242 results on '"Kyritsi, Konstantina"'
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2. Development of Scaffold-Free Three-Dimensional Cholangiocyte Organoids to Study the Progression of Primary Sclerosing Cholangitis
3. Secretin alleviates biliary and liver injury during late-stage primary biliary cholangitis via restoration of secretory processes
4. The Functional Roles of Immune Cells in Primary Liver Cancer
5. Cultural Accommodation of the Strengthening Families Program for Parents and Young Adolescents 10–14: Greek Phase I and II Study
6. Prolonged Administration of Melatonin Ameliorates Liver Phenotypes in Cholestatic Murine Model
7. Kupffer Cells: Inflammation Pathways and Cell-Cell Interactions in Alcohol-Associated Liver Disease
8. Knockout of the Tachykinin Receptor 1 in the Mdr2−/− (Abcb4−/−) Mouse Model of Primary Sclerosing Cholangitis Reduces Biliary Damage and Liver Fibrosis
9. Biliary damage and liver fibrosis are ameliorated in a novel mouse model lacking l-histidine decarboxylase/histamine signaling
10. Amelioration of Large Bile Duct Damage by Histamine-2 Receptor Vivo-Morpholino Treatment
11. Downregulation of hepatic stem cell factor by Vivo-Morpholino treatment inhibits mast cell migration and decreases biliary damage/senescence and liver fibrosis in Mdr2−/− mice
12. Knockdown of vimentin reduces mesenchymal phenotype of cholangiocytes in the Mdr2−/− mouse model of primary sclerosing cholangitis (PSC)
13. Cultural Accommodation of the Strengthening Families Program for Parents and Young Adolescents 10–14: Greek Phase I and II Study
14. Pinealectomy or light exposure exacerbates biliary damage and liver fibrosis in cholestatic rats through decreased melatonin synthesis
15. Knockout of α-calcitonin gene-related peptide attenuates cholestatic liver injury by differentially regulating cellular senescence of hepatic stellate cells and cholangiocytes
16. Knockout of secretin receptor reduces biliary damage and liver fibrosis in Mdr2−/− mice by diminishing senescence of cholangiocytes
17. The Secretin/Secretin Receptor Axis Modulates Ductular Reaction and Liver Fibrosis through Changes in Transforming Growth Factor-β1–Mediated Biliary Senescence
18. Regulation of Cellular Senescence by miR-34a in Alcoholic Liver Injury
19. Knockdown of Hepatic Gonadotropin-Releasing Hormone by Vivo-Morpholino Decreases Liver Fibrosis in Multidrug Resistance Gene 2 Knockout Mice by Down-Regulation of miR-200b
20. 662 INHIBITION OF THE GNRH/GNRHR1 AXIS AMELIORATES THE PHENOTYPES OF PRIMARY BILIARY CHOLANGITIS (PBC).
21. Knockout of microRNA-21 reduces biliary hyperplasia and liver fibrosis in cholestatic bile duct ligated mice
22. Biliary damage and liver fibrosis are ameliorated in a novel mouse model lacking l-histidine decarboxylase/histamine signaling
23. p16 INK4A drives nonalcoholic fatty liver disease phenotypes in high fat diet fed mice through biliary E2F1/FOXO1/IGF-1 signaling
24. Prolonged administration of a secretin receptor antagonist inhibits biliary senescence and liver fibrosis in Mdr2−/− mice
25. Loss of apical sodium bile acid transporter alters bile acid circulation and reduces biliary damage in cholangitis
26. Development of Scaffold-free 3D Cholangiocyte Organoids to Study the Progression of Primary Sclerosing Cholangitis
27. Mast cells selectively target large cholangiocytes during biliary injury via H2HR‐mediated cAMP/pERK1/2 signaling
28. Forkhead box A2 regulates biliary heterogeneity and senescence during cholestatic liver injury in mice‡
29. Prolonged administration of a secretin receptor antagonist inhibits biliary senescence and liver fibrosis in Mdr2 −/− mice.
30. The Effects of Taurocholic Acid on Biliary Damage and Liver Fibrosis Are Mediated by Calcitonin-Gene-Related Peptide Signaling
31. Tu1297: MELATONIN RECEPTOR 1A (MT1) KNOCKOUT DECREASES BILIARY DAMAGE AND LIVER STEATOSIS VIA DOWNREGULATION OF MIR-200B IN HIGH FAT DIET (HFD) MODEL OF NONALCOHOLIC FATTY LIVER DISEASE (NAFLD)
32. Suppression of MT1 and Melatonin Treatment Improves Liver Phenotypes in Mdr2 ‐/‐ mice
33. Tu1316: CROMOLYN SODIUM AMELIORATES MAST CELL(MC)-MEDIATED HEPATIC DAMAGE IN A MURINE MODEL OF NON-ALCOHOLIC FATTY LIVER DISEASE (NAFLD)
34. Su1345: GROWTH HORMONE AND GROWTH HORMONE RECEPTOR SIGNALING AS A NOVEL THERAPEUTIC TARGET FOR PRIMARY SCLEROSING CHOLANGITIS
35. The protective effects of estrogen on biliary and liver damage are independent of ER‐β signaling in female Mdr2 ‐/‐ mice
36. Mast Cell (MC)‐Induced Cholestasis is Dependent on Apical Sodium Bile Acid Transporter (ASBT) Expression
37. Mo1264: INHIBITION OF TRANSFORMING GROWTH FACTOR BETA 1 (TGFβ1) BY VIVO-MORPHOLINO (VM) AMELIORATES CHOLESTASIS IN THE MDR2-/- MOUSE MODEL OF PRIMARY SCLEROSING CHOLANGITIS (PSC)
38. Mast Cells Contribute to Hepatic Neurokinin1 Receptor Signaling, Subsequent Biliary Damage and Peribiliary Fibrosis Via TGF‐β1 Signaling in MDR2‐/‐ Mouse Model of Primary Scelrosing Cholangitis
39. FGF1 Signaling Modulates Biliary Injury and Liver Fibrosis in the Mdr2−/− Mouse Model of Primary Sclerosing Cholangitis
40. Melatonin receptor 1A, but not 1B, knockout decreases biliary damage and liver fibrosis during cholestatic liver injury
41. Mast cells (MCs) induce ductular reaction mimicking liver injury in mice via MC-derived TGF-β1 signaling
42. Mast Cells Regulate Ductular Reaction and Intestinal Inflammation in Cholestasis Through Farnesoid X Receptor Signaling
43. Inhibition of Secretin/Secretin Receptor Axis Ameliorates NAFLD Phenotypes
44. Mast Cells Promote Nonalcoholic Fatty Liver Disease Phenotypes and Microvesicular Steatosis in Mice Fed a Western Diet
45. 233 KNOCKDOWN OF THE MT1 MELATONIN RECEPTOR AMELIORATES THE PHENOTYPES OF THE MDR2-/- MOUSE MODEL OF PRIMARY SCLEROSING CHOLANGITIS (PSC) BY DOWNREGULATION OF ORPHAN GPR50 RECEPTOR-PROMOTED CONSTITUTIVE TGF-β1 RECEPTOR SIGNALING.
46. Sa380 KNOCKDOWN OF P16 IMPROVES MAST CELL (MC)-MEDIATED BILIARY SENESCENCE AND NAFLD PHENOTYPES IN A DIET-INDUCED RODENT MODEL
47. Fr366 ADVANCED 3D HUMAN LIVER ORGANOIDS CREATED BY MULTIPLE-HEPATIC LINEAGE CELLS FOR THE STUDY OF LIVER DISEASES
48. 85 MAST CELLS REGULATE CHOLESTATIC LIVER INJURY AND INTESTINAL BILE ACID SIGNALING VIA MODIFICATION OF FXR/FGF15 AXIS
49. 86 LONG-TERM SECRETIN TREATMENT RESTORES THE DUCTULO-CANALICULAR JUNCTIONS (DCJ) AND AMELIORATES BILIARY DAMAGE AND LIVER FIBROSIS IN A MODEL OF LATE-STAGE PRIMARY BILIARY CHOLANGITIS (PBC).
50. 87 DIFFERENTIAL EFFECTS OF MELATONIN AND IL-33/ST-2/NFκB SIGNALING ON MAST CELL-INDUCED PRIMARY SCLEROSING CHOLANGITIS (PSC)
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