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2. Targeting DNA-PK

4. A Randomized Trial of Combined PD-L1 and CTLA-4 Inhibition with Targeted Low-Dose or Hypofractionated Radiation for Patients with Metastatic Colorectal CancerPD-L1/CTLA-4 Inhibition with Radiation for Colorectal Cancer

7. Concurrent inhibition of CDK2 adds to the anti-tumour activity of CDK4/6 inhibition in GIST

17. Supplementary Data from A Randomized Trial of Combined PD-L1 and CTLA-4 Inhibition with Targeted Low-Dose or Hypofractionated Radiation for Patients with Metastatic Colorectal Cancer

24. 14-3-3 fusion oncogenes in high-grade endometrial stromal sarcoma

26. Abstract A013: CDK2 and CDK4/6 inhibition in GIST: Mechanisms of response and resistance

27. Aberrant AKT activation drives well-differentiated liposarcoma

28. Abstract 5648: Response and resistance to CDK2 and CDK4/6 inhibition in GIST

29. Aurora kinases as prognostic biomarkers in ovarian carcinoma

31. GLI1 Immunohistochemistry Distinguishes Mesenchymal Neoplasms With GLI1Alterations From Morphologic Mimics

32. Correction: A Randomized Trial of Combined PD-L1 and CTLA-4 Inhibition with Targeted Low-dose or Hypofractionated Radiation for Patients with Metastatic Colorectal Cancer

33. A Randomized Trial of Combined PD-L1 and CTLA-4 Inhibition with Targeted Low-Dose or Hypofractionated Radiation for Patients with Metastatic Colorectal Cancer.

34. Relationships between highly recurrent tumor suppressor alterations in 489 leiomyosarcomas

37. Contributors

38. MAX inactivation is an early event in GIST development that regulates p16 and cell proliferation

44. Fusion of the AHRR and NCOA2 genes through a recurrent translocation t(5;8)(p15;q13) in soft tissue angiofibroma results in upregulation of aryl hydrocarbon receptor target genes

47. Contributors

48. MAX inactivation is an early event in GIST development that regulates p16 and cell proliferation

49. Inhibition of Bcl-2 family members sensitises soft tissue leiomyosarcomas to chemotherapy

50. HDACi inhibits liposarcoma via targeting of the MDM2-p53 signaling axis and PTEN, irrespective of p53 mutational status

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