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265 results on '"Maggiore Q"'

2. Dialysis and Bioethics

Author

Maggiore, Q. and Maggiore, Q.

Abstract

No abstract, non disponibile

Published
2018

6. Plasmapheresis and Cryopheresis

Author

Maggiore, Q., L’Abbate, A., Bartolomeo, F., Misefari, V., Caccamo, A., Martorano, C., Zichichi, Antonino, editor, Pavone-Macaluso, Michele, editor, Smith, Philip H., editor, Vercellone, Antonio, editor, Maiorca, Rosario, editor, and Rotolo, Ugo, editor

Published
1981
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10. Phosphate may promote CKD progression and attenuate renoprotective effect of ACE inhibition

Author

Remuzzi, G, Tognoni, G, Ruggenenti, P, Bossini, N, Viola, Bf, Scolari, Francesco, Maiorca, Rosario, Cofano, F, Fellin, G, D'Amico, G, Dissegna, D, Brendolan, A, La Greca, G, Feriozzi, A, Ancarani, E, Gandini, E, D'Amato, I, Giangrande, A, Giannico, G, Vitale, O, Manno, C, Schena, Fp, Mazzi, A, Garini, G, Borghetti, A, Pisoni, R, Mosconi, L, Bertani, T, Scanferla, F, Bazzato, G, Oliva, E, Zoccali, C, Toti, G, Sisca, S, Maggiore, Q, Pignone, E, Boero, R, Piccoli, G, Piperno, R, Rosati, A, Salvadori, M, Ene Iordache, B, Remuzzi, A, Perna, A, Benini, R, Tammuzzo, L, Gaspari, F, Arnoldi, F, Ciocca, I, Signorini, O, Ferrari, S, Gritti, D, Roggeri, A, Del Priore, L, Cattaneo, D, Stucchi, N, Migone, L, Marubini, E, Del Favero, A, Ideo, G, Geraci, E, and Loi, U.

Subjects
ace inhibitors
Published
2011

16. In chronic nephropathies prolonged ACE inhibition can induce remission: dynamics of time-dependent changes in GFR. Investigators of the GISEN Group. Gruppo Italiano Studi Epidemiologici in Nefrologia

Author

Ruggenenti P, Perna A, Benini R, Bertani T, Zoccali C, Maggiore Q, Salvadori M, and Giuseppe Remuzzi

Subjects
Adult, Male, Tissue Survival, Time Factors, Remission Induction, Angiotensin-Converting Enzyme Inhibitors, Middle Aged, Kidney, Cohort Studies, Ramipril, Chronic Disease, Humans, Female, Kidney Diseases, Follow-Up Studies, Glomerular Filtration Rate
Abstract

The Ramipril Efficacy in Nephropathy Core and Follow-Up Study found thator =36 mo of continued ramipril therapy decreased substantially the risk of end-stage renal failure (ESRF) in patients with chronic nephropathies and a urinary protein excretion rateor =3 g/24 h. This study investigates the time-dependent changes in GFR in these patients and in control subjects who were randomized to conventional therapy during the Core period and switched to ramipril during the Follow-Up study. Analyses included 150 patients (continued ramipril: n = 74; switched to ramipril: n = 76) who had at least three GFR measurements (including baseline) during the whole observation period and a subgroup of 43 patients (continued ramipril: n = 26; switched to ramipril: n = 17) who had at least six GFR measurements, including at least three on the Core and at least three on the Follow-Up study. Ramipril (1.25 to 5 mg/d) and conventional therapy were targeted at achieving a diastolic BP below 90 mm Hg. The main efficacy variables were GFR and ESRF (need for dialysis). Analysis was by intention to treat. Throughout the study, the mean +/- SEM rate of GFR decline (deltaGFR) was significantly lower in patients continued on ramipril compared to those switched to ramipril (0.51+/-0.09 versus 0.76+/-0.10 ml/min per 1.73 m2 per mo, P0.03). In patients on continued ramipril who had at least six GFR measured--but not in control subjects--deltaGFR progressively improved with time and, in the cohort with the longest follow-up, decreased from (in ml/min per 1.73 m2 per mo): 0.16+/-0.12 (at 18 mo) to 0.10+/-0.05 (at 60 mo). This rate was about 10-fold slower compared to patients on conventional therapy during the REIN Core study. Analyses of the individual slopes found that at the end of the follow-up, 10 of 26 patients on continued ramipril therapy had a positive deltaGFR and another 10 patients had an improvement of deltaGFR while on ramipril therapy. DeltaGFR significantly improved in parallel with a significant reduction in proteinuria. Changes in deltaGFR (P = 0.0001) and proteinuria (P = 0.04) were significantly different in the two groups. Baseline characteristics and changes in systolic and diastolic BP and 24-h urine urea and sodium excretion were comparable. The present results offer evidence that in chronic nephropathies, the tendency of GFR to decline with time can be effectively halted, even in patients with remarkably severe disease.

Published
1999

17. Linee Guida per la terapia conservativa dell’insufficienza renale cronica

Author

Barsotti, G, Oldrizzi, L, Gentile, Mg, Del Vecchio, L, De Marchi, S, De Pascale, C, De Vecchi, A, Enia, G, Fuiano, G, Gallucci, M, Garibotto, Giacomo, Maggiore, Q, Mangiacapra, S, Panarello, G, Poggi, A, Quintaliani, G, Salvadori, M, Terracciano, V, Timio, M, and Tizianello, A.

Subjects
insufficienza renale cronica, linee guida, insufficienza renale cronica, linee guida
Published
1999

47. The ‘Breakpoint’ Test, A New Statistical Method for Studying Progression of Chronic Renal Failure.

Author

Zoccali, C., Postorino, M., Martorano, C., Salnitro, F., and Maggiore, Q.

Abstract

The application of a new statistical method (‘breakpoint’ test) to the study of the progression of chronic renal failure is described. This test establishes whether the best fit of a series of GFR measurements is linear or broken. Such an approach avoids the analytical constraint of the time of intervention assumed by other methods. Re-analysis by this test of previous studies of low-protein diet suggests that in some cases the effect of the dietary regimen has been overemphasised. [ABSTRACT FROM PUBLISHER]

Published
1989

48. Effect of Ultrafiltration on Plasma Concentrations of Atrial Natriuretic Peptide in Haemodialysis Patients.

Author

Zoccali, C., Ciccarelli, M., Mallamaci, F., Delfino, D., Salnitro, F., Parlongo, S., and Maggiore, Q.

Abstract

We have investigated the influence of body fluid volume status on plasma levels of immunoreactive atrial natriuretic peptide (irANP) in eight uraemic patients on chronic haemodialysis, including two diabetics with severely impaired reflex control of the heart. IrANP was significantly higher in volume-expanded uraemic patients (36±16 pg/ml) than in a group of seven age and sex-matched normal subjects (14±2 pg/ml), and fell consistently, approaching the normal range after the removal of 2.0–4.3 litres of isotonic plasma ultrafiltrate (by isolated ultrafiltration). Plasma levels of the hormone were strictly related to right atrial pressure. The irANP response to ultrafiltration in the two diabetics was similar to that of the other uraemic patients. The results suggest that the elevated irANP levels found in volume-expanded uraemic patients depend largely on fluid overload per Se. The preserved irANP response to ultrafiltration of the two diabetics with severe autonomic neuropathy indicates that in chronic renal failure irANP secretion may be regulated independently from autonomic influences. [ABSTRACT FROM PUBLISHER]

Published
1986

49. Plasma Met-Enkephalin and Leu-Enkephalin in Chronic Renal Failure.

Author

Zoccali, C., Ciccarelli, M., Mallamaci, F., Maggiore, Q., Lotti, M., and Zucchelli, G. C.

Abstract

Plasma met-enkephalin and leu-enkephalin has been measured in a group of 28 patients with chronic renal failure, to discover whether these opioids are affected by standard haemodialysis and haemofiltration. Met-enkephalin was markedly higher (<0.001) in uraemic patients than in a group of 13 normal subjects, and was directly related to plasma creatinine (=0.60; <0.01) and to plasma urea (=0.36; =0.06). In contrast, leuenkephalin was suppressed in uraemic patients (<0.001). Met-enkephalin fell slightly but significantly (<0.02) after both haemodialysis and haemofiltration; however, on average it remained at concentrations four times higher than normal. No changes in plasma leu-enkephalin were observed after haemodialysis and haemofiltration. The cause(s) of the altered plasma concentrations of these opioid substances remains to be clarified. [ABSTRACT FROM PUBLISHER]

Published
1987

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