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185 results on '"Miura, Yoichi"'

9. A case of bilateral vertebral artery dissecting aneurysms presenting with subarachnoid hemorrhage evaluated using computational fluid dynamics: Which is the rupture side?

Author

Tsuji, Masanori, primary, Ishida, Fujimaro, additional, Yasuda, Ryuta, additional, Ichikawa, Tomonori, additional, Sato, Takenori, additional, Furukawa, Kazuhiro, additional, Miura, Yoichi, additional, Umeda, Yasuyuki, additional, Toma, Naoki, additional, and Suzuki, Hidenori, additional

Published
2024
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12. Cilostazol may Improve Outcomes even in Aneurysmal Subarachnoid Hemorrhage Patients Aged 75 and Over: Multicenter Cohort Study and Propensity Score Matched Analyses

Author

Nakajima, Hideki, primary, Okada, Takeshi, additional, Kawakita, Fumihiro, additional, Oinaka, Hiroki, additional, Suzuki, Yume, additional, Nampei, Mai, additional, Kitano, Yotaro, additional, Nishikawa, Hirofumi, additional, Fujimoto, Masashi, additional, Miura, Yoichi, additional, Yasuda, Ryuta, additional, Toma, Naoki, additional, and Suzuki, Hidenori, additional

Published
2023
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23. Effects of New-Generation Antiepileptic Drug Prophylaxis on Delayed Neurovascular Events After Aneurysmal Subarachnoid Hemorrhage

Author

Suzuki, Hidenori, primary, Miura, Yoichi, additional, Yasuda, Ryuta, additional, Yago, Tetsushi, additional, Mizutani, Hisashi, additional, Ichikawa, Tomonori, additional, Miyazaki, Takahiro, additional, Kitano, Yotaro, additional, Nishikawa, Hirofumi, additional, Kawakita, Fumihiro, additional, Fujimoto, Masashi, additional, and Toma, Naoki, additional

Published
2022
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27. Treatment of Aneurysmal Subarachnoid Hemorrhage : Current Status and Prospects

Author

Suzuki, Hidenori, primary, Kawakita, Fumihiro, additional, Asada, Reona, additional, Yamamoto, Atsushi, additional, Miyazaki, Takahiro, additional, Yamanaka, Takuya, additional, Sato, Takenori, additional, Tsuji, Masanori, additional, Nishikawa, Hirofumi, additional, Fujimoto, Masashi, additional, Miura, Yoichi, additional, Yasuda, Ryuta, additional, and Toma, Naoki, additional

Published
2022
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33. Plasma Fibulin-5 Levels as an Independent Predictor of a Poor Outcome after an Aneurysmal Subarachnoid Hemorrhage.

Author

Suzuki, Yume, Oinaka, Hiroki, Nakajima, Hideki, Nampei, Mai, Kawakita, Fumihiro, Miura, Yoichi, Yasuda, Ryuta, Toma, Naoki, and Suzuki, Hidenori

Subjects
SUBARACHNOID hemorrhage, CORONARY vasospasm, CEREBRAL infarction, CEREBRAL ischemia, DRUG target, EXTRACELLULAR matrix proteins
Abstract

Aneurysmal subarachnoid hemorrhage (SAH) is a poor-outcome disease with a delayed neurological exacerbation. Fibulin-5 (FBLN5) is one of matricellular proteins, some of which have been involved in SAH pathologies. However, no study has investigated FBLN5's roles in SAH. This study was aimed at examining the relationships between serially measured plasma FBLN5 levels and neurovascular events or outcomes in 204 consecutive aneurysmal SAH patients, including 77 patients (37.7%) with poor outcomes (90-day modified Rankin Scale 3–6). Plasma FBLN5 levels were not related to angiographic vasospasm, delayed cerebral ischemia, and delayed cerebral infarction, but elevated levels were associated with severe admission clinical grades, any neurological exacerbation and poor outcomes. Receiver-operating characteristic curves indicated that the most reasonable cut-off values of plasma FBLN5, in order to differentiate 90-day poor from good outcomes, were obtained from analyses at days 4–6 for all patients (487.2 ng/mL; specificity, 61.4%; and sensitivity, 62.3%) and from analyses at days 7–9 for only non-severe patient (476.8 ng/mL; specificity, 66.0%; and sensitivity, 77.8%). Multivariate analyses revealed that the plasma FBLN5 levels were independent determinants of the 90-day poor outcomes in both all patients' and non-severe patients' analyses. These findings suggest that the delayed elevation of plasma FBLN5 is related to poor outcomes, and that FBLN5 may be a new molecular target to reveal a post-SAH pathophysiology. [ABSTRACT FROM AUTHOR]

Published
2022
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34. Retrograde Angiography to Detect Dropped Thrombus in Mechanical Thrombectomy.

Author

Aoki, Kazuaki, Miura, Yoichi, Toma, Naoki, Suzuki, Yume, Fujimoto, Masashi, Shiba, Masato, Yasuda, Ryuta, and Suzuki, Hidenori

Subjects
*THROMBOSIS, *THROMBECTOMY, *ANGIOGRAPHY, *ISCHEMIC stroke, *TECHNOLOGICAL innovations, *THERAPEUTIC embolization
Abstract

Objective: The risk of embolization to distal territory or to new territory in mechanical thrombectomy remains a major issue despite advancements in technological device. This condition can be caused by a large and firm dropped thrombus without passing through a guiding catheter during stent retriever or aspiration catheter withdrawal. This report introduced a novel technique referred to as retrograde angiography to detect dropped thrombus. Methods: The retrograde angiography to detect dropped thrombus technique is a kind of retrograde angiography that consists of a contrast medium injection via a distal microcatheter and aspiration through an inflated balloon-guiding catheter. This method was used to detect dropped thrombus at the balloon-guiding catheter tip when back flow was blocked from the balloon-guiding catheter after stent retriever or aspiration catheter withdrawal. We retrospectively reviewed four consecutive patients who underwent the retrograde angiography to detect dropped thrombus technique during mechanical thrombectomy for acute ischemic stroke due to large vessel occlusion in the anterior circulation between January 2018 and January 2021. Results: Three of four patients had dropped thrombus, which was diagnosed with the technique and retrieved completely with subsequent procedures while maintaining the balloon-guiding catheter inflated. None of the patients experienced embolization to distal territory/embolization to new territory, and a successful reperfusion was achieved in all four cases. Conclusions: The retrograde angiography to detect dropped thrombus is a technique to detect a dropped thrombus at the balloon-guiding catheter tip and allows us to retrieve it with subsequent mechanical thrombectomy procedures while maintaining the balloon-guiding catheter inflated and it may be useful for reducing the risk of embolization to distal territory/embolization to new territory. [ABSTRACT FROM AUTHOR]

Published
2022
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47. Treatment of Poor-grade Subarachnoid Hemorrhage

Author

Suzuki, Hidenori, primary, Kawakita, Fumihiro, additional, Kanamaru, Hideki, additional, Asada, Reona, additional, Fujimoto, Masashi, additional, Miura, Yoichi, additional, Shiba, Masato, additional, and Toma, Naoki, additional

Published
2020
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49. Higher Plasma Osteopontin Concentrations Associated with Subsequent Development of Chronic Shunt-Dependent Hydrocephalus After Aneurysmal Subarachnoid Hemorrhage.

Author

Asada, Reona, Nakatsuka, Yoshinari, Kanamaru, Hideki, Kawakita, Fumihiro, Fujimoto, Masashi, Miura, Yoichi, Shiba, Masato, Yasuda, Ryuta, Toma, Naoki, and Suzuki, Hidenori

Abstract

A matricellular protein osteopontin (OPN) is considered to exert neuroprotective and healing effects on neurovascular injuries in an acute phase of aneurysmal subarachnoid hemorrhage (SAH). However, the relationships between OPN expression and chronic shunt-dependent hydrocephalus (SDHC) have never been investigated. In 166 SAH patients (derivation and validation cohorts, 110 and 56, respectively), plasma OPN levels were serially measured at days1−3, 4−6, 7−9, and 10−12 after aneurysmal obliteration. The OPN levels and clinical factors were compared between patients with and without subsequent development of chronic SDHC. Plasma OPN levels in the SDHC patients increased from days 1−3 to days 4−6 and remained high thereafter, while those in the non-SDHC patients peaked at days 4−6 and then decreased over time. Plasma OPN levels had no correlation with serum levels of C-reactive protein (CRP), a systemic inflammatory marker. Univariate analyses showed that age, modified Fisher grade, acute hydrocephalus, cerebrospinal fluid drainage, and OPN and CRP levels at days 10−12 were significantly different between patients with and without SDHC. Multivariate analyses revealed that higher plasma OPN levels at days 10−12 were an independent factor associated with the development of SDHC, in addition to a more frequent use of cerebrospinal fluid drainage and higher modified Fisher grade at admission. Plasma OPN levels at days 10−12 maintained similar discrimination power in the validation cohort and had good calibration on the Hosmer-Lemeshow goodness-of-fit test. Prolonged higher expression of OPN may contribute to the development of post-SAH SDHC, possibly by excessive repairing effects promoting fibrosis in the subarachnoid space. [ABSTRACT FROM AUTHOR]

Published
2021
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