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2. 1258P MET exon14 skipping in non-small cell lung cancer: Clinicopathological characteristics, treatments, and efficacy of crizotinib according to functional analysis: AFonMET - GFPC study

4. A new case of Kaufman Oculocerebrofacial Syndrome caused by two new splicing variants in UBE3B

5. Analyse FONctionnelle des mutations identifiées au voisinage de l’exon 14 du gène MET et efficacité du crizotinib chez des patients atteints d’un carcinome broncho-pulmonaire non à petites cellules de stade avancé (CBNPC) : étude GFPC-AFonMET

14. SABRINA-RT, a distributed DBMS for telecommunications

15. Traitement par ECMO d'une pneumopathie nécrosante à Staphylococcus aureus producteur de la leucocidine de Panton-Valentine [Extracorporeal circuit for Panton-Valentine leukocidin-producing Staphylococcus aureus necrotizing pneumonia]

19. Nramp2 ANALYSIS IN HEMOCHROMATOSIS PROBANDS

21. Genome-wide association study identifies TF as a significant modifier gene of iron metabolism in HFE hemochromatosis

22. Structural and optical investigations of AlGaN MQWs grown on a relaxed AlGaN buffer on AlN templates for emission at 280nm

24. AlGaN-based MQWs grown on a thick relaxed AlGaN buffer on AlN templates emitting at 285 nm

27. Multicentric, retrospective study evaluating the epidemiologic characteristics and outcomes of patients aged 80years or older with non-small-cell lung cancer (NSCLC) harboring egfr mutations treated by EGFR-TKI

30. Comprehensive functional annotation of 18 missense mutations found in suspected hemochromatosis type 4 patients

31. Extraordinary blueshift of a photonic crystal nanocavity by reducing its mode volume with an opaque microtip

41. AlGaN-based MQWs emitting at 280nm for vertical cavity surface emitting lasers

48. Genome-wide association study identifies TF as a significant modifier gene of iron metabolism in HFE hemochromatosis

49. The dual loss and gain of function of the FPN1 iron exporter results in the ferroportin disease phenotype.

50. Targeted RNAseq from patients' urinary cells to validate pathogenic noncoding variants in autosomal dominant polycystic kidney disease genes: a proof of concept.

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