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987 results on '"Kutok, Jeffery"'

1. T-Lymphoblastic Lymphoma Cells Express High Levels of BCL2, S1P1, and ICAM1, Leading to a Blockade of Tumor Cell Intravasation

Author

Feng, Hui, primary, Stachura, David L., additional, White, Richard M., additional, Gutierrez, Alejandro, additional, Zhang, Lu, additional, Sanda, Takaomi, additional, Jette, Cicely A., additional, Testa, Joseph R., additional, Neuberg, Donna S., additional, Langenau, David M., additional, Kutok, Jeffery L., additional, Zon, Leonard I., additional, Traver, David, additional, Fleming, Mark D., additional, Kanki, John P., additional, and Look, A. Thomas, additional

Published
2024
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2. Correction: Corrigendum: PI3Kγ is a molecular switch that controls immune suppression

Author

Kaneda, Megan M, Messer, Karen S, Ralainirina, Natacha, Li, Hongying, Leem, Christopher J, Gorjestani, Sara, Woo, Gyunghwi, Nguyen, Abraham V, Figueiredo, Camila C, Foubert, Philippe, Schmid, Michael C, Pink, Melissa, Winkler, David G, Rausch, Matthew, Palombella, Vito J, Kutok, Jeffery, McGovern, Karen, Frazer, Kelly A, Wu, Xuefeng, Karin, Michael, Sasik, Roman, Cohen, Ezra EW, and Varner, Judith A

Subjects
Biological Sciences, Bioinformatics and Computational Biology, Genetics, Cancer, General Science & Technology
Published
2017

3. Corrigendum: PI3Kγ is a molecular switch that controls immune suppression.

Author

Kaneda, Megan M, Messer, Karen S, Ralainirina, Natacha, Li, Hongying, Leem, Christopher J, Gorjestani, Sara, Woo, Gyunghwi, Nguyen, Abraham V, Figueiredo, Camila C, Foubert, Philippe, Schmid, Michael C, Pink, Melissa, Winkler, David G, Rausch, Matthew, Palombella, Vito J, Kutok, Jeffery, McGovern, Karen, Frazer, Kelly A, Wu, Xuefeng, Karin, Michael, Sasik, Roman, Cohen, Ezra EW, and Varner, Judith A

Subjects
Genetics, Cancer, General Science & Technology
Published
2017

4. The Selective Phosphoinoside-3-Kinase p110δ Inhibitor IPI-3063 Potently Suppresses B Cell Survival, Proliferation, and Differentiation

Author

Chiu, Honyin, Mallya, Sharmila, Nguyen, Phuongthao, Mai, Annie, Jackson, Leandra V, Winkler, David G, DiNitto, Jonathan P, Brophy, Erin E, McGovern, Karen, Kutok, Jeffery L, and Fruman, David A

Subjects
Biochemistry and Cell Biology, Biomedical and Clinical Sciences, Biological Sciences, Immunology, Autoimmune Disease, Development of treatments and therapeutic interventions, 5.1 Pharmaceuticals, Underpinning research, 1.1 Normal biological development and functioning, 2.1 Biological and endogenous factors, Aetiology, Inflammatory and immune system, phosphoinoside-3-kinase, lipid kinase, B cell survival, B cell proliferation, B cell differentiation, Medical Microbiology, Biochemistry and cell biology, Genetics
Abstract

The class I phosphoinoside-3-kinases (PI3Ks) are important enzymes that relay signals from cell surface receptors to downstream mediators driving cellular functions. Elevated PI3K signaling is found in B cell malignancies and lymphocytes of patients with autoimmune disease. The p110δ catalytic isoform of PI3K is a rational target since it is critical for B lymphocyte development, survival, activation, and differentiation. In addition, activating mutations in PIK3CD encoding p110δ cause a human immunodeficiency known as activated PI3K delta syndrome. Currently, idelalisib is the only selective p110δ inhibitor that has been FDA approved to treat certain B cell malignancies. p110δ inhibitors can suppress autoantibody production in mouse models, but limited clinical trials in human autoimmunity have been performed with PI3K inhibitors to date. Thus, there is a need for additional tools to understand the effect of pharmacological inhibition of PI3K isoforms in lymphocytes. In this study, we tested the effects of a potent and selective p110δ inhibitor, IPI-3063, in assays of B cell function. We found that IPI-3063 potently reduced mouse B cell proliferation, survival, and plasmablast differentiation while increasing antibody class switching to IgG1, almost to the same degree as a pan-PI3K inhibitor. Similarly, IPI-3063 potently inhibited human B cell proliferation in vitro. The p110γ isoform has partially overlapping roles with p110δ in B cell development, but little is known about its role in B cell function. We found that the p110γ inhibitor AS-252424 had no significant impact on B cell responses. A novel dual p110δ/γ inhibitor, IPI-443, had comparable effects to p110δ inhibition alone. These findings show that p110δ is the dominant isoform mediating B cell responses and establish that IPI-3063 is a highly potent molecule useful for studying p110δ function in immune cells.

Published
2017

5. PI3Kγ is a molecular switch that controls immune suppression

Author

Kaneda, Megan M, Messer, Karen S, Ralainirina, Natacha, Li, Hongying, Leem, Christopher J, Gorjestani, Sara, Woo, Gyunghwi, Nguyen, Abraham V, Figueiredo, Camila C, Foubert, Philippe, Schmid, Michael C, Pink, Melissa, Winkler, David G, Rausch, Matthew, Palombella, Vito J, Kutok, Jeffery, McGovern, Karen, Frazer, Kelly A, Wu, Xuefeng, Karin, Michael, Sasik, Roman, Cohen, Ezra EW, and Varner, Judith A

Subjects
Cancer, Animals, CCAAT-Enhancer-Binding Protein-beta, Cells, Cultured, Class Ib Phosphatidylinositol 3-Kinase, Female, Humans, Immune Tolerance, Inflammation, Lymphocyte Activation, Macrophages, Male, Mice, Mice, Inbred C57BL, NF-kappa B, Neoplasms, Phosphoinositide-3 Kinase Inhibitors, Programmed Cell Death 1 Receptor, Proto-Oncogene Proteins c-akt, Signal Transduction, T-Lymphocytes, TOR Serine-Threonine Kinases, Tumor Escape, General Science & Technology
Abstract

Macrophages play critical, but opposite, roles in acute and chronic inflammation and cancer. In response to pathogens or injury, inflammatory macrophages express cytokines that stimulate cytotoxic T cells, whereas macrophages in neoplastic and parasitic diseases express anti-inflammatory cytokines that induce immune suppression and may promote resistance to T cell checkpoint inhibitors. Here we show that macrophage PI 3-kinase γ controls a critical switch between immune stimulation and suppression during inflammation and cancer. PI3Kγ signalling through Akt and mTor inhibits NFκB activation while stimulating C/EBPβ activation, thereby inducing a transcriptional program that promotes immune suppression during inflammation and tumour growth. By contrast, selective inactivation of macrophage PI3Kγ stimulates and prolongs NFκB activation and inhibits C/EBPβ activation, thus promoting an immunostimulatory transcriptional program that restores CD8+ T cell activation and cytotoxicity. PI3Kγ synergizes with checkpoint inhibitor therapy to promote tumour regression and increased survival in mouse models of cancer. In addition, PI3Kγ-directed, anti-inflammatory gene expression can predict survival probability in cancer patients. Our work thus demonstrates that therapeutic targeting of intracellular signalling pathways that regulate the switch between macrophage polarization states can control immune suppression in cancer and other disorders.

Published
2016

6. Inhibition of de novo lipogenesis targets androgen receptor signaling in castration-resistant prostate cancer

Author

Zadra, Giorgia, Ribeiro, Caroline F., Chetta, Paolo, Ho, Yeung, Cacciatore, Stefano, Gao, Xueliang, Syamala, Sudeepa, Bango, Clyde, Photopoulos, Cornelia, Huang, Ying, Tyekucheva, Svitlana, Bastos, Debora C., Tchaicha, Jeremy, Lawney, Brian, Uo, Takuma, D’Anello, Laura, Csibi, Alfredo, Kalekar, Radha, Larimer, Benjamin, Ellis, Leigh, Butler, Lisa M., Morrissey, Colm, McGovern, Karen, Palombella, Vito J., Kutok, Jeffery L., Mahmood, Umar, Bosari, Silvano, Adams, Julian, Peluso, Stephane, Dehm, Scott M., Plymate, Stephen R., and Loda, Massimo

Published
2019

8. A Zebrafish bmyb Mutation Causes Genome Instability and Increased Cancer Susceptibility

Author

Shepard, Jennifer L., Amatruda, James F., Stern, Howard M., Subramanian, Aravind, Finkelstein, David, Ziai, James, Finley, K. Rose, Pfaff, Kathleen L., Hersey, Candace, Zhou, Yi, Barut, Bruce, Freedman, Matthew, Lee, Charles, Spitsbergen, Jan, Neuberg, Donna, Weber, Gerhard, Golub, Todd R., Glickman, Jonathan N., Kutok, Jeffery L., Aster, Jon C., Zon, Leonard I., and Kunkel, Louis M.

Published
2005

11. PKC412 Inhibits the Zinc Finger 198-Fibroblast Growth Factor Receptor 1 Fusion Tyrosine Kinase and Is Active in Treatment of Stem Cell Myeloproliferative Disorder

Author

Chen, Jing, DeAngelo, Daniel J., Kutok, Jeffery L., Williams, Ifor R., Lee, Benjamin H., Wadleigh, Martha, Duclos, Nicole, Cohen, Sarah, Adelsperger, Jennifer, Okabe, Rachel, Coburn, Allison, Galinsky, Ilene, Huntly, Brian, Cohen, Pamela S., Meyer, Thomas, Fabbro, Doriano, Roesel, Johannes, Banerji, Lolita, Griffin, James D., Xiao, Sheng, Fletcher, Jonathan A., Stone, Richard M., Gilliland, D. Gary, and Witte, Owen N.

Published
2004

13. EZH2 Cooperates with BRD4-NUT to Drive NUT Carcinoma Growth by Silencing Key Tumor Suppressor Genes

Author

Huang, Yeying, primary, Durall, R. Taylor, additional, Luong, Nhi M., additional, Hertzler, Hans J., additional, Huang, Julianna, additional, Gokhale, Prafulla C., additional, Leeper, Brittaney A., additional, Persky, Nicole S., additional, Root, David E., additional, Anekal, Praju V., additional, Montero Llopis, Paula D.L.M., additional, David, Clement N., additional, Kutok, Jeffery L., additional, Raimondi, Alejandra, additional, Saluja, Karan, additional, Luo, Jia, additional, Zahnow, Cynthia A., additional, Adane, Biniam, additional, Stegmaier, Kimberly, additional, Hawkins, Catherine E., additional, Ponne, Christopher, additional, Le, Quan, additional, Shapiro, Geoffrey I., additional, Lemieux, Madeleine E., additional, Eagen, Kyle P., additional, and French, Christopher A., additional

Published
2023
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14. EZH2 synergizes with BRD4-NUT to drive NUT carcinoma growth through silencing of key tumor suppressor genes

Author

Huang, Yeying, primary, Durall, R Taylor, additional, Luong, Nhi M, additional, Hertzler, Hans J, additional, Huang, Julianna, additional, Gokhale, Prafulla C, additional, Leeper, Brittaney A, additional, Persky, Nicole S, additional, Root, David E, additional, Anekal, Praju V, additional, Montero Llopis, Paula DLM, additional, David, Clement N, additional, Kutok, Jeffery L, additional, Raimondi, Alejandra, additional, Saluja, Karan, additional, Luo, Jia, additional, Zahnow, Cynthia A, additional, Adane, Biniam, additional, Stegmaier, Kimberly, additional, Hawkins, Catherine E, additional, Ponne, Christopher, additional, Le, Quan, additional, Shapiro, Geoffrey I, additional, Lemieux, Madeleine E, additional, Eagen, Kyle P, additional, and French, Christopher A, additional

Published
2023
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15. Bad-Deficient Mice Develop Diffuse Large B Cell Lymphoma

Author

Ranger, Ann M., Zha, Jiping, Harada, Hisashi, Datta, Sandeep Robert, Danial, Nika N., Gilmore, Andrew P., Kutok, Jeffery L., Le Beau, Michelle M., Greenberg, Michael E., and Korsmeyer, Stanley J.

Published
2003

17. Myc-Induced T Cell Leukemia in Transgenic Zebrafish

Author

Langenau, David M., Traver, David, Ferrando, Adolfo A., Kutok, Jeffery L., Aster, Jon C., Kanki, John P., Lin, Shuo, Prochownik, Ed, Trede, Nikolaus S., Zon, Leonard I., and Look, A. Thomas

Published
2003

19. Supplementary Figure S2 from Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Author

Chen, Shih-Shih, primary, Barrientos, Jacqueline C., primary, Ferrer, Gerardo, primary, King-Richards, Morgan, primary, Chen, Yu-Ju, primary, Ravichandran, Priyadarshini, primary, Ibrahim, Michael, primary, Kieso, Yasmine, primary, Waters, Sheila, primary, Kutok, Jeffery L., primary, Peluso, Marisa, primary, Sharma, Sujata, primary, Weaver, David T., primary, Pachter, Jonathan A., primary, Rai, Kanti R., primary, and Chiorazzi, Nicholas, primary

Published
2023
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20. Data from Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Author

Chen, Shih-Shih, primary, Barrientos, Jacqueline C., primary, Ferrer, Gerardo, primary, King-Richards, Morgan, primary, Chen, Yu-Ju, primary, Ravichandran, Priyadarshini, primary, Ibrahim, Michael, primary, Kieso, Yasmine, primary, Waters, Sheila, primary, Kutok, Jeffery L., primary, Peluso, Marisa, primary, Sharma, Sujata, primary, Weaver, David T., primary, Pachter, Jonathan A., primary, Rai, Kanti R., primary, and Chiorazzi, Nicholas, primary

Published
2023
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21. Supplementary Table S1 from Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Author

Chen, Shih-Shih, primary, Barrientos, Jacqueline C., primary, Ferrer, Gerardo, primary, King-Richards, Morgan, primary, Chen, Yu-Ju, primary, Ravichandran, Priyadarshini, primary, Ibrahim, Michael, primary, Kieso, Yasmine, primary, Waters, Sheila, primary, Kutok, Jeffery L., primary, Peluso, Marisa, primary, Sharma, Sujata, primary, Weaver, David T., primary, Pachter, Jonathan A., primary, Rai, Kanti R., primary, and Chiorazzi, Nicholas, primary

Published
2023
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22. Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Author

Chen, Shih-Shih, primary, Barrientos, Jacqueline C., additional, Ferrer, Gerardo, additional, King-Richards, Morgan, additional, Chen, Yu-Ju, additional, Ravichandran, Priyadarshini, additional, Ibrahim, Michael, additional, Kieso, Yasmine, additional, Waters, Sheila, additional, Kutok, Jeffery L., additional, Peluso, Marisa, additional, Sharma, Sujata, additional, Weaver, David T., additional, Pachter, Jonathan A., additional, Rai, Kanti R., additional, and Chiorazzi, Nicholas, additional

Published
2023
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23. Abstract 4933: Duvelisib eliminates CLL B Cells, impairs CLL-supporting cells, and overcomes ibrutinib resistance in a patient-derived xenograft model

Author

Chen, Shih-Shih, primary, Barrientos, Jacqueline C., additional, Ferrer, Gerardo, additional, Ravichandran, Priyadarshini, additional, Ibrahim, Michael, additional, Kieso, Yasmine, additional, Kutok, Jeffery L., additional, Peluso, Marisa, additional, Sharma, Sujata, additional, Weaver, David T., additional, Pachter, Jonathan A., additional, Rai, Kanti R., additional, and Chiorazzi, Nicholas, additional

Published
2023
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24. Supplementary Table 5 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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25. Supplementary Table 1 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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26. Supplementary Table 2 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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27. Supplementary Figure 4 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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28. Interview with Dr. Weinstock from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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29. Supplementary Figure 2 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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30. Supplementary Table 4 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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31. Data from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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32. Supplementary Table 3 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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33. Supplementary Methods, Figure Legends 1-4 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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34. Supplementary Figure 3 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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35. Supplementary Figure 1 from Molecular Ontogeny of Donor-Derived Follicular Lymphomas Occurring after Hematopoietic Cell Transplantation

Author

Weigert, Oliver, primary, Kopp, Nadja, primary, Lane, Andrew A., primary, Yoda, Akinori, primary, Dahlberg, Suzanne E., primary, Neuberg, Donna, primary, Bahar, Anita Y., primary, Chapuy, Bjoern, primary, Kutok, Jeffery L., primary, Longtine, Janina A., primary, Kuo, Frank C., primary, Haley, Terry, primary, Salois, Maura, primary, Sullivan, Timothy J., primary, Fisher, David C., primary, Fox, Edward A., primary, Rodig, Scott J., primary, Antin, Joseph H., primary, and Weinstock, David M., primary

Published
2023
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37. Supplementary Table 1 and 2 from Bim Polymorphisms: Influence on Function and Response to Treatment in Children with Acute Lymphoblastic Leukemia

Author

Gagné, Vincent, primary, Rousseau, Julie, primary, Labuda, Malgorzata, primary, Sharif-Askari, Bahram, primary, Brukner, Ivan, primary, Laverdière, Caroline, primary, Ceppi, Francesco, primary, Sallan, Stephen E., primary, Silverman, Lewis B., primary, Neuberg, Donna, primary, Kutok, Jeffery L., primary, Sinnett, Daniel, primary, and Krajinovic, Maja, primary

Published
2023
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38. Supplementary Data from MLL-Rearranged B Lymphoblastic Leukemias Selectively Express the Immunoregulatory Carbohydrate-Binding Protein Galectin-1

Author

Juszczynski, Przemyslaw, primary, Rodig, Scott J., primary, Ouyang, Jing, primary, O'Donnell, Evan, primary, Takeyama, Kunihiko, primary, Mlynarski, Wojciech, primary, Mycko, Katarzyna, primary, Szczepanski, Tomasz, primary, Gaworczyk, Anna, primary, Krivtsov, Andrei, primary, Faber, Joerg, primary, Sinha, Amit U., primary, Rabinovich, Gabriel A., primary, Armstrong, Scott A., primary, Kutok, Jeffery L., primary, and Shipp, Margaret A., primary

Published
2023
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40. Supplementary Table 1 from Polymorphisms of Asparaginase Pathway and Asparaginase-Related Complications in Children with Acute Lymphoblastic Leukemia

Author

Ben Tanfous, Mohsen, primary, Sharif-Askari, Bahram, primary, Ceppi, Francesco, primary, Laaribi, Haithem, primary, Gagné, Vincent, primary, Rousseau, Julie, primary, Labuda, Malgorzata, primary, Silverman, Lewis B., primary, Sallan, Stephen E., primary, Neuberg, Donna, primary, Kutok, Jeffery L., primary, Sinnett, Daniel, primary, Laverdière, Caroline, primary, and Krajinovic, Maja, primary

Published
2023
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43. Supplementary Tables 1-2 from A Unique Galectin Signature in Human Prostate Cancer Progression Suggests Galectin-1 as a Key Target for Treatment of Advanced Disease

Author

Laderach, Diego J., primary, Gentilini, Lucas D., primary, Giribaldi, Laura, primary, Delgado, Victor Cardenas, primary, Nugnes, Lorena, primary, Croci, Diego O., primary, Al Nakouzi, Nader, primary, Sacca, Paula, primary, Casas, Gabriel, primary, Mazza, Osvaldo, primary, Shipp, Margaret A., primary, Vazquez, Elba, primary, Chauchereau, Anne, primary, Kutok, Jeffery L., primary, Rodig, Scott J., primary, Elola, María T., primary, Compagno, Daniel, primary, and Rabinovich, Gabriel A., primary

Published
2023
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44. Supplementary Table 1 from Serologic Markers of Effective Tumor Immunity against Chronic Lymphocytic Leukemia Include Nonmutated B-Cell Antigens

Author

Marina, Ovidiu, primary, Hainz, Ursula, primary, Biernacki, Melinda A., primary, Zhang, Wandi, primary, Cai, Ann, primary, Duke-Cohan, Jonathan S., primary, Liu, Fenglong, primary, Brusic, Vladimir, primary, Neuberg, Donna, primary, Kutok, Jeffery L., primary, Alyea, Edwin P., primary, Canning, Christine M., primary, Soiffer, Robert J., primary, Ritz, Jerome, primary, and Wu, Catherine J., primary

Published
2023
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45. Supplementary Figure 2 from Serologic Markers of Effective Tumor Immunity against Chronic Lymphocytic Leukemia Include Nonmutated B-Cell Antigens

Author

Marina, Ovidiu, primary, Hainz, Ursula, primary, Biernacki, Melinda A., primary, Zhang, Wandi, primary, Cai, Ann, primary, Duke-Cohan, Jonathan S., primary, Liu, Fenglong, primary, Brusic, Vladimir, primary, Neuberg, Donna, primary, Kutok, Jeffery L., primary, Alyea, Edwin P., primary, Canning, Christine M., primary, Soiffer, Robert J., primary, Ritz, Jerome, primary, and Wu, Catherine J., primary

Published
2023
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47. Supplementary Figure 5 from Proteasome Inhibition Causes Regression of Leukemia and Abrogates BCR-ABL–Induced Evasion of Apoptosis in Part through Regulation of Forkhead Tumor Suppressors

Author

Jagani, Zainab, primary, Song, Keli, primary, Kutok, Jeffery L., primary, Dewar, M. Rajan, primary, Melet, Armelle, primary, Santos, Tanya, primary, Grassian, Alexandra, primary, Ghaffari, Saghi, primary, Wu, Catherine, primary, Ren, Ruibao, primary, Rodin, Heather Yeckes, primary, Miller, Kenneth, primary, and Khosravi-Far, Roya, primary

Published
2023
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48. Supplementary Figure 7 from Proteasome Inhibition Causes Regression of Leukemia and Abrogates BCR-ABL–Induced Evasion of Apoptosis in Part through Regulation of Forkhead Tumor Suppressors

Author

Jagani, Zainab, primary, Song, Keli, primary, Kutok, Jeffery L., primary, Dewar, M. Rajan, primary, Melet, Armelle, primary, Santos, Tanya, primary, Grassian, Alexandra, primary, Ghaffari, Saghi, primary, Wu, Catherine, primary, Ren, Ruibao, primary, Rodin, Heather Yeckes, primary, Miller, Kenneth, primary, and Khosravi-Far, Roya, primary

Published
2023
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49. Supplementary Figure 9 from Proteasome Inhibition Causes Regression of Leukemia and Abrogates BCR-ABL–Induced Evasion of Apoptosis in Part through Regulation of Forkhead Tumor Suppressors

Author

Jagani, Zainab, primary, Song, Keli, primary, Kutok, Jeffery L., primary, Dewar, M. Rajan, primary, Melet, Armelle, primary, Santos, Tanya, primary, Grassian, Alexandra, primary, Ghaffari, Saghi, primary, Wu, Catherine, primary, Ren, Ruibao, primary, Rodin, Heather Yeckes, primary, Miller, Kenneth, primary, and Khosravi-Far, Roya, primary

Published
2023
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50. Supplementary Figure 4 from Proteasome Inhibition Causes Regression of Leukemia and Abrogates BCR-ABL–Induced Evasion of Apoptosis in Part through Regulation of Forkhead Tumor Suppressors

Author

Jagani, Zainab, primary, Song, Keli, primary, Kutok, Jeffery L., primary, Dewar, M. Rajan, primary, Melet, Armelle, primary, Santos, Tanya, primary, Grassian, Alexandra, primary, Ghaffari, Saghi, primary, Wu, Catherine, primary, Ren, Ruibao, primary, Rodin, Heather Yeckes, primary, Miller, Kenneth, primary, and Khosravi-Far, Roya, primary

Published
2023
Full Text
View/download PDF

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