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This experiment aimed to investigate the impact of varying fiber lengths on the flexural strength of coconut fiber composites with epoxy resin. Coconut fiber composite panels were produced using rectangular molds. The effects of various fiber sizes, especially 2, 4, and 6 mm, respectively, were compared with fiberless and continuous fiber polymer composites on the flexural strength of Natural Fiber-reinforced Polymer Composites (NFPC). The lowest flexural strength is 10.04 MPa, was obtained for the NFPC sample with a fiber length of 2 mm, while the highest flexural strength value was 10.70 MPa with a fiber length of 6 mm. Generally, flexural strength is influenced by variations in fiber size but not significantly. The flexural modulus and flexural strength values exhibit the same trend. Fiber pullouts are visible on the fracture surface of the sample, and voids in the form of trapped gas are also apparent on the sample surface. Short fiber size is one of the causes of fiber pullout, which can reduce the flexural strength of short fiber polymers.

NF-protocadherin (NFPC)-mediated cell-- cell adhesion plays a critical role in vertebrate neural tube formation. NFPC is also expressed during the period of axon tract formation, but little is known about its function in axonogenesis. Here we have tested the role of NFPC and its cytosolic cofactor template-activating factor 1 (TAF1) in the emergence of the Xenopus retinotectal projection. NFPC is expressed in the developing retina and optic pathway and is abundant in growing retinal axons. Inhibition of NFPC function in developing retinal ganglion cells (RGCs) severely reduces axon initiation and elongation and suppresses dendrite genesis. Furthermore, an identical phenotype occurs when TAF1 function is blocked. These data provide evidence that NFPC regulates axon initiation and elongation and indicate a conserved role for TAF1, a transcriptional regulator, as a downstream cytosolic effector of NFPC in RGCs. [ABSTRACT FROM AUTHOR]