Search

A Rede Record de Televisão – mídia televisiva da Igreja Universal do Reino de Deus – vem propagando amplamente um mote midiático de cunho religioso, qual seja, Eu sou a Universal. A veiculação desta mensagem parece ser, a um tempo, proselitista e apologética. No entanto, pensamos haver algo mais que se pode depreender acerca deste mote. Nesse fito, o presente ensaio se ocupa de problematizar questões de fundo que subjazem este mote midiático. A partir de três questionamentos, intentamos contribuir para uma reflexão acerca desse fenômeno, cuja chave hermenêutica situa-se no âmbito do paradigma compreensivo, através das lentes da Ciência da Religião. Nesse sentido, não esposamos um único referencial teórico, mas trabalharemos com autores que já lidaram com essa temática, tais como: Paul Freston, Leonildo Silveira Campos, Ricardo Mariano, Magali do Nascimento Cunha, por exemplo. Ademais, além do mote midiático Eu sou a Universal, trabalharemos na análise de representações imagéticas já propagadas que visaram corroborar esse mote

Background: The bed nucleus of the stria terminalis (BNST) is a structure with a peculiar neurochemical composition involved in modulating anxietylike behavior and fear. Aim: The present study investigated the effects on the BNST neurochemical composition and neuronal structure in critical moments of the postnatal period in gestational protein-restricted male rats' offspring. Methods: Dams were maintained during the pregnancy on isocaloric rodent laboratory chow with standard protein content [NP, 17%] or low protein content [LP, 6%]. BNST from male NP and age-matched LP offspring was studied using the isotropic fractionator method, Neuronal 3D reconstruction, dendritic-tree analysis, blotting analysis, and high-performance liquid chromatography. Results: Serum corticosterone levels were higher in male LP offspring than NP rats in 14-day-old offspring, without any difference in 7-day-old progeny. The BNST total cell number and anterodorsal BNST division volume in LP progeny were significantly reduced on the 14th postnatal day compared with NP offspring. The BNST HPLC analysis from 7 days-old LP revealed increased norepinephrine levels compared to NP progeny. The BNST blot analysis from 7-day-old LP revealed reduced levels of GR and BDNF associated with enhanced CRF1 expression compared to NP offspring. 14-day-old LP offspring showed reduced expression of MR and 5HT1A associated with decreased DOPAC and DOPA turnover levels relative to NP rats. In Conclusion, the BNST cellular and neurochemical changes may represent adaptation during development in response to elevated fetal exposure to maternal corticosteroid levels. In this way, gestational malnutrition alters the BNST content and structure and contributes to already-known behavioral changes. [ABSTRACT FROM AUTHOR]

Copyright of Brazilian Journal of Production Engineering / Revista Brasileira de Engenharia de Produção is the property of Brazilian Journal of Production Engineering and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Copyright of Etnobiología is the property of Etnobiologia and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Simple Summary: Wild and captive birds are affected by infectious, metabolic, and neoplastic diseases. Some of these diseases are worrying for public health and the conservation of the affected species, and some diseases may be associated with captivity or with a disturbed environment. A survey of cases was therefore conducted to establish the frequency of diseases affecting wild and captive birds and the relationship with the bird's origin. All cases (necropsy and biopsy) were examined by histopathology, and in some birds, the tissues were subjected to ancillary laboratory tests such as immunohistochemistry, polymerase chain reaction (PCR), and genetic sequencing. A total of 243 birds were examined, and 39.1% were wild birds from illegal trade. The most common primary diseases were infectious diseases caused by parasites (18.1%) or viruses (17.7%). Coinfections were also found in this study (18.1%), mainly in birds confiscated from illegal trade. For exotic birds, the identification of Psittacid alphaherpesvirus 5 (PsAHV 5) raised concerns about the possibility of spreading this virus to native psittaciform species. Due to the infectious nature of some pathogens, it is crucial to diagnose and monitor the health of native and exotic birds to prevent the dissemination of pathogens to avifauna. Native and exotic avian species can act as reservoirs of pathogens, including bacteria and viruses, with conservation and public health implications. A retrospective study on the diagnosis and frequency of diseases in wild and exotic avian species was conducted. The occurrence of particular diseases was associated with the type of captivity or the bird's origin. The investigation included macroscopic and microscopic descriptions and the molecular determination of the causative agent(s). Additional immunohistochemical (IHC) analysis, PCR, and genetic sequencing were conducted. A total of 243 cases were compiled for the study, mainly consisting of native wild species (39.1%) obtained from illegal trade. Primary infectious diseases, mainly parasitic (18.1%) and viral (17.7%), were the most common, although coinfections were substantial (18.1%) in birds rescued from trafficking. Fractures and neoplasms accounted for 3.7% and 3.3% of the cases, respectively. Parasitic and viral diseases were the most common in both exotic and wild birds. Chlamydia psittaci, a lethal and zoonotic bacterium, was an important cause of death, especially in native Psittaciformes. The recent detection of Psittacid alphaherpesvirus 5 (PsAHV 5) in exotic psittacines and the diagnosis of coinfections in trafficked birds highlight the importance of monitoring avian health to control potential pathogens that may endanger conservation efforts. [ABSTRACT FROM AUTHOR]

Gestational protein intake restriction-induced long-lasting harmful outcomes in the offspring's organs and systems. However, few studies have focused on this event's impact on the brain's structures and neurochemical compounds. The present study investigated the effects on the amygdala neurochemical composition and neuronal structure in gestational protein-restricted male rats' offspring. Dams were maintained on isocaloric standard rodent laboratory chow with regular protein [NP, 17%] or low protein content [LP, 6%]. Total cells were quantified using the Isotropic fractionator method, Neuronal 3D reconstruction, and dendritic tree analysis using the Golgi–Cox technique. Western blot and high-performance liquid chromatography performed neurochemical studies. The gestational low-protein feeding offspring showed a significant decrease in birth weight up to day 14, associated with unaltered brain weight in youth or adult progenies. The amygdala cell numbers were unchanged, and the dendrites length and dendritic ramifications 3D analysis in LP compared to age-matched NP progeny. However, the current study shows reduced amygdala content of norepinephrine, epinephrine, and dopamine in LP progeny. These offspring observed a significant reduction in the amygdala glucocorticoid (GR) and mineralocorticoid (MR) receptor protein levels. Also corticotrophin-releasing factor (CRF) amygdala protein content was reduced in 7 and 14-day-old LP rats. The observed amygdala neurochemical changes may represent adaptation during embryonic development in response to elevated fetal exposure to maternal corticosteroid levels. In this way, gestational malnutrition stress can alter the amygdala's neurochemical content and may contribute to known behavioral changes induced by gestational protein restriction. [ABSTRACT FROM AUTHOR]