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3. Cell senescence in pulmonary hypertension

4. Contributors

6. Loss of Pla2r1 decreases cellular senescence and age‐related alterations caused by aging and Western diets

9. Serotonin Transporter and Serotonin Receptors

22. Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease

32. The antioxidant N-acetylcysteine protects from lung emphysema but induces lung adenocarcinoma in mice

33. CCR2/CCR5-mediated macrophage–smooth muscle cell crosstalk in pulmonary hypertension

34. Effects of bone marrow-derived cells on monocrotaline- and hypoxia-induced pulmonary hypertension in mice

35. Targeting CCR2 and CCR5 to inhibit macrophage/pulmonary artery smooth muscle cells cross-talk in pulmonary hypertension

36. New Nitric Oxide Donor NCX 1443: Therapeutic Effects on Pulmonary Hypertension in the SAD Mouse Model of Sickle Cell Disease

37. mTOR pathway activation drives lung cell senescence and emphysema

38. mTOR Pathway Activation Drives Lung-Cell Senescence and Emphysema in Chronic Obstructive Pulmonary Disease

39. Beclin1 circulating levels and accelerated ageing markers in COPD

40. Smoking, telomere length and lung function decline: a longitudinal population-based study

41. Telomere attrition in sickle cell anemia

42. Osteopontin, a Key Mediator Expressed by Senescent Pulmonary Vascular Cells in Pulmonary Hypertension

43. Targeting the mTOR signaling pathway to inhibit lung cell senescence in COPD

44. Selective Tuberous Sclerosis Complex 1 Gene Deletion in Smooth Muscle Activates Mammalian Target of Rapamycin Signaling and Induces Pulmonary Hypertension

45. Extracellular Calpain/Calpastatin Balance Is Involved in the Progression of Pulmonary Hypertension

46. Role of interleukin-1 receptor 1/MyD88 signalling in the development and progression of pulmonary hypertension

47. Telomere Shortening in Middle-Aged Men with Sleep-disordered Breathing

48. Calpastatin overexpression impairs postinfarct scar healing in mice by compromising reparative immune cell recruitment and activation

49. Smoking, telomere length and lung function decline: a longitudinal population-based study.

50. Pulmonary artery smooth muscle cell senescence is a pathogenic mechanism for pulmonary hypertension in chronic lung disease

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