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1. Membrane Atg8ylation, stress granule formation, and MTOR regulation during lysosomal damage

2. ATG5 provides host protection acting as a switch in the atg8ylation cascade between autophagy and secretion

3. Stress granules and mTOR are regulated by membrane atg8ylation during lysosomal damage

4. Mammalian hybrid pre-autophagosomal structure HyPAS generates autophagosomes

6. MERIT, a cellular system coordinating lysosomal repair, removal and replacement

7. Galectin-3 Coordinates a Cellular System for Lysosomal Repair and Removal

8. Galectins control MTOR and AMPK in response to lysosomal damage to induce autophagy

9. Galectins Control mTOR in Response to Endomembrane Damage

13. Membrane Atg8ylation, stress granule formation, and MTOR regulation during lysosomal damage

14. The role of ATG5 beyond Atg8ylation and autophagy.

16. Phosphorylation of Syntaxin 17 by TBK1 Controls Autophagy Initiation

17. Galectins control MTOR and AMPK in response to lysosomal damage to induce autophagy

21. Cellular and molecular mechanism for secretory autophagy

22. DedicatedSNAREs and specializedTRIMcargo receptors mediate secretory autophagy

23. TRIMs and Galectins Globally Cooperate and TRIM16 and Galectin-3 Co-direct Autophagy in Endomembrane Damage Homeostasis

24. Pharmaceutical screen identifies novel target processes for activation of autophagy with a broad translational potential

26. Stress granules and mTOR are regulated by membrane atg8ylation during lysosomal damage.

27. Noncanonical roles of ATG5 and membrane atg8ylation in retromer assembly and function.

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