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1. Peptidomimetics designed to bind to RAS effector domain are promising cancer therapeutic compounds

2. RAD51 is a druggable target that sustains replication fork progression upon DNA replication stress.

3. α4-α5 Helices on Surface of KRAS Can Accommodate Small Compounds That Increase KRAS Signaling While Inducing CRC Cell Death

4. Pleiotropic Roles of Calmodulin in the Regulation of KRas and Rac1 GTPases: Functional Diversity in Health and Disease

5. Modeling and subtleties of K-Ras and Calmodulin interaction.

6. SUMO regulates p21Cip1 intracellular distribution and with p21Cip1 facilitates multiprotein complex formation in the nucleolus upon DNA damage.

7. Detection of Phospho-KRAS by Electrophoretic Mobility Change in Human Cell Lines and in Tumor Samples from Nude Mice Grafts

8. p21 as a transcriptional co-repressor of S-phase and mitotic control genes.

17. Data from Phosphorylation at Ser-181 of Oncogenic KRAS Is Required for Tumor Growth

22. Data from Different S/M Checkpoint Responses of Tumor and Non–Tumor Cell Lines to DNA Replication Inhibition

25. Supplementary Tables 1 - 3 from Phosphorylation at Ser-181 of Oncogenic KRAS Is Required for Tumor Growth

26. Toward understanding calmodulin plasticity by molecular dynamics

27. KRAS phosphorylation regulates cell polarization and tumorigenic properties in colorectal cancer

28. Pleiotropic Roles of Calmodulin in the Regulation of KRas and Rac1 GTPases: Functional Diversity in Health and Disease

29. Nucleotide depletion reveals the impaired ribosomebiogenesis checkpoint as a barrier against DNA damage

30. OZF is a Claspin-interacting protein essential to maintain the replication fork progression rate under replication stress

31. Proteasome Inhibition Reduces Proliferation, Collagen Expression, and Inflammatory Cytokine Production in Nasal Mucosa and Polyp Fibroblasts

32. Protective Effect of Structurally Diverse Grape Procyanidin Fractions against UV-Induced Cell Damage and Death

33. CaM interaction and Ser181 phosphorylation as new K-Ras signaling modulators

34. K-Ras4B phosphorylation at Ser181 is inhibited by calmodulin and modulates K-Ras activity and function

35. A clathrin-dependent pathway leads to KRas signaling on late endosomes en route to lysosomes

36. Increased messenger ribonucleic acid expression of the cyclin-dependent kinase inhibitor p27Kip1 in cleavage-stage human embryos exhibiting developmental arrest

37. Centrosome aberrations in human mammary epithelial cells driven by cooperative interactions between p16 INK4a deficiency and telomere-dependent genotoxic stress

38. Chk1- and Claspin-Dependent but ATR/ATM– and Rad17-Independent DNA Replication Checkpoint Response in HeLa Cells

39. The Diverging Roles of Calmodulin and PKC in the Regulation of p21 Intracellular Localization

40. Heterogeneous nuclear ribonucleoprotein A2 is a SET-binding protein and a PP2A inhibitor

41. Calcium and Calmodulin Function in the Cell Nucleus

42. ATM/ATR-independent inhibition of cyclin B accumulation in response to hydroxyurea in nontransformed cell lines is altered in tumour cell lines

43. Identification of the nuclear localization signal of p21cip1 and consequences of its mutation on cell proliferation

44. Increased expression of the cyclin-dependent kinase inhibitor p27 in cleavage-stage human embryos exhibiting developmental arrest

45. Modulation of the Ras/Raf/MEK/ERK pathway by Ca2+, and Calmodulin

46. Calmodulin Regulates Intracellular Trafficking of Epidermal Growth Factor Receptor and the MAPK Signaling Pathway

47. The p21Cip1protein, a cyclin inhibitor, regulates the levels and the intracellular localization of CDC25A in mice regenerating livers

48. Phosphorylation at ser-181 of oncogenic KRAS is required for tumor growth

49. Calmodulin Binds to K-Ras, but Not to H- or N-Ras, and Modulates Its Downstream Signaling

50. Differential association of p21 Cip1 and p27 Kip1 with cyclin E-CDK2 during rat liver regeneration

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