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134 results on '"Lazarini F"'

9. In vitro migration assays of neural stem cells

Author

Durbec , P., Franceschini , I., Lazarini , F., Dubois-Dalcq , M., Institut de Biologie du Développement de Marseille (IBDM), Aix Marseille Université (AMU)-Centre National de la Recherche Scientifique (CNRS), Institut de Biologie du Développement de Marseille ( IBDM ), and Aix Marseille Université ( AMU ) -Institut National de la Santé et de la Recherche Médicale ( INSERM ) -Centre National de la Recherche Scientifique ( CNRS )

Subjects
[ SDV.BC ] Life Sciences [q-bio]/Cellular Biology, [SDV.BC]Life Sciences [q-bio]/Cellular Biology
Published
2008

11. [Non-conventional infectious agents or PRIONS]

Author

Jj, Hauw, Lazarini F, danielle seilhean, Camilleri S, Mokhtari K, Duyckaerts C, and Hénin D

Subjects
Prions, Animals, Humans, Creutzfeldt-Jakob Syndrome, Prion Diseases
Published
1995

22. Die Kristallstruktur des Hydrazinium-monofluorids.

Author

Golič, L. and Lazarini, F.

Abstract

The hydrazinium monofluoride NHF crystallizes with orthorhombic symmetry, space group P222, a=4.592 Å, b=8.217 Å, c=12.341 Å, and 8 formula units. The structure was determined by the permutation method in projection, and by the three-dimensional Patterson function, refined by Full-Matrix-Least-Squares ( R=0.056). The structure consists of NH- and F-ions, bonded with hydrogen bonds N−H ... N and N−H ... F. Each NH ion is surrounded by four F-ions and two nitrogen atoms of two different NH ions. Each F ion is connected with four different NH ions. [ABSTRACT FROM AUTHOR]

Published
1974
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24. Crystal structure of triiodobismuth(III)-tri-μ-iodo-tris(triphenylarsinoxide)-bismuth(III), C54H45As3Bi2I6O3

Author

Lazarini, F., Golič, L., and Pelizzi, G.

Abstract

The crystals of the title compound are triclinic, with unit-cell dimensionsa =14.804(7),b =18.657(9),c = 22.955(13) Å, a = 90.2(1), ß 106.6(1), ? = 91.0(1) °, space groupP¯1, andZ = 4. The crystal structure has been determined from x-ray diffractometer data by direct methods and Fourier techniques, and refined by least-squares methods using 11,383 independent reflections (I> 2s(I)) to anR value of 0.076. The structure consists of complex molecules with two distorted octahedra sharing a face. The first of the bismuth atoms is coordinated by six iodine atoms, three of them bridging, and the second by three bridging iodine atoms and three oxygen atoms of the triphenylarsinoxides. The orientation of the phenyl rings is the only significant difference between the two complex molecules in the asymmetric unit.

Published
1976
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28. [Diseases transmitted by non-conventional agents ('prions'): nosology and diagnosis]

Author

Jj, Hauw, Lazarini F, Sazdovitch V, danielle seilhean, Suarez S, Ma, Colle, Boularand S, Delasnerie-Lauprêtre N, and Duyckaerts C

Subjects
Diagnosis, Differential, Prions, Humans, Prion Diseases
Abstract

Transmissible non conventional agents are currently called "Prions". This is not a neutral terminology: the attractive Prion hypothesis (the infectious agent being a protein able to replicate in the absence of DNA or RNA) due to Stanley Prusiner is the prevalent one, and has shown to be heuristic, but has not been formally proven and does not easily explain all the data, unless modified and expanded. No simple account has been given for the very unusual physical, chemical, and biological properties of non conventional agents. These infectious agents are associated with degenerative diseases of the nervous system that are either the consequence of a genetic mutation or develop spontaneously in apparently normal individuals, and then can be transmitted to various susceptible hosts, including man. Thus, non conventional agents cannot be considered only as fascinating biological enigmas. They constitute a challenge for public health. The changing characteristics of prion-associated diseases has led to a renewing of their clinical and neuropathological diagnostic criteria. A brief survey of the nosology and neuropathology of prions diseases, with emphasis on new data and on difficulties, is provided. A simple classification based on the familial, sporadic or infectious variety of the disease is suggested. Familial diseases can be named according to the genetic disorder. Sporadic and infectious diseases can be classified following the main clinical symptoms and signs, and the presence or absence of amyloid plaques in the brain, until new tools (analysis of the glycosylation pattern of PrP, strain recognition) allow a more precise nomenclature. The new epidemiology of Prion disorders allowed by these new approaches relies on a full study of Prion diseases affected patients, which necessarily involves their genetic study, and the analysis of brain tissue. This, for practical and ethical reasons, is better achieved by autopsy.

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