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1. Neuroinflammation Plays a Critical Role in Cerebral Cavernous Malformation Disease

2. Mild Hypoxia Accelerates Cerebral Cavernous Malformation Disease Through CX3CR1-CX3CL1 Signaling

3. Inhibition of the HEG1–KRIT1 interaction increases KLF4 and KLF2 expression in endothelial cells

4. β7 Integrin Inhibition Can Increase Intestinal Inflammation by Impairing Homing of CD25hiFoxP3+ Regulatory T Cells

5. Rap1 binding and a lipid-dependent helix in talin F1 domain promote integrin activation in tandem

6. Phenotypic characterization of murine models of cerebral cavernous malformations

7. Lectin-type oxidized LDL receptor-1 as a potential therapeutic target for cerebral cavernous malformations treatment.

8. Thrombospondin1 (TSP1) replacement prevents cerebral cavernous malformations

11. Investigating the role of inflammation-modulated microRNAs and signalling pathways in blood-barrier dysfunction

12. Astrocytes propel neurovascular dysfunction during cerebral cavernous malformation lesion formation

14. Inhibition of the HEG1-KRIT1 interaction increases KLF4 and KLF2 expression in endothelial cells.

17. Non cell-autonomous effect of astrocytes on cerebral cavernous malformations

19. Talin-1 is the principal platelet Rap1 effector of integrin activation

20. Pharmacological inhibition of the heart of glass (HEG1)-Krev interaction trapped protein 1 (KRIT1) protein complex increases Krüppel-like Factors 4 and 2 (KLF4/2) expression in endothelial cells

21. Rap1 binding and a lipid-dependent helix in talin F1 domain promote integrin activation in tandem

24. MicroRNA‐155 negatively affects blood–brain barrier function during neuroinflammation

28. Distinct integrin activation pathways for effector and regulatory T cell trafficking and function

29. Cytokine-induced changes in the gene expression profile of a human cerebral microvascular endothelial cell-line, hCMEC/D3.

30. Cerebral cavernous malformations form an anticoagulant vascular domain in humans and mice

31. MicroRNA-155 negatively affects blood-brain barrier function during neuroinflammation.

32. Regulation of brain endothelial barrier function by microRNAs in health and neuroinflammation

33. Investigating the role of inflammation-modulated microRNAs and signalling pathways in blood-barrier dysfunction

34. Cerebral cavernous malformations form an anticoagulant vascular domain in humans and mice

35. Regulation of brain endothelial barrier function by microRNAs in health and neuroinflammation

36. MicroRNA-155 negatively affects blood-brain barrier function during neuroinflammation.

37. Investigating the role of inflammation-modulated microRNAs and signalling pathways in blood-barrier dysfunction

38. Behavioral impairments are linked to neuroinflammation in mice with Cerebral Cavernous Malformation disease.

39. Isolation and Purification of Mouse Brain Endothelial Cells to Study Cerebral Cavernous Malformation Disease.

40. β7 Integrin Inhibition Can Increase Intestinal Inflammation by Impairing Homing of CD25 hi FoxP3 + Regulatory T Cells.

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