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Copyright of Pediatru.ro is the property of MEDICHUB MEDIA, S.R.L. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of ORL.ro is the property of MEDICHUB MEDIA, S.R.L. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Farmacia is the property of Societatea de Stiinte Farmaceutice Romania and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

One-fourth of the global population suffer from anemia, with iron deficiency being the main reason; it affects both sexes almost equally, with a slight preponderance of women being afflicted. Anemia can be caused by a wide range of conditions and circumstances, and it can present both orally and systemically. Reduced red blood cell count and/or hemoglobin concentration, or disruption in their function, which finally results in reduced oxygen delivery to tissues, are all defining characteristics of anemia. Anemia is linked to decreased well-being, poor cognitive function, and persistent fatigue. The orofacial signs and symptoms include magenta tongue, midfacial overgrowth, angular stomatitis, atrophic glossitis, paresthesia/anesthesia of the mental nerve, dysphagia, osteosclerosis, and conjunctiva and facial pallor. Common orofacial manifestations also include conjunctival hemorrhage, nose bleeding, spontaneous and posttraumatic gingival hemorrhage, and persistent post-extraction bleeding. Patients with anemia require multidisciplinary care. Consequently, the dentist must have a solid awareness of this disorder, with a focus on warning indicators, dental management, and appropriate communication with the patient’s physician. [ABSTRACT FROM AUTHOR]

Copyright of Pediatru.ro is the property of MEDICHUB MEDIA, S.R.L. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Pediatru.ro is the property of MEDICHUB MEDIA, S.R.L. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Medic.ro is the property of MEDICHUB MEDIA, S.R.L. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Introduction. Gynecomastia is the benign development of glandular breast tissue in male patients. Neonatal breast en largement is a common condition due to post natal maternal hormones depletion. Firstly, it should be differentia ted from mastitis and breast abscess. Recent studies in dicate that using progestogen-only contraceptives by breast feeding women appears to be safe for the breastfed infant. Case report. We report the case of a 2-month-old boy who presented for endocrine evaluation for asymmetric gyne comastia, which had been observed since birth, but left breast enlargement worsened in the last two weeks, over lapping the beginning of mother’s treatment with the contraceptive desogestrel. Hormonal evaluation re vealed a slight increase in prolactin values, with normal go nadal, adrenal and thyroid gland hormones. Breast ultra sound showed bilateral glandular breast tissue, without signs of breast abscess. The patient was managed in a multi disciplinary team and had a good clinical evolution, with the resolution of gynecomastia without a particular treatment. Conclusions. We present herein the case of a male infant who was evaluated for bilateral breast enlargement, diagnosed as neonatal gynecomastia, overlapping the mother’s treatment with progestogen-only contraceptive desogestrel, who was followed-up, without a particular treatment, with the resolution of breast enlargement. [ABSTRACT FROM AUTHOR]

Copyright of Farmacia is the property of Societatea de Stiinte Farmaceutice Romania and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Differentiated thyroid cancer (DTC), which includes papillary and follicular cancer, comprises the vast majority (>90%) of all thyroid cancers. Papillary carcinoma of the thyroid gland typically presents as a nodule that is firm and solid on thyroid ultrasound, sometimes with internal calcifications. In multinodular goiter, the cancer is usually a dominant nodule - larger, firmer, and/or with ultrasound characteristics more suspicious than other nodules in the gland. Primary hyperparathyroidism results from the excessive secretion of PTH, and typically produces frank hypercalcemia. Primary hyperparathyroidism is caused by a single parathyroid adenoma in about 80% of cases and by primary hyperplasia of the parathyroids in 10% to 15%. Parathyroid carcinoma is a rare cause of hyperparathyroidism, accounting for 1% to 2% of cases. We report the case of a 67-year-old woman who initially presented herself in our department for clinical and therapeutic evaluation in the context of suspicion of hyperparathyroidism, associating high levels of PTH and serum calcium on test results performed in an outpatient clinic. She is subsequently diagnosed with primary hyperparathyroidism by ultrasound detection of a right parathyroid adenoma, as well as a multinodular goiter with numerous nodules with suspicious aspect. While thyroid function evaluated by repeated dosing of TSH, FT4, T3 has been shown to remain within normal limits, PTH values, as well as total serum calcium and ionic calcium, continued to show elevated values. Phosphorus values remained within normal limits. Further investigations including both parathyroid scintigraphy and contrast CT examination of the cervical region and mediastinum. The patient is redirected for surgical cure of the goiter and parathyroid adenoma, resulting in total thyroidectomy and upper right parathyroidectomy. Histopathologically, the diagnosis of multifocal papillary microcarcinoma and upper right parathyroid adenoma is established. In the context of the detection of thyroid neoplastic pathology, the patient is advised to seek further treatment in the form of radioiodine therapy. The patient should present for periodic clinical and therapeutic follow-up according to indications and guidelines. [ABSTRACT FROM AUTHOR]