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8. Common single-base insertions in the VNTR of the carboxyl ester lipase (CEL) gene are benign and also likely to arise somatically in the exocrine pancreas

9. Characterization of CEL-DUP2: Complete duplication of the carboxyl ester lipase gene is unlikely to influence risk of chronic pancreatitis

11. Congenital hyperinsulinism

12. Genome-wide association meta-analyses combining multiple risk phenotypes provide insights into the genetic architecture of cutaneous melanoma susceptibility

19. Medfødt hyperinsulinisme.

23. Supplementary Data 2 from Generation of Prostate Tumor–Initiating Cells Is Associated with Elevation of Reactive Oxygen Species and IL-6/STAT3 Signaling

24. Data from Generation of Prostate Tumor–Initiating Cells Is Associated with Elevation of Reactive Oxygen Species and IL-6/STAT3 Signaling

25. Supplementary Materials and Methods, Figure Legends, Figures 1-2, and Tables 1-2 from Generation of Prostate Tumor–Initiating Cells Is Associated with Elevation of Reactive Oxygen Species and IL-6/STAT3 Signaling

30. The genetic risk factor CEL-HYB1 causes proteotoxicity and chronic pancreatitis in mice

33. P13-kinase mutation linked to insulin and growth factor resistance in vivo

35. Genome-wide meta-analysis identifies five new susceptibility loci for cutaneous malignant melanoma

36. Long-Range Gene Regulation Links Genomic Type 2 Diabetes and Obesity Risk Regions to HHEX, SOX4, and IRX3

38. The Effect on Melanoma Risk of Genes Previously Associated With Telomere Length

39. Two New Mutations in the CEL Gene Causing Diabetes and Hereditary Pancreatitis: How to Correctly Identify MODY8 Cases

41. Fine mapping of genetic susceptibility loci for melanoma reveals a mixture of single variant and multiple variant regions

43. Protein misfolding in combination with other risk factors in CEL-HYB1-mediated chronic pancreatitis

44. The position of single-base deletions in the VNTR sequence of the carboxyl ester lipase (CEL) gene determines proteotoxicity

47. Carboxyl-ester lipase maturity-onset diabetes of the young is associated with development of pancreatic cysts and upregulated MAPK signaling in secretin-stimulated duodenal fluid

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