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1. Epigenenomic and transcriptomic characterization of NPM1-mutated CN-AML subtypes

2. Genomic analysis of primary and secondary myelofibrosis redefines the prognostic impact of ASXL1 mutations: a FIM study

3. Leukemic evolution of polycythemia vera and essential thrombocythemia: genomic profiles predict time to transformation

4. CALR‐mutated patients with low allele burden represent a specific subtype of essential thrombocythemia: A study on behalf of FIM and GBMHM.

6. Incidence of ATRX mutations in myelodysplastic syndromes, the value of microcytosis

11. Gains of EPOR and ERG genes in adult erythroleukaemia

14. Myeloid malignancies: mutations, models and management

15. Mutations in ASXL1 are associated with poor prognosis across the spectrum of malignant myeloid diseases

16. Sequential mutational evaluation of CALR ‐mutated myeloproliferative neoplasms with thrombocytosis reveals an association between CALR allele burden evolution and disease progression

17. Targeted molecular characterization shows differences between primary and secondary myelofibrosis

18. Common origin of sequential cutaneous CD30+ lymphoproliferations with nodal involvement evidenced by genome-wide clonal evolution

19. Mutation patterns in essential thrombocythemia, polycythemia vera and secondary myelofibrosis

20. Combined mutations of ASXL1, CBL, FLT3, IDH1, IDH2, JAK2, KRAS, NPM1, NRAS, RUNX1, TET2 and WT1 genes in myelodysplastic syndromes and acute myeloid leukemias

21. Sequential mutational evaluation of CALR ‐mutated myeloproliferative neoplasms with thrombocytosis reveals an association between CALR allele burden evolution and disease progression.

23. Number of Mutations and Type of Prior Myeloproliferative Neoplasm Are Prognostic Factors in Acute Myeloid Leukemia Post Myeloproliferative Neoplasms

24. Impact of gene mutations on treatment response and prognosis of acute myeloid leukemia secondary to myeloproliferative neoplasms

25. Genomic analysis of primary and secondary myelofibrosis redefines the prognostic impact of ASXL1mutations: a FIM study

26. Common origin of sequential cutaneous CD30+ lymphoproliferations with nodal involvement evidenced by genome‐wide clonal evolution.

27. Genomic analysis of myeloproliferative neoplasms in chronic and acute phases

28. Syndrome myéloprolifératif avec polyglobulie et translocation t(6;8)(q27;p11) : cas clinique et revue de la littérature

29. Clinical, Cytogenetic and Molecular Characterization of a 96 MDS and AML Cohort with TP53Mutation

30. Role of ASXL1 and TP53 mutations in the molecular classification and prognosis of acute myeloid leukemias with myelodysplasia-related changes

31. Incidence of Atrx Mutations in Myelodysplastic Syndromes (MDS)

32. Differential Association of Calreticulin Type 1 and Type 2 Mutations with Myelofibrosis and Essential Thrombocytemia: Relevance for Disease Evolution

33. Prognostic significance of myelodysplasia-related changes according to the WHO classification among ELN-intermediate-risk AML patients

35. A recurrent chromosome translocation breakpoint in breast and pancreatic cancer cell lines targets the neuregulin/ NRG1 gene

39. Mutation analysis of ASXL1, CBL, DNMT3A, IDH1, IDH2, JAK2, MPL, NF1, SF3B1, SUZ12, and TET2 in myeloproliferative neoplasms

40. Alteration of cohesin genes in myeloid diseases

41. Combined mutations of ASXL1, CBL, FLT3, IDH1, IDH2, JAK2, KRAS, NPM1, NRAS, RUNX1, TET2 and WT1 genes in myelodysplastic syndromes and acute myeloid leukemias

43. Familial Malignant Hematological Disorders: A Systematic Assessment in 216 Consecutive Patients with Acute Leukaemia.

44. Dual lympho-myeloproliferative disorder in a patient with t(8;22) with BCR-FGFR1 gene fusion

45. NACA is a positive regulator of human erythroid-cell differentiation

47. A recurrent chromosome translocation breakpoint in breast and pancreatic cancer cell lines targets the neuregulin/NRG1 gene

48. Reciprocal translocations in breast tumor cell lines: Cloning of a t(3;20) that targets the FHIT gene

49. Combined mutations of ASXL1, CBL, FLT3, IDH1,IDH2, JAK2, KRAS, NPM1, NRAS, RUNX1, TET2 andWT1 genes in myelodysplastic syndromes andacute myeloid leukemias.

50. Venetoclax‐based non‐intensive induction followed by allogenic stem‐cell transplantation in elderly acute myeloid leukemia patients with adverse cytogenetics.

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