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2. Mechanism-centric regulatory network identifies NME2 and MYC programs as markers of Enzalutamide resistance in CRPC

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7. EZH2 inhibition activates a dsRNA–STING–interferon stress axis that potentiates response to PD-1 checkpoint blockade in prostate cancer

8. A MYC and RAS co-activation signature in localized prostate cancer drives bone metastasis and castration resistance

12. ETV4 promotes metastasis in response to activation of PI3-kinase and Ras signaling in a mouse model of advanced prostate cancer

13. Lineage analysis of basal epithelial cells reveals their unexpected plasticity and supports a cell-of-origin model for prostate cancer heterogeneity

14. Tissue-specific biological aging predicts progression in prostate cancer and acute myeloid leukemia

18. Data from OncoLoop: A Network-Based Precision Cancer Medicine Framework

19. Table S4 from OncoLoop: A Network-Based Precision Cancer Medicine Framework

20. Index of Supplementary Data from OncoLoop: A Network-Based Precision Cancer Medicine Framework

21. Detailed Materials and Methods from OncoLoop: A Network-Based Precision Cancer Medicine Framework

22. Supplementary Figures S1-S11 from OncoLoop: A Network-Based Precision Cancer Medicine Framework

23. Editor's Note: Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

24. Data from Transdifferentiation as a Mechanism of Treatment Resistance in a Mouse Model of Castration-Resistant Prostate Cancer

25. Supplementary Tables from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

26. Data from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

27. Dataset 1 from Transdifferentiation as a Mechanism of Treatment Resistance in a Mouse Model of Castration-Resistant Prostate Cancer

28. Supplementary Datasets from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

29. Supplementary Materials and Methods from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

30. Supplementary Data Index from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

31. Supplementary Figures from NKX3.1 Localization to Mitochondria Suppresses Prostate Cancer Initiation

32. Supplementary Figures 1 through 7, Supplementary Tables 1 through 7, and Supplementary Materials and Methods from Transdifferentiation as a Mechanism of Treatment Resistance in a Mouse Model of Castration-Resistant Prostate Cancer

33. Data from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

34. Supplementary Table 8 from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

35. Supplementary Table 7 from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

36. Supplementary Table 6A from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

37. Supplementary Table 6 from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

38. Data from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

39. Supplementary Table 5 from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

40. Supplementary Table 4 from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

41. Supplementary Figure 5B from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

42. Supplementary Table 4 from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

43. Supplementary Figures 1-3, Tables 1-3, Table 7, Legends for Figures 1-3, Tables 1-7 from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

44. Supplementary Table 5A from B-Raf Activation Cooperates with PTEN Loss to Drive c-Myc Expression in Advanced Prostate Cancer

45. Supplementary Figure 1 and Tables 1 - 3 from Dual Targeting of the Akt/mTOR Signaling Pathway Inhibits Castration-Resistant Prostate Cancer in a Genetically Engineered Mouse Model

47. OncoLoop: A Network-Based Precision Cancer Medicine Framework

50. Syntax-Driven Private Evaluation of Quantified Membership Queries