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2. Population Genetics

Author

Lipp, O., Souery, D., Mendlewicz, J., Henn, Fritz, editor, Sartorius, Norman, editor, Helmchen, Hanfried, editor, and Lauter, Hans, editor

Published
2001
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5. Catching up with wonderful women:The women-are-wonderful effect is smaller in more gender egalitarian societies

Author

Kuba, K., Capaldi, C., van Tilburg, W., Lipp, O. V., Bond, M. H., Vauclair, C.-M., Manickam, L. S. S., Dominguez-Espinosa, A., Torres, T, Lun, V. M.-C., Teyssier, J., Miles, L. K., Hansen, K., Park, J., Wagner, W., Yu, A. A., Xing, C., Wise, R., Sun, C.-R., Siddiqui, R. S., Salem, R., Rizwan, M., Pavlopoulos, V., Nader, M., Maricchiolo, F., Malbran, M., Javangwe, G., Isik, I., Igbokwe, D. O., Hur, T., Hassan, A., Gonzalez, A., Fulop, M., Denoux, P., Cenko, E., Chkhaidze, A., Shmeleva, E., Antalíkova, R., Ahmed, R. A., Krys, Kuba, Capaldi, Colin A., van Tilburg, Wijnand, Lipp, Ottmar V., Bond, Michael Harri, Vauclair, C. Melanie, Manickam, L. Sam S., Domínguez Espinosa, Alejandra, Torres, Claudio, Lun, Vivian Miu Chi, Teyssier, Julien, Miles, Lynden K., Hansen, Karolina, Park, Joonha, Wagner, Wolfgang, Yu, Angela Arriola, Xing, Cai, Wise, Ryan, Sun, Chien Ru, Siddiqui, Razi Sultan, Salem, Radwa, Rizwan, Muhammad, Pavlopoulos, Vassili, Nader, Martin, Maricchiolo, Fridanna, Malbran, María, Javangwe, Gwatirera, Işik, Idil, Igbokwe, David O., Hur, Taekyun, Hassan, Arif, Gonzalez, Ana, Fülöp, Márta, Denoux, Patrick, Cenko, Enila, Chkhaidze, Ana, Shmeleva, Eleonora, Antalíková, Radka, and Ahmed, Ramadan A.

Subjects
Cross-Cultural Comparison, Gender stereotypes, Psychology (all), Culture, Gender stereotype, Gender Identity, Ciências Sociais::Psicologia [Domínio/Área Científica], Implicit attitude, humanities, Social cognition, Social Perception, Socioeconomic Factors, Arts and Humanities (miscellaneous), Surveys and Questionnaires, Gender egalitarianism, Humans, Female, Implicit attitudes
Abstract

Inequalities between men and women are common and well-documented. Objective indexes show that men are better positioned than women in societal hierarchies—there is no single country in the world without a gender gap. In contrast, researchers have found that the women-are-wonderful effect—that women are evaluated more positively than men overall—is also common. Cross-cultural studies on gender equality reveal that the more gender egalitarian the society is, the less prevalent explicit gender stereotypes are. Yet, because self-reported gender stereotypes may differ from implicit attitudes towards each gender, we reanalysed data collected across 44 cultures, and (a) confirmed that societal gender egalitarianism reduces the women-are-wonderful effect when it is measured more implicitly (i.e. rating the personality of men and women presented in images) and (b) documented that the social perception of men benefits more from gender egalitarianism than that of women. info:eu-repo/semantics/acceptedVersion

Published
2018

11. In search of anticipation in unipolar affective disorder

Author

Papadimitriou, GN Souery, D Lipp, O Massat, I Mahieu, B and Van Broeckhoven, C Mendlewicz, J

Abstract

Controversial evidence exists regarding the presence of the phenomenon of anticipation in affective disorder. To further evaluate this hypothesis on the unipolar pattern of the disease, we examined 21 two-generation pairs of first and second degree relatives with unipolar recurrent major depression. Biases from index-patient and from unaffected sibs were taken into consideration. A significant difference in the age at onset and episode frequency (as measure of disease severity) between parental and offspring generation was observed. The median age at onset of the parental generation was 37 +/- 8.2 years compared to 22 +/- 8.3 years in the offspring generation (p = 0.001). The offspring generation also experienced an episode frequency two times greater than the parent generation (p = 0.001). Anticipation was demonstrated in 95% of pairs regarding age at onset and in 84% of pairs in episode frequency. However, the observation of a birth cohort effect may possibly explain the differences in age at onset between generations in our sample. (c) 2005 Elsevier B.V. and ECNP. All rights reserved.

Published
2005

13. Linkage of mood disorders with D2, D3 and TH genes: a multicenter study

Author

Serretti, A Macciardi, F Cusin, C Lattuada, E Souery, D and Lipp, O Mahieu, B Van Broeckhoven, C Blackwood, D and Muir, W Aschauer, HN Heiden, AM Ackenheil, M Fuchshuber, S Raeymaekers, P Verheyen, G Kaneva, R Jablensky, A and Papadimitriou, GN Dikeos, DG Stefanis, CN Smeraldi, E and Mendlewicz, J

Abstract

Background: It has been suggested that the dopaminergic system is involved in the pathophysiology of mood disorders. We conducted a multicenter study of families with mood disorders, to investigate a possible linkage with genes coding for dopamine receptor D2, dopamine receptor D3 and tyrosine hydroxylase (TH). Methods: Twenty three mood disorder pedigrees collected within the framework of the European Collaborative Project on Affective Disorders were analyzed with parametric and non-parametric linkage methods. Various potential phenotypes were considered, from a narrow (only bipolar as affected) to a broad (bipolar + major depressive + schizoaffective disorders) definition of affection status. Results: Parametric analyses excluded linkage for all the candidate genes, even though small positive LOD (Limit of Detection) scores were observed for TH in three families. Non-parametric analyses yielded negative results for all markers. Conclusion: The D2 and D3 dopamine receptors were, therefore, not a major liability factor for mood disorders in our sample, whereas TH may play a role in a subgroup of patients. (C) 2000 Elsevier Science B.V. All rights reserved.

Published
2000

14. Tyrosine hydroxylase polymorphism and phenotypic heterogeneity in bipolar affective disorder: A multicenter association study

Author

Souery, D Lipp, O Rivelli, SK Massat, I Serretti, A and Cavallini, C Ackenheil, M Adolfsson, R Aschauer, H and Blackwood, D Dam, H Dikeos, D Fuchshuber, S Heiden, M and Jakovljevic, M Kaneva, R Kessing, L Lerer, B and Lonnqvist, J Mellerup, T Milanova, V Muir, W Nylander, PO Oruc, L Papadimitriou, GN Pekkarinen, P Peltonen, L and Pull, C Raeymaekers, P Shapira, B Smeraldi, E and Staner, L Stefanis, C Verga, M Verheyen, G Macciardi, F and Van Broeckhoven, C Mendlewicz, J

Abstract

Tyrosine hydroxylase (TH), the rate-limiting enzyme in the metabolism of catecholamines, is considered a candidate gene in bipolar affective disorder (BPAD) and has been the subject of numerous linkage and association studies. Taken together, most results do not support a major gene effect for the TH gene in BPAD. Genetic and phenotypic heterogeneity may partially explain the difficulty of confirming the exact role of this gene using both association and linkage methods. Four hundred one BPAD patients and 401 unrelated matched controls were recruited within a European collaborative project (BIOMED1 project in the area of brain research, European Community grant number CT 92-1217, project leader: J. Mendlewicz) involving 14 centers for a case-control association study with a tetranucleotide polymorphism in the TH gene. Patients and controls were carefully matched for geographical origin. Phenotypic heterogeneity was considered and subgroup analyses were performed with relevant variables: age at onset, family history, and diagnostic stability. No association was observed in the total sample or for subgroups according to age at onset (n=172), family history alone (n=159), or high degree of diagnostic stability and a positive family history (n=131). The results of this association study do not confirm the possible implication of TH polymorphism in the susceptibility to BPAD. (C) 1999 Wiley-Liss, Inc.

Published
1999

16. European Collaborative Project on Affective Disorders: interactions between genetic and psychosocial vulnerability factors

Author

Souery, D Lipp, O Serretti, A Mahieu, B Rivelli, SK and Cavallini, C Ackenheil, M Adolfsson, R Aschauer, H and Blackwood, D Dam, H Delcoigne, B Demartelaer, V Dikeos, D Fuchshuber, S Heiden, M Jablensky, A Jakovljevic, M and Kessing, L Lerer, B Macedo, A Mellerup, T Milanova, V Muir, W Nylander, PO Oruc, L Papadimitriou, GN and Pekkarinen, P Peltonen, L de Azevedo, MHP Pull, C and Shapira, B Smeraldi, E Staner, L Stefanis, C Verga, M and Verheyen, G Macciardi, F Van Broeckhoven, C Mendlewicz, J

Abstract

Despite strong evidence provided by genetic epidemiology of genetic involvement in the aetiology of bipolar and unipolar affective disorders, the exact nature of the predisposing gene(s) is still being investigated through linkage and association studies. The interaction of susceptibility genes and environmental factors in these diseases is also of fundamental importance and requires proper investigation. Interesting theories have recently been proposed examining the possible role of various chromosomal regions, candidate genes and mutations in affective disorders. Reliable multicentre-based methodology is currently being employed to examine these theories, with attention given to statistical analysis and the statistical power of the sample. The present article describes the European Collaborative Project on Affective Disorders (ECPAD) ‘Interactions between genetic and psychosocial vulnerability factors’, involving 15 European centres. A description is given of the association and family samples collected for the project and also the methodology used to analyse interactions in the gene-psychosocial environment. This material provides a powerful tool in the search for susceptibility genes in affective disorders and takes into account non-genetic aetiological factors. Psychiatr Genet 8:197-205 (C) 1998 Lippincott Williams & Wilkins.

Published
1998

30. TPH and suicidal behavior: a study in suicide completers

Author

Turecki, G, primary, Zhu, Z, additional, Tzenova, J, additional, Lesage, A, additional, Séguin, M, additional, Tousignant, M, additional, Chawky, N, additional, Vanier, C, additional, Lipp, O, additional, Alda, M, additional, Joober, R, additional, Benkelfat, C, additional, and Rouleau, G A, additional

Published
2000
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31. Linkage of mood disorders with D2, D3 and TH genes: a multicenter study

Author

Serretti, A, primary, Macciardi, F, additional, Cusin, C, additional, Lattuada, E, additional, Souery, D, additional, Lipp, O, additional, Mahieu, B, additional, Van Broeckhoven, C, additional, Blackwood, D, additional, Muir, W, additional, Aschauer, H.N, additional, Heiden, A.M, additional, Ackenheil, M, additional, Fuchshuber, S, additional, Raeymaekers, P, additional, Verheyen, G, additional, Kaneva, R, additional, Jablensky, A, additional, Papadimitriou, G.N, additional, Dikeos, D.G, additional, Stefanis, C.N, additional, Smeraldi, E, additional, and Mendlewicz, J, additional

Published
2000
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35. European Collaborative Project on Affective Disorders

Author

Souery, D., primary, Lipp, O., additional, Serretti, A., additional, Mahieu, B., additional, Rivelli, S. K., additional, Cavallini, C., additional, Ackenheil, M., additional, Adolfsson, R., additional, Aschauer, H., additional, Blackwood, D., additional, Dam, H., additional, Delcoigne, B., additional, Demartelaer, V., additional, Dikeos, D., additional, Fuchshuber, S., additional, Heiden, M., additional, Jablensky, A., additional, Jakovljevic, M., additional, Kessing, L., additional, Lerer, B., additional, Macedo, A., additional, Mellerup, T., additional, Milanova, V., additional, Muir, W., additional, Nylander, P. O., additional, Oruc, L., additional, Papadimitriou, G. N., additional, Pekkarinen, P., additional, Peltonen, L., additional, de Azevedo, Pinto M.H., additional, Pull, C., additional, Shapira, B., additional, Smeraldi, E., additional, Staner, L., additional, Stefanis, C., additional, Verga, M., additional, Verheyen, G., additional, Macciardi, F., additional, Van Broeckhoven, C., additional, and Mendlewicz, J., additional

Published
1998
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37. Repeat expansion detection (red) in neuropsychiatric disorders

Author

Schalling, M., primary, Lindblad, K., additional, Burgess, C.E., additional, Zander, C., additional, Nylander, P.O., additional, Engström, C., additional, Adolfsson, R., additional, Verheyen, G.R., additional, Oruc, L., additional, De bruyn, A., additional, Van Broeckhoven, C., additional, Souery, D., additional, Lipp, O., additional, Mendlewicz, J., additional, Holmgren, G., additional, Hudson, T., additional, Rapoport, J.L., additional, Sidransky, E., additional, and Ginns, E.I., additional

Published
1997
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48. Improving symptoms of tobacco use with people suffering from schizophrenia: a lack of synergic effect in antipsychotic medication.

Author

Légaré N, Potvin S, Mancini-Marie A, Lipp O, Barabé P, Bouchard R, Stip E, and Roy J

Abstract

Copyright of Sante Mentale au Quebec is the property of Revue Sante Mentale au Quebec and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2007

50. Suicide and serotonin: Study of variation at seven serotonin receptor genes in suicide completers

Author

Turecki, G., Sequeira, A., Gingras, Y., Séguin, M., Lesage, A., Tousignant, M., Chawky, N., Vanier, C., Lipp, O., and Benkelfat, C.

Abstract

Suicide is an important public health problem, accounting for a significant proportion of total mortality among young people, particularly males. There is growing and consistent evidence suggesting that genetic factors play an important role in the predisposition to suicide. Based on several lines of evidence supporting a reduced serotonergic neurotransmission in subjects who committed suicide, we investigated variation at genes that code for serotonin receptor 1B (5-HTR1B), 1Dα (5-HTR1Dα), 1E (5-HTR1E), 1F (5-HTR1F), 2C (5-HTR2C), 5A (5-HTR5A), and 6 (5-HTR6) in a total sample of 106 suicide completers and 120 normal controls. No differences were observed in allelic or genotypic distributions between groups for any of the loci investigated. Moreover, further analysis according to suicide method or psychopathology also failed to reveal differences between groups. Our results do not support a substantial role of these serotonergic receptors in suicide completion. © 2003 Wiley-Liss, Inc.

Published
2003
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