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1. Single cell expression and chromatin accessibility of the Toxoplasma gondii lytic cycle identifies AP2XII-8 as an essential ribosome regulon driver

2. Disruption of Plasmodium falciparum kinetochore proteins destabilises the nexus between the centrosome equivalent and the mitotic apparatus

3. The essential malaria protein PfCyRPA targets glycans to invade erythrocytes

4. Development of a Plasmodium vivax biobank for functional ex vivo assays

5. Inhibition of malaria and babesiosis parasites by putative red blood cell targeting small molecules

6. The ecology and epidemiology of malaria parasitism in wild chimpanzee reservoirs

7. Structural organization of erythrocyte membrane microdomains and their relation with malaria susceptibility

8. The molecular basis of antimalarial drug resistance in Plasmodium vivax

9. Plasmodium vivax infection compromises reticulocyte stability

10. A Malaria Parasite Cross Reveals Genetic Determinants of Plasmodium falciparum Growth in Different Culture Media

11. Co-option of Plasmodium falciparum PP1 for egress from host erythrocytes

12. Keras R-CNN: library for cell detection in biological images using deep neural networks

13. Adaptation of Plasmodium falciparum to humans involved the loss of an ape-specific erythrocyte invasion ligand

15. The Modular Circuitry of Apicomplexan Cell Division Plasticity

16. Fussing About Fission: Defining Variety Among Mainstream and Exotic Apicomplexan Cell Division Modes

17. Erythrocytes lacking the Langereis blood group protein ABCB6 are resistant to the malaria parasite Plasmodium falciparum

18. PfCDPK1 mediated signaling in erythrocytic stages of Plasmodium falciparum

19. Author Correction: Adaptation of Plasmodium falciparum to humans involved the loss of an ape-specific erythrocyte invasion ligand

20. Infected erythrocyte-derived extracellular vesicles alter vascular function via regulatory Ago2-miRNA complexes in malaria

21. Ancient human sialic acid variant restricts an emerging zoonotic malaria parasite

22. Bone Marrow Is a Major Parasite Reservoir in Plasmodium vivax Infection

23. International Center of Excellence for Malaria Research for South Asia and Broader Malaria Research in India

24. Deconstructing the parasite multiplication rate of Plasmodium falciparum

25. Plasmodium vivax infection compromises reticulocyte stability

26. Molecular mechanisms of cellular quiescence in apicomplexan parasites

28. Linking nutrient sensing and gene expression in Plasmodium falciparum blood‐stage parasites

29. Comparative single-cell transcriptional atlases of Babesia species reveal conserved and species-specific expression profiles

30. Babesia divergensegress from host cells is orchestrated by essential and druggable kinases and proteases

31. Comparative single-cell transcriptional atlases ofBabesiaspecies reveal conserved and species-specific expression profiles

33. Diverse Malaria Presentations across National Institutes of Health South Asia International Center for Excellence in Malaria Research Sites in India

34. RBC membrane biomechanics and Plasmodium falciparum invasion: probing beyond ligand-receptor interactions

35. Transformative tools for parasitic flatworms

36. Adaptation of Plasmodium falciparum to humans involved the loss of an ape-specific erythrocyte invasion ligand

37. Quantitative comparative analysis of human erythrocyte surface proteins between individuals from two genetically distinct populations

38. The Modular Circuitry of Apicomplexan Cell Division Plasticity

39. A framework for signaling throughout the life cycle of Babesia species

40. Three Signatures of Adaptive Polymorphism Exemplified by Malaria-Associated Genes

41. Bulk segregant analysis reveals environment × genotype interactions determining malaria parasite growth

42. Plasmodium vivax Strains Use Alternative Pathways for Invasion

43. Keras R-CNN: library for cell detection in biological images using deep neural networks

44. Three hallmarks of malaria-induced selection in human genomes

45. Fussing About Fission: Defining Variety Among Mainstream and Exotic Apicomplexan Cell Division Modes

46. Accounting for red blood cell accessibility reveals distinct invasion strategies in Plasmodium falciparum strains

47. Co-option ofPlasmodium falciparumPP1 for egress from host erythrocytes

48. Molecular and cellular interactions defining the tropism of Plasmodium vivax for reticulocytes

49. Epigenetic Variation and Regulation in Malaria Parasites

50. Resistance to Plasmodium falciparum in sickle cell trait erythrocytes is driven by oxygen-dependent growth inhibition

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