265 results on '"Lindén, Sara K"'
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2. Gilthead seabream mucus glycosylation is complex, differs between epithelial sites and carries unusual poly N-acetylhexosamine motifs
3. Aeromonas salmonicida AI-1 and AI-2 quorum sensing pathways are differentially regulated by rainbow trout mucins and during in vivo colonization
4. A Complex Connection Between the Diversity of Human Gastric Mucin O-Glycans, Helicobacter pylori Binding, Helicobacter Infection and Fucosylation
5. Characterization of the rainbow trout (Oncorhynchus mykiss) mucosal glycosphingolipid repertoire and Aeromonas salmonicida binding to neutral glycosphingolipids.
6. In vitro fish mucosal surfaces producing mucin as a model for studying host-pathogen interactions.
7. Evidence for a primate origin of zoonotic Helicobacter suis colonizing domesticated pigs
8. Helicobacter pylori Adapts to Chronic Infection and Gastric Disease via pH-Responsive BabA-Mediated Adherence
9. Carbohydrate-Dependent and Antimicrobial Peptide Defence Mechanisms Against Helicobacter pylori Infections
10. Mass Spectrometry Analysis of Shark Skin Proteins
11. Corrigendum to “Mucin modified SPR interfaces for studying the effect of flow on pathogen binding to Atlantic salmon mucins” [Biosens. Bioelectron. 146 (2019) 111736]
12. Identification of Novel Glycans in the Mucus Layer of Shark and Skate Skin
13. Protein N-glycosylation in the bronchoalveolar space differs between never-smokers and long-term smokers with and without COPD
14. Helicobacter suis infection alters glycosylation and decreases the pathogen growth inhibiting effect and binding avidity of gastric mucins
15. Corrigendum to “Mucin modified SPR interfaces for studying the effect of flow on pathogen binding to Atlantic salmon mucins” [Biosens. Bioelectron. 146 (2019) 111736]
16. Muc2-dependent microbial colonization of the jejunal mucus layer is diet sensitive and confers local resistance to enteric pathogen infection
17. Muc2-dependent microbial colonization of the jejunal mucus layer is diet sensitive and confers local resistance to enteric pathogen infection
18. Differentiation of Gastrointestinal Cell Lines by Culture in Semi-wet Interface
19. Mucin O-glycosylation and pathogen binding ability differ between rainbow trout epithelial sites
20. Aeromonas salmonicida binds α2-6 linked sialic acid, which is absent among the glycosphingolipid repertoires from skin, gill, stomach, pyloric caecum, and intestine
21. Influence of the viscosity of healthy and diseased human mucins on the motility of Helicobacter pylori
22. Carbachol-induced colonic mucus formation requires transport via NKCC1, K+ channels and CFTR
23. Atlantic Salmon Mucins Inhibit LuxS-Dependent A. Salmonicida AI-2 Quorum Sensing in an N-Acetylneuraminic Acid-Dependent Manner
24. Rainbow trout gastrointestinal mucus, mucin production, mucin glycosylation and response to lipopolysaccharide
25. Mucin Binding to Moraxella catarrhalis during Airway Inflammation Is Dependent on Sialic Acid
26. Optimization of Alcian blue pH 1.0 histo-staining protocols to match mass spectrometric quantification of sulfomucins and circumvent false positive results due to sialomucins
27. BabA-mediated adherence of pediatric ulcerogenic H. pylori strains to gastric mucins at neutral and acidic pH
28. Mucin dynamics and enteric pathogens
29. Aeromonas salmonicidabinds α2-6 linked sialic acid, which is absent among the glycosphingolipid repertoires from skin, gill, stomach, pyloric caecum, and intestine
30. Listeria monocytogenes internalins bind to the human intestinal mucin MUC2
31. Glucose Homeostasis in Relation to Neutrophil Mobilization in Smokers with COPD.
32. Stress Impairs Skin Barrier Function and Induces α2-3 Linked N-Acetylneuraminic Acid and Core 1 O-Glycans on Skin Mucins in Atlantic Salmon, Salmo salar
33. IL-36 Cytokines Promote Inflammation in the Lungs of Long-Term Smokers
34. In-Depth Study of Transmembrane Mucins in Association with Intestinal Barrier Dysfunction During the Course of T Cell Transfer and DSS-Induced Colitis
35. Recombinant mucin-type proteins carrying LacdiNAc on different O-glycan core chains fail to support H. pylori binding.
36. Muc2-Dependent Microbial Colonization of the Jejunal Mucus Layer is Diet Sensitive and Confers Resistance to Enteric Pathogen Infection
37. Gill Mucus and Gill Mucin O-glycosylation in Healthy and Amebic Gill Disease-Affected Atlantic Salmon
38. Techniques for Assessment of Interactions of Mucins with Microbes and Parasites In Vitro and In Vivo
39. Presence of terminal N-acetylgalactosamineβ1-4N-acetylglucosamine residues on O-linked oligosaccharides from gastric MUC5AC: Involvement in Helicobacter pylori colonization?
40. The MUC13 cell-surface mucin protects against intestinal inflammation by inhibiting epithelial cell apoptosis
41. Strain-Dependent Proliferation in Response to Human Gastric Mucin and Adhesion Properties of Helicobacter pylori are not Affected by Co-isolated Lactobacillus sp.
42. Increased MUC1 plus a larger quantity and complex size for MUC5AC in the peripheral airway lumen of long-term tobacco smokers
43. Recombinant mucin-type proteins carrying LacdiNAc on different O-glycan core chains fail to support H. pylori binding
44. Mucin modified SPR interfaces for studying the effect of flow on pathogen binding to Atlantic salmon mucins
45. Genomics of host-pathogen interactions: challenges and opportunities across ecological and spatiotemporal scales
46. Optimization of Alcian blue pH 1.0 histo-staining protocols to match mass spectrometric quantification of sulfomucins and circumvent false positive results due to sialomucins.
47. Four Modes of Adhesion are Used During Helicobacter pylori Binding to Human Mucins in the Oral and Gastric Niches
48. Improved In vitro Model Systems for Gastrointestinal Infection by Choice of Cell Line, pH, Microaerobic Conditions, and Optimization of Culture Conditions
49. Fish pathogen binding to mucins from Atlantic salmon and Arctic char differs in avidity and specificity and is modulated by fluid velocity
50. Exploring the Arctic Charr Intestinal Glycome: Evidence of Increased N-Glycolylneuraminic Acid Levels and Changed Host–Pathogen Interactions in Response to Inflammation
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