21 results on '"Mamrosh, Jennifer L."'
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2. Liver receptor homolog‐1 is a critical determinant of methyl‐pool metabolism
3. The fatty liver disease-causing protein PNPLA3-I148M alters lipid droplet-Golgi dynamics
4. Nuclear receptor LRH-1/NR5A2 is required and targetable for liver endoplasmic reticulum stress resolution.
5. p97/VCP promotes degradation of CRBN substrate glutamine synthetase and neosubstrates
6. Quantitative measurement of the requirement of diverse protein degradation pathways in MHC class I peptide presentation
7. A Systematic Interrogation of MHC Class I Peptide Presentation Identifies Constitutive and Compensatory Protein Degradation Pathways
8. A Systematic Interrogation of MHC Class I Peptide Presentation Identifies Constitutive and Compensatory Protein Degradation Pathways
9. LRH-1 is a key regulator of carbon one metabolism and its loss protects from liver injury in the methioninecholine deficient NASH model: 1435
10. A nuclear-receptor-dependent phosphatidylcholine pathway with antidiabetic effects
11. Cand1-Mediated Adaptive Exchange Mechanism Enables Variation in F-Box Protein Expression
12. p97/VCP promotes degradation of CRBN substrate glutamine synthetase and neosubstrates
13. Valosin-containing protein (VCP)–Adaptor Interactions are Exceptionally Dynamic and Subject to Differential Modulation by a VCP Inhibitor
14. Small Heterodimer Partner (NR0B2) Coordinates Nutrient Signaling and the Circadian Clock in Mice
15. Liver receptor homolog‐1 is a critical determinant of methyl‐pool metabolism
16. Correction: Nuclear receptor LRH-1/NR5A2 is required and targetable for liver endoplasmic reticulum stress resolution
17. Using Google Reverse Image Search to Decipher Biological Images
18. p97/VCP promotes degradation of CRBN substrate glutamine synthetase and neosubstrates.
19. Author response: Nuclear receptor LRH-1/NR5A2 is required and targetable for liver endoplasmic reticulum stress resolution
20. The fatty liver disease-causing protein PNPLA3-I148M alters lipid droplet-Golgi dynamics.
21. Quantitative measurement of the requirement of diverse protein degradation pathways in MHC class I peptide presentation.
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