271 results on '"Morgan, Iain M."'
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2. Inhibition of human cytomegalovirus entry into mucosal epithelial cells
3. A new role for human papillomavirus 16 E2: Mitotic activation of the DNA damage response to promote viral genome segregation
4. Identifying predictors of HPV‐related head and neck squamous cell carcinoma progression and survival through patient‐derived models
5. The membrane-associated ubiquitin ligase MARCHF8 stabilizes the human papillomavirus oncoprotein E7 by degrading CUL1 and UBE2L3 in head and neck cancer
6. A human papillomavirus 16 E2-TopBP1 dependent SIRT1-p300 acetylation switch regulates mitotic viral and human protein levels
7. E7-mediated repression of miR-203 promotes LASP1-dependent proliferation in HPV-positive cervical cancer
8. Human Papillomavirus 16 replication converts SAMHD1 into a homologous recombination factor and promotes its recruitment to replicating viral DNA
9. The membrane-associated ubiquitin ligase MARCHF8 stabilizes the human papillomavirus oncoprotein E7 by degrading CUL1 and UBE2L3 in head and neck cancer
10. PTPN14 degradation by high-risk human papillomavirus E7 limits keratinocyte differentiation and contributes to HPV-mediated oncogenesis
11. Aurora kinases are a novel therapeutic target for HPV-positive head and neck cancers
12. HPV16 genome structure analysis in oropharyngeal cancer PDXs identifies tumors with integrated and episomal genomes
13. Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function
14. Patients with integrated HPV16 in head and neck cancer show poor survival
15. Direct interaction with the BRD4 carboxyl-terminal motif (CTM) and TopBP1 is required for human papillomavirus 16 E2 association with mitotic chromatin and plasmid segregation function
16. Figure S5 from Pharmacologic Inhibition of SHP2 Blocks Both PI3K and MEK Signaling in Low-epiregulin HNSCC via GAB1
17. Table S1 from Pharmacologic Inhibition of SHP2 Blocks Both PI3K and MEK Signaling in Low-epiregulin HNSCC via GAB1
18. Data from Pharmacologic Inhibition of SHP2 Blocks Both PI3K and MEK Signaling in Low-epiregulin HNSCC via GAB1
19. HPV upregulates MARCHF8 ubiquitin ligase and inhibits apoptosis by degrading the death receptors in head and neck cancer
20. Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle
21. Failure to interact with Brd4 alters the ability of HPV16 E2 to regulate host genome expression and cellular movement
22. Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response
23. Pharmacologic Inhibition of SHP2 Blocks Both PI3K and MEK Signaling in Low-epiregulin HNSCC via GAB1
24. HPV E6 regulates therapy responses in oropharyngeal cancer by repressing the PGC-1α/ERRα axis
25. HPV E6 upregulates MARCHF8 ubiquitin ligase and inhibits apoptosis by degrading the death receptors in head and neck cancer
26. Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during Mitosis
27. A Critical Role for p53 during the HPV16 Life Cycle
28. Abstract 2329: Variable HPV E6 levels among oropharyngeal cancers govern therapy response via the PGC1/ERR axis
29. Human papilloma virus E6 regulates therapy responses in oropharyngeal cancer by repressing the PGC-1α/ERRα axis
30. Adenovirus 12 E4orf6 inhibits ATR activation by promoting TOPBP1 degradation
31. Interaction with TopBP1 mediates human papillomavirus 16 E2 plasmid segregation/retention function and stability during the viral life cycle
32. A critical role for E2-p53 interaction during the HPV16 life cycle
33. CK2 Phosphorylation of Human Papillomavirus 16 E2 on Serine 23 Promotes Interaction with TopBP1 and Is Critical for E2 Interaction with Mitotic Chromatin and the Viral Life Cycle
34. Expression of TopBP1 in canine mammary neoplasia in relation to histological type, Ki67, ERα and p53
35. Mutations in the Jun Delta Region Suggest an Inverse Correlation Between Transformation and Transcriptional Activation
36. Immunohistochemical expression of TopBP1 in feline mammary neoplasia in relation to histological grade, Ki67, ERα and p53
37. CK2 phosphorylation of human papillomavirus 16 E2 on serine 23 promotes interaction with TopBP1 and is critical for E2 plasmid retention function
38. Restoring the DREAM Complex Inhibits the Proliferation of High-Risk HPV Positive Human Cells
39. Human Papillomavirus 16 (HPV16) E2 Repression of TWIST1 Transcription Is a Potential Mediator of HPV16 Cancer Outcomes
40. UVB irradiation reduces the half-life and transactivation potential of the human papillomavirus 16 E2 protein
41. Werner Syndrome Protein (WRN) Regulates Cell Proliferation and the Human Papillomavirus 16 Life Cycle during Epithelial Differentiation
42. Using a Human Papillomavirus Model to Study DNA Replication and Repair of Wild Type and Damaged DNA Templates in Mammalian Cells
43. Human papillomavirus 16 E2 repression of TWIST1 transcription is a potential mediator of HPV16 cancer outcomes
44. Activating the DNA Damage Response and Suppressing Innate Immunity: Human Papillomaviruses Walk the Line
45. The Relationship between Estrogen-Related Signaling and Human Papillomavirus Positive Cancers
46. Estrogen Attenuates the Growth of Human Papillomavirus-Positive Epithelial Cells
47. Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene Transcription
48. Human papillomavirus 16 E6 and E7 synergistically repress innate immune gene transcription
49. The BPV-4 co-carcinogen quercetin induces cell cycle arrest and up-regulates transcription from the LCR of BPV-4
50. A novel technique with enhanced detection and quantitation of HPV-16 E1- and E2-mediated DNA replication
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