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4. P1284: LIQUID BIOPSY BY ULTRA-DEEP SEQUENCING PLUS PET-TC MONITORING IN REAL-LIFE FOLLICULAR LYMPHOMA PATIENTS

Author

Jiménez-Ubieto, A., primary, Poza Santaella, M., additional, Martin, A., additional, Heredia, Y., additional, Dorado, S., additional, Figaredo, G., additional, Rufian, L., additional, Rodriguez, A., additional, Sarandeses, P., additional, Baumann, T., additional, Grande, C., additional, Bárcena, C., additional, Martinez-Lopez, J., additional, Rosa, J.-M., additional, Rapado, I., additional, Ayala, R., additional, Gallardo, M., additional, Sánchez, R., additional, Carrillo, J., additional, Calbacho, M., additional, Parrilla, L., additional, Juarez, A., additional, Mollejo, M., additional, Casado, L. F., additional, Wang, C., additional, Rodriguez, M., additional, De Toledo, P., additional, and Barrio, S., additional

Published
2022
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5. Whole-exome sequencing in splenic marginal zone lymphoma reveals mutations in genes involved in marginal zone differentiation

Author

Martínez, N, Almaraz, C, Vaqué, J P, Varela, I, Derdak, S, Beltran, S, Mollejo, M, Campos-Martin, Y, Agueda, L, Rinaldi, A, Kwee, I, Gut, M, Blanc, J, Oscier, D, Strefford, J C, Martinez-Lopez, J, Salar, A, Sole, F, Rodriguez-Peralto, J L, Diez-Tascón, C, García, J F, Fraga, M, Sebastián, E, Alvés, J, Menárguez, J, González-Carreró, J, Casado, L F, Bayes, M, Bertoni, F, Gut, I, and Piris, M A

Published
2014
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6. 829MO A gene signature to predict risk of transformation in patients with follicular lymphoma

Author

Fernandez-Miranda, I., primary, Pedrosa, L., additional, Gómez, S., additional, González-Rincón, J., additional, Espinet, B., additional, Climent, F., additional, de la Cruz, F., additional, Martín-Acosta, P., additional, Colomo, L., additional, Salar, A., additional, García-Cosio, M., additional, García-Arroyo, F.R., additional, Llanos, M., additional, Yánguas, N., additional, Sequero Lopez, S., additional, Mercadal, S., additional, Navarro, B., additional, Mollejo, M., additional, Provencio Pulla, M., additional, and Sánchez-Beato, M., additional

Published
2021
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7. Over 30% of patients with splenic marginal zone lymphoma express the same immunoglobulin heavy variable gene: ontogenetic implications

Author

Bikos, V, Darzentas, N, Hadzidimitriou, A, Davis, Z, Hockley, S, Traverse-Glehen, A, Algara, P, Santoro, A, Gonzalez, D, Mollejo, M, Dagklis, A, Gangemi, F, Bosler, D S, Bourikas, G, Anagnostopoulos, A, Tsaftaris, A, Iannitto, E, Ponzoni, M, Felman, P, Berger, F, Belessi, C, Ghia, P, Papadaki, T, Dogan, A, Degano, M, Matutes, E, Piris, M A, Oscier, D, and Stamatopoulos, K

Published
2012
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12. MUTATIONAL ANALYSIS OF THE DIFFERENT NODAL PERIPHERAL T‐CELL LYMPHOMA SUBCLASSES

Author

Tomas‐Roca, L, primary, Rodriguez, M, additional, Alonso‐Alonso, R, additional, Cereceda, L, additional, Rodriguez‐Pinilla, S. M, additional, Borregón, J, additional, Manso, R, additional, Villaescusa, T, additional, Córdoba, R, additional, Sánchez‐Beato, M, additional, Fernández‐Miranda, I, additional, Barcena, C, additional, García, J. F, additional, Mollejo, M, additional, García‐Cosio, M, additional, Martin‐Acosta, P, additional, Climent, F, additional, Caballero, D, additional, and Piris, M. A, additional

Published
2021
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13. A GENE SIGNATURE TO PREDICT RISK OF TRANSFORMATION IN PATIENTS WITH FOLLICULAR LYMPHOMA

Author

Fernandez‐Miranda, I., primary, Dominguez, M., additional, Pedrosa, L., additional, Gomez, S., additional, Gonzalez‐Rincon, J., additional, Espinet, B., additional, Climent, F., additional, Cruz, F. de la, additional, Martin‐Acosta, P., additional, Colomo, L., additional, Salar, A., additional, Garcia‐Cosio, M., additional, Garcia‐Arroyo, F. R., additional, Llanos, M., additional, Yanguas, N., additional, Sequero, S., additional, Mercadal, S., additional, Navarro, B., additional, Piris, M. A., additional, Mollejo, M., additional, Provencio, M., additional, and Sanchez‐Beato, M., additional

Published
2021
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14. GENETIC AND PHENOTYPIC ATTRIBUTES OF SPLENIC MARGINAL ZONE LYMPHOMA

Author

Bonfiglio, F., primary, Bruscaggin, A., additional, Guidetti, F., additional, Terzi di Bergamo, L., additional, Faderl, M., additional, Spina, V., additional, Condoluci, A., additional, Bonomini, L., additional, Forestieri, G., additional, Koch, R., additional, Piffaretti, D., additional, Pini, K., additional, Pirosa, M. C., additional, Cittone, M. G., additional, Arribas, A., additional, Lucioni, M., additional, Ghilardi, G., additional, Wu, W., additional, Arcaini, L., additional, Baptista, M. J., additional, Bastidas, G., additional, Bea, S., additional, Boldorini, R., additional, Broccoli, A., additional, Canzonieri, V., additional, Cascione, L., additional, Ceriani, L., additional, Cogliatti, S., additional, Derenzini, E., additional, Devizzi, L., additional, Dietrich, S., additional, Elia, A. R., additional, Facchetti, F., additional, Gaidano, G., additional, Garcia, J. F., additional, Gerber, B., additional, Ghia, P., additional, Silva, M. G., additional, Gritti, G., additional, Guidetti, A., additional, Hitz, F., additional, Inghirami, G., additional, Ladetto, M., additional, Lopez‐Guillermo, A., additional, Lucchini, E., additional, Maiorana, A., additional, Marasca, R., additional, Matutes, E., additional, Meignin, V., additional, Merli, M., additional, Moccia, A., additional, Mollejo, M., additional, Montalban, C., additional, Novak, U., additional, Oscier, D. G., additional, Passamonti, F., additional, Piazza, F., additional, Pizzolitto, S., additional, Sabattini, E., additional, Salles, G., additional, Santambrogio, E., additional, Scarfó, L., additional, Stathis, A., additional, Stüssi, G., additional, Geyer, J. T., additional, Tapia, G., additional, Thieblemont, C., additional, Tousseyn, T., additional, Tucci, A., additional, Visco, C., additional, Vitolo, U., additional, Zenz, T., additional, Zinzani, P. L., additional, Khiabanian, H., additional, Calcinotto, A., additional, Bertoni, F., additional, Bhagat, G., additional, Campo, E., additional, Leval, L., additional, Dirnhofer, S., additional, Pileri, S. A., additional, Piris, M. Án., additional, Traverse‐Glehen, A., additional, Tzankov, A., additional, Paulli, M., additional, Ponzoni, M., additional, Mazzucchelli, L., additional, Cavalli, F., additional, Zucca, E., additional, and Rossi, D., additional

Published
2021
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20. Disease-biased and shared characteristics of the immunoglobulin gene repertoires in marginal zone B cell lymphoproliferations

Author

Xochelli, A. Bikos, V. Polychronidou, E. Galigalidou, C. Agathangelidis, A. Charlotte, F. Moschonas, P. Davis, Z. Colombo, M. Roumelioti, M. Sutton, L.-A. Groenen, P. van den Brand, M. Boudjoghra, M. Algara, P. Traverse-Glehen, A. Ferrer, A. Stalika, E. Karypidou, M. Kanellis, G. Kalpadakis, C. Mollejo, M. Pangalis, G. Vlamos, P. Amini, R.-M. Pospisilova, S. Gonzalez, D. Ponzoni, M. Anagnostopoulos, A. Giudicelli, V. Lefranc, M.-P. Espinet, B. Panagiotidis, P. Piris, M.A. Du, M.-Q. Rosenquist, R. Papadaki, T. Belessi, C. Ferrarini, M. Oscier, D. Tzovaras, D. Ghia, P. Davi, F. Hadzidimitriou, A. Stamatopoulos, K.

Abstract

The B cell receptor immunoglobulin (Ig) gene repertoires of marginal zone (MZ) lymphoproliferations were analyzed in order to obtain insight into their ontogenetic relationships. Our cohort included cases with MZ lymphomas (n = 488), i.e. splenic (SMZL), nodal (NMZL) and extranodal (ENMZL), as well as provisional entities (n = 76), according to the WHO classification. The most striking Ig gene repertoire skewing was observed in SMZL. However, restrictions were also identified in all other MZ lymphomas studied, particularly ENMZL, with significantly different Ig gene distributions depending on the primary site of involvement. Cross-entity comparisons of the MZ Ig sequence dataset with a large dataset of Ig sequences (MZ-related or not; n = 65 837) revealed four major clusters of cases sharing homologous (‘public’) heavy variable complementarity-determining region 3. These clusters included rearrangements from SMZL, ENMZL (gastric, salivary gland, ocular adnexa), chronic lymphocytic leukemia, but also rheumatoid factors and non-malignant splenic MZ cells. In conclusion, different MZ lymphomas display biased immunogenetic signatures indicating distinct antigen exposure histories. The existence of rare public stereotypes raises the intriguing possibility that common, pathogen-triggered, immune-mediated mechanisms may result in diverse B lymphoproliferations due to targeting versatile progenitor B cells and/or operating in particular microenvironments. Copyright © 2018 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd. Copyright © 2018 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.

Published
2019

22. Disease-biased and shared characteristics of the immunoglobulin gene repertoires in marginal zone B cell lymphoproliferations

Author

Xochelli, A., Bikos, V., Polychronidou, E., Galigalidou, C., Agathangelidis, A., Charlotte, F., Moschonas, P., Davis, Z., Colombo, M., Roumelioti, M., Sutton, L.A., Groenen, P.J., Brand, M. van den, Boudjoghra, M., Algara, P., Traverse-Glehen, A., Ferrer, A., Stalika, E., Karypidou, M., Kanellis, G., Kalpadakis, C., Mollejo, M., Pangalis, G., Vlamos, P., Amini, R.M., Pospisilova, S., Gonzalez, D., Ponzoni, M., Anagnostopoulos, A., Giudicelli, V., Lefranc, M.P., Espinet, B., Panagiotidis, P., Piris, M.A., Du, M.Q., Rosenquist, R., Papadaki, T., Belessi, C., Ferrarini, M., Oscier, D., Tzovaras, D., Ghia, P., Davi, F., Hadzidimitriou, A., Stamatopoulos, K., Xochelli, A., Bikos, V., Polychronidou, E., Galigalidou, C., Agathangelidis, A., Charlotte, F., Moschonas, P., Davis, Z., Colombo, M., Roumelioti, M., Sutton, L.A., Groenen, P.J., Brand, M. van den, Boudjoghra, M., Algara, P., Traverse-Glehen, A., Ferrer, A., Stalika, E., Karypidou, M., Kanellis, G., Kalpadakis, C., Mollejo, M., Pangalis, G., Vlamos, P., Amini, R.M., Pospisilova, S., Gonzalez, D., Ponzoni, M., Anagnostopoulos, A., Giudicelli, V., Lefranc, M.P., Espinet, B., Panagiotidis, P., Piris, M.A., Du, M.Q., Rosenquist, R., Papadaki, T., Belessi, C., Ferrarini, M., Oscier, D., Tzovaras, D., Ghia, P., Davi, F., Hadzidimitriou, A., and Stamatopoulos, K.

Abstract

Contains fulltext : 203155.pdf (Publisher’s version ) (Closed access), The B cell receptor immunoglobulin (Ig) gene repertoires of marginal zone (MZ) lymphoproliferations were analyzed in order to obtain insight into their ontogenetic relationships. Our cohort included cases with MZ lymphomas (n = 488), i.e. splenic (SMZL), nodal (NMZL) and extranodal (ENMZL), as well as provisional entities (n = 76), according to the WHO classification. The most striking Ig gene repertoire skewing was observed in SMZL. However, restrictions were also identified in all other MZ lymphomas studied, particularly ENMZL, with significantly different Ig gene distributions depending on the primary site of involvement. Cross-entity comparisons of the MZ Ig sequence dataset with a large dataset of Ig sequences (MZ-related or not; n = 65 837) revealed four major clusters of cases sharing homologous ('public') heavy variable complementarity-determining region 3. These clusters included rearrangements from SMZL, ENMZL (gastric, salivary gland, ocular adnexa), chronic lymphocytic leukemia, but also rheumatoid factors and non-malignant splenic MZ cells. In conclusion, different MZ lymphomas display biased immunogenetic signatures indicating distinct antigen exposure histories. The existence of rare public stereotypes raises the intriguing possibility that common, pathogen-triggered, immune-mediated mechanisms may result in diverse B lymphoproliferations due to targeting versatile progenitor B cells and/or operating in particular microenvironments. Copyright (c) 2018 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.

Published
2019

24. Advanced‐stage mycosis fungoides: role of the signal transducer and activator of transcription 3, nuclear factor‐κB and nuclear factor of activated T cells pathways

Author

Pérez, C., primary, Mondéjar, R., additional, García‐Díaz, N., additional, Cereceda, L., additional, León, A., additional, Montes, S., additional, Durán Vian, C., additional, Pérez Paredes, M.G., additional, González‐Morán, A., additional, Miguel, V., additional, Sanz Anquela, J.M., additional, Frias, J., additional, Limeres, M.A., additional, González, L.M., additional, Martín Dávila, F., additional, Beltrán, M., additional, Mollejo, M., additional, Méndez, J.R., additional, González, M.A., additional, González García, J., additional, López, R., additional, Gómez, A., additional, Izquierdo, F., additional, Ramos, R., additional, Camacho, C., additional, Rodriguez‐Pinilla, S.M., additional, Martínez, N., additional, Vaqué, J.P., additional, Ortiz‐Romero, P.L., additional, and Piris, M.A., additional

Published
2019
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25. DIFFUSE LARGE B-CELL LYMPHOMA SURVIVAL PROGNOSTICATION, A COMPARATIVE ANALYSIS OF CELL OF ORIGIN VS. MYC/BCL2 EXPRESSION

Author

Rodriguez, M., primary, Fernandez-Miranda, I., additional, Mondejar, R., additional, Capote, J., additional, Rodriguez-Pinilla, S., additional, Cereceda, L., additional, Alonso, R., additional, Cordoba, R., additional, Provencio, M., additional, Martin-Acosta, P., additional, Sanchez, A., additional, Pedrosa, L., additional, Gómez, S., additional, Piris-Villaespesa, M., additional, Garcia-Cosio, M., additional, Quero, C., additional, Llanos, M., additional, Barcena, C., additional, Fraga, M., additional, Camacho, F., additional, Castro, Y., additional, Garcia, J., additional, Mollejo, M., additional, Climent, F., additional, Mayordomo, E., additional, Bacalari, E., additional, Olmedilla, G., additional, Sánchez-Beato, M., additional, and Piris, M., additional

Published
2019
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26. A PTCL GENE SIGNATURE CAPTURING STROMAL AND NEOPLASTIC DATA STRATIFIES PTCL/NOS AND AITL INTO DIFFERENT GROUPS WITH VARIABLE SURVIVAL PROBABILITY

Author

Rodriguez, M., primary, Alonso, R., additional, Rodriguez-Pinilla, S., additional, Cereceda, L., additional, Villaescusa, T., additional, Cordoba, R., additional, Sánchez-Beato, M., additional, Fernandez-Miranda, I., additional, Bárcena, C., additional, García, J., additional, Mollejo, M., additional, Garcia-Cosio, M., additional, Martin-Acosta, P., additional, Climent, F., additional, Caballero, D., additional, Kessler, L., additional, Scholz, C., additional, Gualberto, A., additional, Mondéjar, R., additional, and Piris, M., additional

Published
2019
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27. MULTI-OMICS LANDSCAPE OF SPLENIC MARGINAL ZONE LYMPHOMA (SMZL) - INTERIM ANALYSIS OF IELSG46 STUDY

Author

Bruscaggin, A., primary, Mollejo, M., additional, Tapia, G., additional, Gomes da Silva, M., additional, Novak, U., additional, Dietrich, S., additional, Ponzoni, M., additional, Rambaldi, A., additional, Corradini, P., additional, Vitolo, U., additional, Merli, M., additional, Tzankov, A., additional, Cogliatti, S., additional, Montalban, C., additional, Marasca, R., additional, de Leval, L., additional, Visco, C., additional, Baptista, M.J., additional, Tousseyn, T., additional, Facchetti, F., additional, Paulli, M., additional, Mazzucchelli, L., additional, Bea, S., additional, Oscier, D., additional, Zinzani, P., additional, Bhagat, G., additional, Inghirami, G., additional, Gaidano, G., additional, Traverse-Glehen, A., additional, Thieblemont, C., additional, Piris, M.A., additional, Cavalli, F., additional, Arcaini, L., additional, Zucca, E., additional, and Rossi, D., additional

Published
2019
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30. Advanced‐stage mycosis fungoides: role of the signal transducer and activator of transcription 3, nuclear factor‐κB and nuclear factor of activated T cells pathways.

Author

Pérez, C., Mondéjar, R., García‐Díaz, N., Cereceda, L., León, A., Montes, S., Durán Vian, C., Pérez Paredes, M.G., González‐Morán, A., Miguel, V., Sanz Anquela, J.M., Frias, J., Limeres, M.A., González, L.M., Martín Dávila, F., Beltrán, M., Mollejo, M., Méndez, J.R., González, M.A., and González García, J.

Subjects
T cells, NF-kappa B, CUTANEOUS T-cell lymphoma, PATHOLOGY, MYCOSIS fungoides, TRANSDUCERS, JAK-STAT pathway
Abstract

Summary: Background: The malignant mechanisms that control the development of cutaneous T‐cell lymphoma (CTCL) are beginning to be identified. Recent evidence suggests that disturbances in specific intracellular signalling pathways, such as RAS–mitogen‐activated protein kinase, T‐cell receptor (TCR)–phospholipase C gamma 1 (PLCG1)–nuclear factor of activated T cells (NFAT) and Janus kinase (JAK)–signal transducer and activator of transcription (STAT), may play an essential role in the pathogenesis of CTCL. Objectives: To investigate the mechanisms controlling disease development and progression in mycosis fungoides (MF), the most common form of CTCL. Methods: We collected 100 samples that were submitted for diagnosis of, or a second opinion regarding, MF between 2001 and 2018, 80% of which were in the early clinical stages of the disease. Formalin‐fixed paraffin‐embedded tissues were used for histological review and to measure the expression by immunohistochemistry of surrogate markers of activation of the TCR–PLCG1–NFAT, JAK–STAT and NF‐κB pathways. Folliculotropism and large‐cell transformation were also examined. Results: NFAT and nuclear factor kappa B (NF‐κB) markers showed a comparable activation status in early and advanced stages, while STAT3 activation was more frequent in advanced stages and was associated with large‐cell transformation. Consistently with this observation, STAT3 activation occurred in parallel with MF progression in two initially MF‐negative cases. A significant association of NFAT with NF‐κB markers was also found, reflecting a common mechanism of activation in the two pathways. Genomic studies identified nine mutations in seven genes known to play a potential role in tumorigenesis in T‐cell leukaemia/lymphoma, including PLCG1,JAK3 and STAT3, which underlies the activation of these key cell‐survival pathways. A higher mutational allele frequency was detected in advanced stages. Conclusions: Our results show that STAT3 is activated in advanced cases and is associated with large‐cell transformation, while the activation of NFAT and NF‐κB is maintained throughout the disease. These findings could have important diagnostic and therapeutic implications. What's already known about this topic? Mycosis fungoides is characterized by a clonal expansion of T cells in the skin.The mechanisms controlling disease development and progression are not fully understood. What does this study add? An association of the nuclear factor of activated T cells and nuclear factor kappa B pathways was found, which could reflect a common mechanism of activation. These pathways were activated in early and advanced stages at the same level.Signal transducer and activator of transcription 3 activation was associated with large‐cell transformation and was more frequent in advanced stages.A genomic analysis of cutaneous T‐cell lymphoma‐associated genes was performed. Nine mutations were detected. What is the translational message? These results could have important implications for the treatment of MF in the near future. Linked Comment: Ødum. Br J Dermatol 2020; 182:16–17. [ABSTRACT FROM AUTHOR]

Published
2020
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33. EBV POSITIVE DIFFUSE LARGE B CELL LYMPHOMA OF THE ELDERLY IS AN AGGRESSIVE B CELL NEOPLASM WITH A POST GERMINAL CENTER B CELL PHENOTYPE AND CHARACTERIZED BY NFKB PATHWAY ACTIVATION. EVALUATION OF A WESTERN SERIES OF 47 CASES

Author

Montes Moreno S, Odqvist L, Diaz Perez J, Pajares R, Garcia J, Mollejo M, Ruiz Marcellan C, Adrados M, Ortiz N, Piris M., FRANCO, Renato, Montes Moreno, S, Odqvist, L, Diaz Perez, J, Pajares, R, Garcia, J, Mollejo, M, Ruiz Marcellan, C, Adrados, M, Ortiz, N, Franco, Renato, and Piris, M.

Published
2011

34. Age Related EBV plus Diffuse Large B Cell Lymphoma Is a B Cell Neoplasm Characterized by Prominent NFkB Activation Evaluation of a Series of 47 Cases

Author

Montes Moreno S, Diaz Perez JA, Pajares R, Sanchez Verde L, Garcia JF, Mollejo M, Ruiz Marcellan C, Adrados M, Ortiz N, Menarguez J, Piris MA, FRANCO, Renato, Montes Moreno, S, Diaz Perez, Ja, Pajares, R, Sanchez Verde, L, Garcia, Jf, Mollejo, M, Ruiz Marcellan, C, Adrados, M, Ortiz, N, Franco, Renato, Menarguez, J, and Piris, Ma

Published
2011

36. Array-CGH Identifies Both Common and Subtype-Specific Genomic Aberrations in Marginal Zone Lymphomas. Abstract n. 622

Author

Rinaldi, A, Rancoita, Pmv, Bhagat, G, Novak, U, Nandula, S, Murty, Vv, Ponzoni, M, Govi, S, Ferreri, Ajm, Baldini, L, Gascoyne, R, Soulier, J, Thieblemont, C, Marasca, R, Franceschetti, S, Gaidano, G, Canzonieri, V, Gattei, V, Facchetti, F, Tucci, A, Mollejo, M, Piris, Ma, Uccella, Silvia, Tibiletti, Mg, Dirnhofer, S, Forconi, F, Chigrinova, E, Doglioni, C, Dalla Favera, R, Zucca, E, Kwee, I, and Bertoni, F.

Published
2008

37. Chronic lymphocytic leukemia cells in lymph nodes show frequent NOTCH1 activation

Author

Onaindia, A., primary, Gomez, S., additional, Piris-Villaespesa, M., additional, Martinez-Laperche, C., additional, Cereceda, L., additional, Montes-Moreno, S., additional, Batlle, A., additional, de Villambrosia, S. G., additional, Pollan, M., additional, Martin-Acosta, P., additional, Gonzalez-Rincon, J., additional, Menarguez, J., additional, Alves, J., additional, Rodriguez-Pinilla, S. M., additional, Garcia, J. F., additional, Mollejo, M., additional, Fraga, M., additional, Garcia-Marco, J. A., additional, Piris, M. A., additional, and Sanchez-Beato, M., additional

Published
2014
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39. Whole-exome sequencing in splenic marginal zone lymphoma reveals mutations in genes involved in marginal zone differentiation

Author

Martínez, N, primary, Almaraz, C, additional, Vaqué, J P, additional, Varela, I, additional, Derdak, S, additional, Beltran, S, additional, Mollejo, M, additional, Campos-Martin, Y, additional, Agueda, L, additional, Rinaldi, A, additional, Kwee, I, additional, Gut, M, additional, Blanc, J, additional, Oscier, D, additional, Strefford, J C, additional, Martinez-Lopez, J, additional, Salar, A, additional, Sole, F, additional, Rodriguez-Peralto, J L, additional, Diez-Tascón, C, additional, García, J F, additional, Fraga, M, additional, Sebastián, E, additional, Alvés, J, additional, Menárguez, J, additional, González-Carreró, J, additional, Casado, L F, additional, Bayes, M, additional, Bertoni, F, additional, Gut, I, additional, and Piris, M A, additional

Published
2013
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40. OMICS AND PROGNSTIC MARKERS

Author

Adachi, K., primary, Sasaki, H., additional, Nagahisa, S., additional, Yoshida, K., additional, Hattori, N., additional, Nishiyama, Y., additional, Kawase, T., additional, Hasegawa, M., additional, Abe, M., additional, Hirose, Y., additional, Alentorn, A., additional, Marie, Y., additional, Poggioli, S., additional, Alshehhi, H., additional, Boisselier, B., additional, Carpentier, C., additional, Mokhtari, K., additional, Capelle, L., additional, Figarella-Branger, D., additional, Hoang-Xuan, K., additional, Sanson, M., additional, Delattre, J.-Y., additional, Idbaih, A., additional, Yust-Katz, S., additional, Anderson, M., additional, Olar, A., additional, Eterovic, A., additional, Ezzeddine, N., additional, Chen, K., additional, Zhao, H., additional, Fuller, G., additional, Aldape, K., additional, de Groot, J., additional, Andor, N., additional, Harness, J., additional, Lopez, S. G., additional, Fung, T. L., additional, Mewes, H. W., additional, Petritsch, C., additional, Arivazhagan, A., additional, Somasundaram, K., additional, Thennarasu, K., additional, Pandey, P., additional, Anandh, B., additional, Santosh, V., additional, Chandramouli, B., additional, Hegde, A., additional, Kondaiah, P., additional, Rao, M., additional, Bell, R., additional, Kang, R., additional, Hong, C., additional, Song, J., additional, Costello, J., additional, Nagarajan, R., additional, Zhang, B., additional, Diaz, A., additional, Wang, T., additional, Bie, L., additional, Li, Y., additional, Liu, H., additional, Luyo, W. F. C., additional, Carnero, M. H., additional, Iruegas, M. E. P., additional, Morell, A. R., additional, Figueiras, M. C., additional, Lopez, R. L., additional, Valverde, C. F., additional, Chan, A. K.-Y., additional, Pang, J. C.-S., additional, Chung, N. Y.-F., additional, Li, K. K.-W., additional, Poon, W. S., additional, Chan, D. T.-M., additional, Wang, Y., additional, Ng, H.-a. K., additional, Chaumeil, M., additional, Larson, P., additional, Yoshihara, H., additional, Vigneron, D., additional, Nelson, S., additional, Pieper, R., additional, Phillips, J., additional, Ronen, S., additional, Clark, V., additional, Omay, Z. E., additional, Serin, A., additional, Gunel, J., additional, Omay, B., additional, Grady, C., additional, Youngblood, M., additional, Bilguvar, K., additional, Baehring, J., additional, Piepmeier, J., additional, Gutin, P., additional, Vortmeyer, A., additional, Brennan, C., additional, Pamir, M. N., additional, Kilic, T., additional, Krischek, B., additional, Simon, M., additional, Yasuno, K., additional, Gunel, M., additional, Cohen, A. L., additional, Sato, M., additional, Aldape, K. D., additional, Mason, C., additional, Diefes, K., additional, Heathcock, L., additional, Abegglen, L., additional, Shrieve, D., additional, Couldwell, W., additional, Schiffman, J. D., additional, Colman, H., additional, D'Alessandris, Q. 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F., additional, Mehta, S., additional, Armstrong, B., additional, Peng, S., additional, Bapat, A., additional, Berens, M., additional, Melendez, B., additional, Mollejo, M., additional, Mur, P., additional, Hernandez-Iglesias, T., additional, Fiano, C., additional, Ruiz, J., additional, Rey, J. A., additional, Stadler, V., additional, Schulte, A., additional, Lamszus, K., additional, Schichor, C., additional, Westphal, M., additional, Tonn, J.-C., additional, Morozova, O., additional, Katzman, S., additional, Grifford, M., additional, Salama, S., additional, Haussler, D., additional, Olshen, A., additional, Fouse, S., additional, Nakamizo, S., additional, Sasayama, T., additional, Tanaka, H., additional, Tanaka, K., additional, Mizukawa, K., additional, Yoshida, M., additional, Kohmura, E., additional, Northcott, P., additional, Jones, D., additional, Pfister, S., additional, Otani, R., additional, Takayanagi, S., additional, Saito, K., additional, Tanaka, S., additional, Shin, M., additional, Ozawa, T., additional, Riester, M., additional, Cheng, Y.-K., additional, Huse, J., additional, Helmy, K., additional, Charles, N., additional, Squatrito, M., additional, Michor, F., additional, Holland, E., additional, Perrech, M., additional, Dreher, L., additional, Rohn, G., additional, Goldbrunner, R., additional, Timmer, M., additional, Pollo, B., additional, Palumbo, V., additional, Calatozzolo, C., additional, Patane, M., additional, Nunziata, R., additional, Farinotti, M., additional, Silvani, A., additional, Lodrini, S., additional, Finocchiaro, G., additional, Lopez, E., additional, Rioscovian, A., additional, Ruiz, R., additional, Siordia, G., additional, de Leon, A. 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P., additional, and Koul, D., additional

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2013
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41. Loss of TCR-beta F1 and/or EZRIN expression is associated with unfavorable prognosis in nodal peripheral T-cell lymphomas

Author

Rodríguez-Pinilla, S M, primary, Sánchez, M E C, additional, Rodríguez, J, additional, García, J F, additional, Sánchez-Espiridión, B, additional, Lamana, L F, additional, Sosa, G, additional, Rivero, J C, additional, Menárguez, J, additional, Gómez, I B, additional, Camacho, F I, additional, Guillen, P R, additional, Orduña, C P S, additional, Rodríguez, G, additional, Barrionuevo, C, additional, Franco, R, additional, Mollejo, M, additional, Marco, J F, additional, de Otazu, R D, additional, and Piris, M A, additional

Published
2013
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42. Simultaneous inhibition of pan-phosphatidylinositol-3-kinases and MEK as a potential therapeutic strategy in peripheral T-cell lymphomas

Author

Martin-Sanchez, E., primary, Rodriguez-Pinilla, S. M., additional, Sanchez-Beato, M., additional, Lombardia, L., additional, Dominguez-Gonzalez, B., additional, Romero, D., additional, Odqvist, L., additional, Garcia-Sanz, P., additional, Wozniak, M. B., additional, Kurz, G., additional, Blanco-Aparicio, C., additional, Mollejo, M., additional, Alves, F. J., additional, Menarguez, J., additional, Gonzalez-Palacios, F., additional, Rodriguez-Peralto, J. L., additional, Ortiz-Romero, P. L., additional, Garcia, J. F., additional, Bischoff, J. R., additional, and Piris, M. A., additional

Published
2012
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44. MicroRNA signatures in B-cell lymphomas

Author

Di Lisio, L, primary, Sánchez-Beato, M, additional, Gómez-López, G, additional, Rodríguez, M E, additional, Montes-Moreno, S, additional, Mollejo, M, additional, Menárguez, J, additional, Martínez, M A, additional, Alves, F J, additional, Pisano, D G, additional, Piris, M A, additional, and Martínez, N, additional

Published
2012
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47. Stimulation of the midkine/ALK axis renders glioma cells resistant to cannabinoid antitumoral action

Author

Lorente, M, primary, Torres, S, additional, Salazar, M, additional, Carracedo, A, additional, Hernández-Tiedra, S, additional, Rodríguez-Fornés, F, additional, García-Taboada, E, additional, Meléndez, B, additional, Mollejo, M, additional, Campos-Martín, Y, additional, Lakatosh, S A, additional, Barcia, J, additional, Guzmán, M, additional, and Velasco, G, additional

Published
2011
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50. Aggressive large B-cell lymphoma with plasma cell differentiation: immunohistochemical characterization of plasmablastic lymphoma and diffuse large B-cell lymphoma with partial plasmablastic phenotype

Author

Montes-Moreno, S., primary, Gonzalez-Medina, A.-R., additional, Rodriguez-Pinilla, S.-M., additional, Maestre, L., additional, Sanchez-Verde, L., additional, Roncador, G., additional, Mollejo, M., additional, Garcia, J. F., additional, Menarguez, J., additional, Montalban, C., additional, Ruiz-Marcellan, M. C., additional, Conde, E., additional, and Piris, M. A., additional

Published
2010
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