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Background: Cadence-controlled walking may be a desirable approach for older adults to self-monitor exercise intensity and achieve physical activity guidelines. We examined the acute effects of cadence-controlled walking on cognition and vascular function in physically inactive older adults. Methods: In a randomized crossover design, 26 participants (65% females, 67.8 ± 11.3 years) underwent 30-min acute exercise (walking at 100 steps/min) and control (sitting) conditions. We measured cognition, central blood pressure (BP), and arterial stiffness before, and immediately, after each condition. Results: We observed significant Time × Condition interactions in the Flanker Inhibitory Control and Attention (Flanker) test and Dimensional Change Card Sort (DCCS) test scores, and in central systolic BP, central pulse pressure, and carotid to femoral pulse wave velocity (p <.05). The Flanker and DCCS scores significantly increased after walking (d = 0.4 and 0.5, respectively), but not after sitting. Central systolic BP, central pulse pressure, and carotid to femoral pulse wave velocity significantly increased after sitting but remained unchanged after acute walking (d = 0.4–0.2), with p-values <.05. After walking, significant correlations were observed between DCCS and diastolic BP and central pulse pressure change scores and change scores in central pulse wave velocity, Flanker, and DCCS (rs = −0.45 to −0.52). Conclusion: These findings suggest that a single bout of cadence-controlled walking elicited an immediate improvement in cognition and might have mitigated increases in arterial stiffness and central BP observed in the seated control condition. Further research is needed to examine the association between cognition and vascular function following acute exercise compared to control conditions. Significance: Our findings may have practical implications for developing daily physical activity recommendations for improving the cognitive health for successful aging. [ABSTRACT FROM AUTHOR]

Purpose: Maximal heart rate (MHR) is an important physiologic tool for prescribing and monitoring exercise in both clinical and athletic settings. However, prediction equations developed in adults may have limited accuracy in youth. The purpose of this study was to systematically review and analyze the available evidence regarding the validity of commonly used age-based MHR prediction equations among children and adolescents. Methods: Included articles were peer-reviewed, published in English, and compared measured to predicted MHR in male and female participants <18 years old. The standardized mean difference effect size (ES) was used to quantify the accuracy of age-predicted MHR values and a priori moderators were examined to identify potential sources of variability. Results: The cumulative results of 20 effects obtained from seven articles revealed that prediction equations did not accurately estimate MHR (ES= 0.44, p < 0.05) by 6.3 bpm (bpm). Subgroup analyses indicated that the Fox equation (MHR = 220-age) overestimated MHR by 12.4 bpm (ES = 0.95, p < 0.05), whereas the Tanaka equation (MHR = 208--0.7*age) underestimated MHR by 2.7 bpm (ES = -0.34, p < 0.05). Conclusions: Age-based MHR equations derived from adult populations are not applicable to children. However, if the use of age-based equations cannot be avoided, we recommend using the Tanaka equation, keeping in mind the range of error reported in this study. Future research should control for potential pubertal influences on sympathetic modulation during exercise to facilitate the development of more age-appropriate methods for prescribing exercise intensity.

This study was conducted to investigate the systemic hemodynamic and vascular changes in women during and after two commonly used clinical blood flow restriction (BFR) pressures at rest. There are minimal data regarding the independent effects of BFR on hemodynamic and systemic vascular changes due to pressor response, particularly among women. Therefore, this study investigated BFR‐induced alterations in pressor response and systemic flow redistribution at rest during two commonly used pressures (50% and 80% limb occlusion pressure [LOP]). Fifteen women (22.1 ± 4.2 years) completed two randomised sessions involving 8‐min of bilateral, lower limb restriction at 50% or 80% LOP followed by 8‐min of recovery post‐deflation. Changes in vascular (arterial diameter [DIA], time‐averaged mean velocity [TAMV], volume flow [VF], and area) and hemodynamic (heart rate [HR] and blood pressure) measures over time (pre‐, during, post‐occlusion) and by session (50% vs. 80% LOP) were tested using repeated measures analysis of variance. Repeated measures correlations (rrm) quantified common intraindividual associations between BFR‐induced hemodynamic and vascular responses. HR increased from baseline during 50% LOP and remained elevated during recovery (p < 0.05). HR increased from baseline during 80% LOP, while tibial VF and TAMV decreased (p < 0.03 for all). HR and TAMV values returned to baseline during recovery, while brachial artery VF decreased (p < 0.05). Changes in HR, brachial VF, and brachial TAMV were similar between 50% and 80% LOP (rrm = 0.32–0.70, p < 0.05 for all). At 80% LOP, changes in HR were positively correlated with brachial VF (rrm = 0.38) and TAMV (rrm = 0.43) and negatively correlated with tibial VF (rrm = −0.36) and TAMV (rrm = −0.30) (p < 0.05 for all). Results suggest that BFR at 80% LOP elicits an acute systemic pressor reflex without concomitant increases in brachial arterial flow, while 50% LOP elicits a subdued response. [ABSTRACT FROM AUTHOR]

Introduction: The parasympathetically derived marker of heart rate variability, root mean square of successive R-R differences (RMSSD), and the daily fluctuations as measured by the coefficient of variation (RMSSDCV) may be useful for tracking training adaptations in athletic populations. These vagally derived markers of heart rate variability may be especially pertinent when simultaneously considering a female athlete's menstrual cycle. Purpose: The purpose of this study was to observe the perturbations in RMSSDCV, while considering RMSSD, across a season in the presence and absence of menses with training load in female collegiate rowers. Methods: Thirty-six (20 [1] y, 25.6 [3.4] kg·m−2) National Collegiate Athletic Association Division I female rowers were monitored for 18 consecutive weeks across a full season. Seated, ultrashortened RMSSD measurements were obtained by the rowers on at least 3 mornings per week using a smartphone photoplethysmography device. Following the RMSSD measurement, athletes indicated the presence or absence of menstruation within the application. Individual meters rowed that week and sessions rate of perceived exertion were obtained to quantify training load. Results: Longitudinal mixed-effects modeling demonstrated a significant effect of menses and time, while also considering RMSSD, such that those who were on their period had a significantly greater RMSSDCV than those who were not (11.2% vs 7.5%, respectively; P <.001). These changes were independent of meters rowed, sessions rate of perceived exertion, body mass index, birth-control use, and years of rowing experience, which were all nonsignificant predictors of RMSSDCgV (P >.05). Conclusion: The presence of menses appears to significantly impact RMSSDCV when also considering RMSSD, which may allow coaches to consider individualized training plans accordingly. [ABSTRACT FROM AUTHOR]

Purpose: To assess the agreement of the root mean square of successive R-R interval (RMSSD) values when recorded immediately upon waking to values recorded later in the morning prior to practice, and to determine the associations of the RMSSD recordings with performance outcomes in female rowers. Methods: A total of 31 National Collegiate Athletic Association Division I rowers were monitored for 6 consecutive days. Two seated RMSSD measurements were obtained on at least 3 mornings using a smartphone-based photoplethysmography application. Each 1-minute RMSSD measure was recorded following a 1-minute stabilization period. The first (T1) measurement occurred at the athlete's home following waking, while the second (T2) transpired upon arrival at the team's boathouse immediately before practice. From the measures, the RMSSD mean and coefficient of variation were calculated. Two objective performance assessments were conducted on an indoor rowing ergometer on separate days: 2000-m time trial and distance covered in 30 minutes. Interteam rank was determined by the coaches, based on subjective and objective performance markers. Results: The RMSSD mean (intraclass correlation coefficient =.82; 95% CI,.63 to.92) and RMSSD coefficient of variation (intraclass correlation coefficient =.75; 95% CI,.48 to.88) were strongly correlated at T1 and T2, P <.001. The RMSSD mean at T1 and T2 was moderately associated with athlete rank (r = −.55 and r = −.46, respectively), 30-minute distance (r =.40 and r =.41, respectively), and 2000 m at T1 (r = −.37), P <.05. No significant correlations were observed for the RMSSD coefficient of variation. Conclusion: Ultrashort RMSSD measurements taken immediately upon waking show very strong agreement with those taken later in the morning, at the practice facility. Future research should more thoroughly investigate the relationship between specific performance indices and the RMSSD mean and coefficient of variation for female collegiate rowers. [ABSTRACT FROM AUTHOR]

Certain occupations are associated with greater risk of triggering a sudden cardiac event because of high levels of physical exertion and extreme thermal environments in which they occur. The extent to which sports officials––particularly high school (HS) American football referees––experience these conditions is unknown. Forty-six male HS officials (72% White/Caucasian; age = 48 ± 12 years, body mass index = 31.7 ± 6.6 kg·m−2) were studied to quantify the physiological strain and physical demands of officiating. Referee demographics (e.g., experience, habitual exercise), pre-game urine specific gravity (USG), thermal (peak core temperature [Tcore]) and cardiovascular (average heart rate [HR]) strain, kinematic activity (e.g., total distance, speed, mechanical intensity), and environmental conditions were measured during 10 regular season varsity HS football games (≈2.5 h each) in the Southeastern United States (average wet bulb globe temperature and relative humidity: 18.9 ± 6.0 °C and 78.2% ± 12.1%). Analyses included descriptive statistics, bivariate correlations, and linear regression. Referees covered 5.2 ± 1.2 km per game, eliciting average HR and peak Tcore of 71.5% ± 8.0% HRmax and 38.3 ± 0.5 °C, respectively; 38% began games dehydrated (USG = 1.026 ± 0.004). Multiple regression analyses revealed that obesity (β = 0.34), not participating in regular exercise (β = −0.36), and officiating at lower mechanical intensity (β = −0.33) predicted greater cardiovascular strain (all p ≤ 0.03). White/Caucasian race/ethnicity (β = 0.59), younger age (β = −0.46), and obesity (β = 0.28) predicted greater thermal strain (all p ≤ 0.01). HS football referees experienced elevated levels of physiological strain while officiating, with individual factors modulating the magnitude of strain. Strategies aimed at reducing obesity, increasing exercise participation, and improving cardiovascular health should be emphasized to mitigate strain and prevent cardiac events. [ABSTRACT FROM AUTHOR]

Most blood flow restriction (BFR) devices use a set pressure to occlude flow, and the effects of these devices on post-exercise recovery in the distal musculature have not been thoroughly explored. This study evaluates the effect of BFR on fatigue during resistance exercise in the musculature distal to the occlusion site. Twenty participants (age: 23 ± 4 years) completed two exercise sessions with or without BFR in a crossover-design. The participants performed 3 sets of 15 repetitions of maximum ankle plantarflexion (PF) and dorsiflexion (DF) on an isokinetic dynamometer. Eighty percent limb occlusion pressure (LOP) was applied to the thigh during BFR. Isokinetic dynamometer metrics were evaluated during the exercise. Gastrocnemius cross sectional area (CSA) was measured preexercise and post-exercise. A toe-tap test, average step length test, and counter-movement jump test were employed to evaluate fatigue and recovery from pre-exercise to post-exercise. Repeated measures ANOVA was used to determine significant differences. BFR caused a significant reduction in peak torque (P = 0.009), average force (P<0.001), total work for PF (P<0.001), and elevated RPE (P<0.001) during the exercise compared to the control. A significantly lower (P<0.001) number of toe taps were performed after the BFR (28.9 ± 1.7) condition when compared to the control (42.7 ± 1.3). CSA was increased pre-exercise vs. post-exercise for BFR only and was significantly elevated compared to the control (P = 0.009). BFR appears to induce fatigue, likely through accumulation of metabolic stress as evidenced by the enhanced muscular swelling and rapid recovery rate post-exercise. [ABSTRACT FROM AUTHOR]

Purpose: The aim of this study was to compare the effects of low ([LV]; 4 total sets), moderate ([MV]; 8 total sets), and high set volumes ([HV]; 12 total sets) in acute full-body resistance exercise sessions on post-exercise parasympathetic reactivation measured using RMSSD. Methods: Ten resistance-trained participants (25.8 ± 6.8 yr., 173.4 ± 10.6 cm, 75.4 ± 9.9 kg) performed three resistance exercise sessions. During each session, heart rate variability (HRV) was measured pre- and for 30 min post-exercise, divided into 5-min segments stabilization, Post5-10, Post10-15, Post15-20, Post20-25, and Post25-30. Repeated-measures ANOVA was used to assess differences within and between pre-post exercise natural logarithm RMSSD (LnRMSSD) values. To assess the initial change in LnRMSSD, the delta percent change (ΔLnRMSSD) from pre-exercise to Post5-10 (ΔLnRMSSDpre-post) was calculated for each session. The ΔLnRMSSD was also calculated between Post5-10 and Post25-30 (ΔLnRMSSDpost5-30) to assess recovery. Results: Significant differences were observed between sessions and when comparing pre-exercise values to all post-exercise times across sessions (p ≤.05). The LV session resulted in significantly higher mean LnRMSSD value (3.62) post-exercise compared to both the MV (3.11, effect size [ES] = 3.77) and HV (3.02, ES = 3.92) sessions while the MV and HV sessions produced similar responses. Across sessions no return to baseline occurred and when comparing sessions, no significant differences were found in ΔLnRMSSDpre-post or ΔLnRMSSDpost5-30. Conclusion: Acute bouts of full-body resistance exercise can cause similar reductions in LnRMSSD from pre-exercise levels and can delay parasympathetic reactivation back to baseline values during the same 30-min recovery period despite differences in set volume. [ABSTRACT FROM AUTHOR]