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374 results on '"Nicolae, Alina"'

4. Clinical characteristics and survival outcomes of patients with primary central nervous system lymphoma treated with high-dose methotrexate-based polychemotherapy and consolidation therapies

Author

de Groot, Fleur A., Dekker, Tim J.A., Doorduijn, Jeanette K., Böhringer, Stefan, Brink, Mirian, de Groen, Ruben A.L., de Haan, Lorraine M., Woei-A-Jin, F.J. Sherida H., Noordenbos, Troy, Sijs-Szabo, Aniko, Oudshoorn, Mirjam A., Lam, King H., Diepstra, Arjan, te Boome, Liane C.J., Terpstra, Valeska, Bohmer, Lara H., Nicolae, Alina, Posthuma, Eduardus F.M., Koens, Lianne, Durian, Marc F., Stavast, Jeroen, van der Poel, Marjolein W.M., Hamid, Myrurgia Abdul, Stevens, Wendy B.C., van Rooij, Sjo L.M., Oostvogels, Rimke S., Mühlebner, Angelika, Neelis, Karen J., van den Brand, Michiel, Tousseyn, Thomas, Dierickx, Daan, de Weerdt, Okke, Beeker, Aart, Jansen, Patty M., Kersten, Marie José, Zijlstra, Josée M., Chamuleau, Martine E.D., Veelken, Hendrik, Bromberg, Jacoline C.E., Nijland, Marcel, and Vermaat, Joost S.P.

Published
2024
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8. Nodal cytotoxic peripheral T-cell lymphoma occurs frequently in the clinical setting of immunodysregulation and is associated with recurrent epigenetic alterations

Author

Nicolae, Alina, Bouilly, Justine, Lara, Diane, Fataccioli, Virginie, Lemonnier, François, Drieux, Fanny, Parrens, Marie, Robe, Cyrielle, Poullot, Elsa, Bisig, Bettina, Bossard, Céline, Letourneau, Audrey, Missiaglia, Edoardo, Bonnet, Christophe, Szablewski, Vanessa, Traverse-Glehen, Alexandra, Delfau-Larue, Marie-Hélène, de Leval, Laurence, and Gaulard, Philippe

Published
2022
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11. Frequent mutated B2M, EZH2, IRF8, and TNFRSF14 in primary bone diffuse large B-cell lymphoma reflect a GCB phenotype

Author

de Groen, Ruben A.L., van Eijk, Ronald, Böhringer, Stefan, van Wezel, Tom, Raghoo, Richard, Ruano, Dina, Jansen, Patty M., Briaire-de Bruijn, Inge, de Groot, Fleur A., Kleiverda, Karin, te Boome, Liane, Terpstra, Valeska, Levenga, Henriette, Nicolae, Alina, Posthuma, Eduardus F.M., Focke-Snieders, Isabelle, Hardi, Lizan, den Hartog, Wietske C.E., Bohmer, Lara H., Hogendoorn, Pancras C.W., van den Berg, Anke, Diepstra, Arjan, Nijland, Marcel, Lugtenburg, Pieternella J., Kersten, Marie José, Pals, Steven T., Veelken, Hendrik, Bovée, Judith V.M.G., Cleven, Arjen H.G., and Vermaat, Joost S.P.

Published
2021
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12. Redox Homeostasis and Molecular Biomarkers in Precision Therapy for Cardiovascular Diseases.

Author

Drăgoi, Cristina Manuela, Diaconu, Camelia Cristina, Nicolae, Alina Crenguța, and Dumitrescu, Ion-Bogdan

Subjects
OXIDATIVE stress, REACTIVE oxygen species, MITOCHONDRIAL DNA, HEART diseases, INDIVIDUALIZED medicine
Abstract

Precision medicine is envisioned as the future of cardiovascular healthcare, offering a more tailored and effective method for managing cardiovascular diseases compared to the traditional one-size-fits-all approaches. The complex role of oxidative stress in chronic diseases within the framework of precision medicine was carefully explored, delving into the cellular redox status and its critical involvement in the pathophysiological complexity of cardiovascular diseases (CVDs). The review outlines the mechanisms of reactive oxygen species generation and the function of antioxidants in maintaining redox balance. It emphasizes the elevated reactive oxygen species concentrations observed in heart failure and their detrimental impact on cardiovascular health. Various sources of ROS within the cardiovascular system are examined, including mitochondrial dysfunction, which contributes to oxidative stress and mitochondrial DNA degradation. The article also addresses oxidative stress's role in myocardial remodeling, a process pivotal to the progression of heart diseases. By integrating these aspects, the review underscores the importance of redox homeostasis and identifies molecular biomarkers that can enhance precision therapy for CVDs. The insights provided aim to pave the way for targeted therapeutic strategies that mitigate oxidative stress, thereby improving patient outcomes in cardiovascular medicine. [ABSTRACT FROM AUTHOR]

Published
2024
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13. An Experimental Dynamic Investigation of the Influence of Melatonin, Serotonin and Tryptophan on the Stability of the DNA Structure.

Author

Drăgoi, Cristina Manuela, Zanfirescu, Anca, Dumitrescu, Ion-Bogdan, Ungurianu, Anca, Margină, Denisa Marilena, and Nicolae, Alina-Crenguţa

Subjects
GENE expression, SMALL molecules, DNA structure, ESSENTIAL amino acids, DNA analysis
Abstract

Background: Small molecules play a crucial role in the exploration of physiological pathways and in drug development by targeting deoxyribonucleic acid (DNA). DNA is a central focus for both endogenous and exogenous ligands, which interact directly or indirectly to regulate transcription and replication processes, thus controlling genetic expression in specific cells. Among these molecules, indole derivatives like tryptophan, serotonin, and melatonin are notable for their widespread presence in nature and significant biological effects. Tryptophan, an essential amino acid, serves as a vital structural element in proteins and a precursor for bioactive compounds like serotonin and melatonin, which impact various physiological functions. Methods: Experimental studies have been conducted to reveal the interaction mechanisms of these endogenous indole derivatives with calf thymus DNA (ct-DNA). These investigations involve viscosity measurements and analysis of double-stranded DNA behavior in the presence of indole molecules, using spectrophotometric UV absorption techniques to assess their impact on DNA stability. Additionally, the influence of calcium and magnesium ions on the resulting complexes of these indole derivatives with ct-DNA has been evaluated. Molecular docking validated our findings, offering additional insights into potential DNA–ligand interactions. Utilizing a crystallographic oligomer with an intercalation gap improved docking accuracy, distinguishing intercalation from groove recognition and enhancing assessment precision. Results: Our study offers detailed insights into the interaction patterns of the indole derivatives with DNA and is highly supported by molecular docking analyses: the indole derivatives were predominantly localized between C and G, interacting via π-π interactions and hydrogen bonds and aligning with known data on conventional intercalators. These findings underscore the importance of small compounds' planar structure and appropriate size, facilitating tight insertion between adjacent base pairs and disrupting regular DNA stacking. Conclusions: Indoles' physiological roles and potential as drug candidates targeting specific pathways are highlighted, emphasizing their significance as ubiquitous molecules with the ability to modulate biological effects on DNA structure. [ABSTRACT FROM AUTHOR]

Published
2024
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15. Temporal multiomic modeling reveals a B-cell receptor proliferative program in chronic lymphocytic leukemia

Author

Schleiss, Cedric, Carapito, Raphael, Fornecker, Luc-Matthieu, Muller, Leslie, Paul, Nicodème, Tahar, Ouria, Pichot, Angelique, Tavian, Manuela, Nicolae, Alina, Miguet, Laurent, Mauvieux, Laurent, Herbrecht, Raoul, Cianferani, Sarah, Freund, Jean-Noel, Carapito, Christine, Maumy-Bertrand, Myriam, Bahram, Seiamak, Bertrand, Frederic, and Vallat, Laurent

Published
2021
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18. Gene alterations in epigenetic modifiers and JAK-STAT signaling are frequent in breast implant–associated ALCL

Author

Laurent, Camille, Nicolae, Alina, Laurent, Cécile, Le Bras, Fabien, Haioun, Corinne, Fataccioli, Virginie, Amara, Nadia, Adélaïde, José, Guille, Arnaud, Schiano, Jean-Marc, Tesson, Bruno, Traverse-Glehen, Alexandra, Chenard, Marie-Pierre, Mescam, Lénaïg, Moreau, Anne, Chassagne-Clement, Catherine, Somja, Joan, Escudié, Frédéric, André, Marc, Martin, Nadine, Lacroix, Laetitia, Lemonnier, François, Hamy, Anne-Sophie, Reyal, Fabien, Bannier, Marie, Oberic, Lucie, Prade, Nais, Frénois, François-Xavier, Beldi-Ferchiou, Asma, Delfau-Larue, Marie-Helene, Bouabdallah, Reda, Birnbaum, Daniel, Brousset, Pierre, Xerri, Luc, and Gaulard, Philippe

Published
2020
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19. Morphologic Features of ALK-negative Anaplastic Large Cell Lymphomas With DUSP22 Rearrangements

Author

King, Rebecca L, Dao, Linda N, McPhail, Ellen D, Jaffe, Elaine S, Said, Jonathan, Swerdlow, Steven H, Sattler, Christopher A, Ketterling, Rhett P, Sidhu, Jagmohan S, Hsi, Eric D, Karikehalli, Shridevi, Jiang, Liuyan, Gibson, Sarah E, Ondrejka, Sarah L, Nicolae, Alina, Macon, William R, Dasari, Surendra, Castellar, Edgardo Parrilla, and Feldman, Andrew L

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Oncology and Carcinogenesis, 2.1 Biological and endogenous factors, Aetiology, Adolescent, Adult, Aged, Anaplastic Lymphoma Kinase, Biomarkers, Tumor, Biopsy, Child, Dual-Specificity Phosphatases, Female, Gene Rearrangement, Genetic Predisposition to Disease, Humans, Immunohistochemistry, In Situ Hybridization, Fluorescence, Lymphoma, Large-Cell, Anaplastic, Male, Middle Aged, Mitogen-Activated Protein Kinase Phosphatases, Phenotype, Predictive Value of Tests, Receptor Protein-Tyrosine Kinases, Young Adult, Pathology, Clinical sciences
Abstract

Systemic anaplastic large cell lymphomas (ALCLs) are classified into ALK-positive and ALK-negative types. We recently reported that ALK-negative ALCLs are genetically heterogenous. The largest subset, representing 30% of cases, had rearrangements of the DUSP22 locus. These cases had favorable outcomes similar to ALK-positive ALCL, and superior to other ALK-negative ALCLs. Here, we examined the morphologic features of these cases in more detail. First, we conducted blinded review of hematoxylin and eosin slides of 108 ALCLs from our previous study, scoring cases for the presence of 3 histologic patterns and 5 cell types. Cases then were unblinded and re-reviewed to understand these features further. DUSP22-rearranged ALCLs were more likely than other ALK-negative ALCLs to have so-called doughnut cells (23% vs. 5%; P=0.039), less likely to have pleomorphic cells (23% vs. 49%; P=0.042), and nearly always (95%) had areas with sheet-like growth (common pattern). To examine the reproducibility of these findings, we conducted blinded review of hematoxylin and eosin slides of 46 additional ALK-negative ALCLs using a 0 to 3 scoring system to predict likelihood of DUSP22 rearrangement, the results of which correlated strongly with subsequent findings by fluorescence in situ hybridization (P

Published
2016

21. The 5th edition of the World Health Organization Classification of mature lymphoid and stromal tumors – an overview and update

Author

Attygalle, Ayoma D., primary, Chan, John K. C., additional, Coupland, Sarah E., additional, Du, Ming-Qing, additional, Ferry, Judith A., additional, Jong, Daphne de, additional, Gratzinger, Dita, additional, Lim, Megan S., additional, Naresh, Kikkeri N., additional, Nicolae, Alina, additional, Ott, German, additional, Rosenwald, Andreas, additional, Schuh, Anna, additional, and Siebert, Reiner, additional

Published
2024
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22. Emerging Strategies in Drug Development and Clinical Care in the Era of Personalized and Precision Medicine.

Author

Drăgoi, Cristina Manuela, Nicolae, Alina Crenguța, and Dumitrescu, Ion-Bogdan

Subjects
*GHRELIN receptors, *COMPANION diagnostics, *MEDICAL personnel, *BIOCHEMICAL mechanism of action, *ANGIOTENSIN-receptor blockers, *DEGLUTITION, *CEREBROSPINAL fluid examination
Abstract

This article provides a list of references to scientific articles on various topics related to personalized and precision medicine. The references cover subjects such as cardiovascular risk assessment, biomarkers for cancer, machine learning in healthcare, circadian rhythms, and personalized medicine. [Extracted from the article]

Published
2024
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31. Peripheral T-cell lymphomas of follicular helper T-cell type frequently display an aberrant CD3−/dimCD4+ population by flow cytometry: an important clue to the diagnosis of a Hodgkin lymphoma mimic

Author

Alikhan, Mir, Song, Joo Y, Sohani, Aliyah R, Moroch, Julien, Plonquet, Anne, Duffield, Amy S, Borowitz, Michael J, Jiang, Liuyan, Bueso-Ramos, Carlos, Inamdar, Kedar, Menon, Madhu P, Gurbuxani, Sandeep, Chan, Ernest, Smith, Sonali M, Nicolae, Alina, Jaffe, Elaine S, Gaulard, Philippe, and Venkataraman, Girish

Published
2016
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34. Circadian Rhythms, Chrononutrition, Physical Training, and Redox Homeostasis—Molecular Mechanisms in Human Health.

Author

Drăgoi, Cristina Manuela, Nicolae, Alina Crenguţa, Ungurianu, Anca, Margină, Denisa Marilena, Grădinaru, Daniela, and Dumitrescu, Ion-Bogdan

Subjects
*CIRCADIAN rhythms, *HOMEOSTASIS, *PHYSICAL training & conditioning, *SLEEP interruptions, *BIOLOGICAL rhythms, *LIGHT pollution, *HEALTH behavior
Abstract

A multitude of physiological processes, human behavioral patterns, and social interactions are intricately governed by the complex interplay between external circumstances and endogenous circadian rhythms. This multidimensional regulatory framework is susceptible to disruptions, and in contemporary society, there is a prevalent occurrence of misalignments between the circadian system and environmental cues, a phenomenon frequently associated with adverse health consequences. The onset of most prevalent current chronic diseases is intimately connected with alterations in human lifestyle practices under various facets, including the following: reduced physical activity, the exposure to artificial light, also acknowledged as light pollution, sedentary behavior coupled with consuming energy-dense nutriments, irregular eating frameworks, disruptions in sleep patterns (inadequate quality and duration), engagement in shift work, and the phenomenon known as social jetlag. The rapid evolution of contemporary life and domestic routines has significantly outpaced the rate of genetic adaptation. Consequently, the underlying circadian rhythms are exposed to multiple shifts, thereby elevating the susceptibility to disease predisposition. This comprehensive review endeavors to synthesize existing empirical evidence that substantiates the conceptual integration of the circadian clock, biochemical molecular homeostasis, oxidative stress, and the stimuli imparted by physical exercise, sleep, and nutrition. [ABSTRACT FROM AUTHOR]

Published
2024
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35. Data from The Genetic Basis of Hepatosplenic T-cell Lymphoma

Author

McKinney, Matthew, primary, Moffitt, Andrea B., primary, Gaulard, Philippe, primary, Travert, Marion, primary, De Leval, Laurence, primary, Nicolae, Alina, primary, Raffeld, Mark, primary, Jaffe, Elaine S., primary, Pittaluga, Stefania, primary, Xi, Liqiang, primary, Heavican, Tayla, primary, Iqbal, Javeed, primary, Belhadj, Karim, primary, Delfau-Larue, Marie Helene, primary, Fataccioli, Virginie, primary, Czader, Magdalena B., primary, Lossos, Izidore S., primary, Chapman-Fredricks, Jennifer R., primary, Richards, Kristy L., primary, Fedoriw, Yuri, primary, Ondrejka, Sarah L., primary, Hsi, Eric D., primary, Low, Lawrence, primary, Weisenburger, Dennis, primary, Chan, Wing C., primary, Mehta-Shah, Neha, primary, Horwitz, Steven, primary, Bernal-Mizrachi, Leon, primary, Flowers, Christopher R., primary, Beaven, Anne W., primary, Parihar, Mayur, primary, Baseggio, Lucile, primary, Parrens, Marie, primary, Moreau, Anne, primary, Sujobert, Pierre, primary, Pilichowska, Monika, primary, Evens, Andrew M., primary, Chadburn, Amy, primary, Au-Yeung, Rex K.H., primary, Srivastava, Gopesh, primary, Choi, William W. L., primary, Goodlad, John R., primary, Aurer, Igor, primary, Basic-Kinda, Sandra, primary, Gascoyne, Randy D., primary, Davis, Nicholas S., primary, Li, Guojie, primary, Zhang, Jenny, primary, Rajagopalan, Deepthi, primary, Reddy, Anupama, primary, Love, Cassandra, primary, Levy, Shawn, primary, Zhuang, Yuan, primary, Datta, Jyotishka, primary, Dunson, David B., primary, and Davé, Sandeep S., primary

Published
2023
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36. Supplementary Tables S1 - S10 from The Genetic Basis of Hepatosplenic T-cell Lymphoma

Author

McKinney, Matthew, primary, Moffitt, Andrea B., primary, Gaulard, Philippe, primary, Travert, Marion, primary, De Leval, Laurence, primary, Nicolae, Alina, primary, Raffeld, Mark, primary, Jaffe, Elaine S., primary, Pittaluga, Stefania, primary, Xi, Liqiang, primary, Heavican, Tayla, primary, Iqbal, Javeed, primary, Belhadj, Karim, primary, Delfau-Larue, Marie Helene, primary, Fataccioli, Virginie, primary, Czader, Magdalena B., primary, Lossos, Izidore S., primary, Chapman-Fredricks, Jennifer R., primary, Richards, Kristy L., primary, Fedoriw, Yuri, primary, Ondrejka, Sarah L., primary, Hsi, Eric D., primary, Low, Lawrence, primary, Weisenburger, Dennis, primary, Chan, Wing C., primary, Mehta-Shah, Neha, primary, Horwitz, Steven, primary, Bernal-Mizrachi, Leon, primary, Flowers, Christopher R., primary, Beaven, Anne W., primary, Parihar, Mayur, primary, Baseggio, Lucile, primary, Parrens, Marie, primary, Moreau, Anne, primary, Sujobert, Pierre, primary, Pilichowska, Monika, primary, Evens, Andrew M., primary, Chadburn, Amy, primary, Au-Yeung, Rex K.H., primary, Srivastava, Gopesh, primary, Choi, William W. L., primary, Goodlad, John R., primary, Aurer, Igor, primary, Basic-Kinda, Sandra, primary, Gascoyne, Randy D., primary, Davis, Nicholas S., primary, Li, Guojie, primary, Zhang, Jenny, primary, Rajagopalan, Deepthi, primary, Reddy, Anupama, primary, Love, Cassandra, primary, Levy, Shawn, primary, Zhuang, Yuan, primary, Datta, Jyotishka, primary, Dunson, David B., primary, and Davé, Sandeep S., primary

Published
2023
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37. Supplementary Methods, Figures S1 - S9 from The Genetic Basis of Hepatosplenic T-cell Lymphoma

Author

McKinney, Matthew, primary, Moffitt, Andrea B., primary, Gaulard, Philippe, primary, Travert, Marion, primary, De Leval, Laurence, primary, Nicolae, Alina, primary, Raffeld, Mark, primary, Jaffe, Elaine S., primary, Pittaluga, Stefania, primary, Xi, Liqiang, primary, Heavican, Tayla, primary, Iqbal, Javeed, primary, Belhadj, Karim, primary, Delfau-Larue, Marie Helene, primary, Fataccioli, Virginie, primary, Czader, Magdalena B., primary, Lossos, Izidore S., primary, Chapman-Fredricks, Jennifer R., primary, Richards, Kristy L., primary, Fedoriw, Yuri, primary, Ondrejka, Sarah L., primary, Hsi, Eric D., primary, Low, Lawrence, primary, Weisenburger, Dennis, primary, Chan, Wing C., primary, Mehta-Shah, Neha, primary, Horwitz, Steven, primary, Bernal-Mizrachi, Leon, primary, Flowers, Christopher R., primary, Beaven, Anne W., primary, Parihar, Mayur, primary, Baseggio, Lucile, primary, Parrens, Marie, primary, Moreau, Anne, primary, Sujobert, Pierre, primary, Pilichowska, Monika, primary, Evens, Andrew M., primary, Chadburn, Amy, primary, Au-Yeung, Rex K.H., primary, Srivastava, Gopesh, primary, Choi, William W. L., primary, Goodlad, John R., primary, Aurer, Igor, primary, Basic-Kinda, Sandra, primary, Gascoyne, Randy D., primary, Davis, Nicholas S., primary, Li, Guojie, primary, Zhang, Jenny, primary, Rajagopalan, Deepthi, primary, Reddy, Anupama, primary, Love, Cassandra, primary, Levy, Shawn, primary, Zhuang, Yuan, primary, Datta, Jyotishka, primary, Dunson, David B., primary, and Davé, Sandeep S., primary

Published
2023
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40. Contributors

Author

Arber, Daniel A., Bagg, Adam, Bhagat, Govind, Borowitz, Michael J., Brown, Laura E., Brynes, Russell K., Calvo, Katherine R., Campo, Elias, Caponetti, Gabriel C., Chabot-Richards, Devon, Chan, John K.C., Chen, Yi-Hua, Cherian, Sindhu, Cook, James R., Czader, Magdalena, de Leval, Laurence, Duffield, Amy S., Duncavage, Eric, Egan, Caoimhe, Facchetti, Fabio, Feldman, Andrew L., Fend, Falko, Fernandez-Pol, Sebastian, Ferry, Judith A., Filie, Armando C., Foucar, Kathryn, Gaulard, Philippe, George, Tracy I., Gibson, Sarah E., Gonzalez-Farré, Blanca, Gratzinger, Dita, Hagiya, Ashley S., Hartmann, Sylvia, Hasserjian, Robert P., Horny, Hans-Peter, Hsi, Eric D., Insuasti-Beltran, Giovanni, Jaffe, Elaine S., Jansen, Patty M., Jares, Pedro, Jevremovic, Dragan, Kadin, Marshall E., Kempf, Werner, King, Rebecca L., Klapper, Wolfram, Ko, Young Hyeh, Küppers, Ralf, Kvasnicka, Hans Michael, Lamant-Rochaix, Laurence, Laurent, Camille, LeBoit, Philip E., Linden, Michael A., Llao-Cid, Laura, Lorsbach, Robert B., Louissant, Abner, Jr., Mollejo, Manuela, Morice, William G., II, Mrózek, Krzysztof, Nakashima, Megan, Nicolae, Alina, Orazi, Attilio, Oschlies, Ilske, Ott, German, Petrella, Tony, Pincus, Laura B., Piris, Miguel A., Pittaluga, Stefania, Porwit, Anna, Prakash, Sonam, Quintanilla-Martinez, Leticia, Racke, Frederick, Raffeld, Mark, Rech, Karen L., Reichard, Kaaren K., Rimsza, Lisa M., Rogers, Heesun J., Rosenthal, Nancy S., Said, Jonathan W., Salaverria, Itziar, Schafernak, Kristian T., Schrader, Anne M.R., Scott, David W., Shi, Min, Siebert, Reiner, Silva, Oscar, Sohani, Aliyah R., Sotlar, Karl, Swerdlow, Steven H., Tam, Wayne, Tzankov, Alexandar, Valent, Peter, Venkataraman, Girish, Vergara-Lluri, Maria (Ria) E., Vijayanarayanan, Anjanaa, Wang, Hao-Wei, Wang, Sa A., Weinberg, Olga K., Willemze, Rein, Yoshino, Tadashi, Yuan, Constance M., Zhang, Qian-Yun, and Zukerberg, Lawrence R.

Published
2025
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43. ALK-negative anaplastic large cell lymphoma is a genetically heterogeneous disease with widely disparate clinical outcomes

Author

Parrilla Castellar, Edgardo R., Jaffe, Elaine S., Said, Jonathan W., Swerdlow, Steven H., Ketterling, Rhett P., Knudson, Ryan A., Sidhu, Jagmohan S., Hsi, Eric D., Karikehalli, Shridevi, Jiang, Liuyan, Vasmatzis, George, Gibson, Sarah E., Ondrejka, Sarah, Nicolae, Alina, Grogg, Karen L., Allmer, Cristine, Ristow, Kay M., Wilson, Wyndham H., Macon, William R., Law, Mark E., Cerhan, James R., Habermann, Thomas M., Ansell, Stephen M., Dogan, Ahmet, Maurer, Matthew J., and Feldman, Andrew L.

Published
2014
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46. Melatonin: A Silent Regulator of the Glucose Homeostasis

Author

Dragoi, Cristina Manuela, primary, Arsene, Andreea Letitia, additional, Dinu-Pirvu, Cristina Elena, additional, Dumitrescu, Ion Bogdan, additional, Popa, Daniela Elena, additional, Burcea-Dragomiroiu, George T.A., additional, Udeanu, Denisa Ioana, additional, Timnea, Olivia Carmen, additional, Velescu, Bruno Stefan, additional, and Nicolae, Alina Crenguta, additional

Published
2017
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48. Differential Phosphoproteomics Deciphers Physiopathology of High-Risk Mantle Cell Lymphoma

Author

Fuseau, Charline, primary, Baldacini, Mathieu, additional, Nicolae, Alina, additional, Miguet, Laurent, additional, Mauvieux, Laurent, additional, Vallat, Laurent, additional, Domon-Dell, Claire, additional, Tavian, Manuela, additional, Freund, Jean-Noel, additional, Cianferani, Sarah, additional, Schaeffer-Reiss, Christine, additional, Fornecker, Luc-Matthieu, additional, and Rolland, Delphine C.M., additional

Published
2022
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