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Introduction: In recent years, there is a growing interest in microbiota and how certain dietary patterns affect our brain. We know that diet has an important impact in physical and mental health. The mecanism that underlies is already unknown, but there is emerging evidence that diet modulates brain gut microbiota and has implications in mental problems. Objectives: The aim of this poster is highlight the importance of diet in mental health and the link with microbiota. Methods: Review of recent literature about diet, microbiota and psychiatry. The studies were collected of the electronic databases PubMed. Results: New researches highlight the importance of adequate nutrition for mental health. Several studies link healthy diet with a minor risk of mental illnesses or with the improvement of depressive symptoms. Likewise, poor dietary habits could aggravate cognitive decline and increased risk of developing anxiety, depression or other mental illnesses. It has been shown that a diet rich in fiber, polyphenols and micronutrients improve gut microbial composition and can reduce metabolic endotoxemia and neuroinflammation, and this has been associated with improvements in brain health. Also, prebiotic and probiotics have positive effects. Therefore, dietary interventions could be a complementary therapeutic approach for patients with mental problems. This is what nutritional psychiatry focuses on. Conclusions: Microbiota as a potential therapeutic target for mental illness is a hot topic in psychiatry, but also, its interaction with dietary change or the use of probiotics and prebiotics. This action is easy to implement in our clinical practice and could be part of a biopsychosocial treatment to improve or prevent some psychiatric disorders. Nutritional psychiatry is a new field that needs to be developed and the knowledge in microbiota, diet and mental health could help. Hopefully, the research about this topic continues expanding. Disclosure of Interest: None Declared [ABSTRACT FROM AUTHOR]

Introduction: Pseudologia Fantastica (Delbrück, 1891), also known asmythomania or pathological lying, is a psychological phenomenon where patients represent certain fantasies as real occurences. In contrast to a common lie that pursues a goal, the pseudologue has internalmotives or unconscious gains, so there is no obvious external motive for lying. It has been described in the field for over a century. RichardAsher (1951) published his original observations onMunchausen 's syndrome, but he reflected that the lies, and no medical symptoms, were essential to factitious presentations. Objectives: We present a case based on pathologic lying. We propose the usefulness of Eye Movement Desensitization and Reprocessing (E.M.D.R) therapy. Methods: A systematic review of the literature published in the topic and the discussion of the implications in the differential diagnosis and treatment. Results: The patient is a 34-year-old man with no previous medical history. No substance use disorder or other psychiatric diagnoses, except an anxiety disorder. The pathological lying was persistent since he was an adult and consisted of pretending to be a doctor or a lawyer. In addition he was diagnosed of mayor depressive disorder and insomnia. Conclusions: Although Pseudologia fantastica is not coded in the DSM-5, it has historically been associated with factitious disorder. It is difficult to distinguish factitious disorders from somatisation, conversion and dissociation disorders. In all of them, E.M.D.R therapy might be useful. [ABSTRACT FROM AUTHOR]

Introduction: There is a significant relationship bewtween ischemic heart disease (IHD) and depression or anxiety, with 20% of patients having a depressive disorder after acute myocardial infarction (AMI). The first symptoms of depression appear between 48 and 72 hours after AMI and in most patients disappear within a week. The incidence of depression and anxiety symptoms is important because of the potential impact of these variables on subsequent morbidity and mortality of these patients. Objectives: To describe the cardiac rehabilitation programdesigned in our hospital. Methods: We describe an intensive, multimodal, 8-week cardiac rehabilitation treatment program, for patients who have had a cardiac event. Results: The program is comprised by different professionals: cardiologist, nurse, physiotherapist, psychologist and psychiatrist, who work in a coordinated manner to ensure the optimal and comprehensive treatment of all patients who are part of the program. So far, up to four patients, out of sixty, have required specific treatment with psychotropic drugs for depressive and anxious symptoms. Both at baseline and after 8 weeks of treatment, the Hamilton Depression Rating Scale (HAM-D) and the Montgomery Asberg Depression Rating Scale (MADRS) were applied to patients who showed depressive symptoms, with significant improvement in the scores of both scales at the end of the treatment program. Conclusions: Anxiety and depression should be closely monitored in all patients with cardiovascular disease using simple screening methods, as they are important predictors of the outcome in these kind of patients. [ABSTRACT FROM AUTHOR]

Introduction: The coexistence of psychotic symptoms and obsessive-compulsive symptoms (OCS) has been known since the 19th century, with a high prevalence of bothOCS (25%) and obsessive compulsive disorder (OCD) (12%) in patients with schizophrenia. Objectives: The main objective is to assess the validity of the schizoobsessive disorder as a diagnostic entity for those patients who show symptoms of schizophrenia and OCD. Methods: We present the case of a 61-year-old male with a diagnosis of schizophrenia from the age of 28 and obsessive-compulsive symptoms in the last 5 years. Results: The patient has important negative symptomatology, with ideoaffective impoverishment, short speech, abulia, motor slowing, poor social skills, abandonment of self-care, tendency to isolation, obsessive thoughts and compulsions of doubt and verification. He describes the presence of magical thinking, having to leave the doors opened, with the fear of bad things happening instead, he washes hands ten times a day to get rid of the negative energy of the objects, leaves the shoes in the room always in the same way and in the same place to prevent something bad from happening. He also maintains a delusional ideation of unstructured, chronic damage, without behavioral repercussion. Complementary tests (blood test and cranial CT) were normal. Conclusions: The differential diagnosis is made between schizoobsessive disorder and comorbid schizophrenia with OCD. He is being treated with clozapine, which has shown an exacerbation of obsessive symptoms, which is why amisulpride and sertraline are added. The schizo-obsessive disorder has clinical, neurobiological and neurocognitive features distinct from both schizophrenia and OCD. [ABSTRACT FROM AUTHOR]

Introduction: BD and ADHD are highly prevalent neurodevelopmental disorders, with an early onset age and a chronic course. Comorbidity rates between ADHD and BD vary between 9.5 and 30%. This has been systematically associated with an earlier age at the onset of bipolar disorder and a more chronic and disabling course of the disorder. Objectives: The aim of the study is to shed light on the comorbidity of BD and ADHD in routine clinical practice. Methods: We describe the case of a 20-year-old male patient diagnosed with bipolar disorder (BD), attention deficit hyperactivity disorder (ADHD) and tic disorder. Following the initial evaluation, in which both diagnoses were confirmed, he begun with frequent tics and intense mood swings. Results: The psychopathological examination was characterized by recurring distractions and difficulties in maintaining attention. Slight psychomotor restlessness. Mood swings showing a low mood tendency, without major depressive episode criteria. Impulsiveness. Judgment of reality preserved. Complementary tests (complete blood and urine analysis, electrocardiogram, as well as imaging test consisting of computerized axial tomography) were within the normal range, except for cannabis positivity. The differential diagnosis was made between TBP and ADHD. Valproic acid and guanfacine were prescribed, which has been shown to improve the tics and symptoms of ADHD in a specific group of patients. Conclusions: This particular case made us consider the spectrum of impulsivity, in which many patients with different diagnoses converge. Knowing the high comorbidity rates between BD and ADHD, as well as the different therapeutic options, is essential, since it is the only way to improve the outcome. [ABSTRACT FROM AUTHOR]

Introduction: Psychosis is a frequent complication of Parkinson disease. It is mainly characterized by visual hallucinations and delusions. Hallucinations are the most common manifestation and affect up to 40% of patients, particularly those in an advanced stage of the disease. Objectives: To describe the relationship between Parkinson disease and psychosis. Methods: We present the case of a 75-year-old patient diagnosed with Parkinson disease (stage II) who began with visual hallucinations and then developped delusions of harm. Results: Complementary tests (cranial CT, blood test, ECG) were normal. Ophthalmic pathology was ruled out. Exploration: Hypomimia, monotonous voice. Asymmetrical resting hands tremor, predominantly right, bilateral mild postural tremor. Bradykinesia Right, mild left spontaneous stiffness. Spastic paraparesis Paretoespastic march with support. Conscious and globally oriented. Fluent and spontaneous language, with coherent and structured discourse. Low mood, preserved emotional resonance. Denies autolytic planning. Delusional ideation of harm. Visual and auditory hallucinations. Chronobiological rhythms preserved. Conclusions: The adverse effects of dopamine agonists are probably the most important cause of psychosis in these patients. The differential diagnosis would include a late onset psychosis, Charles Bonnet syndrome and a secondary psychosis. Oral treatment was started with 100mg / day of quetiapine in three doses, with good tolerance and effectiveness on the previously described symptomatology. The recommendations for the management of psychotic symptoms are: 1) Discard intercurrent process. 2)Avoid anticholinergicmedication. 3) Reduce or suspend antiparkinsonian medications. 4) Antipsychotic treatment. [ABSTRACT FROM AUTHOR]