29 results on '"Louradour, Julien"'
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2. Injectable contraceptive Depo-Provera induces erratic beating patterns in patient-specific induced pluripotent stem cell–derived cardiomyocytes with long QT syndrome type 2
3. Activation of hTREK-1 by polyunsaturated fatty acids does not only involve membrane tension
4. Simultaneous assessment of mechanical and electrical function in Langendorff-perfused ex-vivo mouse hearts
5. Activation of hTREK-1 by polyunsaturated fatty acids does not only involve membrane tension
6. Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
7. PO-02-210 AAV9-MEDIATED -SUPPRESSION-REPLACEMENT GENE THERAPY IN TRANSGENIC RABBITS WITH TYPE 1 LONG QT SYNDROME
8. Gene- and variant-specific efficacy of serum/glucocorticoid-regulated kinase 1 inhibition in long QT syndrome types 1 and 2
9. Gene- and variant-specific efficacy of serum/glucocorticoid-regulated kinase 1 inhibition in long QT syndrome types 1 and 2
10. Injectable Contraceptive, Depo-Provera, Produces Erratic Beating Patterns in Patient-Specific Induced Pluripotent Stem Cell-derived Cardiomyocytes with Type 2 Long QT Syndrome
11. Activation of hTREK-1 by polyunsaturated fatty acids does not only involve membrane tension
12. PO-05-191 AAV9-MEDIATED KCNQ1-SUPPRESSION-REPLACEMENT GENE THERAPY IN TRANSGENIC RABBITS WITH TYPE 1 LONG QT SYNDROME (LQT1)
13. EN-482891-004 AAV9-MEDIATED KCNH2-SUPPRESSION-REPLACEMENT GENE THERAPY PROLONGS THE PATHOLOGICALLY SHORTENED QT/APD IN SHORT QT SYNDROME TYPE 1 RABBIT MODELS
14. PO-CES-03 AAV9-MEDIATED KCNQ1-SUPPRESSION-REPLACEMENT GENE THERAPY IN TRANSGENIC RABBITS WITH TYPE 1 LONG QT SYNDROME (LQT1)
15. L-Type Cav1.3 Calcium Channels Are Required for Beta-Adrenergic Triggered Automaticity in Dormant Mouse Sinoatrial Pacemaker Cells
16. Role of L-type Cav1.3 calcium channels in the control of cardiac frequency by catecholamines
17. A Novel Computational Model of Pacemaker Activity in the Mouse Atrioventricular Node Cell
18. Transient Receptor Potential Vanilloid 4 channel participates in mouse ventricular electrical activity - supplemental data Am J phy
19. Transient receptor potential vanilloid 4 channel participates in mouse ventricular electrical activity
20. PO-02-210 AAV9-MEDIATED KCNQ1-SUPPRESSION-REPLACEMENT GENE THERAPY IN TRANSGENIC RABBITS WITH TYPE 1 LONG QT SYNDROME
21. Interplay of electrical and mechanical properties in transgenic rabbit models of long-QT and short-QT syndrome
22. Sex differences in cellular Na+-Ca2+-handling properties in rabbit atrial cardiomyocytes
23. Transient receptor potential vanilloid 4 channel participates in mouse ventricular electrical activity.
24. Sex differences in atrial electrophysiology: IK1is a major factor that is modulated by sex hormones
25. Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity.
26. A Novel Computational Model of Pacemaker Activity in the Mouse Atrioventricular Node Cell
27. KCNQ1 suppression-replacement gene therapy in transgenic rabbits with type 1 long QT syndrome.
28. L-Type Ca v 1.3 Calcium Channels Are Required for Beta-Adrenergic Triggered Automaticity in Dormant Mouse Sinoatrial Pacemaker Cells.
29. Transient receptor potential vanilloid 4 channel participates in mouse ventricular electrical activity.
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