183 results on '"Migliaccio, Enrica"'
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2. Supplementary Table S6 from High-Fat Diet Promotes Acute Promyelocytic Leukemia through PPARδ-Enhanced Self-renewal of Preleukemic Progenitors
3. Supplementary Figure S1 from High-Fat Diet Promotes Acute Promyelocytic Leukemia through PPARδ-Enhanced Self-renewal of Preleukemic Progenitors
4. Data from High-Fat Diet Promotes Acute Promyelocytic Leukemia through PPARδ-Enhanced Self-renewal of Preleukemic Progenitors
5. High-fat diet promotes Acute Promyelocytic Leukemia through PPARδ-enhanced self-renewal of preleukemic progenitors
6. The Influence of Shc Proteins on Life Span in Mice
7. Supplementary Figures and legends from A Rare Subset of Primary Tumor Cells with Concomitant Hyperactivation of Extracellular Matrix Remodeling and dsRNA-IFN1 Signaling Metastasizes in Breast Cancer
8. Data from A Rare Subset of Primary Tumor Cells with Concomitant Hyperactivation of Extracellular Matrix Remodeling and dsRNA-IFN1 Signaling Metastasizes in Breast Cancer
9. Supplementary Table 4_Marker genes all and some per cluster_GO and curated pathways per cluster from A Rare Subset of Primary Tumor Cells with Concomitant Hyperactivation of Extracellular Matrix Remodeling and dsRNA-IFN1 Signaling Metastasizes in Breast Cancer
10. A Rare Subset of Primary Tumor Cells with Concomitant Hyperactivation of Extracellular Matrix Remodeling and dsRNA-IFN1 Signaling Metastasizes in Breast Cancer
11. Expression and Activation of SH2/PTB-Containing ShcA Adaptor Protein Reflects the Pattern of Neurogenesis in the Mammalian Brain
12. Single-cell imaging and transcriptomic analyses of firm adhesion between patient-derived cancer and endothelial cells under shear stress
13. The breast cancer pro-metastatic phenotype requires concomitant hyper-activation of ECM remodeling and dsRNA-IFN1 signaling in rare clone cells
14. Beyond Genetics: Metastasis as an Adaptive Response in Breast Cancer
15. S1P1 expression is controlled by the pro-oxidant activity of p66Shc and is impaired in B-CLL patients with unfavorable prognosis
16. Protein Kinase C β and Prolyl Isomerase 1 Regulate Mitochondrial Effects of the Life-Span Determinant $\text{p}66^{\text{Shc}}$
17. Protein kinase C[beta] and prolyl isomerase 1 regulate mitochondrial effects of the life-span determinant [p66.sup.Shc]
18. p66Shc, Mitochondria, and the Generation of Reactive Oxygen Species
19. Cooperation and selectivity of the two Grb2 binding sites of p52Shc in T-cell antigen receptor signaling to Ras family GTPases and Myc-dependent survival
20. Electron transfer between cytochrome c and p66(super Shc) generates reactive oxygen species that trigger mitochondrial apoptosis
21. Oxidative stress activates a specific p53 transcriptional response that regulates cellular senescence and aging
22. A p53-p66Shc signalling pathway controls intracellular redox status, levels of oxidation-damaged DNA and oxidative stress-induced apoptosis
23. The p66Shc knocked out mice are short lived under natural condition
24. The p66shc adaptor protein controls oxidative stress response and life span in mammals
25. The Shc locus regulates insulin signaling and adiposity in mammals
26. Protein Kinase C β and Prolyl Isomerase 1 Regulate Mitochondrial Effects of the Life-Span Determinant p66Shc
27. Regulatory effects of the mitochondrial energetic status on mitochondrial p66Shc
28. Deletion of p66Shc Longevity Gene Protects Against Experimental Diabetic Glomerulopathy by Preventing Diabetes-Induced Oxidative Stress
29. Not all Shc' roads leads to Ras
30. Erratum. Deletion of p66Shc Longevity Gene Protects Against Experimental Diabetic Glomerulopathy by Preventing Diabetes-Induced Oxidative Stress. Diabetes 2006;55:1642–1650
31. P66SHC deletion improves fertility and progeric phenotype of late-generation TERC-deficient mice but not their short lifespan
32. Modelling the p53/p66Shc Aging Pathway in the Shortest Living Vertebrate Nothobranchius Furzeri
33. The Influence of Shc Proteins on Life Span in Mice
34. The p66shc adaptor protein, controls oxidative stress response and life span in mammals
35. p53 and aging: role of p66Shc
36. Deletion of p66Shc in mice increases the frequency of size-change mutations in the lacZ transgene
37. P66 SHC deletion improves fertility and progeric phenotype of late-generation TERC-deficient mice but not their short lifespan.
38. The p66Shcknockout mice are short lived under natural condition
39. The Shc locus regulates insulin signaling and adiposity in mammals
40. Decreased Superoxide Production in Macrophages of Long-lived p66Shc Knock-out Mice
41. P66Shc signals to age
42. p66Shc-generated Oxidative Signal Promotes Fat Accumulation
43. p66ShcDeletion Confers Vascular Protection in Advanced Atherosclerosis in Hypercholesterolemic Apolipoprotein E Knockout Mice
44. Hydrogen peroxide: a metabolic by-product or a common mediator of ageing signals?
45. Protein Kinase C ß and Prolyl Isomerase 1 Regulate Mitochondrial Effects of the Life-Span Determinant p66 Shc
46. Apoptosis and Aging: Role of p66ShcRedox Protein
47. Genetic Deletion of the p66 Shc Adaptor Protein Protects From Angiotensin II–Induced Myocardial Damage
48. Electron Transfer between Cytochrome c and p66Shc Generates Reactive Oxygen Species that Trigger Mitochondrial Apoptosis
49. Deletion of p66 shc Gene Protects Against Age-Related Endothelial Dysfunction
50. The Life Span Determinant p66Shc Localizes to Mitochondria Where It Associates with Mitochondrial Heat Shock Protein 70 and Regulates Trans-membrane Potential
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