100 results on '"Navari N"'
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2. Fatty Acid Synthase expression promotes the malignant features of cholangiocarcinoma cells and predicts shorter survival in patients
3. Hepatocyte depletion of ERK5 impairs the response to lipotoxic oxidative stress resulting in defective insulin receptor signaling
4. Fatty Acid Synthase expression promotes the malignant features of cholangiocarcinoma cells and predicts shorter survival in patients
5. Macrophage MerTK promotes a profibrogenic cross-talk with hepatic stellate cells via soluble mediators
6. Mitochondrial oxidative metabolism contributes to maintain a cancer stem cell phenotype in cholangiocarcinoma
7. Mitochondrial oxidative metabolism contributes to maintain a cancer stem cell phenotype in cholangiocarcinoma
8. Acquisition of stem-like features in human cholangiocarcinoma is associated with an oxidative metabolism
9. Antitumor Activity of a Novel Fibroblast Growth Factor Receptor Inhibitor for Intrahepatic Cholangiocarcinoma
10. ANTITUMOR ACTIVITY OF A NOVEL FGFR INHIBITOR IN INTRAHEPATIC CHOLANGIOCARCINOMA
11. Acquisition of stem-like features in human cholangiocarcinoma is associated with an oxidative metabolism
12. Acquisition of stem-like features in human cholangiocarcinoma is associated with an oxidative glucose metabolism
13. Adequateness of contact ultrasonic bone analyser in assessing bone density in women: Comparison with dual energy X-ray absorption
14. Contact ultrasound attenuation of the calcaneus in italian women: normative data
15. Involvement of the IDO-1 pathway in experimental NASH
16. Dietary supplementation with olive oil with or without n-3 PUFA differentially affect inflammation and progression of experimental nonalcoholic steatohepatitis
17. Cytoskeletal rearrangement by the mitogen-activated protein kinaseERK5 interferes with the motile and invasive phenotype ofhepatocellular carcinoma cells
18. A NOVEL ROLE FOR THE KYNURENINE PATHWAY IN EXPERIMENTAL STEATOHEPATITIS
19. The mammalian target of rapamycin mediates the angiogenic and chemotactic effects of leptin in human hepatic stellate cells
20. P94 c-Rel AND p27Kip ARE DOWNSTREAM TARGETS OF ERK5 IN HEPATOCELLULAR CARCINOMA (HCC) IN VIVO AND IN VITRO
21. P291 INHIBITION OF INDOLEAMINE 2,3-DIOXYGENASE AMELIORATES INFLAMMATION AND FIBROSIS IN EXPERIMENTAL STEATOHEPATITIS
22. Inhibition of indoleamine 2,3-dioxygenase ameliorates inflammation and fibrosis in experimental steatohepatitis
23. 1039 ACTIVATION OF THE MITOGEN-ACTIVATED PROTEIN KINASE ERK5 REGULATES THE DEVELOPMENT AND GROWTH OF HEPATOCELLULAR CARCINOMA
24. OC-04 Activation of the mitogen-activated protein kinase ERK5 regulates the development and growth of hepatocellular carcinoma
25. 1256 DIETARY SUPPLEMENTATION WITH OLIVE OIL WITH OR WITHOUT N-3 PUFA DIFFERENTIALLY AFFECTS INFLAMMATION AND PROGRESSION OF EXPERIMENTAL NONALCOHOLIC STEATOHEPATITIS
26. F-32 Dietary supplementation with olive oil with or without n-3 PUFA differentially affects inflammation and progression of experimental nonalcoholic steatohepatitis
27. 206 CYTOSKELETAL REARRANGEMENT BY THE MITOGEN-ACTIVATED PROTEIN KINASE ERK5 INTERFERES WITH THE MOTILE AND INVASIVE PHENOTYPE OF HEPATOCELLULAR CARCINOMA CELLS
28. OC-5 Cytoskeletal rearrangement by the mitogen-activated protein kinase ERK5 interferes with the motile and invasive phenotype of hepatocellular carcinoma cells
29. 892 ERK5 IS OVEREXPRESSED IN EXPERIMENTAL LIVER INJURY AND IN HEPATOCELLULAR CARCINOMA (HCC) AND IS MODULATED BY SOLUBLE FACTORS IN HCC CELLS
30. F.N.24 ERK5 IS OVEREXPRESSED IN HEPATOCELLULAR CARCINOMA (HCC), AND IS MODULATED BY SOLUBLE FACTORS IN HCC CELLS
31. Activation of the TSC/m-TOR pathway by PDGF or leptin is required for expression of VEGF by human hepatic stellate cells
32. [99] EXPRESSION OF ANGIOGENIC CYTOKINES BY LEPTIN REQUIRES A TSC/M-TOR PATHWAY AND GENERATION OF REACTIVE OXYGEN SPECIES (ROS) IN HUMAN HEPATIC STELLATE CELLS
33. Expression of angiogenic cytokines by leptin requires the TSC/m-TOR pathway and generation of reactive oxygen species (ROS) in human hepatic stellate cells
34. c-Rel and p27Kip are downstream targets of ERK5 in hepatocellular carcinoma (HCC) in vivo and in vitro
35. Evidence for bone mass and body fat distribution relationship in post-menopausal obese women
36. Cytoskeletal rearrangement by the mitogen-activated protein kinase ERK5 interferes with the motile and invasive phenotype of hepatocellular carcinoma cells
37. ERK5 IS OVEREXPRESSED IN EXPERIMENTAL LIVER INJURY AND IN HEPATOCELLULAR CARCINOMA (HCC) AND IS MODULATED BY SOLUBLE FACTORS IN HCC CELLS
38. 332 Activation of ERK5, a member of the mapk family, differentially regulates proliferation and migration of hepatic stellate cells
39. Mitochondrial oxidative metabolism contributes to a cancer stem cell phenotype in cholangiocarcinoma
40. CXCR7 contributes to the aggressive phenotype of cholangiocarcinoma cells
41. Antitumor Activity of a Novel Fibroblast Growth Factor Receptor Inhibitor for Intrahepatic Cholangiocarcinoma
42. Mitochondrial injury during normothermic regional perfusion (NRP) and hypothermic oxygenated perfusion (HOPE) in a rodent model of DCD liver transplantation.
43. Integrating conventional and complementary treatments in cancer care: The process within the public healthcare system of the region of Tuscany, Italy.
44. Transjugular intrahepatic Porto-systemic shunt positively influences the composition and metabolic functions of the gut microbiota in cirrhotic patients.
45. Cylindrical Implant Versus Tapered Implant: A Comparative Study.
46. Macrophage MerTK promotes profibrogenic cross-talk with hepatic stellate cells via soluble mediators.
47. Extracellular Signal-Regulated Kinase 5 Regulates the Malignant Phenotype of Cholangiocarcinoma Cells.
48. Mitochondrial oxidative metabolism contributes to a cancer stem cell phenotype in cholangiocarcinoma.
49. Antitumor Activity of a Novel Fibroblast Growth Factor Receptor Inhibitor for Intrahepatic Cholangiocarcinoma.
50. CXCR7 contributes to the aggressive phenotype of cholangiocarcinoma cells.
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