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8. Bone marrow‐fibroblast progenitor cell‐derived small extracellular vesicles promote cardiac fibrosis via miR‐21‐5p and integrin subunit αV signalling.

14. Sustained Increases in Cardiomyocyte Protein O ‐Linked β‐N‐Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment

22. Neutrophil-Derived S100A8/A9 Amplify Granulopoiesis After Myocardial Infarction

25. Sustained Increases in Cardiomyocyte Protein O-Linked β-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment.

27. Metabolic labeling of cardiomyocyte‐derived small extracellular‐vesicle (sEV) miRNAs identifies miR‐208a in cardiac regulation of lung gene expression

43. GSK-3α is a central regulator of age-related pathologies in mice

46. GSK-3α directly regulates β-adrenergic signaling and the response of the heart to hemodynamic stress in mice

47. Prevention of liver cancer cachexia-induced cardiac wasting and heart failure

48. Cardiac natriuretic peptide deficiency sensitizes the heart to stress-induced ventricular arrhythmias via impaired CREB signalling.

49. Targeting 5-HT 2B Receptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling After Myocardial Infarction

50. Tom Force (1951–2020)

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