137 results on '"McFarland, Braden C."'
Search Results
2. Exploring Gut Microbiota Alterations with Trimethoprim-Sulfamethoxazole and Dexamethasone in a Humanized Microbiome Mouse Model.
3. Changes in the gut microbiome community of nonhuman primates following radiation injury
4. Tumor Treating Fields Alter the Kinomic Landscape in Glioblastoma Revealing Therapeutic Vulnerabilities
5. Protein Kinase CK2 and Dysregulated Oncogenic Inflammatory Signaling Pathways
6. Data from Therapeutic Potential of AZD1480 for the Treatment of Human Glioblastoma
7. Supplementary Table 1 from Therapeutic Potential of AZD1480 for the Treatment of Human Glioblastoma
8. Data from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
9. Supplementary Figure 2 from Therapeutic Potential of AZD1480 for the Treatment of Human Glioblastoma
10. Supplementary Figure 5 from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
11. Supplementary Figure Legend from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
12. Supplementary Figure 1 from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
13. Supplementary Figure 4 from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
14. Supplementary Figure 2 from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
15. Supplementary Figure 3 from Activation of the NF-κB Pathway by the STAT3 Inhibitor JSI-124 in Human Glioblastoma Cells
16. Supplementary Figure Legends 1-2 from Therapeutic Potential of AZD1480 for the Treatment of Human Glioblastoma
17. Supplementary Figure 1 from Therapeutic Potential of AZD1480 for the Treatment of Human Glioblastoma
18. CCR Translation for This Article from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
19. Supplementary Figure 2 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
20. Supplementary Figure 6 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
21. Supplementary Methods, Figure Legend from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
22. Supplementary Figure 5 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
23. Supplementary Figure 4 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
24. Supplementary Figure 1 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
25. Supplementary Figure 3 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
26. Supplementary Figure 7 from Targeting Protein Kinase CK2 Suppresses Prosurvival Signaling Pathways and Growth of Glioblastoma
27. Supplementary Figure Legends 1-3 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
28. Data from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
29. Supplementary Figure 3 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
30. Supplementary Figure 1 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
31. Supplementary Figure 2 from Plasminogen Kringle 5 Induces Apoptosis of Brain Microvessel Endothelial Cells: Sensitization by Radiation and Requirement for GRP78 and LRP1
32. Cooperativity between H3.3K27M and PDGFRA poses multiple therapeutic vulnerabilities in human iPSC-derived diffuse midline glioma avatars
33. Protein kinase CK2 is important for the function of glioblastoma brain tumor initiating cells
34. Glioma and the gut–brain axis: opportunities and future perspectives
35. NF-κB and STAT3 in glioblastoma: therapeutic targets coming of age
36. Additional file 1 of Changes in the gut microbiome community of nonhuman primates following radiation injury
37. Hedgehog blockade remodels the gut microbiota and the intestinal effector CD8+T cells in a mouse model of mammary carcinoma
38. Hedgehog blockade remodels the gut microbiota and the intestinal effector CD8+ T cells in a mouse model of mammary carcinoma.
39. Figure S1. StrainPhlAn on the mothers-infants data set. from An individualized mosaic of maternal microbial strains are transmitted to the infant gut microbial community
40. Human gut microbial communities dictate efficacy of anti-PD-1 therapy in a humanized microbiome mouse model of glioma
41. Function of SOCS3 on Macrophage Polarization and Inflammation: CS10-4
42. Changes in the Gut Microbiome Community of nonhuman primate following radiation injury
43. An individualized mosaic of maternal microbial strains is transmitted to the infant gut microbial community
44. Reactive astrocytes foster brain metastases via STAT3 signaling
45. Therapeutic Efficacy of Suppressing the JAK/STAT Pathway in Multiple Models of EAE1
46. Attenuation of PKR-like ER Kinase (PERK) Signaling Selectively Controls Endoplasmic Reticulum Stress-induced Inflammation Without Compromising Immunological Responses
47. Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma
48. TMIC-17LOSS OF SOCS3 IN MYELOID CELLS PROMOTES A DECREASED M2 MACROPHAGE PHENOTYPE AND AN INCREASED CYTOTOXIC T-CELL RESPONSE IN A SYNGENEIC MODEL OF GLIOMA
49. SOCS3 Deficiency in Myeloid Cells Promotes Tumor Development: Involvement of STAT3 Activation and Myeloid-Derived Suppressor Cells
50. Loss of tumor suppressive microRNA-31 enhances TRADD/NF-κB signaling in glioblastoma
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.