156 results on '"Marionneau†, Céline"'
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2. C-terminal phosphorylation of NaV1.5 impairs FGF13-dependent regulation of channel inactivation
3. Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp
4. Determinants of iFGF13-mediated regulation of myocardial voltage-gated sodium (NaV) channels in mouse
5. Up-regulation of voltage-gated sodium channels by peptides mimicking S4-S5 linkers reveals a variation of the ligand-receptor mechanism
6. FHF2 phosphorylation and regulation of native myocardial NaV1.5 channels
7. Cardiac Sodium Current Under Sympathetic Control: Protein Phosphatase 2A Regulates Cardiac Na+ Channels
8. Regulation of the cardiac Na+ channel NaV1.5 by post-translational modifications
9. Dysfunction in Ankyrin-B-Dependent Ion Channel and Transporter Targeting Causes Human Sinus Node Disease
10. Fluorescent‐ and tagged‐protoxin II peptides: potent markers of the Na v 1.7 channel pain target
11. Human model of IRX5 mutations reveals key role for this transcription factor in ventricular conduction
12. Human model of IRX5 mutations reveals key role for this transcription factor in ventricular conduction
13. Proteomic and functional mapping of cardiac NaV1.5 channel phosphorylation sites
14. Human model of IRX5 mutations reveals key role for this transcription factor in ventricular conduction
15. Proteomic and functional mapping of cardiac NaV1.5 channel phosphorylation reveals multisite regulation of surface expression and gating
16. Computer modeling of whole-cell voltage-clamp analyses to delineate guidelines for good practice of manual and automated patch-clamp
17. Homeostatic regulation of electrical excitability in physiological cardiac hypertrophy
18. Proteomic analyses of native brain KV4.2 channel complexes
19. Specific pattern of ionic channel gene expression associated with pacemaker activity in the mouse heart
20. Human model of IRX5 mutations reveals key role for this transcription factor in ventricular conduction.
21. Fluorescent- and tagged-protoxin II peptides: potent markers of the Nav 1.7 channel pain target.
22. RRAD mutation causes electrical and cytoskeletal defects in cardiomyocytes derived from a familial case of Brugada syndrome
23. Mutant voltage-gated Na+ channels can exert a dominant negative effect through coupled gating
24. Voltage-gated sodium channels assemble and gate as dimers
25. Dysfunction of the Voltage-Gated K + Channel b2 Subunit in a Familial Case of Brugada Syndrome
26. Mutant voltage-gated Na+ channels can exert a dominant negative effect through coupled gating.
27. Dysfunction of the Voltage‐Gated K + Channel β2 Subunit in a Familial Case of Brugada Syndrome
28. 0062 : C-terminal phosphorylation of Nav1.5 channels impairs FGF13-dependent inactivation: potential impact in heart failure
29. Proteomic analysis of native cerebellar iFGF14 complexes
30. Physiological and Pathophysiological Insights of Nav1.4 and Nav1.5 Comparison
31. 0279: Phosphoproteomic identification of CAMKII- and SGK1-dependent phosphorylation sites on the native cardiac NAV1.5 channel protein in heart failure
32. A Long QT Mutation Substitutes Cholesterol for Phosphatidylinositol-4,5-Bisphosphate in KCNQ1 Channel Regulation
33. In KCNQ1 Channels, a Long QT Mutation Induces a Regulation by Cholesterol Instead of Phosphatidylinositol-4,5-Bisphosphate
34. Phosphoproteomic Identification of CaMKII- and Heart Failure-Dependent Phosphorylation Sites on the Native Cardiac Nav1.5 Channel Protein
35. Physiological and pathophysiological insights of Nav1.4 and Nav1.5 comparison.
36. Mass Spectrometry-Based Identification of Native Cardiac Nav1.5 Channel α Subunit Phosphorylation Sites
37. The Sodium Channel Accessory Subunit Navβ1 Regulates Neuronal Excitability through Modulation of Repolarizing Voltage-Gated K+Channels
38. Proteomic analysis highlights the molecular complexities of native Kv4 channel macromolecular complexes
39. Co-assembly of Kv4 α Subunits with K+ Channel-interacting Protein 2 Stabilizes Protein Expression and Promotes Surface Retention of Channel Complexes*
40. Variable Nav1.5 Protein Expression from the Wild-Type Allele Correlates with the Penetrance of Cardiac Conduction Disease in the Scn5a+/− Mouse Model
41. KChiP2 Stabilizes Kv4 Protein Expression and Cell Surface Retention to Control Cardiac Ito Channel Densities
42. Distinct Cellular and Molecular Mechanisms Underlie Functional Remodeling of Repolarizing K + Currents With Left Ventricular Hypertrophy
43. PPARα-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy
44. Kv4.3 is not required for the generation of functional Ito,f channels in adult mouse ventricles
45. Calmodulin Kinase II Inhibition Shortens Action Potential Duration by Upregulation of K + Currents
46. Chronic heart rate reduction remodels ion channel transcripts in the mouse sinoatrial node but not in the ventricle
47. Targeted Deletion of Kv4.2 Eliminates I to,f and Results in Electrical and Molecular Remodeling, With No Evidence of Ventricular Hypertrophy or Myocardial Dysfunction
48. Mouse Model of SCN5A -Linked Hereditary Lenègre’s Disease
49. Specific pattern of ionic channel gene expression associated with pacemaker activity in the mouse heart
50. Long-Term Amiodarone Administration Remodels Expression of Ion Channel Transcripts in the Mouse Heart
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