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Both strain waves, generated by the shock wave, and expanding gaseous products of the explosion contribute to the destruction of the rocks. The role of these factors is especially obvious at explosion of soft and moderately hard rocks. The article considers some aspects of the phenomena accompanying the explosion of the detonating explosive charge on example of the unworked coal.

When developing coal and ore deposits by underground method, it is necessary to carry out a large volume of preparatory mine workings using drilling and blasting operations. Minerals are found in rocks under various conditions and their physical and mechanical properties change in a wide range. Under such conditions, it is necessary to use drilling machines this different to the principle of action and structural features. The article considers geometric parameters of roc cutters, peculiarities of drilling modes, affecting energy intensity and drilling productivity in rocks with different strength coefficient.

The lipoprotein (LP) fractions VLDL, LDL, HDL2 and HDL3 were prepared by ultracentrifugation of plasma from healthy volunteers and from patients with coronary heart disease (CHD). We investigated the capacity of platelets from healthy volunteers and patients with atherosclerosis to generate thromboxane A2 (TXA2) during spontaneous clotting of whole blood under the influence of the lipoprotein fractions. In our experiments the serum concentration of TXB2, reflecting the capacity of platelets to generate TXA2 during clotting, depends on several factors: the type of LP fraction used, the blood used for generation of TXA2, and for the same LP fraction whether it was taken from plasma of healthy volunteers or patients with CHD. VLDL prepared from plasma of healthy volunteers inhibited but VLDL prepared from plasma of patients with CHD enhanced the TXA2 formation of platelets from healthy volunteers (p less than 0.05, resp.). LDL from CHD patients inhibited the TXA2 formation of platelets from atherosclerotic patients (p less than 0.01). The HDL subfractions HDL2 and HDL3 from healthy volunteers inhibited TXA2 formation by platelets from healthy volunteers as well as those from atherosclerotic patients (p less than 0.05; p less than 0.01, respectively). HDL2 from patients with CHD inhibited only the TXA2 formation of platelets from healthy volunteers (p less than 0.01), whereas HDL3 from CHD patients inhibited only the TXA2 formation of platelets from atherosclerotic patients (p less than 0.01).