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1. Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusions

2. Inhibiting mitochondrial phosphate transport as an unexploited antifungal strategy

3. Deep sequencing of proteotoxicity modifier genes uncovers a Presenilin-2/beta-amyloid-actin genetic risk module shared among alpha-synucleinopathies

5. An HSF1-JMJD6-HSP feedback circuit promotes cell adaptation to proteotoxic stress.

7. Cross-Kingdom Chemical Communication Drives a Heritable, Mutually Beneficial Prion-Based Transformation of Metabolism

10. Mitochondrial metabolism promotes adaptation to proteotoxic stress

11. α-Synuclein Blocks ER-Golgi Traffic and Rab1 Rescues Neuron Loss in Parkinson's Models

31. Structural basis for species-selective targeting of Hsp90 in a pathogenic fungus

32. Green tea (−)-epigallocatechin-gallate modulates early events in huntingtin misfolding and reduces toxicity in Huntington's disease models

33. Green tea (-)-epigallocatechin-gallate modulates early events in huntingtin misfolding and reduces toxicity in Huntington's disease models.

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