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Previous research has shown that “attachment anxiety” is a robust predictor of disinhibited eating behaviours and that this relationship is underpinned by difficulties in managing emotion. Night eating syndrome (NES), a proposed eating disorder characterized by evening hyperphagia, nocturnal awakenings to eat, and morning anorexia, is also associated with eating to manage emotion. Across two studies (N = 276 & N = 486), we considered a relationship between attachment anxiety and NES. In Study 1, we hypothesised (pre-registered) that attachment anxiety would predict NES score and that this relationship would be mediated by disinhibited eating. Participants were asked to complete questionnaire measures of attachment orientation, disinhibited eating (emotional and uncontrolled eating) and NES. Our parallel mediation model confirmed a direct relationship between attachment anxiety and NES (p < .001) and showed an indirect path via both emotional (95% CI: 0.15–0.63) and uncontrolled eating (95% CI: 0.001–0.36). In Study 2, we showed that fear of negative evaluation of eating significantly mediated a reversed relationship between attachment anxiety and NES (95% CI: 0.02–0.04). Finally, across both studies we used a novel tool to assess “eating to cope”. We showed a relationship with emotional eating but failed to show a robust relationship with NES. Attachment orientation may represent a potential intervention target for night eating syndrome. Future research should consider a longitudinal approach to strengthen our understanding of directionality amongst these factors.

Background The Night Eating Diagnostic Questionnaire (NEDQ) is a validated assessment of the night eating syndrome (NES) based on the proposed diagnostic criteria. While past results show that NEDQ is associated with psychopathology and body mass index (BMI), the relationships between the proposed NES diagnostic criteria and psychopathology and BMI have not been investigated. Thus, the relationships between the proposed NES diagnostic criteria and factors previously associated with NES, namely depression, “food addiction” (FA), sleep quality, and BMI were examined. Finally, the NEDQ factor structure was examined for the first time in order to determine whether the NEDQ is assessing NES appropriately. Methods The NEDQ, depression, sleep quality, and FA questionnaires were administered to a sample of older community members (n = 468) and a student sample (n = 254). Principal Components Analysis (PCA) was performed to examine the factor structure of the NEDQ. The relationships between the proposed diagnostic criteria and depression, sleep quality, FA, and BMI were examined using multiple regression. Results The proposed NES criteria were highly intercorrelated. PCA revealed a single factor solution for the NEDQ. In the community sample, depression was associated with the presence of five of nine proposed diagnostic criteria while poor sleep quality and FA were associated with the presence of seven and six criteria respectively. In the community sample, only the sleep problems and impairment/distress criteria were positively associated with BMI. In the student sample, fewer criteria were associated with psychopathology, and no criteria were associated with BMI. Conclusions These findings support the proposed criteria for diagnosis of NES. All proposed criteria were associated with at least one psychopathology measure. BMI was only associated with the sleep problems and impairment/distress criteria in the community sample. The PCA finding of a single factor indicates that the NEDQ is a cohesive assessment of NES, and that the NEDQ is a good measure of NES criteria.

The goal of this chapter is to describe measures of emotions as influences on eating behavior and weight control. We begin with the measurement of eating behavior in response to negative emotions or stress (i.e., emotional eating). Next, we show how the measures of emotions can test theoretical models that underlie emotional eating along with the triggers and relief components of emotional eating. We end this section with an amalgamation of current theoretical and empirical perspectives referred to as the emotional eating cycle, a feed forward loop that generates positive feedback via emotional relief and thereby leads to overeating and weight gain. We then present empirical evidence from research studies that both support and refute these theoretical models and discuss challenges to measurement of emotional eating and related constructs in these contexts, as well as directions for future research. Finally, we address the biological link between theoretical models and measurements of physiological changes induced within the body in response negative emotions or stress, in conjunction with mathematical and statistical model connections to overeating and weight gain, in both clinical and research applications.

Group data means from individuals who self-assess as emotional eaters do not reliably show increased food intake in response to stress or negative emotions. This inconsistency in predictive validity of self-reported emotional eating (EE) could be attributable to unconsidered moderation of the relationship between self-reported EE and behavioral measures of EE. Greater emotional relief from stress by eating may provide enhanced negative reinforcement and promote future EE in response to stress as a form of self-medication. Thus, we predicted that greater emotional relief from stress by eating (decrease in negative affect from stress to post-eating) would moderate the extent to which heightened stress reactivity (measured by systolic blood pressure, SBP) moderates the relationship between self-reported EE and food intake post-stress. We also hypothesized that self-reported EE would not predict greater food consumption on the rest day. 43 undergraduate women completed online assessments of eating behaviors. Participants were given snacks to eat after a mental stress task (TSST) or rest period on separate days in counterbalanced order. Our prediction was supported, as the moderated moderation model (PROCESS model 3) was highly significant on the stress day. Self-reported EE predicted increased food intake post-stress only under conditions of high stress reactivity and high emotional relief. On the rest day, self-reported EE predicted greater snack food intake only when SBP was high. This conditional increased intake substantiates stress as a promoter of snack food consumption for women with greater EE. Overall, our findings identified factors that may distinguish the subset of self-reported emotional eaters who are more likely to display EE behaviors in a laboratory setting, yet further studies are needed to directly test whether negative reinforcement via emotional relief from stress by eating drives enhanced EE following stress.

Food addiction is controversial within the scientific community. However many lay people consider themselves addicted to certain foods. We assessed the prevalence and characteristics of self-perceived “food addiction” and its relationship to a diagnostic measure of “clinical food addiction” in two samples: (1) 658 university students, and (2) 614 adults from an international online crowdsourcing platform. Participants indicated whether they considered themselves to be addicted to food, and then completed the Yale Food Addiction Scale, measures of eating behavior, body image, and explicit and internalized weight stigma. Participants in the community sample additionally completed measures of impulsivity, food cravings, binge eating, and depressive symptomatology. Follow-up data were collected from a subset of 305 students (mean follow-up 280 ± 30 days). Self-perceived “food addiction” was prevalent, and was associated with elevated levels of problematic eating behavior, body image concerns, and psychopathology compared with “non-addicts”, although individuals who also received a positive “diagnosis” on the Yale Food Addiction Scale experienced the most severe symptoms. A clear continuum was evident for all measures despite no differences in body mass index between the three groups. Multinomial logistic regression analyses indicated that perceived lack of self-control around food was the main factor distinguishing between those who did and did not consider themselves addicted to food, whereas severity of food cravings and depressive symptoms were the main discriminating variables between self-classifiers and those receiving a positive “diagnosis” on the Yale Food Addiction Scale. Self-perceived “food addiction” was moderately stable across time, but did not appear predictive of worsening eating pathology. Self-classification as a “food addict” may be of use in identifying individuals in need of assistance with food misuse, loss-of-control eating, and body image issues.

Irrational beliefs (IB) are believed, in cognitive behavioral therapies, to be a prime cause of psychopathologies including anxiety, depression, problem eating, and alcohol misuse. &ldquo, Food addiction&rdquo, (FA), which has been modeled on diagnostic criteria for substance use disorder, and emotional eating (EE) have both been implicated in the rise in overweight and obesity. Both FA and EE are associated with anxiety. Thus, in the present study, the hypothesis that IB is associated with FA and with EE was tested. Furthermore, possible mediation of these relationships by trait anxiety and depression (and EE for IB and FA) was examined. The responses of 239 adult participants to questionnaires measuring FA, IB, EE, depression, trait anxiety, and anthropometrics were recorded. The results revealed that IB was significantly positively correlated with FA and EE (and depression and trait anxiety). Furthermore, only EE mediated the effect of IB on FA and this was not moderated by BMI. Finally, trait anxiety (but not depression) mediated the effect of IB on EE. Exploratory analysis revealed a significant serial mediation such that IB predicted higher FA via elevated trait anxiety and emotional eating in that order. The results of this study suggest that IB may be a source of the anxiety that is associated with EE and FA and suggest that clinicians may find IB a target for treatment of those persons who report experiences of EE and FA. IB may play a role in food misuse that leads to elevated BMI.

Night eating syndrome (NES) and "food addiction" (FA) are associated with elevated body mass index (BMI) and disturbed eating behavior. The present study was conducted to examine whether NES is associated with FA, and whether BMI, depression and sleep quality contribute to any relationship between NES and FA. Two groups were studied: a sample of 254 university students and a sample of 244 older adults. All completed the Yale Food Addiction Scale (YFAS), the Night Eating Questionnaire (NEQ), the Zung Self-report Depression Scale, and the Pittsburgh Sleep Quality Index, and BMI was computed from height and weight. In both samples, higher global NEQ scores were significantly correlated with more FA symptoms, elevated depression, and poorer sleep quality, and these correlations were significantly higher in the older adult sample than in the younger student sample. Higher BMI was significantly correlated with NEQ score only in the older adult sample. The hypothesis that the prediction of NEQ by YFAS was moderated by BMI and group membership (moderated moderation) was tested; while the prediction of NEQ by YFAS was not moderated by BMI, elevated YFAS predicted higher NEQ in the adult sample than it did in the student sample. In addition, multiple regression revealed that "continued use of food despite adverse effects" was the sole FA symptom predictive of NES symptoms in students while in older adults food tolerance was the only predictor of NES. Thus, NES appears to be associated with FA, more strongly in an older community sample; higher food tolerance in NES may contribute to a desire to eat late in the evening and/or when awakening at night.

In the contemporary milieu, the term "addiction" brings to mind issues of physical dependence, uncontrolled behavior, psychoactive substances, and disease. Thus, the use of the term "food addiction" which has become common in research on binge eating and obesity, suggests a disease state characterized by craving, compulsive eating and, possibly, the presence of food constituents with drug-like properties which weaken the will power to abstain from consumption. In this commentary, the case is made that, following the trends in substance use disorder terminology, adoption of "food use disorder" as a term for compulsive eating associated with subjective loss of control may foster continued research in this area without the connotations suggested by "food addiction."

Studies of persons in treatment for substance use disorder (SUD) frequently report significant body weight gain and higher food consumption. It is unclear what psychological variables might mediate a relationship between SUD treatment status and food selection. To examine the association between SUD treatment status and food selection, 212 men and women (104 in treatment for SUD) were asked, in an online survey, to choose from among 16 food images in an imagined all-you-can-eat buffet scenario. Food craving, emotional eating (EE), impulsivity, and drugs used were examined as potential mediators while reward responsiveness (RR) was examined as a potential moderator. The amount of energy from foods selected was the predicted variable. Results indicated that those in treatment for SUD had significantly higher food cravings, positive EE, and attentional and motor impulsivity (but not RR or negative EE) which were (with the exception of RR) not correlated with energy selected from food images. Those in SUD treatment selected more energy (M = 2655.10 kcal, SEM = 156.97) from food images than those in the control group (M = 2221.52 kcal, SEM = 112.29), t(188) = -2.25, p = 0.026. In a multiple regression moderation analysis, SUD treatment status (β = 419.58, p = 0.029) and RR (β = 39.23, p = 0.016) were significant predictors of energy from foods selected. The amount of energy selected from food images predicted by SUD treatment status was dependent on values of RR. Mean BMI was not significantly higher for the SUD group but there were more participants with BMI values over 29 and fewer with BMI between 25 and 29 in the SUD treatment group. While the cause remains unclear, the results show that persons in treatment for SUD and with higher RR do select more energy from food images in a meal scenario.

The results of several behavioral and neurophysiological studies suggest that some obese people respond to foods in a manner that resembles the behavior of drug dependent persons toward drugs. Indeed, there are common mechanisms in the brain responsible for the reinforcement of eating and drug use by food and drugs respectively and for the cue incentives associated with them. There is some evidence that dependence in one modality may influence the other. Active drug use is associated with normal (or even low) body weight but recovery from drug dependence may lead to increased consumption of food and ultimately to weight gain. It is possible that drug abstinence leads to incentive seeking in the food domain, increased food consumption, and weight gain. Thus, those in treatment for substance use disorders may benefit from weight maintenance counseling.

This chapter describes the senior learning community in psychology at Wagner College, which is an example of a discipline-specific curriculum-based learning community (LC). This LC acts both as a capstone to the undergraduate experience and as a transition for students from college to career. We describe the components of the LC, their development, our attempts to integrate them, and how they might be applied in other intradisciplinary LCs.

Night eating syndrome (NES) is commonly assessed using the Night Eating Questionnaire (NEQ), a validated scale of symptom severity, which does not assess all diagnostic criteria. The Night Eating Diagnostic Questionnaire (NEDQ) assesses all diagnostic criteria, but has not been fully validated. The study purpose was to establish convergent validity for the NEDQ with the NEQ. It was also expected that higher NEDQ scores would be associated with elevated depression, poorer sleep quality, "food addiction," and BMI as in other studies of NES. Students (n = 254) and community members (n = 468) were administered the NEQ, NEDQ, Pittsburgh Sleep Quality Index, Zung Self-report Depression Scale (SDS), and the Yale Food Addiction Scale (YFAS). Convergent validity between the NEDQ and the NEQ was demonstrated; the scores were significantly positively correlated. There was good agreement between the NEDQ and the NEQ in diagnosis of NES; 56% of those diagnosed by the NEDQ met the threshold score on the NEQ, while the other 44% did not. Only 5 participants out of 33 who met the NEQ threshold score for NES did not meet the NEDQ diagnostic criteria. MANOVA revealed that higher NEDQ was associated with higher SDS and YFAS scores and poorer sleep quality. Full-syndrome NES by the NEDQ was associated with higher BMI in the community group unlike the student group. Scores on all the other questionnaires were higher in the community group. The discrepancies between NEDQ and NEQ diagnosis may be due to differences in construction of the questionnaires and specifically due to the NEDQ being designed for diagnosis. The NEQ provides a convenient global score for NES severity, whereas the NEDQ, which shows convergent validity with the NEQ, provides clinically useful diagnostic categories.

Alcoholic beverages have long been associated with feasts, celebration and marking special events. Today, it is commonplace to consume alcoholic beverages before, with and/or after a meal. Alcohol provides additional pleasure to the meal and enhances appetite. However, consuming an alcoholic beverage with or before a meal is associated with poor short-term energy compensation; energy from alcohol is additive to total energy intake with the added property of stimulating further eating. Limiting alcohol intake is an obvious means to reduce total energy intake for those who wish to lose weight. However, dieters and restrained eaters drink more and report greater binge drinking than unrestrained eaters despite employing cognitive strategies to reduce their intake. Increased intake may be attributable to greater attentional bias to alcohol related cues as well as to food cues, since these are more salient to those limiting intake. Alcohol increases energy intake in dieters, in part due to abandonment of restraint (disinhibition) and consumption of forbidden items including alcohol exacerbates attempts to resist temptation. Paradoxically, links between binge drinking or increased drinking frequency to overweight and obesity may be mediated by dietary restraint. Efforts to limit food and alcohol intake for weight control appear to be unsuccessful and have the net effect of promoting overconsumption. The potential role of restrained eating in the association between alcohol, appetite and obesity has been overlooked by much of the current research and further investigation of this is therefore warranted.

The Emotional Appetite Questionnaire (EMAQ) comprises ratings of tendency to eat in response to both positive and negative, emotions and situations. To assess construct validity, the responses of 232 male and female participants to the EMAQ subscales were correlated with the subscales of the Dutch Eating Behavior Questionnaire (DEBQ), which has been extensively validated. In addition, the EMAQ scores were correlated with BMI. Convergent validity was demonstrated by a significant positive correlation between the negative emotions and situations scores of the EMAQ and the emotional eating subscale score of the DEBQ (DEBQ-E). Moreover, discriminant validity was demonstrated by low correlations of EMAQ positive emotions and situations scores with the DEBQ-E score. For the study sample, the EMAQ negative scores were significantly positively correlated with BMI, and the EMAQ positive scores were significantly inversely correlated with BMI. As BMI increased so did reported negative emotional and situational eating whereas as BMI decreased, reported positive emotional and situational eating increased. Although causality cannot be inferred from correlations, eating more under negative emotions may contribute to being overweight whereas eating less may contribute to being underweight. The EMAQ was shown to have construct validity, and emotional eating was significantly correlated with BMI.

Sensory specific satiation (SSS) occurs in the absence of postingestive feedback via use of a modified sham feeding procedure (MSF) which can increase satiety and fullness ratings. In the present experiment, less suppression of food intake after MSF than after eating was predicted. Furthermore, since variety tends to increase food intake, it was hypothesized that SSS to one food would suppress intake of the same food in a subsequent course compared to intake of a different food. 23 participants from the US and UK ate lunch in the laboratory in a repeated measures design with MSF and food variety as factors. In the first course, there were no differences in intake among the conditions. Participants ate less in the second course after eating than after MSF. However, those in the MSF condition consumed less total energy for lunch. Participants ate more in the varied second course than in the same condition. There was no difference in SSS for MSF and eating conditions. Thus, changes in the pleasantness of the taste of the eaten food declined whether that food was sham fed or eaten. There was no interaction between MSF and variety conditions on food intake. The results suggest that while MSF can produce SSS, it does not lower subsequent food intake.

Because body weight is largely seen as controllable, weight stigma-the social devaluation of those who are overweight-is not subject to the social norms that condemn open expression of racism and sexism. Indeed, rejection of peers based on perceptions of excess weight is normative. Since weight stigma is internalized, popular views (and often the views of physicians) have suggested that increasing the salience of weight stigma might produce a reduction in overeating and/or an increase in physical activity. However, that perspective is not rooted in scientific evidence. Recent randomized controlled designs demonstrate that stigma may promote overeating. Correlational evidence suggests that self-reported stigma experience is associated with risk for binge eating and decreased interest in physical exercise and dieting, for children and adults. In addition to reviewing these research studies, this paper examines the potential for intersectionality of stigma across multiple social identities and considers alternatives to stigmatizing weight loss interventions.

Objective This study had two objectives. The first was to evaluate the possibility that, in a previous study, a soup preload augmented the reduction of food intake in a test meal induced by an exogenous infusion of cholecystokinin (CCK) because the soup also endogenously released CCK. The second was to compare CCK release by soup between men and women to determine whether the increased satiating effectiveness of soup in women as opposed to men could have been partly attributable to differences in CCK release. Methods By using a bioassay that measures all of its known isoforms, we determined plasma CCK levels at baseline and at several times postprandially in eight healthy, non-obese men and women (four of each sex). Each subject ingested 800 g of tomato soup, which was followed 30 min later by 300 g of a yogurt shake. Appetitive ratings were also collected and related to CCK levels. Results Ingestion of tomato soup significantly increased plasma CCK levels by 3.81 pmol/L (± 1.21 standard error, P = 0.016) over baseline within 30 min in all subjects combined. When CCK concentrations at 5 min after soup and 5 min after yogurt were averaged, the women’s mean averaged concentration was 5.58 pmol/L (± 1.994, t = 2.80, P = 0.0073) higher than the men’s. The elevated levels persisted but did not rise further upon consumption of the yogurt shake. Hunger ratings declined and fullness ratings increased after eating, although patterns of ratings did not match exactly patterns of CCK release. Conclusions A large quantity of tomato soup stimulates significant CCK release; therefore, some of the satiating effects of soup preloads could have been mediated by an elevation in endogenous CCK.

Some bodyweight gain is common with many of the psychotropic drugs frequently used to treat psychiatric disorders. Although in most instances the gain is not clinically significant, that induced by some drugs, such as lithium and atypical antipsychotics, can be quite large and clinically significant. Individual patients may not wish to continue with medications that increase bodyweight, even by amounts that have no significant effect on health. The true prevalence of bodyweight gain is often difficult to discern because of confounding factors within studies that have assessed this effect, such as the simultaneous use of multiple drugs and the absence of data on starting bodyweight. Risk factors for weight increase have not yet been well characterised, although in general the potential for drug-induced bodyweight gain is greatest in individuals with a past and/or family history of obesity. Little is currently known about the mechanisms of bodyweight gain that is induced by any drug.

The formation of a conditioned taste aversion (CTA) in rats results in neural changes at several levels of the gustatory system. In the nucleus of the solitary tract (NTS), the outstanding feature of the response to a CS is a brief burst of activity that is absent in unconditioned animals. The burst occurs about 1 s after stimulus onset and is seen only in neurons that respond well to sugars and the CS (0.0025 M NaSaccharin). We recorded single neuron activity in response to 12 stimuli from taste cells in the NTS of 8 rats, in which a CTA to NaSaccharin had been created and fully extinguished, and in 8 unconditioned controls. The issue was if the neural effects of the CTA in NTS were reversed with extinction. We recorded the activity of 41 neurons in controls and 55 in CTA-extinguished rats. Responses measured across all neurons were not significantly different in spontaneous activity, breadth of tuning, overall response magnitude to each of the 12 stimuli, relationship among stimuli in taste spaces, or time-course. However, cells in the sugar-sensitive subgroup showed a clear vestige of the conditioning experience. They gave a well-defined burst of activity to the CS, though of reduced amplitude and slightly longer latency than in fully conditioned rats. This burst was no longer associated with the conditioned behavior-which was fully extinguished-though it may be a permanent marker for the once-salient CS that can influence subsequent reacquisition of the aversion.

The literature is divided over whether a conditioned taste aversion (CTA) can be fully extinguished. In Experiment 1, we created a powerful aversion in 54 rats by pairing the taste of 0.0025 M NaSaccharin (CS) with intraperitoneal injections of 127 mg/kg LiCl (US) on 3 occasions. We then offered 23-h deprived rats NaSaccharin for 10 min/day to observe the course of recovery. Extinction occurred in three phases: static, dynamic, and asymptotic. During the static phase (mean = 9.6 days), rats consumed the CS at10% of their preconditioned rate. With dynamic recovery (6.0 days), they increased acceptance to80% of preconditioning levels. Finally, they achieved asymptote (3.1 days) at 100% acceptance. In Experiment 2, we used 8 additional conditioned rats and 8 unconditioned controls. We followed the same 1-bottle extinction procedure and, again, obtained 100% acceptance. Then we offered both NaSaccharin and water for 8 days at 23 h/day and monitored lick patterns every 6 s to determine taste preferences. The conditioned animals consumed less NaSaccharin than controls on Day 1, and less NaSaccharin as a percentage of total fluid as late as Day 3. For the last 5 days of 2-bottle preference testing, there were no significant differences between the groups with regard to 1. volume of NaSaccharin or water consumed, 2. percentage of total fluid taken as NaSaccharin, 3. consumption of each fluid associated with a meal or taken spontaneously, 4. intake during the light or dark periods, or 5. the characteristics of ingestion, including number of drinking bouts, duration of bouts, number of licks/bout, and rate of licking. Therefore, a robust CTA is subject to complete behavioral extinction.

Short sleep duration is associated with elevated body mass index (BMI) and increased energy consumption. The present studies were conducted to determine what role emotional eating and stress might play in these relationships. The first was an exploratory questionnaire study in which sleep quality and duration were measured in conjunction with the Dutch Eating Behavior Questionnaire in 184 women. Emotional and external eating scores were significantly higher in those who reported poor sleep quality (but were not related to sleep duration). In a second study of 64 women who were provided with snacks in the laboratory under stressed and control conditions, elevated food consumption was observed in those who scored high on emotional eating and who reported short sleep (a significant stress × emotional eating × sleep duration interaction) but not in those who reported poor sleep quality. No effects were found in liking or wanting of food and few effects were found on appetite. BMI was not related to sleep duration or sleep quality in either study. The results suggest that the relationship between short sleep and elevated food consumption exists in those who are prone to emotional eating. An external stressor elevated consumption in normal sleepers to the level observed in short sleepers, however, it did not significantly elevate consumption in short sleepers. Future examinations of the effects of sleep duration and quality on food consumption should examine emotional eating status.

For small colleges, the use of invertebrates in undergraduate learning laboratory experiments may be a valuable alternative to the use of vertebrate species. This article describes a habituation experiment using terrestrial hermit crabs. All of the materials required are inexpensive and readily available. What makes this experiment unique is that although the instructor sets basic parameters, students have a crucial role in the experiment's design and the analysis of collected data. Assessment of learning outcomes and student evaluations indicate that conducting this experiment aided in students' learning of behavioral techniques, habituation phenomena and theory, and the role of habituation in the lives of organisms.

The night eating syndrome (NES) consists of evening hyperphagia and/or nocturnal eating and has been associated with depressed mood, that worsens in the evening. However, it is not consistently related to elevated BMI. The present study was conducted to examine whether a relationship exists between NES and emotional, external, and restrained eating. BMI and sleep quality were also obtained. A sample of 246 students completed the Night Eating Diagnostic Questionnaire (NEDQ), Night Eating Syndrome History and Inventory (NESHI), Sleep Quality Index (SQI), and Dutch Eating Behavior Questionnaire (DEBQ), containing subscales for emotional, external, and restrained eating. They also provided demographic information, including height and weight. Participants were grouped by severity of NES features using the NEDQ and NESHI: normal, mild night eater, moderate night eater, and full night eater syndrome. MANOVA was used to compare DEBQ subscores for the groups; those in the full syndrome category had significantly higher emotional eating scores and external eating scores than those in the normal and mild categories. There was no difference in restrained eating between the normal and full syndrome groups. Those with moderate and full syndrome NES symptoms also reported significantly lower sleep quality. No significant relationship was found between NES and BMI. The results show that NES is associated with more eating in response to negative mood and in response to food cues.

Higher food consumption, particularly of sweets, has been reported by substance-dependent persons in various stages of treatment but no investigation of substance use and actual food consumption has been conducted in non-dependent persons. Sixty-two male and female college students completed the Core Alcohol and Drug Survey and the amount of each of six snack foods (including both sweet and savory items) they consumed were measured. Participants also rated appetite before and after eating and rated the pleasantness of and desire to eat each food. Hierarchical multivariate regression revealed that, while controlling for the influence of BMI and intermeal interval, frequency and breadth of substance use significantly predicted elevated food consumption and elevated ratings of hunger and desire to eat. Furthermore, the link between breadth of drug use and food consumption was mediated by higher desire to eat and not by pleasantness of the food. The relationship between substance use and eating behavior may not be restricted only to those in recovery or treatment for persons diagnosed with substance use disorders.

Opiate use has been associated with preference for sweets in both humans and animals. In 2002, the food preferences and eating habits of non-institutionalized patients in methadone treatment and controls were measured and compared. Questionnaires were administered to 14 patients and 14 controls with similar demographic characteristics (there were 19 women and 9 men ranging in age from 19 to 59 years). The patients reported higher consumption of sweets, higher eagerness to consume sweet foods, and a wish to consume quantities larger than that desired by controls. Patients had a significantly higher body mass than controls. The study's limitations are noted. These findings suggest a link between methadone and a desire for calorically dense foods. A study of eating behavior is warranted.

Drug use is associated with eating behaviour. Students with past history of broad drug use ate more in a laboratory snack and reported an elevated desire to eat. In an attempt to find potential mediating psychological variables, exploratory research revealed that high impulsivity is linked to both higher pleasantness of eating foods and consuming alcohol and higher frequency of alcohol use. Those with broader drug use, higher drug consumption, and high enjoyment of intoxication also rated the pleasantness of the taste of foods lower in a questionnaire study. Drug use was not associated with emotional or external eating and dietary restraint (DEBQ). In the present study, a possible link of drug use to food addiction and food neophobia was investigated. Seventy-nine university students (65% women)were administered the FoodNeophobia Scale (FNS), the Food Attitude Survey (FAS), the Yale Food Addiction Scale, Core Alcohol and Drug Survey, and appetite measures. Height and weight were also measured to compute BMI (ranged 18–40). Analyses revealed that breadth of drug use was correlated with lower food neophobia (FNS and FAS), low finickiness (FAS), and low general neophobia. Higher alcohol consumption was also associated with both low food and general neophobia. Food addiction was not related to drug use and BMI was correlated only with food addiction. In multiple regression, wider breadth of drug use emerged as the primary predictor of low neophobia. Low food neophobia and higher drug use have been associated with sensation seeking and it is proposed that it underlies the relationships observed in the present study.

Corrigendum to “Night eating is associated with emotional and external eating in college students” [Eating Behaviors (2012) 202–206] Laurence J. Nolan⁎, Allan Geliebter a Department of Psychology, Wagner College, 1 Campus Rd., Staten Island, NY 10301, USA b New York Obesity Nutrition Research Center, Department of Medicine, St. Luke's-Roosevelt Hospital Center and Department of Psychiatry, College of Physicians & Surgeons, Columbia University, 1111 Amsterdam Ave., New York, NY 10025, USA c Psychology Department, Touro College, 27 West 23rd St., New York, NY 10010, USA

The thesis of this chapter is that understanding of the control of eating in animals is essential for understanding control of eating in humans. Animal research has provided four tools for the study of human eating, each of which will be a focus of this paper: (a) the methods, both strategic and practical (b) the observed phenomena (c) the theoretical framework and experimental analysis (d) models for the study of eating disturbances and their treatments.

Much of our knowledge about the determinants and practices of human eating behavior comes through fundamental research intended to identify and characterize operational mechanisms of ingestive behaviors. Opportunities must be taken, however, to build upon this through basic and applied studies with “real” consumers. One objective is to determine why people’s concern about the hazards of dietary fat does not result in a reduction in their fat intake. One possibility is a misplaced sense of optimism about their own behavior. Disinterest in reducing fat intakes may be in part attributable to a perception that intakes are already low or have been lowered significantly (Lloyd et al., 1993). Perceived fat intakes amongst consumers have been found to be wholly uncorrelated with measured fat intake (r=0·04). Indeed, in one study of reduced-fat food use, subjects reported a moderate drop in perceived fat intake even where little actual change in fat intakes had occurred. Although the perceived fat content of individual foods is generally correlated with their true fat content (Mela, 1993), there were a number of common food items for which fat content was significantly misjudged. This may partly represent a confusion between foods considered “fattening” (which may not be high in fat) and high fat foods. To test the eVects of food labels on liking of food, subjects were given a low-fat margarine labeled either “reduced-fat spread 40% fat” or “full-fat margarine 80% fat” and asked to rate sensory attributes and liking of each (Aaron et al., 1994). No label eVects were found in the outcome measures scores unless the subjects were classified according to their beliefs about high- and low-fat spreads. This revealed that sensory judgements were shifted in a manner consistent with previously held beliefs. Therefore, consumer attitudes toward foods should be considered in consumer testing of sensory quality. One way to aVect dietary change is to encourage the selection of foods in which fat is replaced by other macronutrients, although there is little consumer data on the eVectiveness of these foods in changing fat intake or body weight. Men and women who were not users of reduced-fat foods were instructed to purchase these products in their usual shops and freely incorporate them into their normal domestic diet. Almost immediately, there was a drop in the percentage of energy from fat consumed, but no significant change in the total amount of energy consumed, indicating a caloric compensation from other macronutrients. Over the first 4 weeks of the 6-week study period, however, there was a mean weight loss of 1·25 kg in the reduced-fat group (Gatenby et al., 1995). Consumer attitudes and beliefs have useful explanatory power in studies of ingestive behavior. Consumers have positive attitudes toward change in dietary intake but a poor conception of personal intakes and food composition, which can lead to a false perception of their own success in reducing fat intake. Casual use of macronutrient-replaced food by normal-weight consumers can have an eVect on the percentage of energy intake as fat, but with apparently little eVect on body weight. The change in fat intake, however, is important in light of postulated links of fat intake to other human disease processes. These studies are designed to address issues of consumer behavior, not to examine biological aspects of macronutrient selection and their implications for ingestive behavior. Studies oriented toward these diVerent areas would be expected to use quite diVerent approaches and methodologies. Aaron, J. I., Mela, D. J. & Evans, R. E. (1994) The influences of attitudes, beliefs and label