349 results on '"Mancias, Joseph D"'
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2. Coordinated Transcriptional and Catabolic Programs Support Iron-Dependent Adaptation to RAS-MAPK Pathway Inhibition in Pancreatic Cancer.
3. Stereotactic MR-guided on-table adaptive radiation therapy (SMART) for borderline resectable and locally advanced pancreatic cancer: A multi-center, open-label phase 2 study
4. The hepcidin-ferroportin axis modulates liver endothelial cell BMP expression to influence iron homeostasis in mice
5. Quantitative proteomics and RNA-sequencing of mouse liver endothelial cells identify novel regulators of BMP6 by iron
6. A Multi-Institutional Phase 2 Trial of Ablative 5-Fraction Stereotactic Magnetic Resonance-Guided On-Table Adaptive Radiation Therapy for Borderline Resectable and Locally Advanced Pancreatic Cancer
7. Gender-Affirming Surgery and Cancer: Considerations for Radiation Oncologists for Pelvic Radiation in Transfeminine Patients
8. Autophagy promotes immune evasion of pancreatic cancer by degrading MHC-I
9. PTEN-induced kinase PINK1 supports colorectal cancer growth by regulating the labile iron pool
10. Modulation of the HIF2α-NCOA4 axis in enterocytes attenuates iron loading in a mouse model of hemochromatosis
11. The Role of NCOA4-Mediated Ferritinophagy Ferritinophagy in Ferroptosis Ferroptosis
12. Autophagy is required for proper cysteine homeostasis in pancreatic cancer through regulation of SLC7A11
13. De novo pyrimidine biosynthesis inhibition synergizes with BCL-XLtargeting in pancreatic cancer
14. Recharacterization of RSL3 reveals that the selenoproteome is a druggable target in colorectal cancer
15. Reimagining high-throughput profiling of reactive cysteines for cell-based screening of large electrophile libraries
16. Chemical Biology Toolkit for DCLK1 Reveals Connection to RNA Processing
17. Supplementary Data 1 from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
18. Arm B count matrices 4 from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
19. Data from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
20. Arm A count matrices 3 from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
21. Table S1 from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
22. TCR raw data 2 from PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
23. Abstract C053: Autophagy regulates MAT2A in response to hypoxia in pancreatic cancer cells
24. Abstract B052: Defining the lysosome proteome during tumor evolution
25. Stereotactic MR-guided on-table adaptive radiation therapy (SMART) for borderline resectable and locally advanced pancreatic cancer: a multi-center, open-label phase 2 study
26. Multiomic analysis on human cell model of wolfram syndrome reveals changes in mitochondrial morphology and function
27. Discovery of a selective inhibitor of doublecortin like kinase 1
28. Neoadjuvant Therapy is Associated with Improved Survival in Borderline-Resectable Pancreatic Cancer
29. The Role of NCOA4-Mediated Ferritinophagy in Ferroptosis
30. PD-1 Blockade Induces Reactivation of Nonproductive T-Cell Responses Characterized by NF-κB Signaling in Patients with Pancreatic Cancer
31. Endothelial ZIP8 plays a minor role in BMP6 regulation by iron in mice
32. Mutations in RABL3 alter KRAS prenylation and are associated with hereditary pancreatic cancer
33. What Patients Look for When Browsing Online for Pancreatic Cancer: The Bait Behind the Byte
34. Rapid and direct control of target protein levels with VHL-recruiting dTAG molecules
35. Plasticity in binding confers selectivity in ligand-induced protein degradation
36. Autophagy supports mitochondrial metabolism through the regulation of iron homeostasis in pancreatic cancer
37. Supplementary Table from NCOA4-Mediated Ferritinophagy Is a Pancreatic Cancer Dependency via Maintenance of Iron Bioavailability for Iron–Sulfur Cluster Proteins
38. Supplementary Figure from NCOA4-Mediated Ferritinophagy Is a Pancreatic Cancer Dependency via Maintenance of Iron Bioavailability for Iron–Sulfur Cluster Proteins
39. Data from NCOA4-Mediated Ferritinophagy Is a Pancreatic Cancer Dependency via Maintenance of Iron Bioavailability for Iron–Sulfur Cluster Proteins
40. Supplementary Data from NCOA4-Mediated Ferritinophagy Is a Pancreatic Cancer Dependency via Maintenance of Iron Bioavailability for Iron–Sulfur Cluster Proteins
41. Supplementary Data from Coordinated Transcriptional and Catabolic Programs Support Iron-Dependent Adaptation to RAS–MAPK Pathway Inhibition in Pancreatic Cancer
42. Data from Coordinated Transcriptional and Catabolic Programs Support Iron-Dependent Adaptation to RAS–MAPK Pathway Inhibition in Pancreatic Cancer
43. Supplementary Figure from Coordinated Transcriptional and Catabolic Programs Support Iron-Dependent Adaptation to RAS–MAPK Pathway Inhibition in Pancreatic Cancer
44. Data from Selective Modulation of a Pan-Essential Protein as a Therapeutic Strategy in Cancer
45. Supplementary Figure S3 from Selective Modulation of a Pan-Essential Protein as a Therapeutic Strategy in Cancer
46. Data from An In Vivo CRISPR Screening Platform for Prioritizing Therapeutic Targets in AML
47. Supplementary Tables from An In Vivo CRISPR Screening Platform for Prioritizing Therapeutic Targets in AML
48. Supplementary Data from An In Vivo CRISPR Screening Platform for Prioritizing Therapeutic Targets in AML
49. Data from Selective Alanine Transporter Utilization Creates a Targetable Metabolic Niche in Pancreatic Cancer
50. Supplementary Table S1 from Selective Modulation of a Pan-Essential Protein as a Therapeutic Strategy in Cancer
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