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Your search keyword '"Luo, Manqing"' showing total 19 results
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19 results on '"Luo, Manqing"'

7. The predictive value of the neutrophil/platelet ratio on in-hospital adverse events and long-term prognosis in patients with coronary artery disease after percutaneous coronary intervention and its possible internal mechanism

Author

Xiang, Yifei, primary, Zeng, Jilang, additional, Lin, Xueqin, additional, Cai, Xiaoling, additional, Zhang, Liwei, additional, Luo, Manqing, additional, Zhou, Gengyu, additional, Chen, Xiaofang, additional, Lin, Biting, additional, Chen, Junhan, additional, Chen, fan, additional, Huang, Xuan, additional, Guo, Yansong, additional, and Lin, Kai-Yang, additional

Published
2023
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10. A salt stress‐activated GSO1‐SOS2‐SOS1 module protects the Arabidopsis root stem cell niche by enhancing sodium ion extrusion

Author

Chen, Changxi, primary, He, Gefeng, additional, Li, Jianfang, additional, Perez‐Hormaeche, Javier, additional, Becker, Tobias, additional, Luo, Manqing, additional, Wallrad, Lukas, additional, Gao, Junping, additional, Li, Jia, additional, Pardo, José M, additional, Kudla, Jörg, additional, and Guo, Yan, additional

Published
2023
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12. A salt stress-activated GSO1-SOS2-SOS1 module protects the Arabidopsis root stem cell niche by enhancing sodium ion extrusion

Author

National Key Research and Development Program (China), National Natural Science Foundation of China, German Research Foundation, Ministerio de Ciencia, Innovación y Universidades (España), European Commission, China Scholarship Council, Chen, Changxi [0000-0003-2833-3264], Li, Jianfang [0000-0002-2445-677X], Perez-Hormaeche, Javier [0000-0002-1797-9468], Wallrad, Lukas [0000-0002-9153-6331], Li, Jia [0000-0002-3148-6897], Pardo, José M [0000-0003-4510-8624], Kudla, Jörg [0000-0002-8238-767X], Guo, Yan [0000-0002-6955-8008], Chen, Changxi, He, Gefeng, Li, Jianfang, Perez-Hormaeche, Javier, Becker, Tobias, Luo, Manqing, Wallrad, Lukas, Gao, Junping, Li, Jia, Pardo, José M., Kudla, Jörg, Guo, Yan, National Key Research and Development Program (China), National Natural Science Foundation of China, German Research Foundation, Ministerio de Ciencia, Innovación y Universidades (España), European Commission, China Scholarship Council, Chen, Changxi [0000-0003-2833-3264], Li, Jianfang [0000-0002-2445-677X], Perez-Hormaeche, Javier [0000-0002-1797-9468], Wallrad, Lukas [0000-0002-9153-6331], Li, Jia [0000-0002-3148-6897], Pardo, José M [0000-0003-4510-8624], Kudla, Jörg [0000-0002-8238-767X], Guo, Yan [0000-0002-6955-8008], Chen, Changxi, He, Gefeng, Li, Jianfang, Perez-Hormaeche, Javier, Becker, Tobias, Luo, Manqing, Wallrad, Lukas, Gao, Junping, Li, Jia, Pardo, José M., Kudla, Jörg, and Guo, Yan

Abstract

Soil salinity impairs plant growth reducing crop productivity. Toxic accumulation of sodium ions is counteracted by the Salt Overly Sensitive (SOS) pathway for Na+ extrusion, comprising the Na+ transporter SOS1, the kinase SOS2, and SOS3 as one of several Calcineurin-B-like (CBL) Ca2 + sensors. Here, we report that the receptor-like kinase GSO1/SGN3 activates SOS2, independently of SOS3 binding, by physical interaction and phosphorylation at Thr16. Loss of GSO1 function renders plants salt sensitive and GSO1 is both sufficient and required for activating the SOS2-SOS1 module in yeast and in planta. Salt stress causes the accumulation of GSO1 in two specific and spatially defined areas of the root tip: in the endodermis section undergoing Casparian strip (CS) formation, where it reinforces the CIF-GSO1-SGN1 axis for CS barrier formation; and in the meristem, where it creates the GSO1-SOS2-SOS1 axis for Na+ detoxification. Thus, GSO1 simultaneously prevents Na+ both from diffusing into the vasculature, and from poisoning unprotected stem cells in the meristem. By protecting the meristem, receptor-like kinase-conferred activation of the SOS2-SOS1 module allows root growth to be maintained in adverse environments.

Published
2023

19. Assessing the Predictive Value of Different Nutritional Indexes for Contrast-Associated Acute Kidney Injury in Patients Undergoing Percutaneous Coronary Intervention.

Author

Chen L, Zhang S, Luo M, He C, You Z, Zhang L, Zeng J, Chen J, Lin K, and Guo Y

Subjects
Humans, Male, Female, Aged, Middle Aged, Risk Factors, Risk Assessment, Aged, 80 and over, Retrospective Studies, Percutaneous Coronary Intervention adverse effects, Acute Kidney Injury chemically induced, Acute Kidney Injury etiology, Acute Kidney Injury diagnosis, Contrast Media adverse effects, Malnutrition diagnosis, Nutritional Status, Nutrition Assessment, Predictive Value of Tests
Abstract

Background: The link between malnutrition and poor prognosis in cardiovascular disease has been established but the association between malnutrition and contrast-associated acute kidney injury (CA-AKI), a common complication of coronary procedures, remains poorly understood. In this study we investigated the predictive value of 3 nutritional indexes for CA-AKI in patients undergoing percutaneous coronary intervention (PCI)., Methods and results: The study included a total of 6,049 consecutive patients undergoing PCI between May 2012 and September 2020, among whom 352 (5.8%) developed CA-AKI. We used the Controlling Nutritional Status (CONUT) score, the Geriatric Nutritional Risk Index (GNRI), and the Prognostic Nutritional Index (PNI) to assess the association between malnutrition risk and CA-AKI after PCI. Multivariate logistic regression analysis revealed that malnutrition, as identified by GNRI and PNI, was significantly associated with a higher risk of CA-AKI (moderate-severe malnutrition in GNRI: odds ratio [OR]=1.92, [95% confidence interval (CI), 1.27-2.85]; malnutrition in PNI: OR=1.87, [95% CI, 1.39-2.50]), whereas the CONUT score did not demonstrate a significant difference (P>0.05). Furthermore, GNRI (∆AUC=0.115, P<0.001) and PNI (∆AUC=0.101, P<0.001) exhibited superior predictive ability than the CONUT score for CA-AKI and significantly improved reclassification and discrimination in the fully adjusted model., Conclusions: Malnutrition, especially identified by the GNRI and PNI, was associated with a higher risk of CA-AKI after PCI. GNRI and PNI performed better than the CONUT score in predicting CA-AKI.

Published
2024
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