith M., Naidu, Babu, Middleton, Gary, Blyth, Kevin G., Fennell, Dean A., Forster, Martin D., Lee, Siow Ming, Falzon, Mary, Hewish, Madeleine, Shackcloth, Michael J., Lim, Eric, Benafif, Sarah, Russell, Peter, Boleti, Ekaterini, Krebs, Matthew G., Lester, Jason F., Papadatos-Pastos, Dionysis, Ahmad, Tanya, Thakrar, Ricky M., Lawrence, David, Navani, Neal, Janes, Sam M., Dive, Caroline, Blackhall, Fiona H., Summers, Yvonne, Cave, Judith, Marafioti, Teresa, Herrero, Javier, Quezada, Sergio A., Peggs, Karl S., Schwarz, Roland F., Van Loo, Peter, Miedema, Daniël M., Birkbak, Nicolai J., Hiley, Crispin T., Hackshaw, Allan, Zaccaria, Simone, Jamal-Hanjani, Mariam, McGranahan, Nicholas, and Swanton, Charles

Published
2023
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48. Tracking early lung cancer metastatic dissemination in TRACERx using ctDNA

Author

Abbosh, Christopher, Frankell, Alexander M., Harrison, Thomas, Kisistok, Judit, Garnett, Aaron, Johnson, Laura, Veeriah, Selvaraju, Moreau, Mike, Chesh, Adrian, Chaunzwa, Tafadzwa L., Weiss, Jakob, Schroeder, Morgan R., Ward, Sophia, Grigoriadis, Kristiana, Shahpurwalla, Aamir, Litchfield, Kevin, Puttick, Clare, Biswas, Dhruva, Karasaki, Takahiro, Black, James R. M., Martínez-Ruiz, Carlos, Bakir, Maise Al, Pich, Oriol, Watkins, Thomas B. K., Lim, Emilia L., Huebner, Ariana, Moore, David A., Godin-Heymann, Nadia, L’Hernault, Anne, Bye, Hannah, Odell, Aaron, Kalavakur, Paula, Gomes, Fabio, Patel, Akshay J., Manzano, Elizabeth, Hiley, Crispin T., Carey, Nicolas, Riley, Joan, Cook, Daniel E., Hodgson, Darren, Stetson, Daniel, Barrett, J. Carl, Kortlever, Roderik M., Evan, Gerard I., Hackshaw, Allan, Daber, Robert D., Shaw, Jacqui A., Aerts, Hugo J. W. L., Licon, Abel, Stahl, Josh, Jamal-Hanjani, Mariam, Birkbak, Nicolai J., McGranahan, Nicholas, and Swanton, Charles

Published
2023
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49. Genomic–transcriptomic evolution in lung cancer and metastasis

Author

Martínez-Ruiz, Carlos, Black, James R. M., Puttick, Clare, Hill, Mark S., Demeulemeester, Jonas, Larose Cadieux, Elizabeth, Thol, Kerstin, Jones, Thomas P., Veeriah, Selvaraju, Naceur-Lombardelli, Cristina, Toncheva, Antonia, Prymas, Paulina, Rowan, Andrew, Ward, Sophia, Cubitt, Laura, Athanasopoulou, Foteini, Pich, Oriol, Karasaki, Takahiro, Moore, David A., Salgado, Roberto, Colliver, Emma, Castignani, Carla, Dietzen, Michelle, Huebner, Ariana, Al Bakir, Maise, Tanić, Miljana, Watkins, Thomas B. K., Lim, Emilia L., Al-Rashed, Ali M., Lang, Danny, Clements, James, Cook, Daniel E., Rosenthal, Rachel, Wilson, Gareth A., Frankell, Alexander M., de Carné Trécesson, Sophie, East, Philip, Kanu, Nnennaya, Litchfield, Kevin, Birkbak, Nicolai J., Hackshaw, Allan, Beck, Stephan, Van Loo, Peter, Jamal-Hanjani, Mariam, Swanton, Charles, and McGranahan, Nicholas

Published
2023
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50. Evolutionary characterization of lung adenocarcinoma morphology in TRACERx

Author

Karasaki, Takahiro, Moore, David A., Veeriah, Selvaraju, Naceur-Lombardelli, Cristina, Toncheva, Antonia, Magno, Neil, Ward, Sophia, Bakir, Maise Al, Watkins, Thomas B. K., Grigoriadis, Kristiana, Huebner, Ariana, Hill, Mark S., Frankell, Alexander M., Abbosh, Christopher, Puttick, Clare, Zhai, Haoran, Gimeno-Valiente, Francisco, Saghafinia, Sadegh, Kanu, Nnennaya, Dietzen, Michelle, Pich, Oriol, Lim, Emilia L., Martínez-Ruiz, Carlos, Black, James R. M., Biswas, Dhruva, Campbell, Brittany B., Lee, Claudia, Colliver, Emma, Enfield, Katey S. S., Hessey, Sonya, Hiley, Crispin T., Zaccaria, Simone, Litchfield, Kevin, Birkbak, Nicolai J., Cadieux, Elizabeth Larose, Demeulemeester, Jonas, Van Loo, Peter, Adusumilli, Prasad S., Tan, Kay See, Cheema, Waseem, Sanchez-Vega, Francisco, Jones, David R., Rekhtman, Natasha, Travis, William D., Hackshaw, Allan, Marafioti, Teresa, Salgado, Roberto, Le Quesne, John, Nicholson, Andrew G., McGranahan, Nicholas, Swanton, Charles, and Jamal-Hanjani, Mariam

Published
2023
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