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Unmanned aerial systems (UAS) have recently gained popularity, and they are envisioned as an integral parts of the current and future wireless and mobile-radio networks. Despite the exhaustive research on air-to-ground channels, there are insufficient studies, experimental campaigns and general channel models related to air-to-space (A2S) and air-to-air (A2A) wireless links. This paper presents a comprehensive review of the available channel models and path-loss prediction for A2S and A2A communications. Specific case studies attempting to extend current models' parameters and provide important knowledge of the channel behavior in combination with UAV flight characteristics are also provided. A time-series rain-attenuation synthesizer is also presented that describes quite accurately the impact of the troposphere at frequencies above 10 GHz. This specific model can be also applied to both A2S and A2A wireless links. Finally, scientific challenges and gaps that can be used for future research on the upcoming 6G networks are highlighted.

This article introduces a unified and detailed methodology for interference assessment between coexisting fixed satellite service (FSS) and broadcast satellite service (BSS) with spectrum sharing at the Ka-band. The interference analysis is presented along with a step-by-step algorithm for the calculation of the carrier-to-interference ratio (CIR). The proposed procedure takes into consideration the near-field effect of ground-satellite-terminal antennas since these may reside at very close distances. Furthermore, numerical results are delivered so as to assess the CIR in relation to the relative geometry and the technical characteristics of the satellite terminals. A real application scenario is also provided along with measurements so as to validate the recommended methodology. Finally, mitigation techniques are proposed for the protection of the victim stations and operation under harmful interference conditions.

Aims: Cardiac implantable electronic devices (CIEDs) are susceptible to electromagnetic interference (EMI). Smartwatches and their chargers could be a possible source of EMI. We sought to assess whether the latest generation smartwatches and their chargers interfere with proper CIED function., Methods and Results: We included consecutive CIED recipients in two centres. We tested two latest generation smartwatches (Apple Watch and Samsung Galaxy Watch) and their charging cables for potential EMI. The testing was performed under continuous electrocardiogram recording and real-time device telemetry, with nominal and 'worst-case' settings. In vitro magnetic field measurements were performed to assess the emissions from the tested devices, initially in contact with the probe and then at a distance of 10 cm and 20 cm. In total, 171 patients with CIEDs (71.3% pacemakers-28.7% implantable cardioverter-defibrillators) from five manufacturers were enrolled (63.2% males, 74.8 ± 11.4 years), resulting in 684 EMI tests. No EMI was identified in any patient either under nominal or 'worst-case scenario' programming. The peak magnetic flux density emitted by the smartwatches was similar to the background noise level (0.81 μT) even when in contact with the measuring probe. The respective values for the chargers were 4.696 μΤ and 4.299 μΤ for the Samsung and Apple chargers, respectively, which fell at the background noise level when placed at 20 cm and 10 cm, respectively., Conclusion: Two latest generation smartwatches and their chargers resulted in no EMI in CIED recipients. The absence of EMI in conjunction with the extremely low intensity of magnetic fields emitted by these devices support the safety of their use by CIED patients., (Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2020. For permissions, please email: journals.permissions@oup.com.)

This paper presents the experimental configuration and procedure as well as the in-vitro assessment of Jurkat T-cells response to 1966 MHz exposure of modulated and unmodulated electromagnetic signals within a Gigahertz Transverse Electro-Magnetic (GTEM) cell. Different combinations of electric field intensity, exposure duration and modulation schemes were applied. Exposures at continuous wave (CW) signal at low intensity levels (3 V/m) did not induce any significant DNA damage, but a slight increase was observed for extreme stress levels (76.4 V/m). On the other hand, the results indicate that, at both, low and high electric field intensity UMTS (Universal Mobile Telecommunications System) signal could be statistically related to DNA damage in-vitro. Nevertheless, further experiments are required, increasing the statistical number of samples and recruiting more DNA damage endpoints before conclusive statements are drawn.

Valid characterization of carotid atherosclerosis (CA) is a crucial public health issue, which would limit the major risk held by CA for both patient safety and state economies. CA is typically diagnosed and assessed using duplex ultrasonography (US). Elastrography Imaging (EI) is a promising US technique for quantifying tissue elasticity (ES). In this work, we investigated the association between ES of carotid atherosclerotic lesions, derived from EI, and texture indices, calculated from US image analysis. US and EI images of 23 atherosclerotic plaques (16 patients) were analyzed. Texture features derived from US image analysis (Gray-Scale Median (GSM), plaque area (A) and co-occurrence-matrixderived features) were calculated. Statistical analysis revealed associations between US texture features and EI measured indices. This result indicates accordance in US and EI techniques and states the promising role of EI in diagnosis of CA.

Over 1000 monoclonal antibodies (MAB), previously characterized in terms of reactivity with various haemopoietic cells and including representatives from each of the currently known Clusters of Differentiation (CD), have now been evaluated by us for reactivity against human endothelial cells. In the current study, screening was performed on both fixed, and on non-fixed, endothelial cells derived and cultured from umbilical cord veins. Results indicate that endothelial cells co-express a wide variety of antigens that are otherwise characterized by expression on haemopoietic cells. The most intense and consistent degree of reactivity was seen using MAB to CD-9, CD-13, CD-29, CD-31, CD-44, CD-46, CD-51, CD-54 and CD-55, although reactivity was also observed using MAB belonging to CD-41, CD-47, CD-49, CD-61 and CD-71, in addition to a number of non-clustered MAB.

Human endothelial cells, cultured from umbilical cord veins, have been evaluated for expression of a large number of cell surface antigens with known haemopoietic, particularly myeloid, cell distribution. This was achieved by evaluating endothelial reactivity (using non-fixed cells) with groups of monoclonal antibodies (MAB) belonging to distinct Clusters of Differentiation (CD), as defined by the Third International Workshop on Leukocyte Differentiation Antigens (ILWS). Results indicate that many antigens known to be present on haemopoietic cells, including those on platelets, are co-expressed on endothelial cells. The most intense degree of reactivity was seen using MAB to CD-9 and CD-13, although significant reactivity was also observed using MAB to CD-31 and CD-32. Data also suggests weak binding to endothelial cells of MAB belonging to CD-14, CD-15 and CD-16. A number of unclustered MAB reactive with haemopoietic antigens can also be shown to bind to endothelial cells.