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164 results on '"Marr, T."'

11. Genome-wide scan of bipolar disorder in 65 pedigrees: supportive evidence for linkage at 8q24, 18q22, 4q32, 2p12, and 13q12

Author

McInnis, M G, Lan, T-H, Willour, V L, McMahon, F J, Simpson, S G, Addington, A M, MacKinnon, D F, Potash, J B, Mahoney, A T, Chellis, J, Huo, Y, Swift-Scanlan, T, Chen, H, Koskela, R, Colin Stine, O, Jamison, K R, Holmans, P, Folstein, S E, Ranade, K, Friddle, C, Botstein, D, Marr, T, Beaty, T H, Zandi, P, and Raymond DePaulo, J

Published
2003
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20. Metallographic Preparation of Aluminium-Titanium Composites

Author

Romberg, J., primary, Freudenberger, J., additional, Scharnweber, J., additional, Gaitzsch, U., additional, Marr, T., additional, Eschke, A., additional, Kühn, U., additional, Oertel, C.-G., additional, Okulov, I., additional, Petters, R., additional, Skrotzki, W., additional, Eckert, J., additional, and Schultz, L., additional

Published
2013
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31. Insuring the data processing risk

Author

Haack, Marr T.

Subjects
risk management -- Cases, Computers, Property and casualty insurance -- Cases, Business, Insurance
Abstract

The day-to-day operation of every company is related to, or directly dependent upon, data processing. No company can escape the risk of an accident that impairs its dependency on computers. [...]

Published
1984

34. Update from the DANA consortium on the genetics of bipolar disorder

Author

Mclnnis, M. G., primary, Xu, J. F., additional, Simpson, S. G., additional, McMahon, F. J., additional, Friddle, C., additional, Stine, O. C., additional, Koskella, R. J., additional, MacKinnon, D. F., additional, Gschwend, M., additional, Meyers, D. A., additional, Botstein, D., additional, Marr, T. G., additional, and DePaulo, J. R., additional

Published
1996
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37. Tool chest. Outpatient therapeutic recommendations by diabetes educators in a rural Vermont hospital.

Author

Gorson DM, Haytko P, Carpenter P, Marr T, Borders LK, and Probola D

Abstract

Because of the rapid increase in the incidence of Type 2 diabetes that is occurring in southwestern Vermont (which correlates with the national increase), a 'therapeutic guidelines' worksheet was developed to assist primary care providers with the management of Type 2 diabetes patients. Hospital-based diabetes nurse educators access cholesterol and A1C information from the hospital laboratory, assess the patient, and then make recommendations to the primary care provider. The worksheet is designed to be easily used by both the educator and the primary care provider. Feedback has been positive, as the worksheet serves to enhance providers' ability to treat patients and to achieve recommended treatment goals. [ABSTRACT FROM AUTHOR]

Published
2006

38. Alignment of molecular sequences seen as random path analysis

Author

Zhang, M. Q. and Marr, T. G.

Abstract

We propose a generating functional method—random path analysis (RPA)—that generalizes the classical dynamic programming (DP) method widely used in sequence alignments. For a given cost function, DP is a deterministic method that finds an optimal alignment by minimizing the total cost function for all possible alignments. By allowing uncertainty, RPA is a statistical method that weights fluctuating alignments by probabilities. Therefore, DP maybe thought of as the deterministic limit of RPA when the fluctuations approach zero. DP is the method of choice if one is only interested in optimal alignment. But we argue that, when information beyond the optimal alignment is desired, RPA gives a natural extension of DP for biological applications. As an algebraic approach, RPA is computationally intensive for long sequences, but it can provide better parametric control for developing analytical or perturbational results and it is more informative and biologically relevant. The idea of RPA opens up new opportunities for simulational approaches and more importantly it suggests a novel hardware implementation that has the potential of improving the way a sequence alignment is done. Here we focus on deriving a mathematically rigorous solution to RPA both in its combinatorial form and in its graphical representation; this puts DP in logical perspective under a more general conceptual framework.

Published
1995
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39. Rabbit mortality survey

Author

Whitney, J. C., Blackmore, D. K., Townsend, G. H., Parkin, R. J., Hugh-Jones, M. E., Crossman, P. J., Graham-Marr, T., Rowland, A. C., Festing, M. F. W., and Krzysiak, D.

Abstract

In a survey of mortality in 2224 rabbits of up to 12 weeks of age, drawn from 10 production units, neonatal mortality was important with 111 still-births and 175 deaths in the 1st week of life out of a total of 457 deaths in the whole survey. There was a low overall incidence of death from respiratory and enteric disease during the period of study.

Published
1976
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42. Evidence for linkage of bipolar disorder to chromosome 18 with a parent-of-origin effect

Author

Stine, O. C., Xu, J., Koskela, R., Francis McMahon, Gschwend, M., Friddle, C., Clark, C. D., Mclnnis, M. G., Simpson, S. G., Breschel, T. S., Vishio, E., Riskin, K., Feilotter, H., Chen, E., Shen, S., Folstein, S., Meyers, D. A., Botstein, D., Marr, T. G., and Depaulo, J. R.

Subjects
Adult, Male, Bipolar Disorder, Adolescent, Genotype, Genetic Linkage, Chromosome Mapping, Humans, Female, Original Articles, Lod Score, Chromosomes, Human, Pair 18
Abstract

A susceptibility gene on chromosome 18 and a parent-of-origin effect have been suggested for bipolar affective disorder (BPAD). We have studied 28 nuclear families selected for apparent unilineal transmission of the BPAD phenotype, by using 31 polymorphic markers spanning chromosome 18. Evidence for linkage was tested with affected-sib-pair and LOD score methods under two definitions of the affected phenotype. The affected-sibpair analyses indicated excess allele sharing for markers on 18p within the region reported previously. The greatest sharing was at D18S37: 64% in bipolar and recurrent unipolar (RUP) sib pairs (P = .0006). In addition, excess sharing of the paternally, but not maternally, transmitted alleles was observed at three markers on 18q: at D18S41, 51 bipolar and RUP sib pairs were concordant for paternally transmitted alleles, and 21 pairs were discordant (P = 0004). The evidence for linkage to loci on both 18p and 18q was strongest in the 11 paternal pedigrees, i.e., those in which the father or one of the father's sibs is affected. In these pedigrees, the greatest allele sharing (81%; P = .00002) and the highest LOD score (3.51; phi = 0.0) were observed at D18S41. Our results provide further support for linkage of BPAD to chromosome 18 and the first molecular evidence for a parent-of-origin effect operating in this disorder. The number of loci involved, and their precise location, require further study..

48. Vulnerability to hi-tech losses growing

Author

Haack, Marr T.

Subjects
Risk management -- Innovations, Computer crimes -- Prevention, Risk (Insurance) -- Safety and security measures, Computers -- Usage, Business, Insurance
Published
1988

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