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A 5 years old child with pertussis reported to us with a unique complication not mentioned in the literature. He was thoroughly examined and investigated to find out cause for his lower motor neuron (LMN) facial palsy but purtussis seemed to be the sole reason. With treatment of purtussis the patient showed marked improvement of neurological deficit. Bangladesh Journal of Infectious Diseases 2020;7(2):107-109

Facial paralysis associated with parotid disease is usually caused by a malignant process. Facial nerve palsy due to parotid gland abscess is very rare with only about 10 previously reported cases. Parotid abscess with facial palsy may be the first presenting symptom of underlying diabetes mellitus. We report a case of a 35-year-old man, not a known case of diabetes or hypertension, who presented with a right sided parotid abscess and difficulty in mouth opening with grade 4 facial nerve palsy, who on investigation was found to have underlying uncontrolled diabetes mellitus. Parotid abscess is mainly seen in elderly, diabetic and immunocompromised. Facial nerve palsy secondary to parotid abscess is a rare condition but probably underreported. Facial nerve palsy associated with parotid abscess is rare and may be one of the first presenting feature of uncontrolled diabetes mellitus.

A 63-year-old man, who was on treatment with interferon beta-1a for relapsing remitting multiple sclerosis for the last 14 years, presented with a new isolated right-sided lower motor neuron facial palsy. He was diagnosed with a right-sided Bell’s palsy, and subsequently he was commenced on oral

Ramsay Hunt Syndrome is a rare complication of herpes zoster in which reactivation of latent varicella zoster virus infection in the geniculate ganglion causes otalgia, auricular vesicles and lower motor neuron facial palsy. A 52-year-old man presented to us with complaints of vesicular eruptions over the right pinna, otalgia and facial weakness i.e. deviation of the angle of mouth to the left side and inability to close the right eye from last 3 days. A diagnosis of Ramsay Hunt Syndrome with grade V facial palsy was made. Intravenous acyclovir and oral steroids were started. Patient showed marked improvement within 10 days. For facial palsy, rehabilitation therapy was also given.

A 40-year-old woman underwent marsupialisation of a symptomatic retrocerebellar cyst. She developed pseudomeningocoele after the procedure. CT of the brain showed hydrocephalus. Therefore, a right-sided ventriculoperitoneal shunt procedure was performed. Unfortunately, the patient developed lower motor neuron-type facial paralysis immediately after the surgery. We localised the site of injury in the distal fallopian canal probably during tunnelling of the subcutaneous tract for passage of the shunt. We discuss the steps to be followed to avoid such mishaps with a brief review of the literature.

With advances in the neuro‑imaging modalities, diverse manifestations of the cerebral venous sinus thrombosis (CVT) are being recognized. There are very few reports of isolated cranial nerve palsies in CVT. In this case report, we describe a patient of lower motor neuron facial palsy with CVT who was successfully treated with anticoagulation, highlighting the atypical manifestation of the disease.

A síndrome de Pyle é um quadro raro de displasia óssea, de transmissão autossômica recessiva com início na infância.Os autores descrevem o caso de uma paciente de 15 anos de idade, com quadro de paralisia facial periférica bilateral, diminuição progressiva da acuídade auditiva do ouvido direito, saliências das bossas frontais, dilatação das metáfises dos membros inferiores e geno valgo .Discutem-se no presente registro os aspectos clínicos da doença, os exames complementares e o diagnóstico diferencial. Pyle's syndrome is a rare picture of osseous dysplasia with autosomal recessive inheritance beginning in early childhood. The authors report the case of a 15-years-old female patient with bilateral lower motor neuron facial palsy, progressive hearing loss, salience of frontal bone, metaphyseal enlargement of the lower limbs and genu valgus. In the present paper we briefly review the clinical features and the differential diagnosis of Pyle's syndrome.

Bell’s palsv or acute idiopathic lower motor neuron facial palsy is a common cause of facial palsy seen in clinical practice. The cause of this disease is not exactly known though there arc many theories regarding Ils etiology. In this article, we present the theory of ”Tertiary is chacmia “ in eases of pertinent Bell’s palsy. Our concept of Tertiary ischaemia is further supported by histological findings of the nerve sheath biopsy taken during surgical decompression In eases of Bell’ s palsy. The procedure, of performing posterior tympanotomy and improving the exposure of the middle ear structures therein is also explained.

We report an unusual case of sudden onset lower motor neuron facial palsy in a 30-year-old male. It was subsequently diagnosed to be caused by silent mastoiditis of tuberculous aetiology. The diagnosis was based on the histology of granulations found during facial nerve decompression. The facial palsy resolved after initiating anti-tubercular therapy and surgical decompression. Our case is an uncommon case of acute onset infranuclear facial palsy due to tuberculosis, in the absence of any ear findings, resembling Bell's palsy.

A 15-week old male infant presented with bilateral lower motor neuron facial palsy of unknown cause. Subsequently his growth deteriorated and he developed progressively worsening cough and wheeze. A diagnosis of cystic fibrosis was confirmed and hypovitaminosis A detected. Improvement of the facial palsy was noted following standard management of cystic fibrosis including vitamin A supplementation.

A case of malakoplakia, of the temporal bone in a nine-month-old male child is reported.The lesion presented as an aural polyp, associated with a lower motor neuron facial palsy. On exploration, the granuloma was noted to involve the temporal bone, eroding the bony labyrinth. It was successfully treated with surgical debridement, and antibiotics. A review of the relevant literature is presented.

A 22-year-old man developed painful tongue swelling, a right facial rash, and right lower motor neuron facial palsy over 2 weeks. On examination he had a vesicular rash in a mandibular distribution and on the concha of …

We have experienced 540 cases with Kawasaki disease over the past 10 years. Six of them (1.1%) had neurological complications with clinical manifestation. Two infants had central nervous system involvement with remarkable changes of cerebrum on CT scan during an acute stage, and these findings disappeared completely within six months. One of these two patients showed no abnormal changes on cerebral angiography. The other four infants had lower motor neuron facial palsy of acute onset and improved within two months.

The childhood syndrome of lower motor neuron facial palsy associated with refractory otitis media, a mass in the external auditory canal, and/or postauricular swelling should suggest embryonal rhabdomyosarcoma of the middle ear cleft. Two patients are described and 23 previously reported cases are reviewed. Neurologic signs, in addition to facial palsy, usually indicate spread to the posterior fossa. The duration of survival after neurologic signs have developed has been less than one year in 23 of the 25 patients.

This case report describes the clinical course of a patient with Ramsay Hunt Syndrome. Partial recovery of the lower motor neuron facial palsy was associated with decreased hearing and a reduction of the middle ear compliance on voluntary contraction of the facial musculature. It is suggested that this is due to misdirection of regenerating nerve fibres, normally destined for facial muscles, to stapedius muscle.