78 results on '"Liao, V."'
Search Results
2. 480 Dissecting the skin-gut-blood axis: Microbial and immune dysregulation in prurigo nodularis
- Author
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Lee, K.K., Liao, V., Lee, J.J., Bao, A., Ma, E., Kollhoff, A.L., Cornman, H., Marani, M., Pritchard, T., and Kwatra, S.G.
- Published
- 2024
- Full Text
- View/download PDF
3. Temporal expression of CD184(CXCR4) and CD171(L1CAM) identifies distinct early developmental stages of human retinal ganglion cells in embryonic stem cell derived retina
- Author
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Aparicio, J. G., Hopp, H., Choi, A., Comar, Mandayam J., Liao, V. C., Harutyunyan, N., and Lee, T. C.
- Published
- 2017
- Full Text
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4. Long-term monitoring of benthic communities reveals spatial determinants of disturbance and recovery dynamics on coral reefs
- Author
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Moritz, C, primary, Brandl, SJ, additional, Rouzé, H, additional, Vii, J, additional, Pérez-Rosales, G, additional, Bosserelle, P, additional, Chancerelle, Y, additional, Galzin, R, additional, Liao, V, additional, Siu, G, additional, Taiarui, M, additional, Nugues, MM, additional, and Hédouin, L, additional
- Published
- 2021
- Full Text
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5. Elective cancer surgery in COVID-19–Free surgical pathways during the SARS-cov-2 pandemic: An international, multicenter, comparative cohort study
- Author
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James C Glasbey, Dmitri Nepogodiev, Joana Ff Simoes, Omar Omar, Elizabeth Li, Mary L Venn, Mohammad Abou Chaar, Vita Capizzi, Daoud Chaudhry, Anant Desai, Jonathan G Edwards, Jonathan P Evans, Marco Fiore, Jose Flavio Videria, Samuel J Ford, Ian Ganyli, Ewen A Griffiths, Rohan R Gujjuri, Angelos G Kolias, Haytham Ma Kaafarani, Ana Minaya-Bravo, Siobhan C McKay, Helen M Mohan, Keith Roberts, Carlos San Miguel-Méndez, Peter Pockney, Richard Shaw, Neil J Smart, Grant D Stewart, Sudha Sundar, Raghavan Vidya, Aneel A Bhangu, James C Glasbey, Omar Omar, Aneel A Bhangu, Kwabena Siaw-Acheampong, Ruth A Benson, Edward Bywater, Daoud Chaudhry, Brett E Dawson, Jonathan P Evans, James C Glasbey, Rohan R Gujjuri, Emily Heritage, Conor S Jones, Sivesh K Kamarajah, Chetan Khatri, Rachel A Khaw, James M Keatley, Andrew Knight, Samuel Lawday, Elizabeth Li, Harvinder S Mann, Ella J Marson, Kenneth A McLean, Siobhan C McKay, Emily C Mills, Dmitri Nepogodiev, Gianluca Pellino, Maria Picciochi, Elliott H Taylor, Abhinav Tiwari, Joana Ff Simoes, Isobel M Trout, Mary L Venn, Richard Jw Wilkin, Aneel A Bhangu, James C Glasbey, Neil J Smart, Ana Minaya-Bravo, Jonathan P Evans, Gaetano Gallo, Susan Moug, Francesco Pata, Peter Pockney, Salomone Di Saverio, Abigail Vallance, Dale Vimalchandran, Ewen A Griffiths, Sivesh K Kamarajah, Richard Pt Evans, Philip Townend, Keith Roberts, Siobhan McKay, John Isaac, Sohei Satoi, John Edwards, Aman S Coonar, Adrian Marchbank, Edward J Caruana, Georgia R Layton, Akshay Patel, Alessandro Brunelli, Samuel Ford, Anant Desai, Alessandro Gronchi, Marco Fiore, Max Almond, Fabio Tirotta, Sinziana Dumitra, Angelos Kolias, Stephen J Price, Daniel M Fountain, Michael D Jenkinson, Peter Hutchinson, Hani J Marcus, Rory J Piper, Laura Lippa, Franco Servadei, Ignatius Esene, Christian Freyschlag, Iuri Neville, Gail Rosseau, Karl Schaller, Andreas K Demetriades, Faith Robertson, Alex Alamri, Richard Shaw, Andrew G Schache, Stuart C Winter, Michael Ho, Paul Nankivell, Juan Rey Biel, Martin Batstone, Ian Ganly, Raghavan Vidya, Alex Wilkins, Jagdeep K Singh, Dinesh Thekinkattil, Sudha Sundar, Christina Fotopoulou, Elaine Leung, Tabassum Khan, Luis Chiva, Jalid Sehouli, Anna Fagotti, Paul Cohen, Murat Gutelkin, Rahel Ghebre, Thomas Konney, Rene Pareja, Rob Bristow, Sean Dowdy, T S Shylasree, R Kottayasamy Seenivasagam, Joe Ng, Keiiji Fujiwara, Grant D Stewart, Benjamin Lamb, Krishna Narahari, Alan McNeill, Alexandra Colquhoun, John McGrath, Steve Bromage, Ravi Barod, Veeru Kasivisvanathan, Tobias Klatte, Joana Ff Simoes, Tom Ef Abbott, Sadi Abukhalaf, Michel Adamina, Adesoji O Ademuyiwa, Arnav Agarwal, Murat Akkulak, Ehab Alameer, Derek Alderson, Felix Alakaloko, Markus Albertsmeiers, Osaid Alser, Muhammad Alshaar, Sattar Alshryda, Alexis P Arnaud, Knut Magne Augestad, Faris Ayasra, José Azevedo, Brittany K Bankhead-Kendall, Emma Barlow, David Beard, Ruth A Benson, Ruth Blanco-Colino, Amanpreet Brar, Ana Minaya-Bravo, Kerry A Breen, Chris Bretherton, Igor Lima Buarque, Joshua Burke, Edward J Caruana, Mohammad Chaar, Sohini Chakrabortee, Peter Christensen, Daniel Cox, Moises Cukier, Miguel F Cunha, Giana H Davidson, Anant Desai, Salomone Di Saverio, Thomas M Drake, John G Edwards, Muhammed Elhadi, Sameh Emile, Shebani Farik, Marco Fiore, J Edward Fitzgerald, Samuel Ford, Tatiana Garmanova, Gaetano Gallo, Dhruv Ghosh, Gustavo Mendonça Ataíde Gomes, Gustavo Grecinos, Ewen A Griffiths, Madalegna GrÜndl, Constantine Halkias, Ewen M Harrison, Intisar Hisham, Peter J Hutchinson, Shelley Hwang, Arda Isik, Michael D Jenkinson, Pascal Jonker, Haytham Ma Kaafarani, Debby Keller, Angelos Kolias, Schelto Kruijff, Ismail Lawani, Hans Lederhuber, Sezai Leventoglu, Andrey Litvin, Andrew Loehrer, Markus W Löffler, Maria Aguilera Lorena, Maria Marta Modolo, Piotr Major, Janet Martin, Hassan N Mashbari, Dennis Mazingi, Symeon Metallidis, Ana Minaya-Bravo, Helen M Mohan, Rachel Moore, David Moszkowicz, Susan Moug, Joshua S Ng-Kamstra, Mayaba Maimbo, Ionut Negoi, Milagros Niquen, Faustin Ntirenganya, Maricarmen Olivos, Kacimi Oussama, Oumaima Outani, Marie Dione Parreno-Sacdalanm, Francesco Pata, Carlos Jose Perez Rivera, Thomas D Pinkney, Willemijn van der Plas, Peter Pockney, Ahmad Qureshi, Dejan Radenkovic, Antonio Ramos-De la Medina, Keith Roberts, April C Roslani, Martin Rutegård, Juan José Segura-Sampedro, Irène Santos, Sohei Satoi, Raza Sayyed, Andrew Schache, Andreas A Schnitzbauer, Justina O Seyi-Olajide, Neil Sharma, Richard Shaw, Sebastian Shu, Kjetil Soreide, Antonino Spinelli, Grant D Stewart, Malin Sund, Sudha Sundar, Stephen Tabiri, Philip Townend, Georgios Tsoulfas, Gabrielle H van Ramshorst, Raghavan Vidya, Dale Vimalachandran, Oliver J Warren, Duane Wedderburn, Naomi Wright, C Allemand, L Boccalatte, M Figari, M Lamm, J Larrañaga, C Marchitelli, F Noll, D Odetto, M Perrotta, J Saadi, L Zamora, C Alurralde, E L Caram, D Eskinazi, J P Mendoza, M Usandivaras, R Badra, A Esteban, J S García, P M García, J I Gerchunoff, S M Lucchini, M A NIgra, L Vargas, T Hovhannisyan, A Stepanyan, T Gould, R Gourlay, B Griffiths, S Gananadha, M McLaren, J Cecire, N Joshi, S Salindera, A Sutherland, J H Ahn, G Charlton, S Chen, N Gauri, R Hayhurst, S Jang, F Jia, C Mulligan, W Yang, G Ye, H Zhang, M Ballal, D Gibson, D Hayne, J Moss, T Richards, P Viswambaram, U G Vo, J Bennetts, T Bright, M Brooke-Smith, R Fong, B Gricks, Y H Lam, B S Ong, M Szpytma, D Watson, K Bagraith, S Caird, E Chan, C Dawson, D Ho, E Jeyarajan, S Jordan, A Lim, G J Nolan, A Oar, D Parker, H Puhalla, A Quennell, L Rutherford, P Townend, M Von Papen, M Wullschleger, A Blatt, D Cope, N Egoroff, M Fenton, J Gani, N Lott, P Pockney, N Shugg, M Elliott, D Phung, D Phan, D Townend, C Bong, J Gundara, A Frankel, S Bowman, G R Guerra, J Bolt, K Buddingh, N N Dudi-Venkata, S Jog, H M Kroon, T Sammour, R Smith, C Stranz, M Batstone, K Lah, W McGahan, D Mitchell, A Morton, A Pearce, M Roberts, G Sheahan, B Swinson, N Alam, S Banting, L Chong, P Choong, S Clatworthy, D Foley, A Fox, M W Hii, B Knowles, J Mack, M Read, A Rowcroft, S Ward, G Wright, M Lanner, I Königsrainer, M Bauer, C Freyschlag, M Kafka, F Messner, D Öfner, I Tsibulak, K Emmanuel, M Grechenig, R Gruber, M Harald, L Öhlberger, J Presl, A Wimmer, I Namazov, E Samadov, D Barker, R Boyce, S Corbin, A Doyle, A Eastmond, R Gill, A Haynes, S Millar, M O'Shea, G Padmore, N Paquette, E Phillips, S St John, K Walkes, N Flamey, P Pattyn, W Oosterlinck, J Van den Eynde, R Van den Eynde, A Gatti, C Nardi, R Oliva, R De Cicco, I Cecconello, P Gregorio, L Pontual Lima, U Ribeiro Junior, F Takeda, R M Terra, M Sokolov, B Kidane, S Srinathan, M Boutros, N Caminsky, G Ghitulescu, G Jamjoum, J Moon, J Pelletier, T Vanounou, S Wong, M Boutros, S Dumitra, A Kouyoumdjian, B Johnston, C Russell, M Boutros, S Demyttenaere, R Garfinkle, J Abou-Khalil, C Nessim, J Stevenson, F Heredia, A Almeciga, A Fletcher, A Merchan, L O Puentes, J Mendoza Quevedo, G Bacic, D Karlovic, D Krsul, M Zelic, I Luksic, M Mamic, B Bakmaz, I Coza, E Dijan, Z Katusic, J Mihanovic, I Rakvin, K Frantzeskou, N Gouvas, G Kokkinos, P Papatheodorou, I Pozotou, O Stavrinidou, A Yiallourou, L Martinek, M Skrovina, I Szubota, J Žatecký, V Javurkova, J Klat, T Avlund, P Christensen, J L Harbjerg, L H Iversen, D W Kjaer, Hø Kristensen, M Mekhael, A L Ebbehøj, P Krarup, N Schlesinger, H Smith, A Abdelsamed, A Y Azzam, H Salem, A Seleim, A Abdelmajeed, M Abdou, N E Abosamak, M Al Sayed, F Ashoush, R Atta, E Elazzazy, M Elhoseiny, M Elnemr, M S Elqasabi, M E Elsayed Hewalla, I Elsherbini, E Essam, M Eweda, I Ghallab, E Hassan, M Ibrahim, M Metwalli, M Mourad, M S Qatora, M Ragab, A Sabry, H Saifeldin, M Saleh Mesbah Mohamed Elkaffas, A Samih, A Samir Abdelaal, S Shehata, K Shenit, D Attia, N Kamal, N Osman, A M Abbas, Has Abd Elazeem, M M Abdelkarem, S Alaa, A K Ali, A Ayman, M G Azizeldine, H Elkhayat, S M Elghazaly, F A Monib, M A Nageh, M M Saad, M Salah, M Shahine, E A Yousof, A Youssef, A Eldaly, M ElFiky, A Nabil, G Amira, I Sallam, M Sherief, A Sherif, A Abdelrahman, H Aboulkassem, G Ghaly, R Hamdy, A Morsi, H Salem, G Sherif, H Abdeldayem, I Abdelkader Salama, M Balabel, Y Fayed, A E Sherif, D Bekele, J Kauppila, E Sarjanoja, O Helminen, H Huhta, J H Kauppila, C Beyrne, L Jouffret, L Lugans, L Marie-Macron, E Chouillard, B De Simone, J Bettoni, S Dakpé, B Devauchelle, N Lavagen, S Testelin, S Boucher, R Breheret, A Gueutier, A Kahn, J KÜn-Darbois, A Barrabe, Z Lakkis, A Louvrier, S Manfredelli, P Mathieu, A Chebaro, V Drubay, M El Amrani, C Eveno, K Lecolle, G Legault, L Martin, G Piessen, F R Pruvot, S Truant, P Zerbib, Q Ballouhey, B Barrat, J Laloze, H Salle, A Taibi, J Usseglio, D Bergeat, A Merdrignac, Roy B Le, L O Perotto, A Scalabre, A Aimé, A Ezanno, B Malgras, P Bouche, S Tzedakis, E Cotte, O Glehen, V Kepenekian, J Lifante, G Passot, A D'Urso, E Felli, D Mutter, P Pessaux, B Seeliger, J Bardet, R Berry, G Boddaert, S Bonnet, E Brian, C Denet, D Fuks, D Gossot, M Grigoroiu, A Laforest, Y Levy-Zauberman, C Louis-Sylvestre, A Moumen, G Pourcher, A Seguin-Givelet, E Tribillon, E Duchalais, F Espitalier, C Ferron, O Malard, U Bork, M Distler, J Fritzmann, J Kirchberg, C Praetorius, C Riediger, J Weitz, T Welsch, P Wimberger, K Beyer, C Kamphues, J Lauscher, F N Loch, C Schineis, M Albertsmeier, M Angele, A Kappenberger, H Niess, T Schiergens, J Werner, R Becker, J Jonescheit, I Pergolini, D Reim, C Boeker, I Hakami, J Mall, P Liokatis, W Smolka, K Nowak, T Reinhard, F Hölzle, A Modabber, P Winnand, M Knitschke, P Kauffmann, S Wolfer, J Kleeff, K Lorenz, C Michalski, U Ronellenfitsch, R Schneider, E Bertolani, A Königsrainer, M W Löffler, M Quante, C Steidle, L ÜberrÜck, C Yurttas, C S Betz, J Bewarder, A Böttcher, S Burg, C Busch, M Gosau, A Heuer, J Izbicki, T O Klatte, D Koenig, N Moeckelmann, C Nitschke, M Priemel, R Smeets, U Speth, S Thole, F G Uzunoglu, T Vollkommer, N Zeller, M J Battista, K Gillen, A Hasenburg, S Krajnak, V Linz, R Schwab, K Angelou, D Haidopoulos, A Rodolakis, P Antonakis, K Bramis, L Chardalias, I Contis, N Dafnios, D Dellaportas, G Fragulidis, A Gklavas, M Konstadoulakis, N Memos, I Papaconstantinou, A Polydorou, T Theodosopoulos, A Vezakis, M I Antonopoulou, D K Manatakis, N Tasis, N Arkadopoulos, N Danias, P Economopoulou, P Kokoropoulos, A Larentzakis, N Michalopoulos, J Selmani, T Sidiropoulos, V Tsaousis, P Vassiliu, K Bouchagier, S Klimopoulos, D Paspaliari, G Stylianidis, K Baxevanidou, K Bouliaris, P Chatzikomnitsa, M Efthimiou, A Giaglaras, C Kalfountzos, G Koukoulis, A M Ntziovara, K Petropoulos, K Soulikia, I Tsiamalou, K Zervas, S Zourntou, I Baloyiannis, A Diamantis, E Gkrinia, J Hajiioannou, C Korais, O Koukoura, K Perivoliotis, A Saratziotis, C Skoulakis, D Symeonidis, K Tepetes, G Tzovaras, D Zacharoulis, V Alexoudi, K Antoniades, I Astreidis, P Christidis, D Deligiannidis, T Grivas, O Ioannidis, I Kalaitsidou, L Loutzidou, A Mantevas, D Michailidou, K Paraskevopoulos, S Politis, A Stavroglou, D Tatsis, I Tilaveridis, K Vahtsevanos, G Venetis, I Karaitianos, T Tsirlis, A Charalabopoulos, T Liakakos, E Mpaili, D Schizas, E Spartalis, A Syllaios, C Zografos, C Anthoulakis, C Christou, V Papadopoulos, A Tooulias, D Tsolakidis, G Tsoulfas, D Zouzoulas, E Athanasakis, E Chrysos, J Tsiaoussis, S Xenaki, E Xynos, K Futaba, M F Ho, S F Hon, Twc Mak, Ssm Ng, C C Foo, B Banky, N Suszták, M Aremu, A Canas-Martinez, O Cullivan, C Murphy, P Owens, L Pickett, L Akmenkalne, J Byrne, M Corrigan, C Cullinane, A Daly, C Fleming, P Jordan, S Killeen, N Lynch, A McCarthy, H Mustafa, S O'Brien, P O'Leary, Was Syed, L Vernon, D Callanan, L Huang, A Ionescu, P Sheahan, I Balasubramanian, M Boland, K Conlon, D Evoy, N Fearon, T Gallagher, J Geraghty, H Heneghan, N Kennedy, D Maguire, D McCartan, E W McDermott, R S Prichard, D Winter, D Alazawi, C Barry, T Boyle, W Butt, E M Connolly, N Donlon, C Donohue, B A Fahey, R Farrell, C Fitzgerald, J Kinsella, J O Larkin, P Lennon, P J Maguire, P Mccormick, B J Mehigan, H Mohan, T Nugent, H O'Sullivan, N Ravi, J V Reynolds, A Rogers, P Shokuhi, J Smith, L A Smith, C Timon, Y Bashir, G Bass, T Connelly, B Creavin, H Earley, J A Elliott, A Gillis, D Kavanagh, P Neary, J O'riordan, I S Reynolds, D Rice, P Ridgway, M Umair, M Whelan, P Carroll, C Collins, K Corless, L Finnegan, A Fowler, A Hogan, M Kerin, A Lowery, P McAnena, K McKevitt, K Nizami, É Ryan, A Samy, J C Coffey, R Cunningham, M Devine, D Nally, C Peirce, S Tormey, N Hardy, P Neary, S O'Malley, M Ryan, S Macina, N M Mariani, E Opocher, A Pisani Ceretti, F Ferrari, F Odicino, E Sartori, C Cotsoglou, S Granieri, F Bianco, A Camillo, M Colledan, S Tornese, M F Zambelli, G Bissolotti, S Fusetti, F Lemma, M V Marino, A Mirabella, G Vaccarella, C Agostini, G Alemanno, I Bartolini, C Bergamini, A Bruscino, C Checcucci, R De Vincenti, A Di Bella, M Fambrini, L Fortuna, G Maltinti, P Muiesan, F Petraglia, P Prosperi, M N Ringressi, M Risaliti, F Sorbi, A Taddei, R Tucci, C Bassi, L Bortolasi, T Campagnaro, L Casetti, M De Pastena, A Esposito, M Fontana, A Guglielmi, L Landoni, G Malleo, G Marchegiani, S Nobile, S Paiella, C Pedrazzani, S Rattizzato, A Ruzzenente, R Salvia, G Turri, M Tuveri, P Bellora, G D'Aloisio, M Ferrari, E Francone, S Gentilli, H Nikaj, M Bianchini, M Chiarugi, F Coccolini, G Di Franco, N Furbetta, D Gianardi, S Guadagni, L Morelli, M Palmeri, D Tartaglia, G Anania, P Carcoforo, M Chiozza, A De Troia, M Koleva Radica, M Portinari, M G Sibilla, A Urbani, N Fabbri, C V Feo, S Gennari, S Parini, E Righini, L Ampollini, L Bellanti, M Bergonzani, G Bertoli, G Bocchialini, G D'Angelo, D Lanfranco, L Musini, T Poli, G P Santoro, A Varazzani, L Aguzzoli, G Borgonovo, C Castro Ruiz, S Coiro, G Falco, V D Mandato, V Mastrofilippo, M T Montella, V Annessi, M Zizzo, U Grossi, S Novello, M Romano, S Rossi, G Zanus, G Esposito, F Frongia, A Pisanu, M Podda, C Belluco, A Lauretta, G Montori, L Moras, M Olivieri, F Bussu, A G Carta, M L Cossu, P Cottu, A Fancellu, C F Feo, G C Ginesu, G Giuliani, M Madonia, T Perra, A Piras, A Porcu, D Rizzo, A M Scanu, A Tedde, M Tedde, P Delrio, D Rega, G Badalamenti, G Campisi, A Cordova, M Franza, G Maniaci, G Rinaldi, F Toia, M Calabrò, F Farnesi, E G Lunghi, A Muratore, N S Pipitone Federico, F Bàmbina, G D'Andrea, P Familiari, V Picotti, G De Palma, G Luglio, G Pagano, F P Tropeano, L Baldari, G A Beltramini, L Boni, E Cassinotti, A Gianni, L Pignataro, S Torretta, C Abatini, M Baia, D Biasoni, G Bogani, P Cadenelli, V Capizzi, Spb Cioffi, D Citterio, L V Comini, M Cosimelli, M Fiore, S Folli, M Gennaro, L Giannini, A Gronchi, M Guaglio, A Macchi, F Martinelli, V Mazzaferro, A Mosca, S Pasquali, C Piazza, F Raspagliesi, L Rolli, R Salvioni, G Sarpietro, C Sarre, L Sorrentino, A Agnes, S Alfieri, F Belia, A Biondi, V Cozza, A D'Amore, D D'Ugo, V De Simone, A Fagotti, G Gasparini, L Gordini, F Litta, C P Lombardi, L Lorenzon, A A Marra, F Marzi, A Moro, A Parello, E Perrone, R Persiani, C Ratto, F Rosa, G Saponaro, G Scambia, O Scrima, G Sganga, R Tudisco, A Belli, V Granata, F Izzo, R Palaia, R Patrone, F M Carrano, M M Carvello, A De Virgilio, F Di Candido, F Ferreli, F Gaino, G Mercante, V Rossi, A Spinelli, G Spriano, D M Donati, T Frisoni, E Palmerini, A Aprile, F Barra, P Batistotti, S Ferrero, P Fregatti, S Scabini, M Sparavigna, E Asti, D Bernardi, L Bonavina, A Lovece, L Adamoli, M Ansarin, S Cenciarelli, F Chu, R De Berardinis, U Fumagalli Romario, F Mastrilli, G Pietrobon, M Tagliabue, E Badellino, A Ferrero, R Massobrio, A De Manzoni Garberini, P Federico, P Maida, E Marra, G Marte, A Petrillo, T Tammaro, A Tufo, M Berselli, G Borroni, E Cocozza, L Conti, M Desio, L Livraghi, V Quintodei, A Rizzi, A Zullo, C Baldi, C Corbellini, G M Sampietro, P Cellerino, E Baldini, P Capelli, L Conti, S M Isolani, M Ribolla, A Bondurri, F Colombo, L Ferrario, C Guerci, A Maffioli, T Armao, M Ballabio, P Bisagni, A Gagliano, M Longhi, M Madonini, P PizziCni, A M Baietti, M Biasini, P Maremonti, F Neri, G M Prucher, S Ricci, F Ruggiero, A G Zarabini, R Barmasse, S Mochet, L Morelli, A Usai, F Bianco, P Incollingo, S Mancini, L Marino Cosentino, A Sagnotta, R Fruscio, T Grassi, L C Nespoli, N Tamini, A Anastasi, B Bartalucci, A Bellacci, G Canonico, L Capezzuoli, C Di Martino, P Ipponi, C Linari, M Montelatici, T Nelli, G Spagni, L Tirloni, A Vitali, E Abate, M Casati, T Casiraghi, L Laface, M Schiavo, A Arminio, A Cotoia, V Lizzi, F Vovola, R Vergari, S D'Ugo, N Depalma, M G Spampinato, P Bartolucci, G Brachini, P Bruzzaniti, A Chiappini, V Chiarella, F Ciccarone, P M Cicerchia, B Cirillo, G De Toma, A Di Bartolomeo, E Fiori, G B Fonsi, G Franco, A Frati, M Giugliano, I Iannone, F La Torre, P Lapolla, C Leonardo, G Marruzzo, S Meneghini, A Mingoli, D Ribuffo, M Salvati, A Santoro, P Sapienza, A K Scafa, L Simonelli, M Zambon, G T Capolupo, F Carannante, M Caricato, G Mascianà, E Mazzotta, A Gattolin, M Migliore, R Rimonda, D Sasia, E Travaglio, M Cervellera, A Gori, L Sartarelli, V Tonini, M Giacometti, S Zonta, A Chessa, A Fiorini, C Norcini, G Colletti, M Confalonieri, A Costanzi, C Frattaruolo, G Mari, M Monteleone, A Bandiera, L Bocciolone, G Bonavina, M Candiani, G Candotti, P De Nardi, F Gagliardi, M Medone, P Mortini, G Negri, P Parise, M Piloni, P Sileri, A Vignali, A Belvedere, P Bernante, P Bertoglio, S Boussedra, E Brunocilla, R Cipriani, G Cisternino, E De Crescenzo, P De Iaco, G Dondi, F Frio, E Jovine, F Mineo Bianchi, J Neri, D Parlanti, A M Perrone, A P Pezzuto, M Pignatti, V Pinto, G Poggioli, M Ravaioli, M Rottoli, R Schiavina, M Serenari, M Serra, P Solli, M Taffurelli, M Tanzanu, M Tesei, T Violante, S Zanotti, F Borghi, D Cianflocca, S Di Maria Grimaldi, D Donati, E Gelarda, P Geretto, G Giraudo, M C Giuffrida, A Marano, S Palagi, L Pellegrino, C Peluso, V Testa, F Agresta, D Prando, M Zese, F Aquila, C Gambacciani, L Lippa, F Pieri, O S Santonocito, G Armatura, G Bertelli, A Frena, P Marinello, F Notte, S Patauner, G Scotton, S F Fulginiti, G Gallo, G Sammarco, G Vescio, P Balercia, L Catarzi, G Consorti, F Di Marzo, T Fontana, H Daiko, M Ishikawa, K Ishiyama, S Iwata, K Kanematsu, Y Kanemitsu, T Kato, A Kawai, E Kobayashi, M Kobayashi Kato, K Moritani, F Nakatani, J Oguma, Y Tanase, M Uno, M Al Abdallah, F Ayasra, Y Ayasra, A Qasem, F J Abu Za'nouneh, T Fahmawee, A Hmedat, A Ibrahim, K Obeidat, S Abdel Al, R Abdel Jalil, M K Abou Chaar, M Al-Masri, H Al-Najjar, F Alawneh, O Alsaraireh, M Elayyan, R Ghanem, I Lataifeh, G Z Alkadeeki, F S Al Maadany, N Aldokali, O Senossi, M T Subhi, D Burgan, E Kamoka, A I Kilani, A Salamah, M Salem, A Shuwayyah, E Abdulwahed, E Alshareea, N Aribi, S Aribi, M Biala, R Ghamgh, M Morgom, Z Aldayri, I Ellojli, A Kredan, S Bradulskis, E Dainius, E Kubiliute, J Kutkevicius, A Parseliunas, A Subocius, D Venskutonis, F Rasoaherinomenjanahary, J B Razafindrahita, L H Samison, E C Ong, K H Hamdan, M R Ibrahim, J A Tan, M R Thanapal, N Amin Sahid, F Hayati, J 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Aged, 80 and over ,Male ,Critical Care ,SARS-CoV-2 ,International Cooperation ,COVID-19 ,Middle Aged ,Cohort Studies ,Logistic Models ,Postoperative Complications ,Elective Surgical Procedures ,Neoplasms ,Outcome Assessment, Health Care ,Humans ,Female ,Epidemics ,Aged - Abstract
PURPOSE As cancer surgery restarts after the first COVID-19 wave, health care providers urgently require data to determine where elective surgery is best performed. This study aimed to determine whether COVID-19–free surgical pathways were associated with lower postoperative pulmonary complication rates compared with hospitals with no defined pathway. PATIENTS AND METHODS This international, multicenter cohort study included patients who underwent elective surgery for 10 solid cancer types without preoperative suspicion of SARS-CoV-2. Participating hospitals included patients from local emergence of SARS-CoV-2 until April 19, 2020. At the time of surgery, hospitals were defined as having a COVID-19–free surgical pathway (complete segregation of the operating theater, critical care, and inpatient ward areas) or no defined pathway (incomplete or no segregation, areas shared with patients with COVID-19). The primary outcome was 30-day postoperative pulmonary complications (pneumonia, acute respiratory distress syndrome, unexpected ventilation). RESULTS Of 9,171 patients from 447 hospitals in 55 countries, 2,481 were operated on in COVID-19–free surgical pathways. Patients who underwent surgery within COVID-19–free surgical pathways were younger with fewer comorbidities than those in hospitals with no defined pathway but with similar proportions of major surgery. After adjustment, pulmonary complication rates were lower with COVID-19–free surgical pathways (2.2% v 4.9%; adjusted odds ratio [aOR], 0.62; 95% CI, 0.44 to 0.86). This was consistent in sensitivity analyses for low-risk patients (American Society of Anesthesiologists grade 1/2), propensity score–matched models, and patients with negative SARS-CoV-2 preoperative tests. The postoperative SARS-CoV-2 infection rate was also lower in COVID-19–free surgical pathways (2.1% v 3.6%; aOR, 0.53; 95% CI, 0.36 to 0.76). CONCLUSION Within available resources, dedicated COVID-19–free surgical pathways should be established to provide safe elective cancer surgery during current and before future SARS-CoV-2 outbreaks.
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- 2021
6. Isolation of an arsenite-oxidizing bacterium in the arsenic contaminated shallow alluvial aquifer in the Blackfoot disease endemic area
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Liao, V, primary and Kao, A, additional
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- 2012
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7. Differential display analysis of gene expression in invertebrates
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Liao, V. H.-C. and Freedman, J. H.
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- 2002
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8. Arsenite-oxidizing and arsenate-reducing bacteria associated with arsenic-rich groundwater in Taiwan
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Chu, Y, primary, Hsiao, S, additional, Weo, C, additional, Liu, C, additional, Liao, C, additional, Chang, F, additional, Liao, V, additional, and Shen, W, additional
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- 2010
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9. Distributed surgical instrument sterilization using solar powered autoclaves for low-infrastructure settings
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Liao, V. T., Hsu, C., Durazo, J., and Young, A.
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- 2011
10. Chronic exposure to low concentration of di-(2-ethylhexyl) phthalate adversely affects aging indicators in Caenorhabditis elegans
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Liao, V. H.-C., primary, How, C.-M., additional, and Yen, P.-L., additional
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- 2018
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11. Measurement of the cross section for production of bb¯X decaying to muons in pp collisions at √s = 7 TeV
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V., Vinogradov, Azhgirey, A., Bayshev, I., Bitioukov, I., Grishin, S., Kachanov, V., Konstantinov, V., Korablev, D., Krychkine, A., Petrov, V., Ryutin, V., Sobol, R., Tourtchanovitch, A., Troshin, L., Tyurin, S., Uzunian, N., Volkov, A., Adzic, A., Djordjevic, P., Ekmedzic, M., Krpic, M., Milosevic, D., Aguilar, Benitez, Alcaraz, Maestre, Arce, J., Battilana, P., Calvo, C., Cerrada, E., Chamizo, Llatas, Colino, M., De La Cruz, Delgado, Peris, Diez, Pardos, Domínguez, Vázquez, Fernandez, Bedoya, Fernández, Ramos, J. P., Ferrando, Flix, A., Fouz, J., M. C., Garcia, Abia, Gonzalez, Lopez, Goy, Lopez, Hernandez, S., J. M., Josa, M. I., Merino, Puerta, Pelayo, Redondo, J., Romero, I., Santaolalla, L., Soares, J., M. S., Willmott, Albajar, C., Codispoti, C., Trocóniz, De, J. F., Cuevas, Fernandez, Menendez, Folgueras, J., Gonzalez, Caballero, Lloret, Iglesias, Piedra, Gomez, Vizan, Garcia, J. M., Brochero, Cifuentes, J. A., Cabrillo, I. J., Calderon, Chuang, A., S. 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T., Dolen, Erbacher, J., Gardner, R., Houtz, M., Ko, R., Kopecky, W., Lander, A., Mall, R., Miceli, O., Nelson, T., Pellett, R., Robles, D., Rutherford, J., Searle, B., Smith, M., Squires, J., Tripathi, M., Vasquez, Sierra, Andreev, R., Arisaka, V., Cline, K., Cousins, D., Duris, R., Erhan, J., Everaerts, S., Farrell, P., Hauser, C., Ignatenko, J., Jarvis, M., Plager, C., Rakness, C., Schlein, G., Tucker, P., Valuev, J., Weber, V., Babb, M., Clare, J., Ellison, R., Gary, J., J. W., Giordano, Hanson, F., Jeng, G., G. Y., Liu, Long, H., O. R., Luthra, Nguyen, A., Paramesvaran, H., Sturdy, S., Sumowidagdo, J., Wilken, S., Wimpenny, R., Andrews, S., Branson, W., J. G., Cerati, G. 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P., Dinardo, M. E., Drell, B. R., Edelmaier, C. J., Ford, W. T., Gaz, Heyburn, A., Luiggi, Lopez, Nauenberg, E., Smith, U., J. G., Stenson, Ulmer, K., K. A., Wagner, S. R., Zang, S. L., Agostino, Alexander, L., Chatterjee, J., Eggert, A., Gibbons, N., L. K., Heltsley, Hopkins, B., Khukhunaishvili, W., Kreis, A., Mirman, B., Nicolas, Kaufman, Patterson, G., J. R., Ryd, Salvati, A., Sun, E., Teo, W., W. D., Thom, Thompson, J., Vaughan, J., Weng, J., Winstrom, Y., Wittich, L., Biselli, P., Cirino, A., Winn, G., Abdullin, D., Albrow, S., Anderson, M., Apollinari, J., Atac, G., Bakken, M., J. A., Bauerdick, L. A. T., Beretvas, Berryhill, A., Bhat, J., P. C., Bloch, Burkett, I., Butler, K., J. N., Chetluru, Cheung, V., H. W. K., Chlebana, Cihangir, F., Cooper, S., Eartly, W., D. P., Elvira, V. D., Esen, Fisk, S., Freeman, I., Gao, J., Gottschalk, Y., Green, E., Gutsche, D., Hanlon, O., Harris, J., R. M., Hirschauer, Hooberman, J., Jensen, B., Jindariani, H., Johnson, S., Joshi, M., Klima, U., Kunori, B., Kwan, S., Leonidopoulos, S., Lincoln, C., Lipton, D., Lykken, R., Maeshima, J., Marraffino, K., J. M., Maruyama, Mason, S., Mcbride, D., Miao, P., Mishra, T., Mrenna, K., Musienko, S., Newman, Holmes, O'Dell, C., Pivarski, V., Pordes, J., Prokofyev, R., Schwarz, O., Sexton, Kennedy, Sharma, E., Spalding, S., W. J., Spiegel, Tan, L., Taylor, P., Tkaczyk, L., Uplegger, S., Vaandering, L., E. W., Vidal, Whitmore, R., Wu, J., Yang, W., Yumiceva, F., Yun, F., J. C., Acosta, Avery, D., Bourilkov, P., Chen, D., Das, M., Gruttola, De, Giovanni, Di, G. P., Dobur, Drozdetskiy, D., Field, A., R. D., Fisher, Fu, M., Furic, Y., I. K., Gartner, Goldberg, J., Hugon, S., Konigsberg, B., Korytov, J., Kropivnitskaya, A., Kypreos, A., Low, T., J. F., Matchev, Milenovic, K., Mitselmakher, P., Muniz, G., Remington, L., Rinkevicius, R., Schmitt, A., Scurlock, M., Sellers, B., Skhirtladze, P., Snowball, N., Wang, M., Yelton, D., Zakaria, J., Gaultney, M., Lebolo, V., L. M., Linn, Markowitz, S., Martinez, P., Rodriguez, G., J. L., Adams, Askew, T., Bochenek, A., Diamond, J., Gleyzer, B., S. V., Haas, Hagopian, J., Hagopian, S., Jenkins, V., Johnson, M., K. F., Prosper, Sekmen, H., Veeraraghavan, S., Weinberg, V., Baarmand, M., M. M., Dorney, Hohlmann, B., Kalakhety, M., Vodopiyanov, H., Adams, I., M. R., Anghel, I. M., Apanasevich, Bai, L., Bazterra, Y., V. E., Betts, R. R., Callner, Cavanaugh, J., Dragoiu, R., Gauthier, C., Gerber, L., C. E., Hofman, D. J., Khalatyan, Kunde, S., G. J., Lacroix, Malek, F., O'Brien, M., Silkworth, C., Silvestre, C., Strom, C., Varelas, D., Akgun, N., Albayrak, U., E. A., Bilki, Clarida, B., Duru, W., Griffiths, F., Lae, S., C. K., Mccliment, Merlo, E., J. 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C., Twedt, Alver, E., Bauer, B., Bendavid, G., Busza, J., Butz, W., Cali, E., I. A., Chan, Dutta, M., Gomez, Ceballos, Goncharov, G., Hahn, M., K. A., Kim, Klute, Y., Lee, M., Y. J., Li, Luckey, W., P. D., Ma, Nahn, T., Paus, S., Ralph, C., Roland, D., Roland, C., Rudolph, G., Stephans, M., G. S. F., Stöckli, Sumorok, F., Sung, K., Velicanu, K., Wenger, D., E. A., Wolf, Wyslouch, R., Xie, B., Yang, S., Yilmaz, M., Yoon, Y., A. S., Zanetti, Cooper, M., S. I., Cushman, Dahmes, P., Benedetti, De, Franzoni, A., Gude, G., Haupt, A., Kao, J., S. C., Klapoetke, Kubota, K., Mans, Y., Pastika, J., Rekovic, N., Rusack, V., Sasseville, R., Singovsky, M., Tambe, A., Turkewitz, N., Cremaldi, J., L. M., Godang, Kroeger, R., Perera, R., Rahmat, L., Sanders, R., D. A., Summers, Avdeeva, D., Bloom, E., Bose, K., Butt, S., Claes, J., D. R., Dominguez, Eads, A., Jindal, M., Keller, P., Kravchenko, J., Lazo, Flores, Malbouisson, J., Malik, H., Snow, S., G. R., Baur, Godshalk, U., Iashvili, A., Jain, I., Kharchilava, S., Shipkowski, A., S. P., Smith, Wan, K., Alverson, Z., Barberis, G., Baumgartel, E., Chasco, D., Trocino, M., Wood, D., Zhang, D., Anastassov, J., Kubik, A., Mucia, A., Odell, N., Ofierzynski, N., R. A., Pollack, Pozdnyakov, B., Stoynev, M., Velasco, S., Won, M., Antonelli, S., Berry, L., Brinkerhoff, D., Hildreth, A., Jessop, M., Karmgard, C., D. J., Kolb, Lannon, J., Luo, K., Lynch, W., Marinelli, S., Morse, N., D. M., Pearson, Ruchti, T., Slaunwhite, R., Valls, J., Wayne, N., Wolf, M., Ziegler, M., Bylsma, J., Durkin, B., L. S., Hill, Killewald, C., Kotov, P., Ling, K., T. Y., Puigh, Rodenburg, D., Vuosalo, M., Williams, C., Adam, G., Berry, N., Elmer, E., Gerbaudo, P., Halyo, D., Hebda, V., Hegeman, P., Hunt, J., Laird, A., Lopes, Pegna, Lujan, D., Marlow, P., Medvedeva, D., Mooney, T., Olsen, M., Piroué, J., Quan, P., Raval, X., Saka, A., Stickland, H., Tully, D., Werner, C., J. S., Zuranski, Acosta, A., J. 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P., Contreras, Campana, Duggan, E., Ferencek, D., Gershtein, D., Gray, Y., Halkiadakis, R., Hidas, E., Hits, D., Lath, D., Panwalkar, A., Park, S., Patel, M., Richards, R., Rose, A., Salur, K., Schnetzer, S., Seitz, S., Somalwar, C., Stone, S., Thomas, R., Cerizza, S., Hollingsworth, G., Spanier, M., Z. C., York, Eusebi, A., Flanagan, R., Gilmore, W., Kamon, J., Khotilovich, T., Montalvo, V., Osipenkov, R., Pakhotin, I., Perloff, Y., Roe, A., Safonov, J., Sakuma, A., Sengupta, T., Suarez, S., Tatarinov, I., Toback, A., Akchurin, D., Bardak, N., Damgov, C., Dudero, J., P. R., Jeong, Kovitanggoon, C., Lee, K., S. W., Libeiro, Mane, T., Roh, P., Sill, Y., Volobouev, A., Wigmans, I., Appelt, R., Brownson, E., Engh, E., Florez, D., Gabella, C., Gurrola, W., Issah, A., Johns, M., Kurt, W., Maguire, P., Melo, C., Sheldon, A., Snook, P., Tuo, B., Velkovska, S., Arenton, J., M. W., Balazs, Boutle, M., Conetti, S., Cox, S., Francis, B., Goadhouse, B., Goodell, S., Hirosky, J., Ledovskoy, R., Lin, A., Neu, C., Wood, C., Yohay, J., Gollapinni, R., Harr, S., Karchin, R., P. E., Kottachchi Kankanamge Don, Lamichhane, C., Mattson, P., Milstène, M., Sakharov, C., Anderson, A., Bachtis, M., Belknap, M., Bellinger, D., J. N., Bernardini, Borrello, J., Carlsmith, L., Cepeda, D., Dasu, M., Efron, S., Friis, J., Gray, E., Grogg, L., K. S., Grothe, Hall, Wilton, Herndon, R., Hervé, M., Klabbers, A., Klukas, P., Lanaro, J., Lazaridis, A., Leonard, C., Loveless, J., Mohapatra, R., Ojalvo, A., Pierro, I., G. A., Ross, Savin, I., Smith, A., W. H., Swanson, wsky A, J., Shreyber, I, Titov, M, Baffioni, S, Beaudette, F, Benhabib, L, Bianchini, L, Bluj, M, Broutin, C, Busson, P, Charlot, C, Daci, N, Dahms, T, Dobrzynski, L, Elgammal, S, de Cassagnac RG, Haguenauer, M, Mine, P, Mironov, C, Ochando, C, Paganini, P, Sabes, D, Salerno, R, Sirois, Y, Thiebaux, C, Veelken, C, Zabi, A, Agram, Jl, Andrea, J, Bloch, D, Bodin, D, Brom, Jm, Cardaci, M, Chabert, Ec, Collard, C, Conte, E, Drouhin, F, Ferro, C, Fontaine, Jc, Gele, D, Goerlach, U, Greder, S, Juillot, P, Karim, M, Le Bihan AC, Van Hove, P, Fassi, F, Mercier, D, Baty, C, Beauceron, S, Beaupere, N, Bedjidian, M, Bondu, O, Boudoul, G, Boumediene, D, Brun, H, Chasserat, J, Chierici, R, Contardo, D, Depasse, P, El Mamouni, H, Falkiewicz, A, Fay, J, Gascon, S, Gouzevitch, M, Ille, B, Kurca, T, Le Grand, T, Lethuillier, M, Mirabito, L, Perries, S, Sordini, V, Tosi, S, Tschudi, Y, Verdier, P, Viret, S, Lomidze, D, Anagnostou, G, Beranek, S, Edelhoff, M, Feld, L, Heracleous, N, Hindrichs, O, Jussen, R, Klein, K, Merz, J, Ostapchuk, A, Perieanu, A, Raupach, F, Sammet, J, Schael, S, Sprenger, D, Weber, H, Wittmer, B, Zhukov, V, Ata, M, Caudron, J, Dietz Laursonn, E, Erdmann, M, Guth, A, Hebbeker, T, Heidemann, C, Hoepfner, K, Klimkovich, T, Klingebiel, D, Kreuzer, P, Lanske, D, Lingemann, J, Magass, C, Merschmeyer, M, Meyer, A, Olschewski, M, Papacz, P, Pieta, H, Reithler, H, Schmitz, Sa, Sonnenschein, L, Steggemann, J, Teyssier, D, Weber, M, Bontenackels, M, Cherepanov, V, Davids, M, Flugge, G, Geenen, H, Geisler, M, Ahmad, Wh, Hoehle, F, Kargoll, B, Kress, T, Kuessel, Y, Linn, A, Nowack, A, Perchalla, L, Pooth, O, Rennefeld, J, Sauerland, P, Stahl, A, Zoeller, Mh, Martin, Ma, Behrenhoff, W, Behrens, U, Bergholz, M, Bethani, A, Borras, K, Cakir, A, Campbell, A, Castro, E, Dammann, D, Eckerlin, G, Eckstein, D, Flossdorf, A, Flucke, G, Geiser, A, Hauk, J, Jung, H, Kasemann, M, Katsas, P, Kleinwort, C, Kluge, H, Knutsson, A, Kramer, M, Krucker, D, Kuznetsova, E, Lange, W, Lohmann, W, Lutz, B, Mankel, R, Marfin, I, Marienfeld, M, Melzer Pellmann IA, Meyer, Ab, Mnich, J, Mussgiller, A, Naumann Emme, S, Olzem, J, Petrukhin, A, Pitzl, D, Raspereza, A, Cipriano, Pmr, Rosin, M, Salfeld Nebgen, J, Schmidt, R, Schoerner Sadenius, T, Sen, N, Spiridonov, A, Stein, M, Tomaszewska, J, Walsh, R, Wissing, C, Autermann, C, Blobel, V, Bobrovskyi, S, Draeger, J, Enderle, H, Erfle, J, Gebbert, U, Gorner, M, Hermanns, T, Kaschube, K, Kaussen, G, Kirschenmann, H, Klanner, R, Lange, J, Mura, B, and Nowak, F.
- Subjects
Hadron-Hadron scattering ,Nuclear and High Energy Physics ,CMS experiment ,LHC ,High Energy Physics::Experiment ,Nuclear Experiment - Published
- 2012
12. Shape, transverse size, and charged hadron multiplicity of jets in pp collisions at 7 TeV
- Author
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Chatrchyan, S., Khachatryan, V., Sirunyan, A. M., Tumasyan, A., Adam, W., Bergauer, T., Dragicevic, M., Erö, J., Fabjan, C., Friedl, M., Frühwirth, R., Ghete, V. M., Hammer, J., Hoch, M., Hörmann, N., Hrubec, J., Jeitler, M., Kiesenhofer, W., Krammer, M., Liko, D., Mikulec, I., Pernicka, M., Rahbaran, B., Rohringer, C., Rohringer, H., Schöfbeck, R., Strauss, J., Taurok, A., Teischinger, F., Wagner, P., Waltenberger, W., Walzel, G., Widl, E., Wulz, C. E., Mossolov, V., Shumeiko, N., Suarez, Gonzalez, Bansal, J., Benucci, S., Wolf, De, E. A., Janssen, Luyckx, X., Maes, S., Mucibello, T., Ochesanu, L., Roland, S., Rougny, B., Selvaggi, R., Van, Haevermaet, Van, Mechelen, Van, Remortel, Van, Spilbeeck, Blekman, A., Blyweert, F., D'Hondt, S., Gonzalez, Suarez, Kalogeropoulos, R., Maes, A., Olbrechts, M., Van, Doninck, Van, Mulders, Van, Onsem, G. P., Villella, Charaf, I., Clerbaux, O., Lentdecker, De, Dero, G., Gay, V., A. P. R., Hammad, G. H., Hreus, Léonard, T., Marage, A., P. E., Thomas, Vander, Velde, Vanlaer, C., Wickens, P., Adler, J., Beernaert, V., Cimmino, K., Costantini, A., Grunewald, S., Klein, M., Lellouch, B., Marinov, J., Mccartin, A., Ryckbosch, J., Strobbe, D., Thyssen, N., Tytgat, F., Vanelderen, M., Verwilligen, L., Walsh, P., Zaganidis, S., Basegmez, N., Bruno, S., Caudron, G., Ceard, J., De Favereau De Jeneret, Delaere, J., Favart, C., Forthomme, D., Giammanco, L., Grégoire, A., Hollar, G., Lemaitre, J., Liao, V., Militaru, J., Nuttens, O., Pagano, C., Pin, D., Piotrzkowski, A., Schul, K., Beliy, N., Caebergs, N., Daubie, T., Alves, E., G. A., De Jesus Damiao, Pol, D., M. E., Souza, M. H. G., Aldá, J. r., W. L., Carvalho, Custódio, W., Costa, Da, E. M., De Oliveira Martins, Fonseca De Souza, Matos, Figueiredo, Mundim, D., Nogima, L., Oguri, H., Prado Da Silva, W. L., Santoro, Silva Do Amaral, S. M., Sznajder, Anjos, A., T. S., Bernardes, C. A., Dias, F. A., Fernandez Perez Tomei, T. R., Gregores, M., Lagana, E., Marinho, C., Mercadante, F., P. G., Novaes, S. F., Padula, S. S., Darmenov, Genchev, N., Iaydjiev, V., Piperov, P., Rodozov, S., Stoykova, M., Sultanov, S., Tcholakov, G., Trayanov, V., Vutova, R., Dimitrov, M., Hadjiiska, A., Karadzhinova, R., Kozhuharov, A., Litov, V., Pavlov, L., Petkov, B., Bian, P., J. G., Chen, G. M., Chen, H. S., Jiang, C. H., Liang, Liang, D., Meng, S., Tao, X., Wang, J., Wang, X., Xiao, Z., Xu, H., Zang, M., Zhang, J., Ban, Z., Guo, Y., Guo, S., Li, Y., Mao, W., Qian, Y., S. J., Teng, Wang, H., Zhu, S., Zou, B., Cabrera, W., Gomez, Moreno, Ocampo, Rios, A. A., Osorio, Oliveros, A. F., Sanabria, J. C., Godinovic, Lelas, N., Plestina, D., Polic, R., Puljak, D., Antunovic, I., Dzelalija, Z., Kovac, M., Brigljevic, M., Duric, V., Kadija, S., Luetic, K., Morovic, J., Attikis, S., Galanti, A., Mousa, M., Nicolaou, J., Ptochos, C., Razis, F., P. A., Finger, Finger, J. r., Assran, M., Ellithi, Kamel, Khalil, A., Mahmoud, S., M. A., Radi, Hektor, A., Kadastik, A., Müntel, M., Raidal, M., Rebane, M., Tiko, L., Azzolini, A., Eerola, V., Fedi, P., Voutilainen, G., Czellar, M., Härkönen, S., Heikkinen, J., Karimäki, A., Kinnunen, V., Kortelainen, R., M. J., Lampén, Lassila, Perini, Lehti, K., Lindén, S., Luukka, T., Mäenpää, P., Tuominen, T., Tuominiemi, E., Tuovinen, J., Ungaro, E., Wendland, D., Banzuzi, L., Karjalainen, K., Korpela, A., Tuuva, A., Sillou, T., Besancon, D., Choudhury, M., Dejardin, S., Denegri, M., Fabbro, D., Faure, B., J. L., Ferri, Ganjour, F., Givernaud, S., Gras, A., Hamel De Monchenault, Jarry, G., Locci, P., Malcles, E., Marionneau, J., Millischer, M., Rander, L., Rosowsky, J., Shreyber, A., Titov, I., Baffoni, M., Beaudette, S., Benhabib, F., Bianchini, L., Bluj, L., Broutin, M., Busson, C., Charlot, P., Daci, C., Dahms, N., Dobrzynski, T., Elgammal, L., Granier De Cassagnac, Haguenauer, R., Miné, M., Mironov, P., Ochando, C., Paganini, C., Sabes, P., Salerno, D., Sirois, R., Thiebaux, Y., Veelken, C., Zabi, C., Agram, A., J. L., Andrea, Bloch, J., Bodin, D., Brom, D., J. M., Cardaci, Chabert, M., E. C., Collard, Conte, C., Drouhin, E., Ferro, F., Fontaine, C., J. C., Gelé, Goerlach, D., Greder, U., Juillot, S., Karim, P., Bihan, Le, A. C., Van, Hove, Fassi, P., Mercier, F., Baty, D., Beauceron, C., Beaupere, S., Bedjidian, N., Bondu, M., Boudoul, O., Boumediene, G., Brun, D., Chasserat, H., Chierici, J., Contardo, R., Depasse, D., Mamouni, El, Falkiewicz, H., Fay, A., Gascon, J., Ille, S., Kurca, B., Grand, Le, Lethuillier, T., Mirabito, M., Perries, L., Sordini, S., Tosi, V., Tschudi, S., Verdier, Y., Viret, P., Lomidze, S., Anagnostou, D., Beranek, G., Edelhoff, S., Feld, M., Heracleous, L., Hindrichs, N., Jussen, O., Klein, R., Merz, K., Ostapchuk, J., Perieanu, A., Raupach, A., Sammet, F., Schael, J., Sprenger, S., Weber, D., Weber, H., Wittmer, M., Zhukov, B., Ata, V., Dietz, Laursonn, Erdmann, E., Hebbeker, M., Heidemann, T., Hinzmann, C., Hoepfner, A., Klimkovich, K., Klingebiel, T., Kreuzer, D., Lanske, P., Lingemann, D., Magass, J., Merschmeyer, C., Meyer, M., Papacz, A., Pieta, P., Reithler, H., Schmitz, H., S. A., Sonnenschein, Steggemann, L., Teyssier, J., Bontenackels, D., Cherepanov, M., Davids, V., Flügge, M., Geenen, G., Haj, Ahmad, Hoehle, W., Kargoll, F., Kress, B., Kuessel, T., Linn, Y., Nowack, A., Perchalla, A., Pooth, L., Rennefeld, O., Sauerland, J., Stahl, P., Tornier, A., Zoeller, D., M. H., Aldaya, Martin, Behrenhoff, M., Behrens, W., Bergholz, U., Bethani, M., Borras, A., Cakir, K., Campbell, A., Castro, A., Dammann, E., Eckerlin, D., Eckstein, G., Flossdorf, D., Flucke, A., Geiser, G., Hauk, A., Jung, J., Kasemann, H., Katsas, M., Kleinwort, P., Kluge, C., Knutsson, H., Krämer, A., Krücker, M., Kuznetsova, D., Lange, E., Lohmann, W., Lutz, W., Mankel, B., Marfin, R., Marienfeld, I., Melzer, Pellmann, I. A., Meyer, A. 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R., Baur, Godshalk, U., Iashvili, A., Jain, I., Kharchilava, S., Smith, A., Wan, K., Alverson, Z., Barberis, G., Baumgartel, E., Chasco, D., Trocino, M., Wood, D., Zhang, D., Anastassov, J., Kubik, A., Mucia, A., Odell, N., Ofierzynski, N., R. A., Pollack, Pozdnyakov, B., Stoynev, M., Velasco, S., Won, M., Antonelli, S., Berry, L., Brinkerhoff, D., Hildreth, A., Jessop, M., Karmgard, C., D. J., Kolb, Kolberg, J., Lannon, T., Luo, K., Lynch, W., Marinelli, S., Morse, N., D. M., Pearson, Ruchti, T., Slaunwhite, R., Valls, J., Wayne, N., Ziegler, M., Bylsma, J., Durkin, B., L. S., Hill, Killewald, C., Kotov, P., Ling, K., T. Y., Rodenburg, Vuosalo, M., Williams, C., Adam, G., Berry, N., Elmer, E., Gerbaudo, P., Halyo, D., Hebda, V., Hunt, P., Laird, A., Lopes, Pegna, Lujan, D., Marlow, P., Medvedeva, D., Mooney, T., Olsen, M., Piroué, J., Quan, P., Raval, X., Saka, A., Stickland, H., Tully, D., Werner, C., J. S., Zuranski, Acosta, A., J. G., Huang, X. T., Lopez, Mendez, A., Oliveros, H., Ramirez, Vargas, J. E., Zatserklyaniy, Alagoz, A., Barnes, E., V. E., Benedetti, Bolla, D., Borrello, G., Bortoletto, L., Mattia, De, Everett, M., Gutay, A., Hu, L., Jones, Z., Koybasi, M., Kress, O., Laasanen, M., A. T., Leonardo, Maroussov, N., Merkel, V., Miller, P., D. H., Neumeister, Shipsey, N., Silvers, I., Svyatkovskiy, D., Vidal, Marono, Yoo, M., H. D., Zablocki, Zheng, J., Guragain, Y., Parashar, S., Adair, N., Boulahouache, A., Cuplov, C., Ecklund, V., K. M., Geurts, F. J. M., Padley, B. P., Redjimi, Roberts, R., Zabel, J., Betchart, J., Bodek, B., Chung, A., Y. S., Covarelli, Barbaro, De, Demina, P., Eshaq, R., Flacher, Y., Garcia, Bellido, Goldenzweig, A., Gotra, P., Han, Y., Harel, J., Miner, A., D. C., Petrillo, Sakumoto, G., Vishnevskiy, W., Zielinski, D., Bhatti, M., Ciesielski, A., Demortier, R., Goulianos, L., Lungu, K., Malik, G., Mesropian, S., Arora, C., Atramentov, S., Barker, O., Chou, A., J. P., Contreras, Campana, Duggan, E., Ferencek, D., Gershtein, D., Gray, Y., Halkiadakis, R., Hidas, E., Hits, D., Lath, D., Panwalkar, A., Park, S., Patel, M., Richards, R., Rose, A., Salur, K., Schnetzer, S., Somalwar, S., Stone, S., Thomas, R., Cerizza, S., Hollingsworth, G., Spanier, M., Z. C., York, Eusebi, A., Flanagan, R., Gilmore, W., Kamon, J., Khotilovich, T., Montalvo, V., Osipenkov, R., Pakhotin, I., Perloff, Y., Roe, A., Safonov, J., Sengupta, A., Suarez, S., Tatarinov, I., Toback, A., Akchurin, D., Bardak, N., Damgov, C., Dudero, J., P. R., Jeong, Kovitanggoon, C., Lee, K., S. W., Libeiro, Mane, T., Roh, P., Sill, Y., Volobouev, A., Wigmans, I., Yazgan, R., Appelt, E., Brownson, E., Engh, E., Florez, D., Gabella, C., Gurrola, W., Issah, A., Johns, M., Johnston, W., Kurt, C., Maguire, P., Melo, C., Sheldon, A., Snook, P., Tuo, B., Velkovska, S., Arenton, J., M. W., Balazs, Boutle, M., Conetti, S., Cox, S., Francis, B., Goadhouse, B., Goodell, S., Hirosky, J., Ledovskoy, R., Lin, A., Neu, C., Wood, C., Yohay, J., Gollapinni, R., Harr, S., Karchin, R., P. E., Kottachchi Kankanamge Don, Lamichhane, C., Mattson, P., Milstène, M., Sakharov, C., Anderson, A., Bachtis, M., Belknap, M., Bellinger, D., J. N., Bernardini, Carlsmith, J., Cepeda, D., Dasu, M., Efron, S., Friis, J., Gray, E., Grogg, L., K. S., Grothe, Hall, Wilton, Herndon, R., Hervé, A., Klabbers, P., Klukas, J., Lanaro, A., Lazaridis, C., Leonard, J., Loveless, R., Mohapatra, A., Ojalvo, I., Pierro, G. A., Ross, I., Savin, A., Smith, W. H., Swanson, J., Weinberg, eiden A, M., Seifert, F, Seixas, Jm, Sekhniaidze, G, Seliverstov, Dm, Sellden, B, Sellers, G, Seman, M, Semprini Cesari, N, Serfon, C, Serin, L, Seuster, R, Severini, H, Sevior, Me, Sfyrla, A, Shabalina, E, Shamim, M, Shan, Ly, Shank, Jt, Shao, Qt, Shapiro, M, Shatalov, Pb, Shaver, L, Shaw, K, Sherman, D, Sherwood, P, Shibata, A, Shichi, H, Shimizu, S, Shimojima, M, Shin, T, Shiyakova, M, Shmeleva, A, Shochet, Mj, Short, D, Shrestha, S, Shupe, Ma, Sicho, P, Sidoti, A, Siebel, A, Siegert, F, Sijacki, D, Silbert, O, Silva, J, Silver, Y, Silverstein, D, Silverstein, Sb, Simak, V, Simard, O, Simic, L, Simion, S, Simmons, B, Simonyan, M, Sinervo, P, Sinev, Nb, Sipica, V, Siragusa, G, Sircar, A, Sisakyan, An, Sivoklokov, Sy, Sjolin, J, Sjursen, Tb, Skinnari, La, Skottowe, Hp, Skovpen, K, Skubic, P, Skvorodnev, N, Slater, M, Slavicek, T, Sliwa, K, Sloper, J, Smakhtin, V, Smirnov, Sy, Smirnova, Ln, Smirnova, O, Smith, Bc, Smith, D, Smith, Km, Smizanska, M, Smolek, K, Snesarev, Aa, Snow, Sw, Snow, J, Snuverink, J, Snyder, S, Soares, M, Sobie, R, Sodomka, J, Soffer, A, Solans, Ca, Solar, M, Solc, J, Soldatov, E, Soldevila, U, Camillocci, Es, Solodkov, Aa, Solovyanov, Ov, Sondericker, J, Soni, N, Sopko, V, Sopko, B, Sosebee, M, Soualah, R, Soukharev, A, Spagnolo, S, Spano, F, Spighi, R, Spigo, G, Spila, F, Spiwoks, R, Spousta, M, Spreitzer, T, Spurlock, B, St Denis RD, Stahl, T, Stahlman, J, Stamen, R, Stanecka, E, Stanek, Rw, Stanescu, C, Stapnes, S, Starchenko, Ea, Stark, J, Staroba, P, Starovoitov, P, Staude, A, Stavina, P, Stavropoulos, G, Steele, G, Steinbach, P, Steinberg, P, Stekl, I, Stelzer, B, Stelzer, Hj, Stelzer Chilton, O, Stenzel, H, Stevenson, K, Stewart, Ga, Stillings, Ja, Stockton, Mc, Stoerig, K, Stoicea, G, Stonjek, S, Strachota, P, Stradling, Ar, Straessner, A, Strandberg, J, Strandberg, S, Strandlie, A, Strang, M, Strauss, E, Strauss, M, Strizenec, P, Strohmer, R, Strom, Dm, Strong, Ja, Stroynowski, R, Strube, J, Stugu, B, Stumer, I, Stupak, J, Sturm, P, Soh, Da, Su, D, Subramania, Hs, Succurro, A, Sugaya, Y, Sugimoto, T, Suhr, C, Suita, K, Suk, M, Sulin, Vv, Sultansoy, S, Sumida, T, Sun, X, Sundermann, Je, Suruliz, K, Sushkov, S, Susinno, G, Sutton, Mr, Suzuki, Y, Svatos, M, Sviridov, Ym, Swedish, S, Sykora, I, Sykora, T, Szeless, B, Sanchez, J, Ta, D, Tackmann, K, Taffard, A, Tafirout, R, Taiblum, N, Takahashi, Y, Takai, H, Takashima, R, Takeda, H, Takeshita, T, Talby, M, Talyshev, A, Tamsett, Mc, Tanaka, J, Tanaka, R, Tanaka, S, Tanaka, Y, Tani, K, Tannoury, N, Tappern, Gp, Tapprogge, S, Tardif, D, Tarem, S, Tarrade, F, Tartarelli, Gf, Tas, P, Tasevsky, M, Tassi, E, Tatarkhanov, M, Tayalati, Y, Taylor, C, Taylor, Fe, Taylor, Gn, Taylor, W, Teinturier, M, Castanheira, Mtd, Teixeira Dias, P, Temming, Kk, Ten Kate, H, Teng, Pk, Terada, S, Terashi, K, Terron, J, Terwort, M, Testa, M, Teuscher, Rj, Thadome, J, Therhaag, J, Laboratoire d'Annecy de Physique des Particules (LAPP), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Savoie Mont Blanc (USMB [Université de Savoie] [Université de Chambéry])-Centre National de la Recherche Scientifique (CNRS), Institut de Recherches sur les lois Fondamentales de l'Univers (IRFU), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Université Paris-Saclay, Laboratoire Leprince-Ringuet (LLR), Centre National de la Recherche Scientifique (CNRS)-École polytechnique (X)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Département Recherches Subatomiques (DRS-IPHC), Institut Pluridisciplinaire Hubert Curien (IPHC), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique 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(LAPP/Laboratoire d'Annecy-le-Vieux de Physique des Particules), Centre National de la Recherche Scientifique (CNRS)-Université Savoie Mont Blanc (USMB [Université de Savoie] [Université de Chambéry])-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-École polytechnique (X)-Centre National de la Recherche Scientifique (CNRS), Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA), Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), UAM. Departamento de Física Teórica, V. KHACHATRYAN, A.M. SIRUNYAN, A. TUMASYAN, W. ADAM, T. BERGAUER, M. DRAGICEVIC, J. ER¨O, C. FABJAN, M. FRIEDL, R. FRUHWIRTH, V.M. GHETE, J. HAMMER, S. H¨ANSEL, C. HARTL, M. HOCH, N. H N(ORMANN, J. HRUBEC, M. JEITLER, G. KASIECZKA, W. KIESENHOFER, M. KRAMMER, D. LIKO, I. MIKULEC, M. PERNICKA, H. ROHRINGER, R. SCH N(OFBECK, J. STRAUSS, A. TAUROK, F. TEISCHINGER, W.WALTENBERGER, G.WALZEL, E.WIDL, C.-E.WULZ, V. MOSSOLOV, N. SHUMEIKO, J. SUAREZ GONZALEZ, L. BENUCCI, L. CEARD, E.A. DE WOLF, X. JANSSEN, T. MAES, L. MUCIBELLO, S. OCHESANU, B. ROLAND, R. ROUGNY, M. SELVAGGI, H. VAN HAEVERMAET, P. VAN MECHELEN, N. VAN REMORTEL, V. ADLER, S. BEAUCERON, S. BLYWEERT, J. D $B!G (BHONDT, O. DEVROEDE, A. KALOGEROPOULOS, J. MAES, M. MAES, S. TAVERNIER, W. VAN DONINCK, P. VAN MULDERS, I. VILLELLA, E.C. CHABERT, O. CHARAF, B. CLERBAUX, G. DE LENTDECKER, V. DERO, A.P.R. GAY, G.H. HAMMAD, T. HREUS, P.E. MARAGE, C. VANDER VELDE, P. VANLAER, J.WICKENS, S. COSTANTINI, M. GRUNEWALD, B. KLEIN, A. MARINOV, D. RYCKBOSCH, F. THYSSEN, M. TYTGAT, L. VANELDEREN, P. VERWILLIGEN, S.WALSH, N. ZAGANIDIS, S. BASEGMEZ, G. BRUNO, J. CAUDRON, J. DE FAVEREAU DE JENERET, C. DELAERE, P. DEMIN, D. FAVART, A. GIAMMANCO, G. GR N4EGOIRE, J. HOLLAR, V. LEMAITRE, O. MILITARU, S. OVYN, D. PAGANO, A. PIN, K. PIOTRZKOWSKI, L. QUERTENMONT, N. SCHUL, N. BELIY, T. CAEBERGS, E. DAUBIE, G.A. ALVES, D. DE JESUS DAMIAO, M.E. POL, M.H.G. SOUZA, W. CARVALHO, E.M. DA COSTA, C. DE OLIVEIRA MARTINS, S. FONSECA DE SOUZA, L. MUNDIM, H. NOGIMA, V. OGURI, J.M. OTALORA GOICOCHEA, W.L. PRADO DA SILVA, A. SANTORO, S.M. SILVA DO, AMARAL, A. SZNAJDER, F. TORRES DA SILVA DE ARAUJO, F.A. DIAS, M.A.F. DIAS, T.R. FERNANDEZ PEREZ TOMEI, E. M. GREGORES2, F. MARINHO, S.F. NOVAES, SANDRA S. PADULA, N. DARMENOV, L. DIMITROV, V. GENCHEV, P. IAYDJIEV, S. PIPEROV, M. RODOZOV, S. STOYKOVA, G. SULTANOV, V. TCHOLAKOV, R. TRAYANOV, I. VANKOV, M. DYULENDAROVA, R. HADJIISKA, V. KOZHUHAROV, L. LITOV, E. MARINOVA, M. MATEEV, B. PAVLOV, P. PETKOV, J.G. BIAN, G.M. CHEN, H.S. CHEN, C.H. JIANG, D. LIANG, S. LIANG, J. WANG, X. WANG, Z.WANG, M. YANG, J. ZANG, Z. ZHANG, Y. BAN, S. GUO, Z. HU, W. LI, Y. MAO, S.J. QIAN, H. TENG, B. ZHU, A. CABRERA, B. GOMEZ MORENO, A.A. OCAMPO RIOS, A.F. OSORIO OLIVEROS, J.C. SANABRIA, N. GODINOVIC, D. LELAS, K. LELAS, R. PLESTINA3, D. POLIC, I. PULJAK, Z. ANTUNOVIC, M. DZELALIJA, V. BRIGLJEVIC, S. DURIC, K. KADIJA, S. MOROVIC, A. ATTIKIS, R. FEREOS, M. GALANTI, J. MOUSA, C. NICOLAOU, F. PTOCHOS, P.A. RAZIS, H. RYKACZEWSKI, Y. ASSRAN, M.A. MAHMOUD, A. HEKTOR, M. KADASTIK, K. KANNIKE, M. M N(UNTEL, M. RAIDAL, L. REBANE, V. AZZOLINI, P. EEROLA, S. CZELLAR, J. H N(ARK N(ONEN, A. HEIKKINEN, V. KARIM N(AKI, R. KINNUNEN, J. KLEM, M.J. KORTELAINEN, T. LAMP N4EN, K. LASSILA-PERINI, S. LEHTI, T. LIND N4EN, P. LUUKKA, T. M N(AENP N(A N(A, E. TUOMINEN, J. TUOMINIEMI, E. TUOVINEN, D. UNGARO, L.WENDLAND, K. BANZUZI, A. KORPELA, T. TUUVA, D. SILLOU, M. BESANCON, M. DEJARDIN, D. 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MOHANTY1, L.M. PANT, P. SHUKLA, P. SUGGISETTI, T. AZIZ, M. GUCHAIT12, A. GURTU, M. MAITY13, D. MAJUMDER, G. MAJUMDER, K. MAZUMDAR, G.B. MOHANTY, A. SAHA, K. SUDHAKAR, N.WICKRAMAGE, S. BANERJEE, S. DUGAD, N.K. MONDAL, H. ARFAEI, H. BAKHSHIANSOHI, S.M. ETESAMI, A. FAHIM, M. HASHEMI, A. JAFARI, M. KHAKZAD, A. MOHAMMADI, M. MOHAMMADI NAJAFABADI, S. PAKTINAT MEHDIABADI, B. SAFARZADEH, M. ZEINALI, M. ABBRESCIA, L. BARBONE, C. CALABRIA, A. COLALEO, D. CREANZA, N. DE FILIPPIS, M. DE, PALMA, A. DIMITROV, F. FEDELE, L. FIORE, G. IASELLI, L. LUSITO, G. MAGGI, M. MAGGI, N. MANNA, B. MARANGELLI, S. MY, S. NUZZO, G.A. PIERRO, A. POMPILI, G. PUGLIESE, F. ROMANO, G. ROSELLI, G. SELVAGGI, L. SILVESTRIS, R. TRENTADUE, S. TUPPUTI, G. ZITO, G. ABBIENDI, A.C. BENVENUTI, D. BONACORSI, S. BRAIBANT, P. CAPILUPPI, A. CASTRO, F.R. CAVALLO, M. CUFFIANI, G.M. DALLAVALLE, F. FABBRI, A. FANFANI, D. FASANELLA, P. GIACOMELLI, M. GIUNTA, C. GRANDI, S. MARCELLINI, G. MASETTI, M. MENEGHELLI, A. MONTANARI, F.L. NAVARRIA, F. ODORICI, A. PERROTTA, A.M. ROSSI, T. ROVELLI, G. SIROLI, R. TRAVAGLINI, S. ALBERGO, G. CAPPELLO, M. CHIORBOLI, S. COSTA, A. TRICOMI, C. TUVE, G. BARBAGLI, G. BROCCOLO, V. CIULLI, C. CIVININI, R. D'ALESSANDRO, E. FOCARDI, S. FROSALI, E. GALLO, P. LENZI, M. MESCHINI, S. PAOLETTI, G. SGUAZZONI, A. TROPIANO, L. BENUSSI, S. BIANCO, S. COLAFRANCESCHI, D. PICCOLO, P. FABBRICATORE, R. MUSENICH, A. BENAGLIA, G.B. CERATI, F. DE GUIO, L. DI MATTEO, A. GHEZZI, P. GOVONI, M. MALBERTI, S. MALVEZZI, A. MARTELLI, A. MASSIRONI, D. MENASCE, V. MICCIO, L. MORONI, M. PAGANONI, D. PEDRINI, S. RAGAZZI, N. REDAELLI, S. SALA, T. TABARELLI DE, FATIS, V. TANCINI, S. BUONTEMPO, C.A. CARRILLO MONTOYA, A. CIMMINO, A. DE COSA, M. DE GRUTTOLA, F. FABOZZI, A.O.M. IORIO, L. LISTA, P. NOLI, P. PAOLUCCI, P. AZZI, N. BACCHETTA, P. BELLAN, D. BISELLO, A. BRANCA, R. CARLIN, P. CHECCHIA, E. CONTI, M. DE MATTIA, T. DORIGO, U. DOSSELLI, F. FANZAGO, F. GASPARINI, U. GASPARINI, P. GIUBILATO, A. GRESELE, S. 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SIBILLE, A. STARODUMOV, L. CAMINADA, Z. CHEN, S. CITTOLIN, G. DISSERTORI, M. DITTMAR, J. EUGSTER, K. FREUDENREICH, C. GRAB, A. HERV N4E, W. HINTZ, P. LECOMTE, W. LUSTERMANN, C. MARCHICA27, P. MARTINEZ RUIZ, DEL ARBOL, P. MERIDIANI, P. MILENOVIC, F. MOORTGAT, A. NARDULLI, P. NEF, F. NESSI-TEDALDI, L. PAPE, F. PAUSS, T. PUNZ, A. RIZZI, F.J. RONGA, L. SALA, A.K. SANCHEZ, M.-C. SAWLEY, B. STIEGER, L. TAUSCHERY, A. THEA, K. THEOFILATOS, D. TREILLE, C. URSCHELER, R. WALLNY, M. WEBER, L. WEHRLI, J. WENG, E. AGUIL´O, C. AMSLER, V. CHIOCHIA, S. DE VISSCHER, C. FAVARO, M. IVOVA RIKOVA, A. JAEGER, B. MILLAN MEJIAS, C. REGENFUS, P. ROBMANN, T. ROMMERSKIRCHEN, A. SCHMIDT, H. SNOEK, L.WILKE, Y.H. CHANG, K.H. CHEN, W.T. CHEN, S. DUTTA, A. GO, C.M. KUO, S.W. LI, W. LIN, M.H. LIU, Z.K. LIU, Y.J. LU, J.H.WU, S.S. YU, P. BARTALINI, P. CHANG, Y.W. CHANG, Y. CHAO, K.F. CHEN, W.-S. HOU, Y. HSIUNG, K.Y. KAO, Y.J. LEI, R.-S. LU, J.G. SHIU, Y.M. TZENG, M.WANG, J.T.WEI, A. ADIGUZEL, M.N. BAKIRCI, S. CERCI, Z. DEMIR, C. DOZEN, I. DUMANOGLU, E. ESKUT, S. GIRGIS, G. G N(OKBULUT, Y. G N( ULER, E. GURPINAR, I. HOS, E.E. KANGAL, T. KARAMAN, A. KAYIS TOPAKSU, A. NART, G. O N( NENGU N( T, K. OZDEMIR, S. OZTURK, A. POLATO N(Z, K. SOGUT, B. TALI, H. TOPAKLI, D. UZUN, L.N. VERGILI, M. VERGILI, C. ZORBILMEZ, I.V. AKIN, T. ALIEV, S. BILMIS, M. DENIZ, H. GAMSIZKAN, A.M. GULER, K. OCALAN, A. OZPINECI, M. SERIN, R. SEVER, U.E. SURAT, E. YILDIRIM, M. ZEYREK, M. DELIOMEROGLU, D. DEMIR, E. GU¨LMEZ, A. HALU, B. ISILDAK, M. KAYA, O. KAYA, M. O¨ZBEK, S. OZKORUCUKLU, N. SONMEZ, L. LEVCHUK, P. BELL, F. BOSTOCK, J.J. BROOKE, T.L. CHENG, D. CUSSANS, R. FRAZIER, J. GOLDSTEIN, M. GRIMES, G.P. HEATH, H.F. HEATH, C. HILL, B. HUCKVALE, J. JACKSON, L. KRECZKO, S. METSON, D.M. NEWBOLD, K. NIRUNPONG, A. POLL, V.J. SMITH, S.WARD, L. BASSO, K.W. BELL, A. BELYAEV, C. BREW, R.M. BROWN, B. CAMANZI, D.J.A. COCKERILL, J.A. COUGHLAN, K. HARDER, S. HARPER, B.W. KENNEDY, E. OLAIYA, D. PETYT, B.C. RADBURN-SMITH, C.H. 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W N(URTHWEIN, A. YAGIL, D. BARGE, R. BELLAN, C. CAMPAGNARI, M. D'ALFONSO, T. DANIELSON, P. GEFFERT, J. INCANDELA, C. JUSTUS, P. KALAVASE, S.A. KOAY, D. KOVALSKYI, V. KRUTELYOV, S. LOWETTE, N. MCCOLL, V. PAVLUNIN, F. REBASSOO, J. RIBNIK, J. RICHMAN, R. ROSSIN, D. STUART, W. TO, J.R. VLIMANT, M.WITHERELL, A. BORNHEIM, J. BUNN, Y. CHEN, M. GATAULLIN, D. KCIRA, V. LITVINE, Y. MA, A. MOTT, H.B. NEWMAN, C. ROGAN, K. SHIN, V. TIMCIUC, P. TRACZYK, J. VEVERKA, R.WILKINSON, Y. YANG, R.Y. ZHU, B. AKGUN, A. CALAMBA, R. CARROLL, T. FERGUSON, Y. IIYAMA, D.W. JANG, S.Y. JUN, Y.F. LIU, M. PAULINI, J. RUSS, N. TERENTYEV, H. VOGEL, I. VOROBIEV, J.P. CUMALAT, M.E. DINARDO, B.R. DRELL, C.J. EDELMAIER, W.T. FORD, B. HEYBURN, E. LUIGGI LOPEZ, U. NAUENBERG, J.G. SMITH, K. STENSON, K.A. ULMER, S.R.WAGNER, S.L. ZANG, L. AGOSTINO, J. ALEXANDER, F. BLEKMAN, A. CHATTERJEE, S. DAS, N. EGGERT, L.J. FIELDS, L.K. GIBBONS, B. HELTSLEY, K. HENRIKSSON, W. HOPKINS, A. KHUKHUNAISHVILI, B. KREIS, V. KUZNETSOV, Y. LIU, G. NICOLAS KAUFMAN, J.R. PATTERSON, D. PUIGH, D. RILEY, A. RYD, M. SAELIM, X. SHI, W. SUN, W.D. TEO, J. THOM, J. THOMPSON, J. VAUGHAN, Y.WENG, P.WITTICH, A. BISELLI, G. CIRINO, D. WINN, S. ABDULLIN, M. ALBROW, J. ANDERSON, G. APOLLINARI, M. ATAC, J.A. BAKKEN, L.A.T. BAUERDICK, A. BERETVAS, J. BERRYHILL, P.C. BHAT, I. BLOCH, F. BORCHERDING, K. BURKETT, J.N. BUTLER, V. CHETLURU, H.W.K. CHEUNG, F. CHLEBANA, S. CIHANGIR, M. DEMARTEAU, D.P. EARTLY, V.D. ELVIRA, I. FISK, J. FREEMAN, Y. GAO, E. GOTTSCHALK, D. GREEN, K. GUNTHOTI, O. GUTSCHE, A. HAHN, J. HANLON, R.M. HARRIS, J. HIRSCHAUER, E. JAMES, H. JENSEN, M. JOHNSON, U. JOSHI, R. KHATIWADA, B. KILMINSTER, B. KLIMA, K. KOUSOURIS, S. KUNORI, S. KWAN, P. LIMON, R. LIPTON, J. LYKKEN, K. MAESHIMA, J.M. MARRAFFINO, D. MASON, P. MCBRIDE, T. MCCAULEY, T. MIAO, K. MISHRA, S. MRENNA, Y. MUSIENKO, C. NEWMAN-HOLMES, V. O $B!G (BDELL, S. POPESCU, R. PORDES, O. PROKOFYEV, N. SAOULIDOU, E. SEXTON-KENNEDY, S. SHARMA, A. SOHA, W.J. SPALDING, L. SPIEGEL, P. TAN, L. TAYLOR, S. TKACZYK, L. UPLEGGER, E.W. VAANDERING, R. VIDAL, J. WHITMORE, W. WU, F. YANG, F. YUMICEVA, J.C. YUN, D. ACOSTA, P. AVERY, D. BOURILKOV, M. CHEN, G.P. DI GIOVANNI, D. DOBUR, A. DROZDETSKIY, R.D. FIELD, M. FISHER, Y. FU, I.K. FURIC, J. GARTNER, S. GOLDBERG, B. KIM, S. KLIMENKO, J. KONIGSBERG, A. KORYTOV, K. KOTOV, A. KROPIVNITSKAYA, T. KYPREOS, K. MATCHEV, G. MITSELMAKHER, L. MUNIZ, Y. PAKHOTIN, M. PETTERSON, C. PRESCOTT, R. REMINGTON, M. SCHMITT, B. SCURLOCK, P. SELLERS, M. SNOWBALL, D.WANG, J. YELTON, M. ZAKARIA, C. CERON, V. GAULTNEY, L. KRAMER, L.M. LEBOLO, S. LINN, P. MARKOWITZ, G. MARTINEZ, D. MESA, J.L. RODRIGUEZ, T. ADAMS, A. ASKEW, J. BOCHENEK, J. CHEN, B. DIAMOND, S.V. GLEYZER, J. HAAS, S. HAGOPIAN, V. HAGOPIAN, M. JENKINS, K.F. JOHNSON, H. PROSPER, S. SEKMEN, V. VEERARAGHAVAN, M.M. BAARMAND, B. DORNEY, S. GURAGAIN, M. HOHLMANN, H. KALAKHETY, R. RALICH, I. VODOPIYANOV, M.R. ADAMS, I.M. ANGHEL, L. APANASEVICH, Y. BAI, V.E. BAZTERRA, R.R. BETTS, J. CALLNER, R. CAVANAUGH, C. DRAGOIU, E.J. GARCIA-SOLIS, C.E. GERBER, D.J. HOFMAN, S. KHALATYAN, F. LACROIX, C. O $B!G (BBRIEN, E. SHABALINA, C. SILVESTRE, A. SMORON, D. STROM, N. VARELAS, U. AKGUN, E.A. ALBAYRAK, B. BILKI, K. CANKOCAK, W. CLARIDA, F. DURU, C.K. LAE, E. MCCLIMENT, J.-P. MERLO, H. MERMERKAYA, A. MESTVIRISHVILI, A. MOELLER, J. NACHTMAN, C.R. NEWSOM, E. NORBECK, J. OLSON, Y. ONEL, F. OZOK, S. SEN, J.WETZEL, T. YETKIN, K. YI, B.A. BARNETT, B. BLUMENFELD, A. BONATO, C. ESKEW, D. FEHLING, G. GIURGIU, A.V. GRITSAN, Z.J. GUO, G. HU, P. MAKSIMOVIC, S. RAPPOCCIO, M. SWARTZ, N.V. TRAN, A. WHITBECK, P. BARINGER, A. BEAN, G. BENELLI, O. GRACHOV, M. MURRAY, D. NOONAN, V. RADICCI, S. SANDERS, J.S.WOOD, V. ZHUKOVA, D. BANDURIN, T. BOLTON, I. CHAKABERIA, A. IVANOV, M. MAKOUSKI, Y. MARAVIN, S. SHRESTHA, I. SVINTRADZE, Z.WAN, J. GRONBERG, D. LANGE, D. WRIGHT, A. BADEN, M. BOUTEMEUR, S.C. ENO, D. FERENCEK, J.A. GOMEZ, N.J. HADLEY, R.G. KELLOGG, M. KIRN, Y. LU, A.C. MIGNEREY, K. ROSSATO, P. RUMERIO, F. SANTANASTASIO, A. SKUJA, J. TEMPLE, M.B. TONJES, S.C. TONWAR, E. TWEDT, B. ALVER, G. BAUER, J. BENDAVID, W. BUSZA, E. BUTZ, I.A. CALI, M. CHAN, V. DUTTA, P. EVERAERTS, G. GOMEZ CEBALLOS, M. GONCHAROV, K.A. HAHN, P. HARRIS, Y. KIM, M. KLUTE, Y.-J. LEE, C. LOIZIDES, P.D. LUCKEY, T. MA, S. NAHN, C. PAUS, C. ROLAND, G. ROLAND, M. RUDOLPH, G.S.F. STEPHANS, K. SUMOROK, K. SUNG, E.A. WENGER, B. WYSLOUCH, S. XIE, Y. YILMAZ, A.S. YOON, M. ZANETTI, P. COLE, S.I. COOPER, P. CUSHMAN, B. DAHMES, A. DE BENEDETTI, P.R. DUDERO, G. FRANZONI, J. HAUPT, K. KLAPOETKE, Y. KUBOTA, J. MANS, V. REKOVIC, R. RUSACK, M. SASSEVILLE, A. SINGOVSKY, L.M. CREMALDI, R. GODANG, R. KROEGER, L. PERERA, R. RAHMAT, D.A. SANDERS, D. SUMMERS, K. BLOOM, S. BOSE, J. BUTT, D.R. CLAES, A. DOMINGUEZ, M. EADS, J. KELLER, T. KELLY, I. KRAVCHENKO, J. LAZO-FLORES, C. LUNDSTEDT, H. MALBOUISSON, S. MALIK, G.R. SNOW, U. BAUR, A. GODSHALK, I. IASHVILI, A. KHARCHILAVA, K. SMITH, J. ZENNAMO, G. ALVERSON, E. BARBERIS, D. BAUMGARTEL, O. BOERIU, M. CHASCO, K. KAADZE, S. REUCROFT, J. SWAIN, D. WOOD, J. ZHANG, A. ANASTASSOV, A. KUBIK, N. ODELL, R.A. OFIERZYNSKI, B. POLLACK, A. POZDNYAKOV, S. STOYNEV, M. VELASCO, S.WON, L. ANTONELLI, D. BERRY, M. HILDRETH, C. JESSOP, D.J. KARMGARD, J. KOLB, T. KOLBERG, K. LANNON, W. LUO, S. LYNCH, N. MARINELLI, D.M. MORSE, T. PEARSON, R. RUCHTI, J. SLAUNWHITE, N. VALLS, J. WARCHOL, M.WAYNE, J. ZIEGLER, B. BYLSMA, L.S. DURKIN, J. GU, P. KILLEWALD, T.Y. LING, M. RODENBURG, G.WILLIAMS, N. ADAM, E. BERRY, P. ELMER, D. GERBAUDO, V. HALYO, P. HEBDA, A. HUNT, J. JONES, E. LAIRD, D. LOPES PEGNA, D. MARLOW, T. MEDVEDEVA, M. MOONEY, J. OLSEN, P. PIROU´E, H. SAKA, D. STICKLAND, C. TULLY, J.S.WERNER, A. ZURANSKI, J.G. ACOSTA, X.T. HUANG, A. LOPEZ, H. MENDEZ, S. OLIVEROS, J.E. RAMIREZ VARGAS, A. ZATSERKLYANIY, E. ALAGOZ, V.E. BARNES, G. BOLLA, L. BORRELLO, D. BORTOLETTO, A. EVERETT, A.F. GARFINKEL, Z. GECSE, L. GUTAY, M. JONES, O. KOYBASI, A.T. LAASANEN, N. LEONARDO, C. LIU, V. MAROUSSOV, M. MEIER, P. MERKEL, D.H. MILLER, N. NEUMEISTER, K. POTAMIANOS, I. SHIPSEY, D. SILVERS, A. SVYATKOVSKIY, H.D. YOO, J. ZABLOCKI, Y. ZHENG, P. JINDAL, N. PARASHAR, C. BOULAHOUACHE, V. CUPLOV, K.M. ECKLUND, F.J.M. GEURTS, J.H. LIU, J. MORALES, B.P. PADLEY, R. REDJIMI, J. ROBERTS, J. ZABEL, B. BETCHART, A. BODEK, Y.S. CHUNG, P. DE BARBARO, R. DEMINA, Y. ESHAQ, H. FLACHER, A. GARCIA, BELLIDO, P. GOLDENZWEIG, Y. GOTRA, J. HAN, A. HAREL, D.C. MINER, D. ORBAKER, G. PETRILLO, D. VISHNEVSKIY, M. ZIELINSKI, A. BHATTI, L. DEMORTIER, K. GOULIANOS, G. LUNGU, C. MESROPIAN, M. YAN, O. ATRAMENTOV, A. BARKER, D. DUGGAN, Y. GERSHTEIN, R. GRAY, E. HALKIADAKIS, D. HIDAS, D. HITS, A. LATH, S. PANWALKAR, R. PATEL, A. RICHARDS, K. ROSE, S. SCHNETZER, S. SOMALWAR, R. STONE, S. THOMAS, G. CERIZZA, M. HOLLINGSWORTH, S. SPANIER, Z.C. YANG, A. YORK, J. ASAADI, R. EUSEBI, J. GILMORE, A. GURROLA, T. KAMON, V. KHOTILOVICH, R. MONTALVO, C.N. NGUYEN, J. PIVARSKI, A. SAFONOV, S. SENGUPTA, A. TATARINOV, D. TOBACK, M.WEINBERGER, N. AKCHURIN, C. BARDAK, J. DAMGOV, C. JEONG, K. KOVITANGGOON, S.W. LEE, P. MANE, Y. ROH, A. SILL, I. VOLOBOUEV, R.WIGMANS, E. YAZGAN, E. APPELT, E. BROWNSON, D. ENGH, C. FLOREZ, W. GABELLA, W. JOHNS, P. KURT, C. MAGUIRE, A. MELO, P. SHELDON, J. VELKOVSKA, M.W. ARENTON, M. BALAZS, S. BOUTLE, M. BUEHLER, S. CONETTI, B. COX, B. FRANCIS, R. HIROSKY, A. LEDOVSKOY, C. LIN, C. NEU, T. PATEL, R. YOHAY, S. GOLLAPINNI, R. HARR, P.E. KARCHIN, V. LOGGINS, M. MATTSON, C. MILST`ENE, A. SAKHAROV, M. ANDERSON, M. BACHTIS, J.N. BELLINGER, D. CARLSMITH, S. DASU, J. EFRON, L. GRAY, K.S. GROGG, M. GROTHE, R. HALL-WILTON, M. HERNDON, P. KLABBERS, J. KLUKAS, A. LANARO, C. LAZARIDIS, J. LEONARD, J. LIU, D. LOMIDZE, R. LOVELESS, A. MOHAPATRA, W. PARKER, D. REEDER, I. ROSS, A. SAVIN, W.H. SMITH, J. SWANSON, M.WEINBERG, S.CHATRCHYAN, F. PRIMAVERA, L. BRIGLIADORI, Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), Yerevan Phys Inst, Inst Hochenergiephys OeAW, Natl Ctr Particle & High Energy Phys, Univ Antwerp, Vrije Univ Brussel, Univ Libre Brussels, Univ Ghent, Catholic Univ Louvain, Univ Mons, Centro Brasileiro de Pesquisas Físicas (CBPF), Universidade do Estado do Rio de Janeiro (UERJ), Universidade Estadual Paulista (Unesp), Bulgarian Acad Sci, Univ Sofia, Inst High Energy Phys, Peking Univ, Universidad de los Andes, Tech Univ Split, Univ Split, Rudjer Boskovic Inst, 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Particle physics ,Nuclear and High Energy Physics ,Astrophysics::High Energy Astrophysical Phenomena ,Monte Carlo method ,Hadron ,HERA ,FOS: Physical sciences ,ComputingMilieux_LEGALASPECTSOFCOMPUTING ,Hadron-hadron scattering ,7. Clean energy ,01 natural sciences ,PARTICLE PHYSICS ,LARGE HADRON COLLIDER ,CMS ,High Energy Physics - Experiment ,Nuclear physics ,DEEP-INELASTIC SCATTERING ,High Energy Physics - Experiment (hep-ex) ,0103 physical sciences ,[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex] ,DISTRIBUTIONS ,ddc:530 ,Multiplicity (chemistry) ,010306 general physics ,Nuclear Experiment ,deep-inelastic scattering ,hadron-hadron scattering ,hera ,distributions ,Quantum chromodynamics ,Physics ,high energy collider ,Large Hadron Collider ,010308 nuclear & particles physics ,Física ,Deep inelastic scattering ,HEP ,Transverse plane ,FIS/01 - FISICA SPERIMENTALE ,Physique des particules élémentaires ,High Energy Physics::Experiment ,LHC ,Particle Physics - Experiment - Abstract
Open Access: This article is distributed under the terms of the Creative Commons Attribution License.-- CMS Collaboration: et al., Measurements of jet characteristics from inclusive jet production in proton-proton collisions at a centre-of-mass energy of 7 TeV are presented. The data sample was collected with the CMS detector at the LHC during 2010 and corresponds to an integrated luminosity of 36 inverse picobarns. The mean charged hadron multiplicity, the differential and integral jet shape distributions, and two independent moments of the shape distributions are measured as functions of the jet transverse momentum for jets reconstructed with the anti-kT algorithm. The measured observables are corrected to the particle level and compared with predictions from various QCD Monte Carlo generators., European Commission; Federal Ministry of Science, Research and Economy (Austria); National Fund for Scientific Research (Belgium); Research Foundation – Flanders; Conselho Nacional de Desenvolvimento Científico e Tecnológico (Brasil); Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (Brasil); Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro; Fundação de Amparo à Pesquisa do Estado de São Paulo; Bulgarian Ministry of Education and Science; Centre National de la Recherche Scientifique (France); Commissariat à l'énergie atomique et aux énergies alternatives (France); Ministry of Science and Technology of the People's Republic of China; Chinese Academy of Sciences; National Natural Science Foundation of China; Colciencias (Colombia); Croatian Ministry of Science, Education and Sport; Academy of Finland; Helsinki Institute of Physics; Bundesministerium für Bildung und Forschung (Deutschland); Deutsche Forschungsgemeinschaft; Helmholtz Association; General Secretariat of Research and Technology (Greece); Hungarian Scientific Research Fund; National Office for Research and Technology (Hungary); Department of Atomic Energy (India); Department of Science and Technology (India); Council of Science and Industrial Research (India); Institute for Research in Fundamental Sciences (Iran); Science Foundation Ireland; Istituto Nazionale di Fisica Nucleare (Italia); Korean Ministry of Education, Science and Technology; Lithuanian Academy of Sciences; Centro de Investigación y de Estudios Avanzados del Instituto Politécnico Nacional (México); Consejo Nacional de Ciencia y Tecnología (México); Secretaría de Educación Pública (México); Universidad Autónoma de San Luis Potosí; Ministry of Science and Innovation (New Zealand); Ministry of Science and Higher Education and the National Science Centre (Poland); Fundação para a Ciência e a Tecnologia (Portugal); Joint Institute for Nuclear Research (Russia); Russian Academy of Sciences; Russian Foundation for Basic Research; Ministry of Education, Science and Technological Development (Serbia); Ministerio de Ciencia e Innovación (España); Swiss Funding Agencies; Swiss National Science Foundation; National Science Council (Taiwan); The Scientific and Technological Research Council of Turkey; Turkish Atomic Energy Authority; Science and Technology Facilities Council (UK); Department of Energy (US); National Science Foundation (US); A. G. Leventis Foundation; Alfred P. Sloan Foundation; Alexander von Humboldt Foundation; Foundation for Polish Science
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13. Study of W boson production in PbPb and pp collisions at √sNN=2.76 TeV
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Chatrchyan, S., Khachatryan, V., Sirunyan, A. M., Tumasyan, A., Adam, W., Bergauer, T., Dragicevic, M., Erö, J., Fabjan, C., Friedl, M., Frühwirth, R., Ghete, V. M., Hammer, J., Hörmann, N., Hrubec, J., Jeitler, M., Kiesenhofer, W., Knünz, V., Krammer, M., Liko, D., Mikulec, I., Pernicka, M., Rahbaran, B., Rohringer, C., Rohringer, H., Schöfbeck, R., Strauss, J., Taurok, A., Wagner, P., Waltenberger, W., Walzel, G., Widl, E., Wulz, C. E., Mossolov, V., Shumeiko, N., Suarez, Gonzalez, Bansal, J., Cornelis, S., Wolf, De, E. A., Janssen, Luyckx, X., Maes, S., Mucibello, T., Ochesanu, L., Roland, S., Rougny, B., Selvaggi, R., Staykova, M., Van, Haevermaet, Van, Mechelen, Van, Remortel, Van, Spilbeeck, Blekman, A., Blyweert, F., D'Hondt, S., Gonzalez, Suarez, Kalogeropoulos, R., Maes, A., Olbrechts, M., Van, Doninck, Van, Mulders, Van, Onsem, G. P., Villella, Charaf, I., Clerbaux, O., Lentdecker, De, Dero, G., Gay, V., A. P. R., Hreus, Léonard, T., Marage, A., P. 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S., Zhukova, Barfuss, V., A. F., Bolton, Chakaberia, T., Ivanov, I., Makouski, S., Maravin, M., Shrestha, Y., Svintradze, S., Gronberg, I., Lange, J., Wright, D., Baden, D., Boutemeur, A., Calvert, M., Eno, B., S. C., Gomez, J. A., Hadley, N. J., Kellogg, R. G., Kirn, Kolberg, M., Lu, T., Marionneau, Y., Mignerey, M., A. C., Pedro, Peterman, K., Skuja, A., Temple, A., Tonjes, J., M. B., Tonwar, S. C., Twedt, Bauer, E., Bendavid, G., Busza, J., Butz, W., Cali, E., I. A., Chan, Dutta, M., Gomez, Ceballos, Goncharov, G., Hahn, M., K. A., Kim, Klute, Y., Li, M., Luckey, W., P. D., Ma, Nahn, T., Paus, S., Ralph, C., Roland, D., Roland, C., Rudolph, G., Stephans, M., G. S. F., Stöckli, Sumorok, F., Sung, K., Velicanu, K., Wenger, D., E. A., Wolf, Wyslouch, R., Xie, B., Yang, S., Yilmaz, M., Yoon, Y., A. S., Zanetti, Cooper, M., S. I., Dahmes, Benedetti, De, Franzoni, A., Gude, G., Kao, A., S. C., Klapoetke, Kubota, K., Mans, Y., Pastika, J., Rusack, N., Sasseville, R., Singovsky, M., Tambe, A., Turkewitz, N., Cremaldi, J., L. M., Kroeger, Perera, R., Rahmat, L., Sanders, R., D. A., Avdeeva, Bloom, E., Bose, K., Butt, S., Claes, J., D. R., Dominguez, Eads, A., Jindal, M., Keller, P., Kravchenko, J., Lazo, Flores, Malbouisson, J., Malik, H., Snow, S., G. R., Baur, Godshalk, U., Iashvili, A., Jain, I., Kharchilava, S., Shipkowski, A., S. P., Smith, Alverson, K., Barberis, G., Baumgartel, E., Chasco, D., Haley, M., Nash, J., Trocino, D., Wood, D., Zhang, D., Anastassov, J., Kubik, A., Mucia, A., Odell, N., Ofierzynski, N., R. A., Pollack, Pozdnyakov, B., Schmitt, A., Stoynev, M., Velasco, S., Won, M., Antonelli, S., Berry, L., Brinkerhoff, D., Hildreth, A., Jessop, M., Karmgard, C., D. J., Kolb, Lannon, J., Luo, K., Lynch, W., Marinelli, S., Morse, N., D. M., Pearson, Ruchti, T., Slaunwhite, R., Valls, J., Wayne, N., Wolf, M., Bylsma, M., Durkin, B., L. S., Hart, Hill, A., Hughes, C., Kotov, R., Ling, K., T. Y., Puigh, Rodenburg, D., Vuosalo, M., Williams, C., Winer, G., B. L., Adam, Berry, N., Elmer, E., Gerbaudo, P., Halyo, D., Hebda, V., Hegeman, P., Hunt, J., Lopes, Pegna, Lujan, D., Marlow, P., Medvedeva, D., Mooney, T., Olsen, M., Piroué, J., Quan, P., Raval, X., Saka, A., Stickland, H., Tully, D., Werner, C., J. S., Zuranski, Acosta, A., J. G., Brownson, Huang, E., X. T., Lopez, Mendez, A., Oliveros, H., Ramirez, Vargas, J. E., Zatserklyaniy, Alagoz, A., Barnes, E., V. E., Benedetti, Bolla, D., Bortoletto, G., Mattia, De, Everett, M., Hu, A., Jones, Z., Koybasi, M., Kress, O., Laasanen, M., A. T., Leonardo, Maroussov, N., Merkel, V., Miller, P., D. H., Neumeister, Shipsey, N., Silvers, I., Svyatkovskiy, D., Vidal, Marono, Yoo, M., H. D., Zablocki, Zheng, J., Guragain, Y., Parashar, S., Adair, N., Boulahouache, A., Cuplov, C., Ecklund, V., K. M., Geurts, F. J. M., Padley, B. P., Redjimi, Roberts, R., Zabel, J., Betchart, J., Bodek, B., Chung, A., Y. S., Covarelli, Barbaro, De, Demina, P., Eshaq, R., Garcia, Bellido, Goldenzweig, A., Han, P., Harel, J., Miner, A., D. C., Vishnevskiy, Zielinski, D., Bhatti, M., Ciesielski, A., Demortier, R., Goulianos, L., Lungu, K., Malik, G., Mesropian, S., Arora, C., Barker, S., Chou, A., Contreras, Campana, Duggan, E., Ferencek, D., Gershtein, D., Gray, Y., Halkiadakis, R., Hidas, E., Lath, D., Panwalkar, A., Park, S., Patel, M., Rekovic, R., Richards, V., Robles, A., Rose, J., Salur, K., Schnetzer, S., Seitz, S., Somalwar, C., Stone, S., Thomas, R., Cerizza, S., Hollingsworth, G., Spanier, M., Z. C., York, Eusebi, A., Flanagan, R., Gilmore, W., Kamon, J., Khotilovich, T., Montalvo, V., Osipenkov, R., Pakhotin, I., Perloff, Y., Roe, A., Safonov, J., Sakuma, A., Sengupta, T., Suarez, S., Tatarinov, I., Toback, A., Akchurin, D., Damgov, N., Dudero, J., P. R., Jeong, Kovitanggoon, C., Lee, K., S. W., Libeiro, Roh, T., Volobouev, Y., Appelt, I., Engh, E., Florez, D., Greene, C., Gurrola, S., Johns, A., Johnston, W., Kurt, C., Maguire, P., Melo, C., Sheldon, A., Snook, P., Tuo, B., Velkovska, S., Arenton, J., M. W., Balazs, Boutle, M., Cox, S., Francis, B., Goodell, B., Hirosky, J., Ledovskoy, R., Lin, A., Neu, C., Wood, C., Yohay, J., Gollapinni, R., Harr, S., Karchin, R., P. E., Kottachchi Kankanamge Don, Lamichhane, C., Sakharov, P., Anderson, A., Bachtis, M., Belknap, M., Borrello, D., Carlsmith, L., Cepeda, D., Dasu, M., Gray, S., Grogg, L., K. S., Grothe, Hall, Wilton, Herndon, R., Hervé, M., Klabbers, A., Klukas, P., Lanaro, J., Lazaridis, A., Leonard, C., Loveless, J., Mohapatra, R., Ojalvo, A., Palmonari, I., Pierro, F., G. A., Ross, Savin, I., Smith, A., W. H., Swanson, Guo S, J., Guo, Y, Li, W, Mao, Y, Qian, Sj, Teng, H, Zhang, L, Zhu, B, Zou, W, Cabrera, A, Moreno, Bg, Rios, Aao, Oliveros, Afo, Sanabria, Jc, Godinovic, N, Lelas, D, Lelas, K, Plestina, R, Polic, D, Puljak, I, Antunovic, Z, Dzelalija, M, Brigljevic, V, Duric, S, Kadija, K, Morovic, S, Attikis, A, Galanti, M, Mousa, J, Nicolaou, C, Ptochos, F, Razis, Pa, Rykaczewski, H, Assran, Y, Mahmoud, Ma, Hektor, A, Kadastik, M, Kannike, K, Muentel, M, Raidal, M, Rebane, L, Azzolini, V, Eerola, P, Czellar, S, Harkonen, J, Heikkinen, A, Karimaki, V, Kinnunen, R, Klem, J, Kortelainen, Mj, Lampen, T, Lassila Perini, K, Lehti, S, Linden, T, Luukka, P, Maenpaa, T, Tuominen, E, Tuominiemi, J, Tuovinen, E, Ungaro, D, Wendland, L, Banzuzi, K, Korpela, A, Tuuva, T, Sillou, D, Besancon, M, Choudhury, S, Dejardin, M, Denegri, D, Fabbro, B, Faure, Jl, Ferri, F, Ganjour, S, Gentit, Fx, Givernaud, A, Gras, P, de Monchenault GH, Jarry, P, Locci, E, Malcles, J, Marionneau, M, Millischer, L, Rander, J, Rosowsky, A, Shreyber, I, Titov, M, Verrecchia, P, Baffioni, S, Beaudette, F, Bianchini, L, Bluj, M, Broutin, C, Busson, P, Charlot, C, Dahms, T, Dobrzynski, L, de Cassagnac RG, Haguenauer, M, Mine, P, Mironov, C, Ochando, C, Paganini, P, Sabes, D, Salerno, R, Sirois, Y, Thiebaux, C, Wyslouch, B, Zabi, A, Agram, Jl, Andrea, J, Besson, A, Bloch, D, Bodin, D, Brom, Jm, Cardaci, M, Chabert, Ec, Collard, C, Conte, E, Drouhin, F, Ferro, C, Fontaine, Jc, Gele, D, Goerlach, U, Greder, S, Juillot, P, Karim, M, Le Bihan AC, Mikami, Y, Van Hove, P, Fassi, F, Mercier, D, Baty, C, Beaupere, N, Bedjidian, M, Bondu, O, Boudoul, G, Boumediene, D, Brun, H, Chanon, N, Chierici, R, Contardo, D, Depasse, P, El Mamouni, H, Falkiewicz, A, Fay, J, Gascon, S, Ille, B, Kurca, T, Le Grand, T, Lethuillier, M, Mirabito, L, Perries, S, Sordini, V, Tosi, S, Tschudi, Y, Verdier, P, Xiao, H, Roinishvili, V, Lomidze, D, Anagnostou, G, Edelhoff, M, Feld, L, Heracleous, N, Hindrichs, O, Jussen, R, Klein, K, Merz, J, Mohr, N, Ostapchuk, A, Perieanu, A, Raupach, F, Sammet, J, Schael, S, Sprenger, D, Weber, H, Weber, M, Wittmer, B, Ata, M, Bender, W, Erdmann, M, Frangenheim, J, Hebbeker, T, Hinzmann, A, Hoepfner, K, Hof, C, Klimkovich, T, Klingebiel, D, Kreuzer, P, Lanske, D, Magass, C, Masetti, G, Merschmeyer, M, Meyer, A, Papacz, P, Pieta, H, Reithler, H, Schmitz, Sa, Sonnenschein, L, Steggemann, J, Teyssier, D, Bontenackels, M, Davids, M, Duda, M, Flugge, G, Geenen, H, Giffels, M, Ahmad, Wh, Heydhausen, D, Kress, T, Kuessel, Y, Linn, A, Nowack, A, Perchalla, L, Pooth, O, Rennefeld, J, Sauerland, P, Stahl, A, Thomas, M, Tornier, D, Zoeller, Mh, Martin, Ma, Behrenhoff, W, Behrens, U, Bergholz, M, Borras, K, Cakir, A, Campbell, A, Castro, E, Dammann, D, Eckerlin, G, Eckstein, D, Flossdorf, A, Flucke, G, Geiser, A, Glushkov, I, Hauk, J, Jung, H, Kasemann, M, Katkov, I, Katsas, P, Kleinwort, C, Kluge, H, Knutsson, A, Krucker, D, Kuznetsova, E, Lange, W, Lohmann, W, Mankel, R, Marienfeld, M, Melzer Pellmann IA, Meyer, Ab, Mnich, J, Mussgiller, A, Olzem, J, Parenti, A, Raspereza, A, Raval, A, Schmidt, R, Schoerner Sadenius, T, Sen, N, Stein, M, Tomaszewska, J, Volyanskyy, D, Walsh, R, Wissing, C, Autermann, C, Bobrovskyi, S, Draeger, J, Enderle, H, Gebbert, U, Kaschube, K, Kaussen, G, Klanner, R, Lange, J, Mura, B, Naumann Emme, S, Nowak, F, Pietsch, N, Sander, C, Schettler, H, Schleper, P, Schroder, M, Schum, T, Schwandt, J, Srivastava, Ak, Stadie, H, Steinbruck, G, Thomsen, J, Wolf, R, Barth, C, Bauer, J, Buege, V, Chwalek, T, De Boer, W, Dierlamm, A, Dirkes, G, Feindt, M, and Gruschke, J.
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Heavy-ions ,Nuclear and High Energy Physics ,Heavy Ion collision ,CMS experiment ,ROOT-S=7 TEV ,Physics::Instrumentation and Detectors ,CMS ,Physics ,ATLAS DETECTOR ,W bosons ,HEAVY-ION COLLISIONS ,PRODUCTION CROSS-SECTION ,Physics and Astronomy ,High Energy Physics::Experiment ,LHC ,Nuclear Experiment - Abstract
A measurement is presented of W-boson production in PbPb collisions carried out at a nucleon-nucleon (NN) centre-of-mass energy root S-NN of 2.76 TeV at the LHC using the CMS detector. In data corresponding to an integrated luminosity of 7.3 mu b(-1), the number of W -> mu v(mu) decays is extracted in the region of muon pseudorapidity vertical bar eta mu vertical bar < 2.1 and transverse momentum p(T)(mu) > 25 GeV/c. Yields of muons found per unit of pseudorapidity correspond to (159 +/- 10(stat.) +/- 12(syst.)) x 10(-8) W and (154 +/- 10(stat.) +/- 12(syst.)) x 10(-8) W- bosons per minimum-bias PbPb collision. The dependence of W production on the centrality of PbPb collisions is consistent with a scaling of the yield by the number of incoherent NN collisions. The yield of W bosons is also studied in a sample of pp interactions at root S = 2.76 TeV corresponding to an integrated luminosity of 231 nb(-1). The individual W+ and W- yields in PbPb and pp collisions are found to agree, once the neutron and proton content in Pb nuclei is taken into account. Likewise, the difference observed in the dependence of the positive and negative muon production on pseudorapidity is consistent with next-to-leading-order perturbative QCD calculations.
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- 2012
14. Neural rhythms of change: Long-term improvement after successful treatment in children with disruptive behavior problems
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Woltering, S., Liao, V., Liu, Z.X., Granic, I., Woltering, S., Liao, V., Liu, Z.X., and Granic, I.
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Contains fulltext : 143353.pdf (publisher's version ) (Open Access), Neural changes were investigated for children with disruptive behavior problems one year after a treatment program ended. Thirty-nine children and their parents visited the research lab before, after, and a year after treatment ended. During those lab visits, electroencephalography (EEG) was recorded during a challenging Go/No-go task. Treatment consisted of intensive 14-week combined cognitive behavioral therapy and parent management training sessions. For the analysis, participants were divided into long-term improvers (IMPs) and long-term nonimprovers (NIMPs) based on changes in their externalizing problem scores. The results showed early no-go theta power (4-8Hz, 100-250ms) decreased for long-term IMPs compared to NIMPs. When participants were divided based on changes in their comorbid internalizing symptoms, effects were stronger and reductions in theta power were found for early as well as later phases (250-650ms). We provided preliminary evidence that theta power is a useful neural measure to trace behavioral change linked to improved self-regulation even up to a year after treatment ended. Results may have implications for the characterization of children with disruptive behavior problems and may lead to the development of novel markers of treatment success.
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- 2015
15. Arsenite exerts transgenerational reproduction defects in Caenorhabditis elegans
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Liao, V., primary and Yu, C.-W., additional
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- 2015
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16. Curcumin-mediated oxidative stress resistance inCaenorhabditis elegansis modulated byage-1, akt-1, pdk-1, osr-1, unc-43, sek-1, skn-1, sir-2.1, andmev-1
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Yu, C.-W., primary, Wei, C.-C., additional, and Liao, V. H.-C., additional
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- 2014
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17. MedtableTM: An EMR-Based Tool to Support Collaborative Planning for Medication Use
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Liao, V., primary, Chin, C.-L., additional, McKeever, S., additional, Kopren, K., additional, Morrow, D., additional, Davis, K., additional, Wilson, E. A. H., additional, Kaiser, D., additional, Wolf, M., additional, Conner-Garcia, T., additional, and Graumlich, J., additional
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- 2011
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18. Identification of a novel, cadmium-inducible, integral membrane protein from C. elegans
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Hsiu-Chuan Liao, V., primary, Dong, J., additional, and Freedman, J.H., additional
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- 2000
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19. Curcumin-mediated oxidative stress resistance in Caenorhabditis elegans is modulated by age-1, akt-1, pdk-1, osr-1, unc-43, sek-1, skn-1, sir-2.1, and mev-1.
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Yu, C.-W., Wei, C.-C., and Liao, V. H.-C.
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CURCUMIN ,OXIDATIVE stress ,CAENORHABDITIS elegans ,ANTI-inflammatory agents ,ANTIOXIDANTS ,CELLULAR signal transduction ,REACTIVE oxygen species - Abstract
Curcumin (diferuloylmethane), a pharmacologically active substance derived from turmeric, exhibits anti-inflammatory, anticarcinogenic, and antioxidant properties. We examined the modulation of oxidative-stress resistance and associated regulatory mechanisms by curcumin in a Caenorhabditis elegans model. Our results showed that curcumin-treated wild-type C. elegans exhibited increased survival during juglone-induced oxidative stress compared with the control treatment. In addition, curcumin reduced the levels of intracellular reactive oxygen species in C. elegans. Moreover, curcumin induced the expression of the gst-4 and hsp-16.2 stress response genes. Lastly, our findings from the mechanistic study in this investigation suggest that the antioxidative effect of curcumin is mediated via regulation of age-1, akt-1, pdk-1, osr-1, unc-43, sek-1, skn-1, sir-2.1, and mev-1. Our study elucidates the diverse modes of action and signaling pathways that underlie the antioxidant activity exhibited by curcumin in vivo. [ABSTRACT FROM AUTHOR]
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- 2014
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20. Cadmium-regulated genes from the nematode Caenorhabditis elegans. Identification and cloning of new cadmium-responsive genes by differential display.
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Liao, V H and Freedman, J H
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The transition metal cadmium is a pervasive and persistent environmental contaminant that has been shown to be both a human toxicant and carcinogen. To inhibit cadmium-induced damage, cells respond by increasing the expression of genes encoding stress-response proteins. In most cases, the mechanism by which cadmium affects the expression of these genes remains unknown. It has been demonstrated in several instances that cadmium activates gene transcription through signal transduction pathways, mediated by protein kinase C, cAMP-dependent protein kinase, or calmodulin. A codicil is that cadmium should influence the expression of numerous genes. To investigate the ability of cadmium to affect gene transcription, the differential display technique was used to analyze gene expression in the nematode Caenorhabditis elegans. Forty-nine cDNAs whose steady-state levels of expression change 2-6-fold in response to cadmium exposure were identified. The nucleotide sequences of the majority of the differentially expressed cDNAs are identical to those of C. elegans cosmids, yeast artificial chromosomes, expressed sequence tags, or predicted genes. The translated amino acid sequences of several clones are identical to C. elegans metallothionein-1, HSP70, collagens, and rRNAs. In addition, C. elegans homologues of pyruvate carboxylase, DNA gyrase, beta-adrenergic receptor kinase, and human hypothetical protein KIAA0174 were identified. The translated amino acid sequences of the remaining differentially expressed cDNAs encode novel proteins.
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- 1998
21. Early life long-term exposure to aflatoxin B1 induces aging and alters innate immunity associated with SKN-1/Nrf2 in Caenorhabditis elegans.
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Chang TT, Chang CH, and Hsiu-Chuan Liao V
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- Animals, NF-E2-Related Factor 2 metabolism, NF-E2-Related Factor 2 genetics, Longevity drug effects, Caenorhabditis elegans drug effects, Caenorhabditis elegans immunology, Caenorhabditis elegans metabolism, Aflatoxin B1 toxicity, Immunity, Innate drug effects, Caenorhabditis elegans Proteins metabolism, Caenorhabditis elegans Proteins genetics, Aging, Transcription Factors metabolism, Transcription Factors genetics, Pseudomonas aeruginosa drug effects, DNA-Binding Proteins genetics, DNA-Binding Proteins metabolism
- Abstract
Aflatoxin B1 (AFB1), a known human carcinogen, represents the most toxic aflatoxin metabolite. Exposure to AFB1 causes increased oxidative stress and immunotoxicity, which are important factors contributing to aging. However, the role of AFB1-induced toxicity in altered innate immunity and aging remains largely unclear. The nematode Caenorhabditis elegans is a suitable model organism for studying aging and toxicology due to its well-studied molecular mechanisms and short life cycle. Effects of AFB1 at 1, 2.5, and 5 μM (312, 781, and 1561 μg/L) on growth, reproduction, and lifespan were examined. The Pseudomonas aeruginosa PA14 slow-killing assay was performed to investigate innate immunity, followed by studying the possible mechanisms using transgenic strains and qPCR analysis. The results showed that early life long-term AFB1 exposure (2.5 and 5 μM) delayed development, reduced reproduction, and shortened lifespan in C. elegans. Furthermore, in aged worms, AFB1 exposure caused a dose-dependent decrease in survival of C. elegans against P. aeruginosa PA14 infection. At adulthood day 4 in the presence of live Escherichia coli OP50, AFB1 (2.5 μM) significantly increased lipofuscin levels (a hallmark of aging) compared to adult day 0, whereas no increase in lipofuscin was observed in nematodes (adulthood day 4) fed with dead E. coli OP50. Additionally, the increased lipofuscin was abolished in the skn-1 mutant with either live or dead E. coli OP50. Furthermore, AFB1 suppressed intestinal SKN-1::GFP translocation. Two-way ANOVA analysis revealed that the activity of E. coli OP50 and AFB1 interactively affected the expression of genes: skn-1, gst-4, hsp-16.1, hsp-16.49, and hsp-70. Our findings highlight the role of AFB1-induced toxicity in altered innate immunity and aging through the involvement of the transcription factor SKN-1/Nrf2., Competing Interests: Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper., (Copyright © 2024 Elsevier B.V. All rights reserved.)
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- 2025
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22. Comprehensive plasma cytokine profiling reveals a distinct endotype of Type 2 Inflammation in Chronic Pruritus of Unknown Origin.
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Manjunath J, Bao A, Gonzalez W, Vojvoda E, D'Amiano AJ, Patel S, Liao V, Sivaloganathan D, Bhatt S, Ahmadi M, and Kwatra SG
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- 2025
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23. Cancer and left atrial enlargement in patients with ischemic stroke.
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Beyeler M, Pawar A, Buffle E, Zhang C, Liao V, Liberman AL, Pabst T, Berger MD, Jung S, Kamel H, and Navi BB
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- Humans, Male, Female, Aged, Cross-Sectional Studies, Retrospective Studies, Middle Aged, Risk Factors, Aged, 80 and over, Risk Assessment, Atrial Remodeling, Embolic Stroke etiology, Embolic Stroke epidemiology, Embolic Stroke diagnosis, Cardiomegaly diagnostic imaging, Cardiomegaly epidemiology, Cardiomegaly etiology, New York epidemiology, New York City epidemiology, Prognosis, Ischemic Stroke epidemiology, Ischemic Stroke diagnosis, Ischemic Stroke etiology, Ischemic Stroke diagnostic imaging, Neoplasms complications, Neoplasms epidemiology, Heart Atria diagnostic imaging, Heart Atria physiopathology, Atrial Function, Left
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Background: Cancer is associated with an increased risk of atrial fibrillation. Whether cancer is also associated with atrial cardiopathy, another atrial pathology associated with heightened ischemic stroke risk, is uncertain., Methods: We conducted a retrospective cross-sectional study among consecutive patients hospitalized with acute ischemic stroke at a quaternary care center in New York, United States from 2011 through 2016. The study exposure was active cancer. The study outcome was atrial cardiopathy, defined as a left atrial volume index ≥35 mL/m
2 on echocardiography. We used multivariable logistic regression, adjusting for baseline characteristics, to evaluate the relationship between cancer (active or historical) and atrial cardiopathy. We performed a subgroup analysis among patients with embolic stroke of undetermined source (ESUS)., Results: The final cohort included 1104 patients with acute ischemic stroke, of whom 10 % had active cancer and 47 % had atrial cardiopathy. Patients with atrial cardiopathy, compared to those without, were older (median age, 77 versus 68 years), and more frequently had hypertension, coronary disease, and atrial fibrillation. Active cancer was present in 9.6 % of patients with atrial cardiopathy (n = 50/520) and 10.4 % of patients without (n = 61/584). There was no association between active cancer and atrial cardiopathy among the overall ischemic stroke cohort (adjusted odds ratio [OR], 0.91; 95 % confidence interval [CI], 0.60-1.37) nor in patients with ESUS (aOR, 0.64; 95 % CI, 0.30-1.36). When the cancer exposure was broadened to include any history of cancer (n = 236, 21.4 %), there still was no significant association with atrial cardiopathy (aOR, 0.93; 95 % CI, 0.68-1.25)., Conclusions: When defining atrial cardiopathy by left atrial volume, we did not find an association between cancer and atrial cardiopathy in patients with ischemic stroke, including among those with ESUS. Future studies, evaluating other atrial cardiopathy biomarkers and settings, are needed to further investigate any potential link between cancer and atrial cardiopathy., Competing Interests: Declaration of competing interest Dr. Beyeler has received research support from the University of Bern, Switzerland. Dr. Navi has received personal fees for serving on an adjudication committee for MindRhythm Inc. Dr. Kamel reports serving as a PI for the ARCADIA trial (NIH/NINDSU01NS095869), which received in-kind study drug from the BMS-Pfizer Alliance for Eliquis® and ancillary study support from Roche Diagnostics; other funding from NIH (R01HL144541, R01NS123576, U01NS106513); serving as Deputy Editor for JAMA Neurology; serving on clinical trial steering/executive committees for Medtronic, Janssen, and Javelin Medical; serving on endpoint adjudication committees for AstraZeneca, Novo Nordisk, and Boehringer Ingelheim; and household ownership interests in TETMedical, Spectrum Plastics Group, and Burke Porter Group. The other authors declare no competing interests., (Copyright © 2024 The Author(s). Published by Elsevier Inc. All rights reserved.)- Published
- 2024
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24. Risk factors and temporal associations of progression of the atopic march in children with early-onset atopic dermatitis.
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Choi UE, Deng J, Parthasarathy V, Liao V, D'Amiano A, Taylor M, Bordeaux ZA, Kambala A, Cornman HL, Canner JK, Drucker AM, and Kwatra SG
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Background: Risk factors and the temporal relationship between atopic dermatitis (AD) and atopic march remain understudied., Objective: Determine risk factors for atopic march in early-onset AD patients and the temporality between AD and atopic march., Methods: We used the MarketScan Research Database for our retrospective cohort analysis from 2010 to 2018, comparing infants diagnosed with AD before age 1 with controls without early-onset AD. Primary outcomes were hazard ratios (HRs) for the development of asthma, allergic rhinitis, and food allergy., Results: Compared to 55,174 controls, higher proportions of the 27,228 AD patients developed asthma (19.21% vs 8.65%, P < .001), allergic rhinitis (28.27% vs 12.62%, P < .001), food allergy (16.00% vs 2.27%, P < .001), and all atopic triad conditions (10.69% vs 0.71%, P < .001). Among AD patients, higher proportions developed the atopic triad if they were male (HR 1.66, 95% confidence interval [1.45-1.90]), had severe disease (HR 3.16, [2.77-3.60]), or had family atopy history (HR > 3.40, P < .001 for all comparisons). Among AD patients, 20.1% developed allergic rhinitis., Limitations: Our study was based on health care claims data., Conclusion: Early-onset AD is associated with higher rates of developing atopic march conditions compared to controls. Particular attention should be paid toward risk factors and atopic march screening in early-onset AD patients., Competing Interests: Conflicts of interest Dr Drucker has received compensation from the British Journal of Dermatology (reviewer and Section Editor), American Academy of Dermatology (guidelines writer), and National Eczema Association (grant reviewer). Dr Kwatra is an advisory board member/consultant for Abbvie, Amgen, Arcutis Biotherapeutics, Aslan Pharmaceuticals, Cara Therapeutics, Castle Biosciences, Celldex Therapeutics, Galderma, Genzada Pharmaceuticals, Incyte Corporation, Johnson & Johnson, Leo Pharma, Novartis Pharmaceuticals Corporation, Pfizer, Regeneron Pharmaceuticals, Kiniksa Pharmaceuticals, and Sanofi and has served as an investigator for Galderma, Kiniksa Pharmaceuticals, Incyte, Pfizer, and Sanofi outside of the submitted work. Drs Choi, Deng, Parthasarathy, Liao, D'Amiano, Taylor, Bordeaux, Kambala, Cornman, and Canner have no conflicts of interest to declare., (Copyright © 2024 American Academy of Dermatology, Inc. Published by Elsevier Inc. All rights reserved.)
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- 2024
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25. A computational method for rapid analysis polymer structure and inverse design strategy (RAPSIDY).
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Liao V, Myers T, and Jayaraman A
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Tailoring polymers for target applications often involves selecting candidates from a large design parameter space including polymer chemistry, molar mass, sequence, and architecture, and linking each candidate to their assembled structures and in turn their properties. To accelerate this process, there is a critical need for inverse design of polymers and fast exploration of the structures they can form. This need has been particularly challenging to fulfill due to the multiple length scales and time scales of structural arrangements found in polymers that together give rise to the materials' properties. In this work, we tackle this challenge by introducing a computational framework called RAPSIDY - Rapid Analysis of Polymer Structure and Inverse Design strategY. RAPSIDY enables inverse design of polymers by accelerating the evaluation of stability of multiscale structure for any given polymer design (sequence, composition, length). We use molecular dynamics simulations as the base method and apply a guiding potential to initialize polymers chains of a selected design within target morphologies. After initialization, the guiding potential is turned off, and we allow the chains and structure to relax. By evaluating similarity between the target morphology and the relaxed morphology for that polymer design, we can screen many polymer designs in a highly parallelized manner to rank designs that are likely to remain in that target morphology. We demonstrate how this method works using an example of a symmetric, linear pentablock, A
x By Az By Ax , copolymer system for which we determine polymer sequences that exhibit stable double gyroid morphology. Rather than trying to identify the global free-energy minimum morphology for a specific polymer design, we aim to identify candidates of polymer design parameter space that are more stable in the desired morphology than others. Our approach reduces computational costs for design parameter exploration by up to two orders-of-magnitude compared to traditional MD methods, thus accelerating design and engineering of novel polymer materials for target applications.- Published
- 2024
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26. Rates of Acute Brain Injury Among Severely Hypertensive Patients Seen in the Emergency Department.
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Ishak A, Kamel H, Kaiser JH, Liao V, Rotblat J, Pawar A, Lappin R, Razzak J, White H, Ng C, and Liberman AL
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Background: Identifying patients with acute brain injury among patients who present to the Emergency Department (ED) with severe hypertension can be challenging. We explored rates of brain injury in a cohort of ED patients with severe hypertension in whom acute target-organ damage was or was not initially suspected., Methods: We conducted a retrospective chart-review study at two different hospitals within the same urban health system. Consecutive adult patients seen from 10/1/2020 to 5/1/2022 with a systolic blood pressure ≥180 mmHg or diastolic blood pressure ≥120 mmHg were included., Results: A total of 972 patients met our eligibility criteria. The average age was 59 years (SD: 16.5), 454 (46.7%) were women, and 582 (59.9%) were non-White. A total of 186/972 (19.1%) patients were initially thought to have acute target-organ damage in the ED with 97/186 (52.2%) diagnosed with stroke or TIA. The remaining 786/972 (80.9%) patients were not initially diagnosed with target-organ damage (hypertensive urgency [HU]). Of the 786 patients with HU, a head CT was obtained in 216/786 (27.7%) and brain MRI in 74/786 (9.4%). Of the HU patients with a brain MRI, 23/74 (31.1%) had acute abnormalities; most abnormalities on brain MRI (17/23 [73.9%]) were not seen on preceding head CT., Conclusions: In a multicenter study of ED patients, nearly 1 in 5 patients though to have HU who eventually underwent brain MRI had unexpected acute neurological findings, though brain MR was obtained in only 9%., Competing Interests: The author(s) declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article., (© The Author(s) 2024.)
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- 2024
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27. Risk of Major Adverse Cardiovascular Events After Emergency Department Visits for Hypertensive Urgency.
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Liberman AL, Razzak J, Lappin RI, Navi BB, Bruce SS, Liao V, Kaiser JH, Ng C, Segal AZ, and Kamel H
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- Humans, Male, Female, Middle Aged, Aged, Cardiovascular Diseases epidemiology, Cross-Over Studies, Hospitalization statistics & numerical data, Risk Factors, Risk Assessment methods, Adult, Emergency Room Visits, Hypertensive Crisis, Hypertension epidemiology, Hypertension complications, Emergency Service, Hospital statistics & numerical data
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Background: Chronic hypertension is an established long-term risk factor for major adverse cardiovascular events (MACEs). However, little is known about short-term MACE risk after hypertensive urgency, defined as an episode of acute severe hypertension without evidence of target-organ damage. We sought to evaluate the short-term risk of MACE after an emergency department (ED) visit for hypertensive urgency resulting in discharge to home., Methods: We performed a case-crossover study using deidentified administrative claims data. Our case periods were 1-week intervals from 0 to 12 weeks before hospitalization for MACE. We compared ED visits for hypertensive urgency during these case periods versus equivalent control periods 1 year earlier. Hypertensive urgency and MACE components were all ascertained using previously validated International Classification of Diseases , Tenth Revision Clinical Modification codes. We used McNemar test for matched data to calculate risk ratios., Results: Among 2 225 722 patients with MACE, 1 893 401 (85.1%) had a prior diagnosis of hypertension. There were 4644 (0.2%) patients who had at least 1 ED visit for hypertensive urgency during the 12 weeks preceding their MACE hospitalization. An ED visit for hypertensive urgency was significantly more common in the first week before MACE compared with the same chronological week 1 year earlier (risk ratio, 3.5 [95% CI, 2.9-4.2]). The association between hypertensive urgency and MACE decreased in magnitude with increasing temporal distance from MACE and was no longer significant by 11 weeks before MACE (risk ratio, 1.2 [95% CI, 0.99-1.6])., Conclusions: ED visits for hypertensive urgency were associated with a substantially increased short-term risk of subsequent MACE., Competing Interests: Disclosures J. Razzak serves as primary investigator for the National Institutes of Health (NIH)–funded grants R21HD103049, D43TW007292, and R21TW012210. B.B. Navi and A.Z. Segal have received personal fees for serving as a neurology expert witness. S.S. Bruce is supported by National Center for Advancing Translational Sciences research grant KL2TR2385. H. Kamel reports: PI for the ARCADIA (Atrial Cardiopathy and Antithrombotic Drugs In prevention After cryptogenic stroke) trial (NIH/NINDS U01NS095869), which receives in-kind study drug from the Bristol-Myers Squibb-Pfizer Alliance for Eliquis and ancillary study support from Roche Diagnostics; other funding from NIH (R01HL144541, R01NS123576, U01NS106513); deputy editor for JAMA Neurology; clinical trial steering/executive committees for Medtronic, Janssen, and Javelin Medical; end point adjudication committees for AstraZeneca, Novo Nordisk, and Boehringer Ingelheim; and household ownership interests in TETMedical, Spectrum Plastics Group, and Burke Porter Group. The other authors report no conflicts.
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- 2024
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28. Atrial fibrillation and short-term outcomes after cancer-related ischemic stroke.
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Wahbeh F, Zhang C, Beyeler M, Kaiser JH, Liao V, Pawar A, Kamel H, and Navi BB
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Introduction: Atrial fibrillation (AF) and cancer are each associated with worse outcomes in patients with acute ischemic stroke (AIS). Few studies have evaluated the impact of AF on outcomes of cancer-related stroke., Patients and Methods: We conducted a retrospective cross-sectional study using the 2016-2019 National Inpatient Sample, identifying all hospitalizations with diagnosis codes for cancer and AIS. The primary exposure was a diagnosis of AF. The primary outcome was in-hospital mortality. The secondary outcomes were length-of-stay and discharge to non-home locations. We used multiple logistic and linear regression models, adjusted for age, gender, race-ethnicity, and the Charlson Comorbidity Index, to examine the association between AF and study outcomes., Results: Among 150,200 hospitalizations with diagnoses of cancer and AIS (mean age 72 years, 53% male), 40,084 (26.7%) included comorbid AF. Compared to hospitalizations without AF, hospitalizations with AF had higher rates of in-hospital mortality (14.8% [95% CI, 14.0%-15.6%] vs 12.1% [95% CI, 11.6%-12.5%]) and non-home discharge disposition (83.5% [95% CI, 82.7%-84.3%] vs 75.1% [95% CI, 74.5%-75.7%]) as well as longer mean length-of-stay (8.4 days [95% CI, 8.2-8.6 days] vs 8.2 days [95% CI, 8.0-8.3 days]). In multivariable analyses, AF remained independently associated with higher odds of in-hospital mortality (adjusted odds ratio [aOR], 1.34; 95% CI, 1.24-1.46), non-home discharge disposition (aOR, 1.32; 95% CI, 1.23-1.42), and longer length-of-stay (adjusted mean difference, 13.7%; 95% CI, 10.9%-16.7%)., Discussion and Conclusion: In cancer-related AIS, comorbid AF is associated with worse short-term outcomes, including higher odds for in-hospital mortality, poor discharge disposition, and longer hospital stays., Competing Interests: Declaration of conflicting interestsThe author(s) declared the following potential conflicts of interest with respect to the research, authorship, and/or publication of this article: Dr. Navi has received personal fees for serving on an adjudication committee for MindRhythm Inc and for serving as a neurology expert witness. Dr. Kamel reports serving as a PI for the ARCADIA trial (NIH/NINDS U01NS095869), which received in-kind study drug from the BMS-Pfizer Alliance for Eliquis® and ancillary study support from Roche Diagnostics; other funding from NIH (R01HL144541, R01NS123576, U01NS106513); serving as Deputy Editor for JAMA Neurology; serving on clinical trial steering/executive committees for Medtronic, Janssen, and Javelin Medical; serving on endpoint adjudication committees for AstraZeneca, Novo Nordisk, and Boehringer Ingelheim; and household ownership interests in TETMedical, Spectrum Plastics Group, and Burke Porter Group. The other authors declare that there is no conflict of interest.
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- 2024
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29. Cancer and the risk of perioperative arterial ischaemic events.
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Navi BB, Zhang C, Kaiser JH, Liao V, Cushman M, Kasner SE, Elkind MSV, Tagawa ST, Guntupalli SR, Gaudino MFL, Lee AYY, Khorana AA, and Kamel H
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- Humans, Female, Male, Middle Aged, Risk Factors, United States epidemiology, Aged, Postoperative Complications epidemiology, Incidence, Retrospective Studies, Risk Assessment methods, Follow-Up Studies, Neoplasms epidemiology, Neoplasms surgery
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Background and Aims: Most cancer patients require surgery for diagnosis and treatment. This study evaluated whether cancer is a risk factor for perioperative arterial ischaemic events., Methods: The primary cohort included patients registered in the National Surgical Quality Improvement Program (NSQIP) between 2006 and 2016. The secondary cohort included Healthcare Cost and Utilization Project (HCUP) claims data from 11 US states between 2016 and 2018. Study populations comprised patients who underwent inpatient (NSQIP, HCUP) or outpatient (NSQIP) surgery. Study exposures were disseminated cancer (NSQIP) and all cancers (HCUP). The primary outcome was a perioperative arterial ischaemic event, defined as myocardial infarction or stroke diagnosed within 30 days after surgery., Results: Among 5 609 675 NSQIP surgeries, 2.2% involved patients with disseminated cancer. The perioperative arterial ischaemic event rate was 0.96% among patients with disseminated cancer vs. 0.48% among patients without (hazard ratio [HR], 2.01; 95% confidence interval [CI], 1.90-2.13). In Cox analyses adjusting for demographics, functional status, comorbidities, surgical specialty, anesthesia type, and clinical factors, disseminated cancer remained associated with higher risk of perioperative arterial ischaemic events (HR, 1.37; 95% CI, 1.28-1.46). Among 1 341 658 surgical patients in the HCUP cohort, 11.8% had a diagnosis of cancer. A perioperative arterial ischaemic event was diagnosed in 0.74% of patients with cancer vs. 0.54% of patients without cancer (HR, 1.35; 95% CI, 1.27-1.43). In Cox analyses adjusted for demographics, insurance, comorbidities, and surgery type, cancer remained associated with higher risk of perioperative arterial ischaemic events (HR, 1.31; 95% CI, 1.21-1.42)., Conclusion: Cancer is an independent risk factor for perioperative arterial ischaemic events., (© The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology.)
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- 2024
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30. Association between elevated fibrosis-4 index of liver fibrosis and risk of hemorrhagic stroke.
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Parikh NS, Zhang C, Bruce SS, Murthy SB, Rosenblatt R, Liberman AL, Liao V, Kaiser JH, Navi BB, Iadecola C, and Kamel H
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Background: Cirrhosis is associated with an increased risk of hemorrhagic stroke. Liver fibrosis, typically a silent condition, is antecedent to cirrhosis. The objective of this study was to test the hypothesis that elevated Fibrosis-4 (FIB-4) index, indicating a high probability of liver fibrosis, is associated with an increased risk of hemorrhagic stroke., Methods: We performed a cohort analysis of the prospective United Kingdom Biobank cohort study. Participants 40-69 years old were enrolled between 2007 and 2010 and had available follow-up data until March 1, 2018. We excluded participants with prevalent hemorrhagic stroke or thrombocytopenia. High probability of liver fibrosis was defined as having a value >2.67 of the validated FIB-4 index. The primary outcome was hemorrhagic stroke (intracerebral or subarachnoid hemorrhage), defined based on hospitalization and death registry data. Secondary outcomes were intracerebral and subarachnoid hemorrhage, separately. We used Cox proportional hazards models to evaluate the association of FIB-4 index >2.67 with hemorrhagic stroke while adjusting for potential confounders including hypertension, alcohol use, and antithrombotic use., Results: Among 452,994 participants (mean age, 57 years; 54% women), approximately 2% had FIB-4 index >2.67, and 1241 developed hemorrhagic stroke. In adjusted models, FIB-4 index >2.67 was associated with an increased risk of hemorrhagic stroke (HR, 2.0; 95% CI, 1.6-2.6). Results were similar for intracerebral hemorrhage (HR, 2.0; 95% CI, 1.5-2.7) and subarachnoid hemorrhage (HR, 2.2; 95% CI, 1.5-3.5) individually., Conclusions: Elevated FIB-4 index was associated with an increased risk of hemorrhagic stroke., Competing Interests: Declaration of conflicting interestThe author(s) declared the following potential conflicts of interest with respect to the research, authorship, and/or publication of this article: Drs. Parikh and Navi have received personal compensation for medicolegal consulting on stroke. Dr. Parikh is now employed by Alnylam Pharmaceuticals, Inc.; his contribution to this work occurred while employed at Weill Cornell Medicine and does not reflect the views of his current employer. Dr. Murthy reports research support from the NIH outside the submitted work, and has received personal compensation for medicolegal consulting on neurological disorders and stroke. Dr. Bruce reports research support from the NIH (KL2-TR-2385) outside the submitted work. Dr. Liberman reports research support from the NIH outside the submitted work. Dr. Kamel serves as a PI for the NIH-funded ARCADIA trial (NINDS U01NS095869) which receives in-kind study drug from the BMS-Pfizer Alliance for Eliquis and ancillary study support from Roche Diagnostics, serves as Deputy Editor for JAMA Neurology, serves as a steering committee member of Medtronic’s Stroke AF trial, serves on a trial executive committee for Janssen, and serves on an endpoint adjudication committee for a trial of empagliflozin for Boehringer-Ingelheim. Dr. Iadecola serves on the Scientific Advisory Board of Broadview Ventures.
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- 2024
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31. Prurigo nodularis: new insights into pathogenesis and novel therapeutics.
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Liao V, Cornman HL, Ma E, and Kwatra SG
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- Humans, Antibodies, Monoclonal, Humanized therapeutic use, Janus Kinase Inhibitors therapeutic use, Pyrimidines therapeutic use, Nitriles therapeutic use, Receptors, Interleukin antagonists & inhibitors, Cytokines metabolism, Pyrazoles, Prurigo etiology, Prurigo drug therapy
- Abstract
Prurigo nodularis (PN) is an inflammatory skin condition characterized by intensely pruritic nodules on the skin. Patients with PN suffer from an intractable itch-scratch cycle leading to impaired sleep, psychosocial distress and a significant disruption in quality of life. The pathogenesis of PN is associated with immune and neural dysregulation, mediated by inflammatory cytokines [such as interleukin (IL)-4, -13, -17, -22 and -31] and neuropeptides (such as substance P and calcitonin gene-related peptide). There is a role for type 2 inflammation in PN in addition to T-helper (Th)17 and Th22-mediated inflammation. The neuroimmune feedback loop in PN involves neuropeptides released from nerve fibres that cause vasodilation and further recruitment of inflammatory cells. Inflammatory cells, particularly mast cells and eosinophils, degranulate and release neurotoxins, as well as nerve growth factor, which may contribute to the neuronal hyperplasia seen in the dermis of patients with PN and neural sensitization. Recent studies have also indicated underlying genetic susceptibility to PN in addition to environmental factors, the existence of various disease endotypes centred around degrees of type 2 inflammation or underlying myelopathy or spinal disc disease, and significant race and ethnicity-based differences, with African Americans having densely fibrotic skin lesions. Dupilumab became the first US Food and Drug Administration-approved therapeutic for PN, and there are several other agents currently in development. The anti-IL-31 receptor A inhibitor nemolizumab is in late-stage development with positive phase III data reported. In addition, the oral Janus kinase (JAK) 1 inhibitors, abrocitinib and povorcitinib, are in phase II trials while a topical JAK1/2 inhibitor, ruxolitinib, is in phase III studies., Competing Interests: Conflicts of interest S.G.K. is an advisory board member/consultant for AbbVie, Amgen, Arcutis Biotherapeutics, Aslan Pharmaceuticals, Cara Therapeutics, Castle Biosciences, Celldex Therapeutics, Galderma, Genzada Pharmaceuticals, Incyte Corporation, Johnson & Johnson, LEO Pharma, Novartis Pharmaceuticals Corporation, Pfizer, Regeneron Pharmaceuticals and Sanofi, and has served as an investigator for Galderma, Incyte, Pfizer and Sanofi. Specifically to prurigo nodularis, S.G.K. has received honoraria for serving as a consultant and advisory board member for the following relevant conflicts of interest: Sanofi-Regeneron, which has developed dupilumab for the treatment of prurigo nodularis (PN); Galderma, which is developing nemolizumab for the treatment of PN; Incyte, which is developing topical ruxolitinib and povorcitinib for the treatment of PN; and Celldex therapeutics, which is developing barzolvolimab for PN., (© The Author(s) 2024. Published by Oxford University Press on behalf of British Association of Dermatologists. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com.)
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- 2024
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32. Immune stimulus exposure as a trigger for the development of chronic pruritus and circulating blood type 2 inflammation.
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Manjunath J, Liao V, Kambala A, Bao A, Kollhoff AL, Ma EZ, Imo BU, Cornman H, Reddy SV, Lee KK, Lu W, Yossef SM, Kwatra MM, and Kwatra SG
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Background: Chronic pruritus (CP) is a poorly characterized condition associated with intense pruritus without a primary skin eruption. This condition tends to emerge more commonly in older adults, and there is limited research on triggering factors., Objective: To explore the clinical characteristics and pathophysiology of CP following exposure to an immune stimulus., Methods: Clinical characteristics and plasma samples were collected from 15 patients who developed CP following an immune stimulus such as checkpoint inhibitors or vaccination. A multiplex panel was used to analyze plasma cytokine concentrations within these patients., Results: Most immunotherapy-treated patients experienced CP during treatment or after 21 to 60 days of receiving treatment, while vaccine-stimulated patients developed pruritus within a week of vaccination. Plasma cytokine analysis revealed elevated levels of 12 cytokines in patients with immune-stimulated CP compared to healthy controls. Notably, T helper 2 (Th2) related cytokines interleukin (IL)-5 (fold change 2.65; q < 0.25) and thymic stromal lymphopoietin (fold change 1.61 q < 0.25) were upregulated., Limitations: Limitations of this study include limited sample size, particularly in the plasma cytokine assay., Conclusions and Relevance: This study reveals triggers of CP development and describes alterations in blood Th2 markers in patients with CP, including IgE, increased blood eosinophils, and cytokines IL-5 and thymic stromal lymphopoietin., Competing Interests: Dr Shawn Kwatra is an advisory board member or consultant for AbbVie, Celldex Therapeutics, Incyte Corporation, Galderma, Pfizer Inc, Regeneron Pharmaceuticals, and Menlo Therapeutics; and is a recipient of a Dermatology Foundation Medical Dermatology Career Development Award., (© 2024 by the American Academy of Dermatology, Inc. Published by Elsevier Inc.)
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- 2024
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33. Association Between Hematoma Volume and Risk of Subsequent Ischemic Stroke: A MISTIE III and ATACH-2 Analysis.
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Harris W, Kaiser JH, Liao V, Avadhani R, Iadecola C, Falcone GJ, Sheth KN, Qureshi AI, Goldstein JN, Awad IA, Hanley DF, Kamel H, Ziai WC, and Murthy SB
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- Female, Humans, Male, Middle Aged, Antihypertensive Agents, Cerebral Hemorrhage diagnostic imaging, Cerebral Hemorrhage epidemiology, Cerebral Hemorrhage complications, Hematoma diagnostic imaging, Hematoma epidemiology, Hematoma complications, Tissue Plasminogen Activator therapeutic use, Treatment Outcome, Ischemic Stroke complications, Stroke diagnostic imaging, Stroke epidemiology, Stroke etiology
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Background: Nontraumatic intracerebral hemorrhage (ICH) is independently associated with a long-term increased risk of major arterial ischemic events. While the relationship between ICH location and ischemic risk has been studied, whether hematoma volume influences this risk is poorly understood., Methods: We pooled individual patient data from the MISTIE III (Minimally Invasive Surgery Plus Alteplase for Intracerebral Hemorrhage Evacuation Phase 3) and the ATACH-2 (Antihypertensive Treatment of Acute Cerebral Hemorrhage-2) trials. The exposure was hematoma volume, treated as a continuous measure in the primary analysis, and dichotomized by the median in the secondary analyses. The outcome was a symptomatic, clinically overt ischemic stroke, adjudicated centrally within each trial. We evaluated the association between hematoma volume and the risk of an ischemic stroke using Cox regression analyses after adjustment for demographics, vascular comorbidities, and ICH characteristics., Results: Of 1470 patients with ICH, the mean age was 61.7 (SD, 12.8) years, and 574 (38.3%) were female. The median hematoma volume was 17.3 mL (interquartile range, 7.2-35.7). During a median follow-up of 107 days (interquartile range, 91-140), a total of 30 ischemic strokes occurred, of which 22 were in patients with a median ICH volume of ≥17.3 mL and a cumulative incidence of 4.6% (95% CI, 3.1-7.1). Among patients with a median ICH volume <17.3 mL, there were 8 ischemic strokes with a cumulative incidence of 3.1% (95% CI, 1.7-6.0). In primary analyses using adjusted Cox regression models, ICH volume was associated with an increased risk of ischemic stroke (hazard ratio, 1.02 per mL increase [95% CI, 1.01-1.04]). In secondary analyses, ICH volume of ≥17.3 mL was associated with an increased risk of ischemic stroke (hazard ratio, 2.5 [95% CI, 1.1-7.2]), compared with those with an ICH volume <17.3 mL., Conclusions: In a heterogeneous cohort of patients with ICH, initial hematoma volume was associated with a heightened short-term risk of ischemic stroke., Competing Interests: Disclosures Dr Harris reports stock holdings in Eli LIlly and stock holdings in GlaxoSmithKline. Dr Iadecola has served on the scientific advisory board for Broadview Ventures. Dr Falcone has received grants from the National Institutes of Health (NIH) and the American Heart Association outside of the submitted work. Dr Sheth reports compensation from Cerevasc for consultant services; service as President for Advanced Innovation in Medicine; a patent pending for Stroke wearables licensed to Alva Health. Dr Goldstein reports compensation from Cayuga for consultant services; compensation from Prothya for consultant services; compensation from AstraZeneca for consultant services; and compensation from Lumosa for consultant services. Dr Qureshi reports compensation from Chiesi Farmaceutici for consultant services and compensation from Chiesi USA, Inc, for consultant services. Dr Awad reports compensation from Medicoegal consulting for expert witness services. Dr Hanley reports grants from US Department of Defense; grants from National Institute of Neurological Disorders and Stroke (NINDS); grants from NINDS to other; grants from NINDS; compensation from Neurelis, Inc, for consultant services; grants from National Center for Advancing Translational Sciences; grants from NINDS; gifts from Jeffrey and Harriet Legum Professorship in Acute Neurological Medicine at Johns Hopkins University. Dr Kamel is a co-principal investigator for the ARCADIA trial ([Atrial Cardiopathy and Antithrombotic Drugs in Prevention After Cryptogenic Stroke]; NIH/NINDS U01NS095869), which received in-kind study drug from the Bristol Myers Squibb-Pfizer Alliance for Eliquis and ancillary study support from Roche Diagnostics; other funding from NIH (R01HL144541, R01NS123576, and U01NS106513); Deputy Editor for JAMA Neurology; clinical trial steering/executive committees for Medtronic, Janssen, and Javelin Medical; end point adjudication committees for AstraZeneca, Novo Nordisk, and Boehringer Ingelheim; and household ownership interests in TETMedical, Spectrum Plastics Group, and Burke Porter Group. Dr Ziai has received grants from the NIH, serves as the Associate Editor of Neurocritical Care. Dr Murthy has received grant support from the NIH. The other authors report no conflicts.
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- 2024
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34. Cognitive impairment and liver fibrosis in non-alcoholic fatty liver disease.
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Parikh NS, Wahbeh F, Tapia C, Ianelli M, Liao V, Jaywant A, Kamel H, Kumar S, and Iadecola C
- Abstract
Background: Data regarding the prevalence and phenotype of cognitive impairment in non-alcoholic fatty liver disease (NAFLD) are limited., Objective: We assessed the prevalence and nature of cognitive deficits in people with NAFLD and assessed whether liver fibrosis, an important determinant of outcomes in NAFLD, is associated with worse cognitive performance., Methods: We performed a prospective cross-sectional study. Patients with NAFLD underwent liver fibrosis assessment with transient elastography and the following assessments: Cognitive Change Index, Eight-Item Informant Interview to Differentiate Aging and Dementia Questionnaire (AD8), Montreal Cognitive Assessment (MoCA), EncephalApp minimal hepatic encephalopathy test and a limited National Institutes of Health Toolbox battery (Flanker Inhibitory Control and Attention Test, Pattern Comparison Test and Auditory Verbal Learning Test). We used multiple linear regression models to examine the association between liver fibrosis and cognitive measures while adjusting for relevant covariates., Results: We included 69 participants with mean age 50.4 years (SD 14.4); 62% were women. The median liver stiffness was 5.0 kilopascals (IQR 4.0-6.9), and 25% had liver fibrosis (≥7.0 kilopascals). Cognitive deficits were common in people with NAFLD; 41% had subjective cognitive impairment, 13% had an AD8 >2, 32% had MoCA <26 and 12% had encephalopathy detected on the EncephalApp test. In adjusted models, people with liver fibrosis had modestly worse performance only on the Flanker Inhibitory Control and Attention Task (β=-0.3; 95% CI -0.6 to -0.1)., Conclusion: Cognitive deficits are common in people with NAFLD, among whom liver fibrosis was modestly associated with worse inhibitory control and attention., Competing Interests: Competing interests: NSP has unrelated research support from the Florence Gould Endowment for Discovery in Stroke, has received personal fees for medicolegal consulting, and provides blinded endpoint assessment for the Embolization of the Middle Meningeal Artery With ONYX Liquid Embolic System for Subacute and Chronic Subdural Hematoma trial (Medtronic) for participants enrolled at his institution. HK reports serving as PI for the ARCADIA trial (NIH/NINDS U01NS095869), which receives in-kind study drug from the BMS-Pfizer Alliance for Eliquis and ancillary study support from Roche Diagnostics, and other funding from NIH (R01HL144541, R01NS123576, U01NS106513); Deputy Editor for JAMA Neurology; clinical trial steering/executive committees for Medtronic, Janssen and Javelin Medical; endpoint adjudication committees for AstraZeneca, Novo Nordisk and Boehringer Ingelheim; and household ownership interests in TETMedical, Spectrum Plastics Group and Burke Porter Group., (© Author(s) (or their employer(s)) 2024. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.)
- Published
- 2024
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35. Early-life long-term ibuprofen exposure reduces reproductive capacity involved in spermatogenesis impairment and associated with the transcription factor DAF-5 in Caenorhabditis elegans.
- Author
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Yu CW, Yen PL, How CM, Kuo YH, and Hsiu-Chuan Liao V
- Subjects
- Animals, Male, Female, Semen, Reproduction, Spermatogenesis, Transcription Factors, Caenorhabditis elegans, Ibuprofen toxicity
- Abstract
Pharmaceuticals and personal care products (PPCPs) are emerging environmental contaminants and have raised significant concern due to their potential adverse impact on the environment. Ibuprofen is one of the most extensively used non-steroidal anti-inflammatory drugs (NSAIDs) and is also considered an environmental contaminant. The negative impact of ibuprofen on non-target organisms has been documented; however, the molecular mechanisms behind its reproductive toxicity remain unclear. We investigated the impact of early-life long-term ibuprofen exposure on reproductive capacity and its involvement of spermiogenesis in the non-target model organism Caenorhabditis elegans. Hermaphrodites were exposed to various ibuprofen concentrations (0.1, 1, 10, and 100 mg/L), resulting in a dose-dependent inhibition of reproduction. In addition, the lowest observed adverse effect concentration (LOAEC) for ibuprofen exposure on the total brood size of C. elegans was 0.1 mg/L, a concentration that falls within the environmentally relevant range for ibuprofen. Outcross progeny assays revealed a significant 47% reduction in total brood size for larval males (him-5) exposed to ibuprofen, while females (fog-2) exhibited only a minor effect. We found that early-life long-term ibuprofen exposure impairs spermatogenesis. The number of mitotic cells significantly reduced by 31%. The rate of sperm malformation in exposed males was 63%, much higher than in unexposed males (11%). Additionally, the percentage of sperm activation decreased from 89% to 39% in ibuprofen-exposed worms. Mechanistic insights indicated that ibuprofen downregulated mRNA levels of genes related to spermatogenesis and DAF-7/TGF-β signaling. RNAi assays provided evidence for the crucial role of the transcription factor DAF-5 in mediating the spermatogenesis impairment by ibuprofen. Our study provides insight into the environmental impacts of pharmaceutical contaminants, such as ibuprofen, on both male and female reproductive systems to safeguard environmental health., Competing Interests: Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper., (Copyright © 2023 Elsevier Ltd. All rights reserved.)
- Published
- 2024
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36. Plasma-Enabled Ligand Removal for Improved Catalysis: Furfural Conversion on Pd/SiO 2 .
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Nguyen DK, Vargheese V, Liao V, Dimitrakellis P, Sourav S, Zheng W, and Vlachos DG
- Abstract
A nonthermal, atmospheric He/O
2 plasma (NTAP) successfully removed polyvinylpyrrolidone (PVP) from Pd cubic nanoparticles supported on SiO2 quickly and controllably. Transmission electron microscopy (TEM) revealed that the shape and size of Pd nanoparticles remain intact during plasma treatment, unlike mild calcination, which causes sintering and polycrystallinity. Using Fourier transform infrared (FTIR) spectroscopy and X-ray photoelectron spectroscopy (XPS), we demonstrate the quantitative estimation of the PVP plasma removal rate and control of the nanoparticle synthesis. First-principles calculations of the XPS and CO FTIR spectra elucidate electron transfer from the ligand to the metal and allow for estimates of ligand coverages. Reactivity testing indicated that PVP surface crowding inhibits furfural conversion but does not alter furfural selectivity. Overall, the data demonstrate NTAP as a more efficient method than traditional calcination for organic ligand removal in nanoparticle synthesis.- Published
- 2023
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37. Modulation of Neuroimmune and Epithelial Dysregulation in Patients With Moderate to Severe Prurigo Nodularis Treated With Nemolizumab.
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Deng J, Liao V, Parthasarathy V, Cornman HL, Kambala A, Kwatra MM, Ständer S, Piketty C, Chaskar P, Krishnaswamy JK, Julia V, and Kwatra SG
- Subjects
- Adult, Male, Humans, Female, Middle Aged, Cohort Studies, Pruritus etiology, Pruritus complications, Biomarkers, Prurigo drug therapy, Prurigo complications
- Abstract
Importance: Prurigo nodularis (PN) is a debilitating skin disease characterized by intense pruritus and hyperkeratotic skin nodules. Nemolizumab, a monoclonal antibody targeting interleukin 31 receptor α, is a promising novel therapy for the treatment of moderate to severe PN. The biological mechanisms by which nemolizumab promotes improvement of itch and skin lesions in PN are unknown., Objective: To characterize changes in plasma protein biomarkers associated with clinical response to nemolizumab in patients with PN., Design, Setting, and Participants: This multicenter cohort study included patients recruited from Austria, France, Germany, Poland, and the US from a phase 2 clinical trial. Adults diagnosed with moderate to severe PN with severe pruritus for at least 6 months were included in the original trial. Patients in the nemolizumab group were included in the present study if they achieved at least a 4-point decrease in the Peak Pruritus Numerical Rating Scale (PP-NRS) from baseline to week 12 during nemolizumab treatment. Placebo controls did not experience a 4-point decrease in PP-NRS. Mass spectrometry with tandem mass tags to enrich skin-specific protein detection was used to characterize changes in plasma protein expression in nemolizumab and placebo groups. Data were collected from November 2, 2017, to September 26, 2018, and analyzed from December 6, 2019, to April 8, 2022., Intervention: As part of the clinical trial, patients were treated with 3 doses of nemolizumab or placebo at 0, 4, and 8 weeks., Main Outcomes and Measures: Changes in plasma and epidermal protein expression in nemolizumab-treated patients compared with the placebo group at 0, 4, and 12 weeks., Results: Among the 38 patients included in the analysis (22 women and 16 men; mean [SD] age, 55.8 [15.8] years), enrichment analysis of canonical pathways, biological functions, and upstream regulators showed downregulation of terms involving inflammation (IL-6, acute-phase response, signal transducer and activator of transcription 3, and interferon γ), neural processes (synaptogenesis signaling and neuritogenesis), tissue remodeling and fibrosis (transforming growth factor β1 and endothelin-1), and epidermal differentiation (epithelial mesenchymal transition) in the plasma of nemolizumab group., Conclusions and Relevance: In this cohort study, differences between nemolizumab and placebo groups included modulation of inflammatory signaling, neural development, and epithelial differentiation, suggesting a promising potential approach for clinical management of PN.
- Published
- 2023
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38. Larval dispersal of pearl oysters Pinctada margaritifera in the Gambier Islands (French Polynesia) and exploring options for adult restocking using in situ data and numerical modelling.
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Bruyère O, Chauveau M, Le Gendre R, Liao V, and Andréfouët S
- Subjects
- Animals, Larva, Islands, Polynesia, Aquaculture, Pinctada
- Abstract
Black pearl farming is the second source of French Polynesia income after tourism, and Gambier Islands are the main farming sites. Gambier main lagoon contains several sub-lagoons critical for pearl oyster rearing and spat collecting (SC). The Rikitea lagoon, traditionally had good SC rates in the warm season which ensured steady supplies of oysters for black pearl production. However, since 2018, SC has abruptly decreased. To assess the factors affecting SC, Gambier lagoon hydrodynamics was investigated in 2019-2020 to calibrate a hydrodynamic model and simulate larval dispersal around the SC areas. The model shows the strong wind influence on larval dispersal and accumulation patterns and suggests that windy months in the warm season as it can occur during La Niña episodes can explain recent poor SC. Larval dispersal scenarios also informed on best locations to perform adult oyster restocking, a practice that can also enhance SC on the long term., Competing Interests: Declaration of competing interest The authors have no conflict of interests., (Copyright © 2023 Elsevier Ltd. All rights reserved.)
- Published
- 2023
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39. Robust Losses for Learning Value Functions.
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Patterson A, Liao V, and White M
- Abstract
Most value function learning algorithms in reinforcement learning are based on the mean squared (projected) Bellman error. However, squared errors are known to be sensitive to outliers, both skewing the solution of the objective and resulting in high-magnitude and high-variance gradients. To control these high-magnitude updates, typical strategies in RL involve clipping gradients, clipping rewards, rescaling rewards, or clipping errors. While these strategies appear to be related to robust losses-like the Huber loss-they are built on semi-gradient update rules which do not minimize a known loss. In this work, we build on recent insights reformulating squared Bellman errors as a saddlepoint optimization problem and propose a saddlepoint reformulation for a Huber Bellman error and Absolute Bellman error. We start from a formalization of robust losses, then derive sound gradient-based approaches to minimize these losses in both the online off-policy prediction and control settings. We characterize the solutions of the robust losses, providing insight into the problem settings where the robust losses define notably better solutions than the mean squared Bellman error. Finally, we show that the resulting gradient-based algorithms are more stable, for both prediction and control, with less sensitivity to meta-parameters.
- Published
- 2023
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40. Deducing subnanometer cluster size and shape distributions of heterogeneous supported catalysts.
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Liao V, Cohen M, Wang Y, and Vlachos DG
- Abstract
Infrared (IR) spectra of adsorbate vibrational modes are sensitive to adsorbate/metal interactions, accurate, and easily obtainable in-situ or operando. While they are the gold standards for characterizing single-crystals and large nanoparticles, analogous spectra for highly dispersed heterogeneous catalysts consisting of single-atoms and ultra-small clusters are lacking. Here, we combine data-based approaches with physics-driven surrogate models to generate synthetic IR spectra from first-principles. We bypass the vast combinatorial space of clusters by determining viable, low-energy structures using machine-learned Hamiltonians, genetic algorithm optimization, and grand canonical Monte Carlo calculations. We obtain first-principles vibrations on this tractable ensemble and generate single-cluster primary spectra analogous to pure component gas-phase IR spectra. With such spectra as standards, we predict cluster size distributions from computational and experimental data, demonstrated in the case of CO adsorption on Pd/CeO
2 (111) catalysts, and quantify uncertainty using Bayesian Inference. We discuss extensions for characterizing complex materials towards closing the materials gap., (© 2023. The Author(s).)- Published
- 2023
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41. The International Classification of Diseases, 10 th Edition, Clinical Modification (ICD-10-CM) Code I16.0 Accurately Identifies Patients with Hypertensive Urgency.
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Kaiser J, Liao V, Kamel H, Ng C, Lappin RI, and Liberman AL
- Abstract
Objective: Hypertensive urgency, defined as acutely elevated BP without target organ damage, is associated with an increased risk of adverse cardiovascular events and accounts for a substantial proportion of national emergency department (ED) visits. To advance research in this space, we sought to validate the new ICD-10-CM diagnostic code for hypertensive urgency within a single healthcare system., Methods: We performed a retrospective chart-review study of ED encounters at Weill Cornell Medicine from 2016 â€" 2021. We randomly selected 25 encounters with the ICD-10-CM code I16.0 as the primary discharge diagnosis and 25 encounters with primary ICD-10-CM discharge diagnosis codes for benign headache disorders. A single board-certified vascular neurologist reviewed all 50 encounters while blinded to the assigned ICD-10-CM codes to identify cases of hypertensive urgency. We calculated the sensitivity, specificity, and positive predictive values of the ICD-10-CM code I16.0 with 95% confidence intervals (CI)., Results: Out of 50 randomly selected ED encounters, 24 were adjudicated as hypertensive urgency. All encounters adjudicated as hypertensive urgency had been assigned the ICD-10-CM discharge diagnosis code of I16.0. All 25 of the encounters adjudicated as headache were assigned an ICD-10-CM discharge diagnosis code for a benign headache disorder. The ICD-10-CM code for hypertensive urgency, I16.0, was thus found to have a sensitivity of 100% (95% CI: 86-100%), specificity of 96% (95% CI: 80-100%), and positive predictive value of 96% (95% CI: 78-99%)., Conclusion: We found that the new ICD-10-CM code for hypertensive urgency, I16.0, can reliably identify patients with this condition.
- Published
- 2023
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42. Natural stocks of Pinctada margaritifera pearl oysters in Tuamotu and Gambier lagoons: New assessments, temporal evolutions, and consequences for the French Polynesia pearl farming industry.
- Author
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Bionaz O, Le Gendre R, Liao V, and Andréfouët S
- Subjects
- Agriculture, Animals, Aquaculture, Polynesia, Water, Ostreidae, Pinctada
- Abstract
Knowledge of the status of the black-lipped pearl oyster Pinctada margaritifera populations is critical for the sustainability of the French Polynesia black pearl farming industry. Indeed, this activity relies on collection of spat settling out of the water column, which is inherently related to the abundance of in situ benthic stocks and their reproductive capacities. From surveys performed between 2016 and 2021, we present new stock assessments of natural wild oyster populations from four contrasted pearl farming lagoons (Gambier, Takapoto, Raroia, Takume). Also using Ahe atoll historical data, we first highlight how results vary with the methods (Direct, Zonal and Cokriging) used to scale in situ density measurements to lagoon-scale stocks. Takapoto and Gambier populations were also previously surveyed at least twice since the 1980s, allowing to demonstrate with field data clear changes in stock distributions and population structures. The consequences of our findings are discussed to provide recommendations for stock monitoring and management in the future., Competing Interests: Declaration of competing interest The authors have no conflict of interests., (Copyright © 2022 Elsevier Ltd. All rights reserved.)
- Published
- 2022
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43. Characteristics of Patients with Unrecognized Sleep Apnea Requiring Postoperative Oxygen Therapy.
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Seet E, Waseem R, Chan MTV, Wang CY, Liao V, Suen C, and Chung F
- Abstract
Surgical patients with obstructive sleep apnea (OSA) have increased risk of perioperative complications. The primary objective is to determine the characteristics of surgical patients with unrecognized OSA requiring oxygen therapy for postoperative hypoxemia. The secondary objective is to investigate the characteristics of patients who were responsive to oxygen therapy. This was a post-hoc multicenter study involving patients with cardiovascular risk factors undergoing major non-cardiac surgery. Patients ≥45 years old underwent Type 3 sleep apnea testing and nocturnal oximetry preoperatively. Responders to oxygen therapy were defined as individuals with ≥50% reduction in oxygen desaturation index (ODI) on postoperative night 1 versus preoperative ODI. In total, 624 out of 823 patients with unrecognized OSA required oxygen therapy. These were mostly males, had larger neck circumferences, higher Revised Cardiac Risk Indices, higher STOP-Bang scores, and higher ASA physical status, undergoing intraperitoneal or vascular surgery. Multivariable regression analysis showed that the preoperative longer cumulative time SpO2 < 90% or CT90% (adjusted p = 0.03), and lower average overnight SpO2 (adjusted p < 0.001), were independently associated with patients requiring oxygen therapy. Seventy percent of patients were responders to oxygen therapy with ≥50% ODI reduction. Preoperative ODI (19.0 ± 12.9 vs. 14.1 ± 11.4 events/h, p < 0.001), CT90% (42.3 ± 66.2 vs. 31.1 ± 57.0 min, p = 0.038), and CT80% (7.1 ± 22.6 vs. 3.6 ± 8.7 min, p = 0.007) were significantly higher in the responder than the non-responder. Patients with unrecognized OSA requiring postoperative oxygen therapy were males with larger neck circumferences and higher STOP-Bang scores. Those responding to oxygen therapy were likely to have severe OSA and worse preoperative nocturnal hypoxemia. Preoperative overnight oximetry parameters may help in stratifying patients.
- Published
- 2022
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44. Plicidentine in the oral fangs of parrotfish (Scarinae, Labriformes).
- Author
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Viviani J, LeBlanc A, Rurua V, Mou T, Liao V, Lecchini D, Galzin R, and Viriot L
- Subjects
- Animals, Biological Evolution, Calcium Carbonate, Perciformes anatomy & histology, Tooth anatomy & histology
- Abstract
Parrotfish play important ecological roles in coral reef and seagrass communities across the globe. Their dentition is a fascinating object of study from an anatomical, functional and evolutionary point of view. Several species maintained non-interlocked dentition and browse on fleshy algae, while others evolved a characteristic beak-like structure made of a mass of coalesced teeth that they use to scrape or excavate food off hard limestone substrates. While parrotfish use their highly specialized marginal teeth to procure their food, they can also develop a series of large fangs that protrude from the upper jaw, and more rarely from the lower jaw. These peculiar fangs do not participate in the marginal dentition and their function remains unclear. Here we describe the morphology of these fangs and their developmental relationship to the rest of the oral dentition in the marbled parrotfish (Leptoscarus vaigiensis), the star-eye parrotfish (Calotomus carolinus), and the palenose parrotfish (Scarus psittacus). Through microtomographic and histological analyses, we show that some of these fangs display loosely folded plicidentine along their bases, a feature that has never been reported in parrotfish. Plicidentine is absent from the marginal teeth and is therefore exclusive to the fangs. Parrotfish fangs develop a particular type of simplexodont plicidentine with a pulpal infilling of alveolar bone at later stages of dental ontogeny. The occurrence of plicidentine and evidence of extensive tooth wear, and even breakage, lead us to conclude that the fangs undergo frequent mechanical stress, despite not being used to acquire food. This strong mechanical stress undergone by fangs could be linked either to forced contact with congeners or with the limestone substrate during feeding. Finally, we hypothesize that the presence of plicidentine in parrotfish is not derived from a labrid ancestor, but is probably a recently evolved trait in some parrotfish taxa, which may even have evolved convergently within this subfamily., (© 2022 Anatomical Society.)
- Published
- 2022
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45. Chronic exposure to environmentally relevant levels of di(2-ethylhexyl) phthalate (DEHP) disrupts lipid metabolism associated with SBP-1/SREBP and ER stress in C. elegans.
- Author
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How CM and Hsiu-Chuan Liao V
- Subjects
- Animals, Caenorhabditis elegans drug effects, Caenorhabditis elegans metabolism, Fatty Acids metabolism, Humans, Lipid Metabolism drug effects, Caenorhabditis elegans Proteins metabolism, Diethylhexyl Phthalate toxicity, Endoplasmic Reticulum Stress drug effects, Sterol Regulatory Element Binding Proteins metabolism, Transcription Factors metabolism
- Abstract
DEHP is commonly found in the environment, biota, food, and humans, raising significant health concerns. Whether developmental stage and exposure duration modify the obesogenic effects of DEHP is unclear, especially the underlying mechanisms by which chronic exposure to DEHP as well as its metabolites remain largely unknown. This study investigated the obesogenic effects of chronic DEHP exposure, with levels below environmentally-relevant amounts and provide the mechanism in Caenorhabditis elegans. We show that early-life DEHP exposure resulted in an increased lipid and triglyceride (TG) accumulation mainly attributed to DEHP itself, not its metabolite mono-2-ethylhexyl phthalate (MEHP). In addition, developmental stage and exposure timing influence DEHP-induced TG accumulation and chronic DEHP exposure resulted in the most significant effect. Analysis of fatty acid composition shows that chronic DEHP exposure altered fatty acid composition and TG, resulting in an increased ω-6/ω-3 ratio. The increased TG content by chronic DEHP exposure required lipogenic genes fat-6, fat-7, pod-2, fasn-1, and sbp-1. Moreover, chronic DEHP exposure induced XBP-1-mediated endoplasmic reticulum (ER) stress which might lead to up-regulation of sbp-1. This study suggests the possible involvement of ER stress and SBP-1/SREBP-mediated lipogenesis in chronic DEHP-induced obesogenic effects. Results from this study implies that chronic exposure to DEHP disrupts lipid metabolism, which is likely conserved across species due to evolutionary conservation of molecular mechanisms, raising concerns in ecological and human health., (Copyright © 2022 Elsevier Ltd. All rights reserved.)
- Published
- 2022
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46. Periodicity of wave-driven flows and lagoon water renewal for 74 Central Pacific Ocean atolls.
- Author
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Andréfouët S, Desclaux T, Buttin J, Jullien S, Aucan J, Le Gendre R, and Liao V
- Subjects
- Agriculture, Animals, Pacific Ocean, Polynesia, Water, Aquaculture, Pinctada
- Abstract
French Polynesia atolls are spread on a vast 2300 by 1200 km Central Pacific Ocean area exposed to spatially and temporally dependent wave forcing. They also have a wide range of closed to open morphologies and several have been suitable to develop from black-lipped pearl oysters a substantial pearl farming activity in the past 30 years, representing nowadays the 2nd source of income for French Polynesia. Considering here only the component of lagoon renewal that is driven by waves, we investigate for 74 atolls different lagoon renewal metrics using 20 years of wave model data at 0.05° spatial resolution. Wavelet spectral analyses highlight that atolls, even in close vicinity, can be exposed to different and characteristic periodicities in wave-driven flows and water renewal. These characteristics are discussed in relation to pearl farming atolls, including atolls known to be efficient oyster spat producers, a critical activity for pearl farming sustainability., (Copyright © 2022 Elsevier Ltd. All rights reserved.)
- Published
- 2022
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47. The MANA (MANagement of Atolls, 2017-2022) project for pearl oyster aquaculture management in the Central Pacific Ocean using modelling approaches: Overview of first results.
- Author
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Andréfouët S, Lo-Yat A, Lefebvre S, Bionaz O, and Liao V
- Subjects
- Animals, Aquaculture methods, Pacific Ocean, Polynesia, Pinctada
- Abstract
This editorial presents results of the MANA (MANagement of Atolls) project compiled in the form of a Marine Pollution Bulletin collection of 14 articles. MANA is a project funded by the French Agence National pour la Recherche that specifically addresses the development of knowledge and management tools for pearl farming atolls, with a focus on the spat collecting activity in French Polynesia. The 14 papers cover the range of thematic tasks described in the initial project, including atoll geomorphology and bathymetry, climate forcing, atoll lagoon and rim hydrodynamics, typology of atolls, evaluation of remote sensing data for monitoring atoll lagoons, and development of numerical models and spatially-explicit tools that altogether have contributed to the applied objectives. In addition, this editorial draws an update on the pearl farming industry in French Polynesia with the latest statistics, and discusses the next targeted priorities for research programs focusing on pearl farming atolls., (Copyright © 2022 Elsevier Ltd. All rights reserved.)
- Published
- 2022
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48. A systematic prioritization approach for identifying suitable pearl oyster restocking zones following a mass mortality event in Takaroa Atoll, French Polynesia.
- Author
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André LV, Chinain M, Gatti CMI, Liao V, Van Wynsberge S, Tedesco P, and Andréfouët S
- Subjects
- Agriculture, Animals, Aquaculture, Ecosystem, Hydrodynamics, Polynesia, Pinctada
- Abstract
Oyster farming for black pearl production is central in French Polynesia. It is the second source of national income and provides substantial job opportunities, notably in remote atolls. However, this sector has been undermined by successive crises, such as mass-mortalities of wild and farmed oyster stocks that have impacted entire lagoons. An option to revive the activity consists of reintroducing oysters in strategic benthic locations selected to maximize reproduction and dispersal of larvae throughout the lagoon, hence promoting recolonization and spat collection for farming. For Takaroa, a Tuamotu atoll recently impacted by mortalities, a systematic prioritization approach identified these restocking sites, using environmental and socio-economic criteria such as: location of suitable habitats for oyster settlement, larval connectivity estimated from hydrodynamic circulation model, farming waste accumulation, and opportunity cost to fishers and farmers who lose access to restocking areas. This approach provides managers with a portfolio of restocking options., (Copyright © 2022 Elsevier Ltd. All rights reserved.)
- Published
- 2022
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49. Tide and wave driven flow across the rim reef of the atoll of Raroia (Tuamotu, French Polynesia).
- Author
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Aucan J, Desclaux T, Le Gendre R, Liao V, and Andréfouët S
- Subjects
- Physical Phenomena, Polynesia, Coral Reefs
- Abstract
The currents flowing across the rim of the atoll of Raroia were investigated with a 1 year-long dataset of wave, water level and currents. Offshore waves break on the edge of the reef outside the atoll's rim and drive current into the lagoon, through the shallow hoa that cut across the rim. The additional water volume generated by this wave driven flow induces an elevation of water level throughout the atoll's lagoon and is evacuated back into the open ocean through a deep reef pass. The water level inside the atoll is also driven by astronomical tides, which enter the lagoon thought the reef pass, after undergoing a ~50% decrease in amplitude and a ~4 hour lag. Using a simple parametric model with three calibrated coefficients, we show that currents across the atoll's rim can be estimated as a function of the offshore wave conditions and the water level difference between the ocean and the lagoon., (Copyright © 2021 Elsevier Ltd. All rights reserved.)
- Published
- 2021
- Full Text
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50. Maternal cecal microbiota transfer rescues early-life antibiotic-induced enhancement of type 1 diabetes in mice.
- Author
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Zhang XS, Yin YS, Wang J, Battaglia T, Krautkramer K, Li WV, Li J, Brown M, Zhang M, Badri MH, Armstrong AJS, Strauch CM, Wang Z, Nemet I, Altomare N, Devlin JC, He L, Morton JT, Chalk JA, Needles K, Liao V, Mount J, Li H, Ruggles KV, Bonneau RA, Dominguez-Bello MG, Bäckhed F, Hazen SL, and Blaser MJ
- Subjects
- Animals, Autoimmune Diseases, Bacteria classification, Bacteria drug effects, Disease Models, Animal, Female, Gene Expression, Histone Code, Intestines immunology, Male, Metabolic Networks and Pathways, Metagenome, Mice, Mice, Inbred NOD, MicroRNAs, Anti-Bacterial Agents pharmacology, Cecum immunology, Cecum microbiology, Diabetes Mellitus, Type 1 immunology, Gastrointestinal Microbiome drug effects, Gastrointestinal Microbiome physiology
- Abstract
Early-life antibiotic exposure perturbs the intestinal microbiota and accelerates type 1 diabetes (T1D) development in the NOD mouse model. Here, we found that maternal cecal microbiota transfer (CMT) to NOD mice after early-life antibiotic perturbation largely rescued the induced T1D enhancement. Restoration of the intestinal microbiome was significant and persistent, remediating the antibiotic-depleted diversity, relative abundance of particular taxa, and metabolic pathways. CMT also protected against perturbed metabolites and normalized innate and adaptive immune effectors. CMT restored major patterns of ileal microRNA and histone regulation of gene expression. Further experiments suggest a gut-microbiota-regulated T1D protection mechanism centered on Reg3γ, in an innate intestinal immune network involving CD44, TLR2, and Reg3γ. This regulation affects downstream immunological tone, which may lead to protection against tissue-specific T1D injury., Competing Interests: Declaration of interests Z.W. and S.L.H. report being named as co-inventors on pending and issued patents held by the Cleveland Clinic relating to cardiovascular diagnostics and therapeutics and being eligible to receive royalty payments for inventions or discoveries related to cardiovascular diagnostics or therapeutics from Cleveland HeartLab, Quest Diagnostics, and Procter & Gamble. S.L.H. also reports being a paid consultant for Procter & Gamble, having received research funds from Procter & Gamble and Roche Diagnostics, and being eligible to receive royalty payments for inventions or discoveries related to cardiovascular diagnostics or therapeutics from Cleveland HeartLab, Quest Diagnostics, and Procter & Gamble. The other authors report that they have no relationships relevant to the contents of this paper to disclose., (Copyright © 2021 Elsevier Inc. All rights reserved.)
- Published
- 2021
- Full Text
- View/download PDF
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