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19. A Nuclear Receptor-mediated Choleretic Action of Fibrates is Associated with Enhanced Canalicular Membrane Fluidity and Transporter Activity Mediating Bile Acid-independent Bile Secretion

20. Unique anti-apoptotic action of biliary lecithin against bile acid-induced cholangiocyte apoptosis is based primarily upon the inhibition asbt-mediated uptake and Mrp3-mediated wash-out of bile acids

21. Angiotensin II type la receptor deficient mice show slow progression of liver fibrosis induced by carbon tetrachloride: A direct evidence for fibrogenetic role of renin-angiotensin system

22. A ligand for peroxisome prolifertor-activated receptor gamma inhibits human cholangioma cell growth in association with G1 arrest of cell-cycle and apoptosis: A potential molecular-targetting strategy for cholangioma

23. Cytoprotective action of hydrophilic bile salts is associated with anti-apoptotic effects on cholangiocyte apoptosis induced by cytotoxic bile salts: Implication to clinical use for cholestatic disorder

24. Hydrophilic bile salts cytoprotectively function against cyclosporine A caused cholestasis through enhacing canalicular membrane fluidity and transporter activity

25. Choleretic action of diosgenin is based upon the increases in canalicurar membrane fluidity and transporter activity mediating bile acid independent bile flow

29. A Combination Therapy With Simvastatin and Ursodeoxycholic Acid Is More Effective for Cholesterol Gallstone Dissolution Than Is Ursodeoxycholic Acid Monotherapy

31. Effects of cerivastatin sodium, a new HMG-CoA reductase inhibitor, on biliary lipid metabolism in patients with hypercholesterolemia

32. A Nuclear Receptor Ligand Down-Regulates Cytosolic Phospholipase A2 Expression to Reduce Bile Acid–Induced Cyclooxygenase 2 Activity in Cholangiocytes: Implications of Anticarcinogenic Action of Farnesoid X Receptor Agonists

33. 391 HCV core protein directly modulates hepatic fatty acid metabolism through enhancing carnitine palmitoyl transferase 1A activity to cause steatosis: relation to compensatory overexpression of rescue molecules, nuclear receptor PPARA and ABC transporter MDR3

36. [Mechanism of HCV cell entry mediated by envelope and receptor proteins].

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