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1. Alpha-lipoic acid supplementation corrects pathological alterations in cellular models of pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels

4. Mitochondrial Quality Control via Mitochondrial Unfolded Protein Response (mtUPR) in Ageing and Neurodegenerative Diseases

5. Actin Polymerization Defects Induce Mitochondrial Dysfunction in Cellular Models of Nemaline Myopathies

7. Antioxidants Prevent Iron Accumulation and Lipid Peroxidation, but Do Not Correct Autophagy Dysfunction or Mitochondrial Bioenergetics in Cellular Models of BPAN

8. Patient-Derived Cellular Models for Polytarget Precision Medicine in Pantothenate Kinase-Associated Neurodegeneration

9. Patient-Derived Cellular Models for Polytarget Precision Medicine in Pantothenate Kinase-Associated Neurodegeneration

10. Neurodegeneration, Mitochondria, and Antibiotics

11. Actin Polymerization Defects Induce Mitochondrial Dysfunction in Cellular Models of Nemaline Myopathies

12. mtUPR Modulation as a Therapeutic Target for Primary and Secondary Mitochondrial Diseases

13. Vicious cycle of lipid peroxidation and iron accumulation in neurodegeneration

14. Pantothenate and L-Carnitine Supplementation Improves Pathological Alterations in Cellular Models of KAT6A Syndrome

15. Alpha-Lipoic acid supplementation corrects pathological alterations in cellular models of pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels

16. Pantothenate and L-carnitine Supplementation Corrects Pathological Alterations in Cellular Models of KAT6A Syndrome

17. Additional file 1 of Alpha-lipoic acid supplementation corrects pathological alterations in cellular models of pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels

19. Activation of the Mitochondrial Unfolded Protein Response: A New Therapeutic Target?

20. Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels.

21. Vitamin E prevents lipid peroxidation and iron accumulation in PLA2G6-Associated Neurodegeneration

22. Pterostilbene in Combination With Mitochondrial Cofactors Improve Mitochondrial Function in Cellular Models of Mitochondrial Diseases

23. Additional file 1 of UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases

24. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases

25. Additional file 1 of Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels

26. Mitochondria and Antibiotics: For Good or for Evil?

27. Additional file 1 of Down regulation of the expression of mitochondrial phosphopantetheinyl-proteins in pantothenate kinase-associated neurodegeneration: pathophysiological consequences and therapeutic perspectives

28. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases.

29. From Mitochondria to Atherosclerosis: The Inflammation Path

30. Coenzyme Q10 Analogues: Benefits and Challenges for Therapeutics

32. Down Regulation Of The Expression Of Mitochondrial Phosphopantetheinyl-Proteins In Pantothenate Kinase-Associated Neurodegeneration: Pathophysiological Consequences and Therapeutic Perspectives

33. Precision Medicine in Rare Diseases

34. Coenzyme Q10: Novel Formulations and Medical Trends

35. Coenzyme Q 10 Analogues: Benefits and Challenges for Therapeutics.

36. Coenzyme Q 10 : Novel Formulations and Medical Trends.

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