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2. CD32 expressing doublets in HIV-infected gut-associated lymphoid tissue are associated with a T follicular helper cell phenotype

3. Impact of antiretroviral therapy in primary HIV infection on natural killer cell function and the association with viral rebound and HIV DNA following treatment interruption

6. Human MAIT cells respond to and suppress HIV-1

7. Epigenetic Features of HIV-Induced T-Cell Exhaustion Persist Despite Early Antiretroviral Therapy

9. A targeted reactivation of latent HIV-1 using an activator vector in patient samples from acute infection

10. Author response: Human MAIT cells respond to and suppress HIV-1

11. Human MAIT cells respond to and suppress HIV-1.

12. HIV infection impairs Th1 and Th17 Mycobacterium tuberculosis-specific T cell responses

13. Levels of Human Immunodeficiency Virus DNA Are Determined Before ART Initiation and Linked to CD8 T-Cell Activation and Memory Expansion

14. Maintenance of Functional CD57+ Cytolytic CD4+ T Cells in HIV+ Elite Controllers

15. Virological remission after antiretroviral therapy interruption in female African HIV seroconverters

16. C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stress

17. Lack of effect of Maraviroc intensification on blood and gut reservoir

18. Levels of Human Immunodeficiency Virus DNA Are Determined Before ART Initiation and Linked to CD8 T-Cell Activation and Memory Expansion.

19. CD32-Expressing CD4 T Cells Are Phenotypically Diverse and Can Contain Proviral HIV DNA

20. Post-treatment control or treated controllers? Viral remission in treated and untreated primary HIV infection

21. Exhaustion of Activated CD8 T Cells Predicts Disease Progression in Primary HIV-1 Infection

23. Enrichment of the HIV reservoir in CD32+ CD4 T cells occurs early in blood and tissue

24. Short Communication: Lack of Effect of Maraviroc Intensification on Blood and Gut Reservoir

25. Immunological biomarkers predict HIV-1 viral rebound after treatment interruption

26. Immunological biomarkers predict HIV-1 viral rebound after treatment interruption

27. Deregulation of subcellular biometal homeostasis through loss of the metal transporter, Zip7, in a childhood neurodegenerative disorder

28. Kinase Inhibitor Screening Identifies Cyclin-Dependent Kinases and Glycogen Synthase Kinase 3 as Potential Modulators of TDP-43 Cytosolic Accumulation during Cell Stress

29. Increased Zinc and Manganese in Parallel with Neurodegeneration, Synaptic Protein Changes and Activation of Akt/GSK3 Signaling in Ovine CLN6 Neuronal Ceroid Lipofuscinosis

30. Copper modulates the large dense core vesicle secretory pathway in PC12 cells

31. Exhaustion of Activated CD8 T Cells Predicts Disease Progression in Primary HIV-1 Infection.

32. Inhibition of TDP-43 Accumulation by Bis(thiosemicarbazonato)-Copper Complexes

34. C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stress

40. CD32-Expressing CD4 T Cells Are Phenotypically Diverse and Can Contain Proviral HIV DNA

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