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1. Gemini: A Family of Highly Capable Multimodal Models

2. Towards practical reinforcement learning for tokamak magnetic control

3. Optimizing Memory Mapping Using Deep Reinforcement Learning

4. Upregulation of ALOX12−12-HETE pathway impairs AMPK-dependent modulation of vascular metabolism in ApoE/LDLR−/− mice

9. Learning to Design Efficient Logic Circuits

10. Association of polymorphism in genes encoding κB inhibitors (IκB) with susceptibility to and phenotype of Graves' disease: a case-control study

12. Cytokine-Mediated Degradation of the Transcription Factor ERG Impacts the Pulmonary Vascular Response to Systemic Inflammatory Challenge

14. Optimizing Memory Mapping Using Deep Reinforcement Learning.

22. Chronic heart failure induces early defenestration of liver sinusoidal endothelial cells (LSECs) in mice.

25. Vascular protein disulfide isomerase A1 mediates endothelial dysfunction induced by angiotensin II in mice.

26. microRNAs in Human Adipose Tissue Physiology and Dysfunction

30. Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

31. Supplementary Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

32. Cytokine-Mediated Degradation of the Transcription Factor ERG Impacts the Pulmonary Vascular Response to Systemic Inflammatory Challenge

34. Supplementary Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

35. Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

36. Cytokine-Mediated Degradation of the Transcription Factor ERG Impacts the Pulmonary Vascular Response to Systemic Inflammatory Challenge

38. Obesity in Caucasian Seniors on the Rise: Is It Truly Harmful? Results of the PolSenior2 Study

43. The role of diet in the management of MAFLD—why does a new disease require a novel, individualized approach?

44. Abstract LB134: Type 1 calreticulin mutations differentially activate the IRE1α-XBP1 pathway of the unfolded protein response to drive myeloproliferative neoplasms

46. Sexual function in women with androgen excess disorders: classic forms of congenital adrenal hyperplasia and polycystic ovary syndrome

47. Estrogens in Adipose Tissue Physiology and Obesity-Related Dysfunction

49. Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

50. Endocrine Disorders Accompanying Obesity - Effect or Cause?

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