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2. Organ-on-a-chip model of vascularized human bone marrow niches

4. Soil and Seed: Coconspirators in Therapy-Induced Myeloid Neoplasms

5. Genomic landscape of TP53-mutated myeloid malignancies

6. Immunosuppression and outcomes in adult patients with de novo acute myeloid leukemia with normal karyotypes

8. Proteomic and phosphoproteomic landscapes of acute myeloid leukemia

15. Molecular responses in decitabine- and decitabine/venetoclax-treated patients with acute myeloid leukemia and myelodysplastic syndromes

16. ArCH: improving the performance of clonal hematopoiesis variant calling and interpretation

17. Plasma Proteomic Signature Predicts Myeloid Neoplasm Risk

19. MicroRNA-142 Is Critical for the Homeostasis and Function of Type 1 Innate Lymphoid Cells

21. TGF-[beta] signaling in myeloproliferative neoplasms contributes to myelofibrosis without disrupting the hematopoietic niche

22. Bone marrow dendritic cells regulate hematopoietic stem/progenitor cell trafficking

23. Targeting VLA4 integrin and CXCR2 mobilizes serially repopulating hematopoietic stem cells

24. Somatic mutations and clonal hematopoiesis in congenital neutropenia

26. Plasma Proteomic Signature Predicts Risk of Myeloid Neoplasm

27. A Pilot Study of High-Throughput, Sequence-Based Mutational Profiling of Primary Human Acute Myeloid Leukemia Cell Genomes

28. Comprehensive discovery of noncoding RNAs in acute myeloid leukemia cell transcriptomes

29. Author Correction: Mutant p53 drives clonal hematopoiesis through modulating epigenetic pathway

30. CpG Island Hypermethylation Mediated by DNMT3A Is a Consequence of AML Progression

31. Comprehensive genomic analysis reveals FLT3 activation and a therapeutic strategy for a patient with relapsed adult B-lymphoblastic leukemia

35. Mutant p53 drives clonal hematopoiesis through modulating epigenetic pathway

37. Supplementary Table from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

38. Supplementary Table from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

39. Supplementary Figure from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

40. Data from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

41. Supplementary Data from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

42. Supplementary Data from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

43. Data from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

44. Supplementary table 1 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations

45. Supplementary table 3 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations

46. Supplementary figures and legends from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations

47. Supplementary methods from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations

48. Supplementary table 2 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations

49. Data from MIR142 Loss-of-Function Mutations Derepress ASH1L to Increase HOXA Gene Expression and Promote Leukemogenesis

50. Supplementary files from MIR142 Loss-of-Function Mutations Derepress ASH1L to Increase HOXA Gene Expression and Promote Leukemogenesis

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