16 results on '"Miller, Tracy K."'
Search Results
2. Facility-Wide Testing for SARS-CoV-2 in Nursing Homes — Seven U.S. Jurisdictions, March–June 2020
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Hatfield, Kelly M., Reddy, Sujan C., Forsberg, Kaitlin, Korhonen, Lauren, Garner, Kelley, Gulley, Trent, James, Allison, Patil, Naveen, Bezold, Carla, Rehman, Najibah, Sievers, Marla, Schram, Benjamin, Miller, Tracy K., Howell, Molly, Youngblood, Claire, Ruegner, Hannah, Radcliffe, Rachel, Nakashima, Allyn, Torre, Michael, Donohue, Kayla, Meddaugh, Paul, Staskus, Mallory, Attell, Brandon, Biedron, Caitlin, Boersma, Peter, Epstein, Lauren, Hughes, Denise, Lyman, Meghan, Preston, Leigh E., Sanchez, Guillermo V., Tanwar, Sukarma, Thompson, Nicola D., Vallabhaneni, Snigdha, Vasquez, Amber, and Jernigan, John A.
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- 2020
3. Severe Human Metapneumovirus Infections — North Dakota, 2016
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Midgley, Claire M., Baber, Jill K., Biggs, Holly M., Singh, Twila, Feist, Michelle, Miller, Tracy K., Kruger, Kirby, Gerber, Susan I., Watson, John T., and Howell, Molly A.
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- 2017
4. Hepatitis C virus transmission in a skilled nursing facility, North Dakota, 2013
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Calles, Dinorah L., Collier, Melissa G., Khudyakov, Yury, Mixson-Hayden, Tonya, VanderBusch, Lindsey, Weninger, Sarah, and Miller, Tracy K.
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- 2017
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5. Enhanced Surveillance for Coccidioidomycosis, 14 US States, 2016
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Benedict, Kaitlin, Ireland, Malia, Weinberg, Meghan P., Gruninger, Randon J., Weigand, Jenna, Chen, Lei, Perez-Lockett, Katharine, Bledsoe, Catherine, Denny, Lynn, Cibulskas, Katie, Gibbons-Burgener, Suzanne, Kocharian, Anna, DeBess, Emilio, Miller, Tracy K., Lepp, Alicia, Cronquist, Laura, Warren, Kimberly, Serrano, Jose Antonio, Loveland, Cody, Turabelidze, George, McCotter, Orion, and Jackson, Brendan R.
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Privacy ,Coccidioidomycosis -- Development and progression ,Epidemiology ,Privacy issue ,Health ,Council of State and Territorial Epidemiologists - Abstract
Coccidioidomycosis is a fungal infection caused by inhalation of soil-dwelling Coccidioides spp. organisms. Symptomatic infection occurs in [approximately equal to] 40% of cases and usually presents as a self-limiting, influenza-like [...]
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- 2018
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6. Human infection with Ehrlichia muris-like pathogen, United States, 2007-2013
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Johnson, Diep K. Hoang, Schiffman, Elizabeth K., Davis, Jeffrey P., Neitzel, David F., Sloan, Lynne M., Nicholson, William L., Fritsche, Thomas R., Steward, Christopher R., Ray, Julie A., Miller, Tracy K., Feist, Michelle A., Uphoff, Timothy S., Franson, Joni J., Livermore, Amy L., Deedon, Alecia K., Theel, Elitza S., and Pritt, Bobbi S.
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Ehrlichiosis -- Risk factors -- Diagnosis -- Research -- Genetic aspects ,Polymerase chain reaction -- Usage ,Health - Abstract
Ehrlichiosis and anaplasmosis are emerging tickborne zoonoses caused by Ehrlichia spp. and Anaplasma phagocytophilum, respectively (1-4). These gram-negative obligate intracellular bacteria infect leukocytes and cause febrile illness in humans (1,3). [...]
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- 2015
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7. Epidemiology of Hepatitis C virus infection among incarcerated populations in North Dakota
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Chandra Deb, Liton, primary, Hove, Hannah, additional, Miller, Tracy K., additional, Pinks, Kodi, additional, Njau, Grace, additional, Hagan, John J., additional, and Jansen, Rick J., additional
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- 2022
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8. Emergence of Ciprofloxacin-resistant Neisseria meningitidis in North America
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Wu, Henry M., Harcourt, Brian H., Hatcher, Cynthia P., Novak, Ryan T., Wei, Stanley C., Wang, Xin, Juni, Billie A., Glennen, Anita, Boxrud, David J., Rainbow, Jean, Schmink, Susanna, Mair, Raydel D., Theodore, Jordan, Sander, Molly A., Miller, Tracy K., Kruger, Kirby, Cohn, Amanda C., Clark, Thomas A., Messonnier, Nancy E., Mayer, Leonard W., and Lynfield, Ruth
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Neisseria meningitidis -- Risk factors ,Meningococcal infections -- Causes of - Abstract
The article discusses the emergence of Ciprofloxacin-resistant Neisseria meningitidis in North America, with one case being reported in North Dakota and two cases in Minnesota. They indicate the growing public health threat from antimicrobial resistance among bacterial pathogens and the need to focus more on wider surveillance.
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- 2009
9. Notes from the Field: Severe Human Metapneumovirus Infections — North Dakota, 2016
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Midgley, Claire M., primary, Baber, Jill K., additional, Biggs, Holly M., additional, Singh, Twila, additional, Feist, Michelle, additional, Miller, Tracy K., additional, Kruger, Kirby, additional, Gerber, Susan I., additional, Watson, John T., additional, and Howell, Molly A., additional
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- 2017
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10. Borrelia mayonii sp. nov., a member of the Borrelia burgdorferi sensu lato complex, detected in patients and ticks in the upper midwestern United States
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Pritt, Bobbi S., primary, Respicio-Kingry, Laurel B., additional, Sloan, Lynne M., additional, Schriefer, Martin E., additional, Replogle, Adam J., additional, Bjork, Jenna, additional, Liu, Gongping, additional, Kingry, Luke C., additional, Mead, Paul S., additional, Neitzel, David F., additional, Schiffman, Elizabeth, additional, Hoang Johnson, Diep K., additional, Davis, Jeffrey P., additional, Paskewitz, Susan M., additional, Boxrud, David, additional, Deedon, Alecia, additional, Lee, Xia, additional, Miller, Tracy K., additional, Feist, Michelle A., additional, Steward, Christopher R., additional, Theel, Elitza S., additional, Patel, Robin, additional, Irish, Cole L., additional, and Petersen, Jeannine M., additional
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- 2016
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11. Identification of a novel pathogenic Borrelia species causing Lyme borreliosis with unusually high spirochaetaemia: a descriptive study
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Pritt, Bobbi S, primary, Mead, Paul S, additional, Johnson, Diep K Hoang, additional, Neitzel, David F, additional, Respicio-Kingry, Laurel B, additional, Davis, Jeffrey P, additional, Schiffman, Elizabeth, additional, Sloan, Lynne M, additional, Schriefer, Martin E, additional, Replogle, Adam J, additional, Paskewitz, Susan M, additional, Ray, Julie A, additional, Bjork, Jenna, additional, Steward, Christopher R, additional, Deedon, Alecia, additional, Lee, Xia, additional, Kingry, Luke C, additional, Miller, Tracy K, additional, Feist, Michelle A, additional, Theel, Elitza S, additional, Patel, Robin, additional, Irish, Cole L, additional, and Petersen, Jeannine M, additional
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- 2016
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12. Human Infection withEhrlichia muris–like Pathogen, United States, 2007–20131
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Johnson, Diep K. Hoang, primary, Schiffman, Elizabeth K., additional, Davis, Jeffrey P., additional, Neitzel, David F., additional, Sloan, Lynne M., additional, Nicholson, William L., additional, Fritsche, Thomas R., additional, Steward, Christopher R., additional, Ray, Julie A., additional, Miller, Tracy K., additional, Feist, Michelle A., additional, Uphoff, Timothy S., additional, Franson, Joni J., additional, Livermore, Amy L., additional, Deedon, Alecia K., additional, Theel, Elitza S., additional, and Pritt, Bobbi S., additional
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- 2015
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13. Birth outcomes following West Nile virus infection of pregnant women in the United States: 2003-2004
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O'Leary, Daniel R., Kuhn, Stephanie, Kniss, Krista L., Hinckley, Alison F., Rasmussen, Sonja A., Pape, W. John, Kightlinger, Lon K., Beecham, Brady D., Miller, Tracy K., Neitzel, David F., Michaels, Sarah R., Campbell, Grant L., Lanciotti, Robert S., and Hayes, Edward B.
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West Nile fever -- Risk factors ,West Nile fever -- Care and treatment ,Pregnant women -- Health aspects ,Birth defects -- Causes of - Abstract
BACKGROUND. Congenital West Nile virus (WNV) infection was first described in a single case in 2002. The proportion of maternal WNV infections resulting in congenital infection and clinical consequences of such infections are unknown. METHODS. In 2003 and 2004, women in the United States who acquired WNV infection during pregnancy were reported to the Centers for Disease Control and Prevention by state health departments. Data on pregnancy outcomes were collected. One of the maternal WNV infections was identified retrospectively after the infant was born. Maternal sera, placenta, umbilical cord tissue, and cord serum were tested for WNV infection by using serologic assays and reverse-transcription polymerase chain reaction. Infant health was assessed at delivery and through 12 months of age. RESULTS. Seventy-seven women infected with WNV during pregnancy were clinically followed in 16 states. A total of 71 women delivered 72 live infants; 4 women had miscarriages, and 2 had elective abortions. Of the 72 live infants, 67 were born at term, and 4 were preterm; gestational age was unknown for 1. Of 55 live infants from whom cord serum was available, 54 tested negative for anti-WNV IgM. One infant born with umbilical hernia and skin tags had anti-WNV IgM in cord serum but not in peripheral serum at age 1 month. An infant who had no anti-WNV IgM in cord blood, but whose mother had WNV illness 6 days prepartum, developed WNV meningitis at age 10 days. Another infant, whose mother had acute WNV illness at delivery, was born with a rash and coarctation of the aorta and had anti-WNV IgM in serum at 1 month of age; cord serum was not available. A fourth infant, whose mother had onset of WNV illness 3 weeks prepartum that was not diagnosed until after delivery, had WNV encephalitis and underlying lissencephaly detected at age 17 days and subsequently died; cord serum was not available. The following major malformations were noted among live-born infants: aortic coarctation (n = 1); cleft palate (n = 1); Down syndrome (n = 1); lissencephaly (n = 1); microcephaly (n = 2); and polydactyly (n = 1). One infant had glycogen storage disease type 1. Abnormal growth was noted in 8 infants. CONCLUSIONS. Of 72 infants followed to date in 2003 and 2004, almost all seemed normal, and none had conclusive laboratory evidence of congenital WNV infection. Three infants had WNV infection that could have been congenitally acquired. Seven infants had major malformations, but only 3 of these had defects that could have been caused by maternal WNV infection based on the timing of the infections and the sensitive developmental period for the specific malformations, and none had any conclusive evidence of WNV etiology. However, the sensitivity and specificity of IgM testing of cord blood to detect congenital WNV infection are currently unknown, and congenital WNV infection among newborns with IgM-negative serology cannot be ruled out. Prospective studies comparing pregnancy outcomes of WNV-infected and -uninfected women are needed to better define the outcomes of WNV infection during pregnancy. KEY WORDS. West Nile virus, congenital infection, birth outcomes, pregnancy, development, growth, pediatrics, birth defects, microcephaly., URL: [...]
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- 2006
14. Human Infection with Ehrlichia muris-like Pathogen, United States, 2007-2013(1).
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Hoang Johnson, Diep K., Schiffman, Elizabeth K., Davis, Jeffrey P., Neitzel, David F., Sloan, Lynne M., Nicholson, William L., Fritsche, Thomas R., Steward, Christopher R., Ray, Julie A., Miller, Tracy K., Feist, Michelle A., Uphoff, Timothy S., Franson, Joni J., Livermore, Amy L., Deedon, Alecia K., Theel, Elitza S., Pritt, Bobbi S., and Johnson, Diep K Hoang
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An Ehrlichia muris-like (EML) pathogen was detected among 4 patients in Minnesota and Wisconsin during 2009. We characterized additional cases clinically and epidemiologically. During 2004-2013, blood samples from 75,077 patients from all 50 United States were tested by PCR from the groEL gene for Ehrlichia spp. and Anaplasma phagocytophilum. During 2007-2013, samples from 69 (0.1%) patients were positive for the EML pathogen; patients were from 5 states: Indiana (1), Michigan (1), Minnesota (33), North Dakota (3), and Wisconsin (31). Most (64%) patients were male; median age was 63 (range 15-94) years; and all 69 patients reported likely tick exposure in Minnesota or Wisconsin. Fever, malaise, thrombocytopenia, and lymphopenia were the most common symptoms. Sixteen (23%) patients were hospitalized (median 4 days); all recovered, and 96% received doxycycline. Infection with the EML pathogen should be considered for persons reporting tick exposure in Minnesota or Wisconsin. [ABSTRACT FROM AUTHOR]
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- 2015
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15. COVID-19 Mortality Among American Indian and Alaska Native Persons - 14 States, January-June 2020.
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Arrazola J, Masiello MM, Joshi S, Dominguez AE, Poel A, Wilkie CM, Bressler JM, McLaughlin J, Kraszewski J, Komatsu KK, Peterson Pompa X, Jespersen M, Richardson G, Lehnertz N, LeMaster P, Rust B, Keyser Metobo A, Doman B, Casey D, Kumar J, Rowell AL, Miller TK, Mannell M, Naqvi O, Wendelboe AM, Leman R, Clayton JL, Barbeau B, Rice SK, Rolland SJ, Warren-Mears V, Echo-Hawk A, Apostolou A, and Landen M
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- Adult, Female, Humans, Male, Middle Aged, United States epidemiology, Young Adult, Alaska Natives statistics & numerical data, COVID-19 ethnology, COVID-19 mortality, Health Status Disparities, American Indian or Alaska Native statistics & numerical data
- Abstract
American Indian/Alaska Native (AI/AN) persons experienced disproportionate mortality during the 2009 influenza A(H1N1) pandemic (1,2). Concerns of a similar trend during the coronavirus disease 2019 (COVID-19) pandemic led to the formation of a workgroup* to assess the prevalence of COVID-19 deaths in the AI/AN population. As of December 2, 2020, CDC has reported 2,689 COVID-19-associated deaths among non-Hispanic AI/AN persons in the United States.
† A recent analysis found that the cumulative incidence of laboratory-confirmed COVID-19 cases among AI/AN persons was 3.5 times that among White persons (3). Among 14 participating states, the age-adjusted AI/AN COVID-19 mortality rate (55.8 deaths per 100,000; 95% confidence interval [CI] = 52.5-59.3) was 1.8 (95% CI = 1.7-2.0) times that among White persons (30.3 deaths per 100,000; 95% CI = 29.9-30.7). Although COVID-19 mortality rates increased with age among both AI/AN and White persons, the disparity was largest among those aged 20-49 years. Among persons aged 20-29 years, 30-39 years, and 40-49 years, the COVID-19 mortality rates among AI/AN were 10.5, 11.6, and 8.2 times, respectively, those among White persons. Evidence that AI/AN communities might be at increased risk for COVID-19 illness and death demonstrates the importance of documenting and understanding the reasons for these disparities while developing collaborative approaches with federal, state, municipal, and tribal agencies to minimize the impact of COVID-19 on AI/AN communities. Together, public health partners can plan for medical countermeasures and prevention activities for AI/AN communities., Competing Interests: All authors have completed and submitted the International Committee of Medical Journal Editors form for disclosure of potential conflicts of interest. No potential conflicts of interest were disclosed.- Published
- 2020
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16. Facility-Wide Testing for SARS-CoV-2 in Nursing Homes - Seven U.S. Jurisdictions, March-June 2020.
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Hatfield KM, Reddy SC, Forsberg K, Korhonen L, Garner K, Gulley T, James A, Patil N, Bezold C, Rehman N, Sievers M, Schram B, Miller TK, Howell M, Youngblood C, Ruegner H, Radcliffe R, Nakashima A, Torre M, Donohue K, Meddaugh P, Staskus M, Attell B, Biedron C, Boersma P, Epstein L, Hughes D, Lyman M, Preston LE, Sanchez GV, Tanwar S, Thompson ND, Vallabhaneni S, Vasquez A, and Jernigan JA
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- Aged, COVID-19, COVID-19 Testing, Coronavirus Infections diagnosis, Coronavirus Infections epidemiology, Coronavirus Infections transmission, Health Personnel, Humans, Infectious Disease Transmission, Patient-to-Professional prevention & control, Infectious Disease Transmission, Professional-to-Patient prevention & control, Pneumonia, Viral epidemiology, Pneumonia, Viral transmission, United States epidemiology, Clinical Laboratory Techniques, Coronavirus Infections prevention & control, Nursing Homes, Pandemics prevention & control, Pneumonia, Viral prevention & control
- Abstract
Undetected infection with SARS-CoV-2, the virus that causes coronavirus disease 2019 (COVID-19) contributes to transmission in nursing homes, settings where large outbreaks with high resident mortality have occurred (1,2). Facility-wide testing of residents and health care personnel (HCP) can identify asymptomatic and presymptomatic infections and facilitate infection prevention and control interventions (3-5). Seven state or local health departments conducted initial facility-wide testing of residents and staff members in 288 nursing homes during March 24-June 14, 2020. Two of the seven health departments conducted testing in 195 nursing homes as part of facility-wide testing all nursing homes in their state, which were in low-incidence areas (i.e., the median preceding 14-day cumulative incidence in the surrounding county for each jurisdiction was 19 and 38 cases per 100,000 persons); 125 of the 195 nursing homes had not reported any COVID-19 cases before the testing. Ninety-five of 22,977 (0.4%) persons tested in 29 (23%) of these 125 facilities had positive SARS-CoV-2 test results. The other five health departments targeted facility-wide testing to 93 nursing homes, where 13,443 persons were tested, and 1,619 (12%) had positive SARS-CoV-2 test results. In regression analyses among 88 of these nursing homes with a documented case before facility-wide testing occurred, each additional day between identification of the first case and completion of facility-wide testing was associated with identification of 1.3 additional cases. Among 62 facilities that could differentiate results by resident and HCP status, an estimated 1.3 HCP cases were identified for every three resident cases. Performing facility-wide testing immediately after identification of a case commonly identifies additional unrecognized cases and, therefore, might maximize the benefits of infection prevention and control interventions. In contrast, facility-wide testing in low-incidence areas without a case has a lower proportion of test positivity; strategies are needed to further optimize testing in these settings., Competing Interests: All authors have completed and submitted the International Committee of Medical Journal Editors form for disclosure of potential conflicts of interest. Kayla Donohue reports full-time employment at United Way of Northwest Vermont with temporary assignment to COVID-19 response at the Vermont Department of Health, which supported her work related to this publication. No other potential conflicts of interest were disclosed.
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- 2020
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